Pisa syndrome without neuroleptic exposure in a patient with Parkinson's disease: case report.
Identifieur interne : 002E72 ( PubMed/Curation ); précédent : 002E71; suivant : 002E73Pisa syndrome without neuroleptic exposure in a patient with Parkinson's disease: case report.
Auteurs : Mattia Gambarin [Italie] ; Angelo Antonini ; Giuseppe Moretto ; Paolo Bovi ; Silvia Romito ; Antonio Fiaschi ; Michele TinazziSource :
- Movement disorders : official journal of the Movement Disorder Society [ 0885-3185 ] ; 2006.
English descriptors
- KwdEn :
- Aged, Antiparkinson Agents (administration & dosage), Antiparkinson Agents (adverse effects), Antipsychotic Agents (administration & dosage), Cerebral Cortex (physiopathology), Corpus Striatum (physiopathology), Dopamine Plasma Membrane Transport Proteins (physiology), Dose-Response Relationship, Drug, Drug Administration Schedule, Dystonia Musculorum Deformans (diagnosis), Dystonia Musculorum Deformans (physiopathology), Female, Humans, Levodopa (administration & dosage), Levodopa (adverse effects), Parkinson Disease (diagnosis), Parkinson Disease (drug therapy), Parkinson Disease (physiopathology), Syndrome, Tomography, Emission-Computed, Single-Photon.
- MESH :
- chemical , administration & dosage : Antiparkinson Agents, Antipsychotic Agents, Levodopa.
- chemical , adverse effects : Antiparkinson Agents, Levodopa.
- chemical , physiology : Dopamine Plasma Membrane Transport Proteins.
- diagnosis : Dystonia Musculorum Deformans, Parkinson Disease.
- drug therapy : Parkinson Disease.
- physiopathology : Cerebral Cortex, Corpus Striatum, Dystonia Musculorum Deformans, Parkinson Disease.
- Aged, Dose-Response Relationship, Drug, Drug Administration Schedule, Female, Humans, Syndrome, Tomography, Emission-Computed, Single-Photon.
Abstract
We report on a patient affected by Parkinson's disease who developed over a period of a few weeks a tonic deviation of her head, neck, and trunk fitting the typical description of Pisa syndrome (PS). This patient was under stable levodopa and pramipexole treatment and had never been exposed to any psychotropic or antiemetic drugs before or at the time she developed the postural abnormality. Because dopamine transporter imaging revealed bilateral and symmetrical reduction of striatal uptake, we suggest that PS is not primarily related to side differences in dopaminergic denervation or drug exposure.
DOI: 10.1002/mds.20711
PubMed: 16161148
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pubmed:16161148Le document en format XML
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<author><name sortKey="Antonini, Angelo" sort="Antonini, Angelo" uniqKey="Antonini A" first="Angelo" last="Antonini">Angelo Antonini</name>
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<term>Antipsychotic Agents (administration & dosage)</term>
<term>Cerebral Cortex (physiopathology)</term>
<term>Corpus Striatum (physiopathology)</term>
<term>Dopamine Plasma Membrane Transport Proteins (physiology)</term>
<term>Dose-Response Relationship, Drug</term>
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<front><div type="abstract" xml:lang="en">We report on a patient affected by Parkinson's disease who developed over a period of a few weeks a tonic deviation of her head, neck, and trunk fitting the typical description of Pisa syndrome (PS). This patient was under stable levodopa and pramipexole treatment and had never been exposed to any psychotropic or antiemetic drugs before or at the time she developed the postural abnormality. Because dopamine transporter imaging revealed bilateral and symmetrical reduction of striatal uptake, we suggest that PS is not primarily related to side differences in dopaminergic denervation or drug exposure.</div>
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<Abstract><AbstractText>We report on a patient affected by Parkinson's disease who developed over a period of a few weeks a tonic deviation of her head, neck, and trunk fitting the typical description of Pisa syndrome (PS). This patient was under stable levodopa and pramipexole treatment and had never been exposed to any psychotropic or antiemetic drugs before or at the time she developed the postural abnormality. Because dopamine transporter imaging revealed bilateral and symmetrical reduction of striatal uptake, we suggest that PS is not primarily related to side differences in dopaminergic denervation or drug exposure.</AbstractText>
<CopyrightInformation>Copyright (c) 2005 Movement Disorder Society.</CopyrightInformation>
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