Further case of paroxysmal exercise-induced dystonia and some insights into pathogenesis.
Identifieur interne : 003888 ( PubMed/Curation ); précédent : 003887; suivant : 003889Further case of paroxysmal exercise-induced dystonia and some insights into pathogenesis.
Auteurs : Michael H. Barnett [Royaume-Uni] ; Paul R. Jarman ; Simon J R. Heales ; Kailash P. BhatiaSource :
- Movement disorders : official journal of the Movement Disorder Society [ 0885-3185 ] ; 2002.
English descriptors
- KwdEn :
- MESH :
- chemical , cerebrospinal fluid : Homovanillic Acid, Hydroxyindoleacetic Acid.
- chemical , physiology : Dopamine.
- diagnosis : Dystonia.
- physiology : Exercise, Synaptic Transmission.
- physiopathology : Dystonia.
- Adult, Humans, Male, Neurologic Examination.
Abstract
Cerebrospinal fluid (CSF) analysis of pterin and monamine metabolites was performed before and after an attack in a patient with paroxysmal exercise-induced dystonia. A twofold increase in CSF homovanillic acid and 5-hydroxyindoleacetic acid after an attack was measured. This finding lends support to the hypothesis that increased dopaminergic transmission contributes to the clinical features of the hyperkinetic movement disorders.
DOI: 10.1002/mds.10291
PubMed: 12465091
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Heales</name></author><author><name sortKey="Bhatia, Kailash P" sort="Bhatia, Kailash P" uniqKey="Bhatia K" first="Kailash P" last="Bhatia">Kailash P. Bhatia</name></author></titleStmt><publicationStmt><idno type="wicri:source">PubMed</idno><date when="2002">2002</date><idno type="RBID">pubmed:12465091</idno><idno type="pmid">12465091</idno><idno type="doi">10.1002/mds.10291</idno><idno type="wicri:Area/PubMed/Corpus">003888</idno><idno type="wicri:Area/PubMed/Curation">003888</idno></publicationStmt><sourceDesc><biblStruct><analytic><title xml:lang="en">Further case of paroxysmal exercise-induced dystonia and some insights into pathogenesis.</title><author><name sortKey="Barnett, Michael H" sort="Barnett, Michael H" uniqKey="Barnett M" first="Michael H" last="Barnett">Michael H. 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A twofold increase in CSF homovanillic acid and 5-hydroxyindoleacetic acid after an attack was measured. This finding lends support to the hypothesis that increased dopaminergic transmission contributes to the clinical features of the hyperkinetic movement disorders.</div></front></TEI><pubmed><MedlineCitation Owner="NLM" Status="MEDLINE"><PMID Version="1">12465091</PMID><DateCreated><Year>2002</Year><Month>12</Month><Day>04</Day></DateCreated><DateCompleted><Year>2003</Year><Month>03</Month><Day>24</Day></DateCompleted><DateRevised><Year>2013</Year><Month>11</Month><Day>21</Day></DateRevised><Article PubModel="Print"><Journal><ISSN IssnType="Print">0885-3185</ISSN><JournalIssue CitedMedium="Print"><Volume>17</Volume><Issue>6</Issue><PubDate><Year>2002</Year><Month>Nov</Month></PubDate></JournalIssue><Title>Movement disorders : official journal of the Movement Disorder Society</Title><ISOAbbreviation>Mov. 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