Myoclonus and extrapyramidal diseases.
Identifieur interne : 005972 ( Main/Exploration ); précédent : 005971; suivant : 005973Myoclonus and extrapyramidal diseases.
Auteurs : L. Defebvre [France]Source :
- Neurophysiologie clinique = Clinical neurophysiology [ 0987-7053 ]
English descriptors
- KwdEn :
- Basal Ganglia Diseases (complications), Basal Ganglia Diseases (physiopathology), Cerebral Cortex (physiopathology), Dystonic Disorders (complications), Dystonic Disorders (physiopathology), Electroencephalography, Evoked Potentials, Somatosensory, Humans, Multiple System Atrophy (complications), Multiple System Atrophy (physiopathology), Myoclonus (etiology), Myoclonus (physiopathology), Parkinson Disease (complications), Parkinson Disease (physiopathology), Tauopathies (complications), Tauopathies (physiopathology).
- MESH :
- complications : Basal Ganglia Diseases, Dystonic Disorders, Multiple System Atrophy, Parkinson Disease, Tauopathies.
- etiology : Myoclonus.
- physiopathology : Basal Ganglia Diseases, Cerebral Cortex, Dystonic Disorders, Multiple System Atrophy, Myoclonus, Parkinson Disease, Tauopathies.
- Electroencephalography, Evoked Potentials, Somatosensory, Humans.
Abstract
Parkinsonism or dystonia are associated with myoclonus in several extrapyramidal diseases. Although the latter symptom is not always prominent, stimulus-sensitive, distal, or focal reflex myoclonus is frequently observed. This review will consider the clinical and electrophysiological features of myoclonus in Parkinson's disease, multiple system atrophy, corticobasal degeneration, progressive supranuclear palsy, Huntington's disease, dentatorubral-pallidoluysian atrophy, Lewy body dementia, and myoclonus with dystonia. The evidence of a long-latency reflex response, the presence of giant somatosensory evoked potentials, and the demonstration of a back-averaged premyoclonus focal cortical EEG activity often lead to classify myoclonus as a cortical phenomenon. However, a subcortical origin cannot always be ruled out.
DOI: 10.1016/j.neucli.2006.11.003
PubMed: 17336776
Affiliations:
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Defebvre</name><affiliation wicri:level="3"><nlm:affiliation>Department of Neurology and Movement Disorders, EA2683, IFR114, Lille University Medical Centre, Hôpital Roger-Salengro, 59037 Lille cedex, France. ldefebvre@chru-lille.fr</nlm:affiliation><country xml:lang="fr">France</country><wicri:regionArea>Department of Neurology and Movement Disorders, EA2683, IFR114, Lille University Medical Centre, Hôpital Roger-Salengro, 59037 Lille cedex</wicri:regionArea><placeName><region type="region" nuts="2">Hauts-de-France</region><region type="old region" nuts="2">Nord-Pas-de-Calais</region><settlement type="city">Lille</settlement></placeName></affiliation></author></titleStmt><publicationStmt><idno type="wicri:source">PubMed</idno><date when="????"><PubDate><MedlineDate>2006 Sep-Dec</MedlineDate></PubDate></date><idno type="RBID">pubmed:17336776</idno><idno type="pmid">17336776</idno><idno type="doi">10.1016/j.neucli.2006.11.003</idno><idno type="wicri:Area/PubMed/Corpus">000E48</idno><idno type="wicri:explorRef" wicri:stream="PubMed" wicri:step="Corpus" wicri:corpus="PubMed">000E48</idno><idno type="wicri:Area/PubMed/Curation">000E08</idno><idno type="wicri:explorRef" wicri:stream="PubMed" wicri:step="Curation">000E08</idno><idno type="wicri:Area/PubMed/Checkpoint">000E08</idno><idno type="wicri:explorRef" wicri:stream="Checkpoint" wicri:step="PubMed">000E08</idno><idno type="wicri:Area/Ncbi/Merge">000705</idno><idno type="wicri:Area/Ncbi/Curation">000705</idno><idno type="wicri:Area/Ncbi/Checkpoint">000705</idno><idno type="wicri:doubleKey">0987-7053::Defebvre L:myoclonus:and:extrapyramidal</idno><idno type="wicri:Area/Main/Merge">006509</idno><idno type="wicri:Area/Main/Curation">005972</idno><idno type="wicri:Area/Main/Exploration">005972</idno></publicationStmt><sourceDesc><biblStruct><analytic><title xml:lang="en">Myoclonus and extrapyramidal diseases.</title><author><name sortKey="Defebvre, L" sort="Defebvre, L" uniqKey="Defebvre L" first="L" last="Defebvre">L. Defebvre</name><affiliation wicri:level="3"><nlm:affiliation>Department of Neurology and Movement Disorders, EA2683, IFR114, Lille University Medical Centre, Hôpital Roger-Salengro, 59037 Lille cedex, France. ldefebvre@chru-lille.fr</nlm:affiliation><country xml:lang="fr">France</country><wicri:regionArea>Department of Neurology and Movement Disorders, EA2683, IFR114, Lille University Medical Centre, Hôpital Roger-Salengro, 59037 Lille cedex</wicri:regionArea><placeName><region type="region" nuts="2">Hauts-de-France</region><region type="old region" nuts="2">Nord-Pas-de-Calais</region><settlement type="city">Lille</settlement></placeName></affiliation></author></analytic><series><title level="j">Neurophysiologie clinique = Clinical neurophysiology</title><idno type="ISSN">0987-7053</idno></series></biblStruct></sourceDesc></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Basal Ganglia Diseases (complications)</term><term>Basal Ganglia Diseases (physiopathology)</term><term>Cerebral Cortex (physiopathology)</term><term>Dystonic Disorders (complications)</term><term>Dystonic Disorders (physiopathology)</term><term>Electroencephalography</term><term>Evoked Potentials, Somatosensory</term><term>Humans</term><term>Multiple System Atrophy (complications)</term><term>Multiple System Atrophy (physiopathology)</term><term>Myoclonus (etiology)</term><term>Myoclonus (physiopathology)</term><term>Parkinson Disease (complications)</term><term>Parkinson Disease (physiopathology)</term><term>Tauopathies (complications)</term><term>Tauopathies (physiopathology)</term></keywords><keywords scheme="MESH" qualifier="complications" xml:lang="en"><term>Basal Ganglia Diseases</term><term>Dystonic Disorders</term><term>Multiple System Atrophy</term><term>Parkinson Disease</term><term>Tauopathies</term></keywords><keywords scheme="MESH" qualifier="etiology" xml:lang="en"><term>Myoclonus</term></keywords><keywords scheme="MESH" qualifier="physiopathology" xml:lang="en"><term>Basal Ganglia Diseases</term><term>Cerebral Cortex</term><term>Dystonic Disorders</term><term>Multiple System Atrophy</term><term>Myoclonus</term><term>Parkinson Disease</term><term>Tauopathies</term></keywords><keywords scheme="MESH" xml:lang="en"><term>Electroencephalography</term><term>Evoked Potentials, Somatosensory</term><term>Humans</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Parkinsonism or dystonia are associated with myoclonus in several extrapyramidal diseases. Although the latter symptom is not always prominent, stimulus-sensitive, distal, or focal reflex myoclonus is frequently observed. This review will consider the clinical and electrophysiological features of myoclonus in Parkinson's disease, multiple system atrophy, corticobasal degeneration, progressive supranuclear palsy, Huntington's disease, dentatorubral-pallidoluysian atrophy, Lewy body dementia, and myoclonus with dystonia. The evidence of a long-latency reflex response, the presence of giant somatosensory evoked potentials, and the demonstration of a back-averaged premyoclonus focal cortical EEG activity often lead to classify myoclonus as a cortical phenomenon. However, a subcortical origin cannot always be ruled out.</div></front></TEI><affiliations><list><country><li>France</li></country><region><li>Hauts-de-France</li><li>Nord-Pas-de-Calais</li></region><settlement><li>Lille</li></settlement></list><tree><country name="France"><region name="Hauts-de-France"><name sortKey="Defebvre, L" sort="Defebvre, L" uniqKey="Defebvre L" first="L" last="Defebvre">L. Defebvre</name></region></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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