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Early life exposure to the 1918 influenza pandemic and old-age mortality by cause of death.

Identifieur interne : 000A39 ( PubMed/Checkpoint ); précédent : 000A38; suivant : 000A40

Early life exposure to the 1918 influenza pandemic and old-age mortality by cause of death.

Auteurs : Mikko Myrskyl [Allemagne] ; Neil K. Mehta ; Virginia W. Chang

Source :

RBID : pubmed:23678911

Descripteurs français

English descriptors

Abstract

We sought to analyze how early exposure to the 1918 influenza pandemic is associated with old-age mortality by cause of death.

DOI: 10.2105/AJPH.2012.301060
PubMed: 23678911


Affiliations:


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pubmed:23678911

Le document en format XML

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<div type="abstract" xml:lang="en">We sought to analyze how early exposure to the 1918 influenza pandemic is associated with old-age mortality by cause of death.</div>
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<Month>08</Month>
<Day>13</Day>
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<Year>2018</Year>
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<AbstractText Label="OBJECTIVES" NlmCategory="OBJECTIVE">We sought to analyze how early exposure to the 1918 influenza pandemic is associated with old-age mortality by cause of death.</AbstractText>
<AbstractText Label="METHODS" NlmCategory="METHODS">We analyzed the National Health Interview Survey (n = 81,571; follow-up 1989-2006; 43,808 deaths) and used year and quarter of birth to assess timing of pandemic exposure. We used Cox proportional and Fine-Gray competing hazard models for all-cause and cause-specific mortality, respectively.</AbstractText>
<AbstractText Label="RESULTS" NlmCategory="RESULTS">Cohorts born during pandemic peaks had excess all-cause mortality attributed to increased noncancer mortality. We found evidence for a trade-off between noncancer and cancer causes: cohorts with high noncancer mortality had low cancer mortality, and vice versa.</AbstractText>
<AbstractText Label="CONCLUSIONS" NlmCategory="CONCLUSIONS">Early disease exposure increases old-age mortality through noncancer causes, which include respiratory and cardiovascular diseases, and may trigger a trade-off in the risk of cancer and noncancer causes. Potential mechanisms include inflammation or apoptosis. The findings contribute to our understanding of the causes of death behind the early disease exposure-later mortality association. The cancer-noncancer trade-off is potentially important for understanding the mechanisms behind these associations.</AbstractText>
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<ForeName>Mikko</ForeName>
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