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La maladie de Parkinson en France (serveur d'exploration) - Merge (Accueil)

Index « Auteurs » - entrée « Claude Forni »
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Claude Feuerstein < Claude Forni < Claude Gallinari  Facettes :

List of bibliographic references

Number of relevant bibliographic references: 6.
Ident.Authors (with country if any)Title
001039 (2012-07-30) Claude Forni ; Olivier Mainard ; Christophe Melon [France] ; Didier Goguenheim [France] ; Lydia Kerkerian-Le Goff [France] ; Pascal Salin [France]Portable microstimulator for chronic deep brain stimulation in freely moving rats.
002323 (2009) Quentin Barraud [France] ; Virginie Lambrecq [France] ; Claude Forni [France] ; Steve Mcguire [Royaume-Uni] ; Michael Hill [Royaume-Uni] ; Bernard Bioulac [France] ; Emmanuel Balzamo [France] ; Erwan Bezard [France] ; François Tison [France] ; Imad Ghorayeb [France]Sleep disorders in Parkinson's disease: The contribution of the MPTP non-human primate model
002A01 (2007) Christelle Baunez [France] ; Anastasia Christakou [Royaume-Uni] ; Yogita Chudasama [Royaume-Uni] ; Claude Forni [France] ; Trevor W. Robbins [Royaume-Uni]Bilateral high‐frequency stimulation of the subthalamic nucleus on attentional performance: transient deleterious effects and enhanced motivation in both intact and parkinsonian rats
003531 (2003) Yassine Darbaky [France] ; Claude Forni ; Marianne Amalric ; Christelle BaunezHigh frequency stimulation of the subthalamic nucleus has beneficial antiparkinsonian effects on motor functions in rats, but less efficiency in a choice reaction time task.
003611 (2003) Yassine Darbaky [France] ; Claude Forni [France] ; Marianne Amalric [France] ; Christelle Baunez [France]High frequency stimulation of the subthalamic nucleus has beneficial antiparkinsonian effects on motor functions in rats, but less efficiency in a choice reaction time task
003838 (2002) Pascal Salin [France] ; Christine Manrique ; Claude Forni ; Lydia Kerkerian-Le GoffHigh-frequency stimulation of the subthalamic nucleus selectively reverses dopamine denervation-induced cellular defects in the output structures of the basal ganglia in the rat.

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