Physiology of human posthypoxic myoclonus.
Identifieur interne : 003F96 ( PubMed/Corpus ); précédent : 003F95; suivant : 003F97Physiology of human posthypoxic myoclonus.
Auteurs : M. HallettSource :
- Movement disorders : official journal of the Movement Disorder Society [ 0885-3185 ] ; 2000.
English descriptors
- KwdEn :
- Animals, Brain Stem (physiopathology), Cerebral Cortex (physiopathology), Disease Models, Animal, Electroencephalography, Electromyography, Epilepsies, Myoclonic (physiopathology), Humans, Hypoxia, Brain (physiopathology), Myoclonus (physiopathology), Reflex, Abnormal (physiology), Reticular Formation (physiopathology).
- MESH :
- physiology : Reflex, Abnormal.
- physiopathology : Brain Stem, Cerebral Cortex, Epilepsies, Myoclonic, Hypoxia, Brain, Myoclonus, Reticular Formation.
- Animals, Disease Models, Animal, Electroencephalography, Electromyography, Humans.
Abstract
There are two types of posthypoxic myoclonus, acute and chronic. The acute type has not been intensively studied but is likely to be brain stem in origin. Chronic posthypoxic myoclonus is most commonly cortical reflex myoclonus, but reticular reflex myoclonus and exaggerated startle may also occur. These three conditions have characteristic physiological features. In assessing the relevance of an animal model, it would be appropriate to identify the myoclonus with one of these three patterns.
PubMed: 10755266
Links to Exploration step
pubmed:10755266Le document en format XML
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Hallett</name><affiliation><nlm:affiliation>Human Motor Control Section, NINDS, NIH, Bethesda, Maryland 20892-1428, USA.</nlm:affiliation></affiliation></author></analytic><series><title level="j">Movement disorders : official journal of the Movement Disorder Society</title><idno type="ISSN">0885-3185</idno><imprint><date when="2000" type="published">2000</date></imprint></series></biblStruct></sourceDesc></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Animals</term><term>Brain Stem (physiopathology)</term><term>Cerebral Cortex (physiopathology)</term><term>Disease Models, Animal</term><term>Electroencephalography</term><term>Electromyography</term><term>Epilepsies, Myoclonic (physiopathology)</term><term>Humans</term><term>Hypoxia, Brain (physiopathology)</term><term>Myoclonus (physiopathology)</term><term>Reflex, Abnormal (physiology)</term><term>Reticular Formation (physiopathology)</term></keywords><keywords scheme="MESH" qualifier="physiology" xml:lang="en"><term>Reflex, Abnormal</term></keywords><keywords scheme="MESH" qualifier="physiopathology" xml:lang="en"><term>Brain Stem</term><term>Cerebral Cortex</term><term>Epilepsies, Myoclonic</term><term>Hypoxia, Brain</term><term>Myoclonus</term><term>Reticular Formation</term></keywords><keywords scheme="MESH" xml:lang="en"><term>Animals</term><term>Disease Models, Animal</term><term>Electroencephalography</term><term>Electromyography</term><term>Humans</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">There are two types of posthypoxic myoclonus, acute and chronic. The acute type has not been intensively studied but is likely to be brain stem in origin. Chronic posthypoxic myoclonus is most commonly cortical reflex myoclonus, but reticular reflex myoclonus and exaggerated startle may also occur. These three conditions have characteristic physiological features. In assessing the relevance of an animal model, it would be appropriate to identify the myoclonus with one of these three patterns.</div></front></TEI><pubmed><MedlineCitation Owner="NLM" Status="MEDLINE"><PMID Version="1">10755266</PMID><DateCreated><Year>2000</Year><Month>05</Month><Day>25</Day></DateCreated><DateCompleted><Year>2000</Year><Month>05</Month><Day>25</Day></DateCompleted><DateRevised><Year>2005</Year><Month>11</Month><Day>16</Day></DateRevised><Article PubModel="Print"><Journal><ISSN IssnType="Print">0885-3185</ISSN><JournalIssue CitedMedium="Print"><Volume>15 Suppl 1</Volume><PubDate><Year>2000</Year></PubDate></JournalIssue><Title>Movement disorders : official journal of the Movement Disorder Society</Title><ISOAbbreviation>Mov. Disord.</ISOAbbreviation></Journal><ArticleTitle>Physiology of human posthypoxic myoclonus.</ArticleTitle><Pagination><MedlinePgn>8-13</MedlinePgn></Pagination><Abstract><AbstractText>There are two types of posthypoxic myoclonus, acute and chronic. The acute type has not been intensively studied but is likely to be brain stem in origin. Chronic posthypoxic myoclonus is most commonly cortical reflex myoclonus, but reticular reflex myoclonus and exaggerated startle may also occur. These three conditions have characteristic physiological features. In assessing the relevance of an animal model, it would be appropriate to identify the myoclonus with one of these three patterns.</AbstractText></Abstract><AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Hallett</LastName><ForeName>M</ForeName><Initials>M</Initials><AffiliationInfo><Affiliation>Human Motor Control Section, NINDS, NIH, Bethesda, Maryland 20892-1428, USA.</Affiliation></AffiliationInfo></Author></AuthorList><Language>eng</Language><PublicationTypeList><PublicationType UI="D016428">Journal Article</PublicationType><PublicationType UI="D016454">Review</PublicationType></PublicationTypeList></Article><MedlineJournalInfo><Country>UNITED STATES</Country><MedlineTA>Mov Disord</MedlineTA><NlmUniqueID>8610688</NlmUniqueID><ISSNLinking>0885-3185</ISSNLinking></MedlineJournalInfo><CitationSubset>IM</CitationSubset><MeshHeadingList><MeshHeading><DescriptorName MajorTopicYN="N" UI="D000818">Animals</DescriptorName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D001933">Brain Stem</DescriptorName><QualifierName MajorTopicYN="N" UI="Q000503">physiopathology</QualifierName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D002540">Cerebral Cortex</DescriptorName><QualifierName MajorTopicYN="N" UI="Q000503">physiopathology</QualifierName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D004195">Disease Models, Animal</DescriptorName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D004569">Electroencephalography</DescriptorName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D004576">Electromyography</DescriptorName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D004831">Epilepsies, Myoclonic</DescriptorName><QualifierName MajorTopicYN="Y" UI="Q000503">physiopathology</QualifierName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D006801">Humans</DescriptorName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D002534">Hypoxia, Brain</DescriptorName><QualifierName MajorTopicYN="Y" UI="Q000503">physiopathology</QualifierName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D009207">Myoclonus</DescriptorName><QualifierName MajorTopicYN="Y" UI="Q000503">physiopathology</QualifierName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D012021">Reflex, Abnormal</DescriptorName><QualifierName MajorTopicYN="N" UI="Q000502">physiology</QualifierName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D012154">Reticular Formation</DescriptorName><QualifierName MajorTopicYN="N" UI="Q000503">physiopathology</QualifierName></MeshHeading></MeshHeadingList><NumberOfReferences>30</NumberOfReferences></MedlineCitation><PubmedData><History><PubMedPubDate PubStatus="pubmed"><Year>2000</Year><Month>4</Month><Day>8</Day><Hour>9</Hour><Minute>0</Minute></PubMedPubDate><PubMedPubDate PubStatus="medline"><Year>2000</Year><Month>6</Month><Day>8</Day><Hour>9</Hour><Minute>0</Minute></PubMedPubDate><PubMedPubDate PubStatus="entrez"><Year>2000</Year><Month>4</Month><Day>8</Day><Hour>9</Hour><Minute>0</Minute></PubMedPubDate></History><PublicationStatus>ppublish</PublicationStatus><ArticleIdList><ArticleId IdType="pubmed">10755266</ArticleId></ArticleIdList></PubmedData></pubmed></record>Pour manipuler ce document sous Unix (Dilib)
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