Intracortical inhibition is reduced in a patient with a lesion in the posterolateral thalamus
Identifieur interne : 002834 ( PascalFrancis/Corpus ); précédent : 002833; suivant : 002835Intracortical inhibition is reduced in a patient with a lesion in the posterolateral thalamus
Auteurs : Alexander Münchau ; Michael Orth ; John C. Rothwell ; Vincenzo Di Lazzaro ; Antonio Oliviero ; Paolo Profice ; Pietro Tonali ; Peter P. Pramstaller ; Kailash P. BhatiaSource :
- Movement disorders [ 0885-3185 ] ; 2002.
Descripteurs français
- Pascal (Inist)
English descriptors
- KwdEn :
Abstract
We describe a patient who developed a complex movement disorder following an ischemic lesion in the right posterolateral thalamus. Transcranial magnetic stimulation showed a shortening of the cortical silent period and deficient cortico-cortical inhibition using paired magnetic pulses on the affected side, indicating reduced effectiveness of intracortical inhibitory mechanisms.
Notice en format standard (ISO 2709)
Pour connaître la documentation sur le format Inist Standard.
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Format Inist (serveur)
NO : | PASCAL 02-0204641 INIST |
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ET : | Intracortical inhibition is reduced in a patient with a lesion in the posterolateral thalamus |
AU : | MÜNCHAU (Alexander); ORTH (Michael); ROTHWELL (John C.); DI LAZZARO (Vincenzo); OLIVIERO (Antonio); PROFICE (Paolo); TONALI (Pietro); PRAMSTALLER (Peter P.); BHATIA (Kailash P.) |
AF : | Institute of Neurology, The National Hospital for Neurology and Neurosurgery, Queen Square/London/Royaume-Uni (1 aut., 2 aut., 9 aut.); MRC Human Movement and Balance Unit, The National Hospital for Neurology and Neurosurgery, Queen Square/London/Royaume-Uni (3 aut.); Istitutio di Neurologia, Universita Cattolica/Rome/Italie (4 aut., 5 aut., 6 aut., 7 aut.); Divisione Neurologica Ospedale Regionale Bolzano/Italie (8 aut.) |
DT : | Publication en série; Niveau analytique |
SO : | Movement disorders; ISSN 0885-3185; Etats-Unis; Da. 2002; Vol. 17; No. 1; Pp. 208-212; Bibl. 33 ref. |
LA : | Anglais |
EA : | We describe a patient who developed a complex movement disorder following an ischemic lesion in the right posterolateral thalamus. Transcranial magnetic stimulation showed a shortening of the cortical silent period and deficient cortico-cortical inhibition using paired magnetic pulses on the affected side, indicating reduced effectiveness of intracortical inhibitory mechanisms. |
CC : | 002B16B |
FD : | Ramollissement cérébral; Thalamus; Mouvement involontaire; Stimulus magnétique; Voie transcrânienne; Etude cas; Complication; Pathogénie; Adulte; Mâle |
FG : | Homme; Système nerveux pathologie; Système nerveux central pathologie; Encéphale pathologie; Cérébrovasculaire pathologie; Appareil circulatoire pathologie; Vaisseau sanguin pathologie; Trouble neurologique; Electrodiagnostic |
ED : | Cerebral infarction; Thalamus; Involuntary movement; Magnetic stimulus; Transcranial route; Case study; Complication; Pathogenesis; Adult; Male |
EG : | Human; Nervous system diseases; Central nervous system disease; Cerebral disorder; Cerebrovascular disease; Cardiovascular disease; Vascular disease; Neurological disorder; Electrodiagnosis |
SD : | Infarto cerebral; Tálamo; Movimiento involuntario; Estímulo magnético; Vía transcraneana; Estudio caso; Complicación; Patogenia; Adulto; Macho |
LO : | INIST-20953.354000100147290390 |
ID : | 02-0204641 |
Links to Exploration step
Pascal:02-0204641Le document en format XML
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<front><div type="abstract" xml:lang="en">We describe a patient who developed a complex movement disorder following an ischemic lesion in the right posterolateral thalamus. Transcranial magnetic stimulation showed a shortening of the cortical silent period and deficient cortico-cortical inhibition using paired magnetic pulses on the affected side, indicating reduced effectiveness of intracortical inhibitory mechanisms.</div>
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</fC07>
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<s5>39</s5>
</fC07>
<fC07 i1="04" i2="X" l="SPA"><s0>Encéfalo patología</s0>
<s5>39</s5>
</fC07>
<fC07 i1="05" i2="X" l="FRE"><s0>Cérébrovasculaire pathologie</s0>
<s5>40</s5>
</fC07>
<fC07 i1="05" i2="X" l="ENG"><s0>Cerebrovascular disease</s0>
<s5>40</s5>
</fC07>
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<s5>40</s5>
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<fC07 i1="06" i2="X" l="FRE"><s0>Appareil circulatoire pathologie</s0>
<s5>41</s5>
</fC07>
<fC07 i1="06" i2="X" l="ENG"><s0>Cardiovascular disease</s0>
<s5>41</s5>
</fC07>
<fC07 i1="06" i2="X" l="SPA"><s0>Aparato circulatorio patología</s0>
<s5>41</s5>
</fC07>
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<s5>42</s5>
</fC07>
<fC07 i1="07" i2="X" l="ENG"><s0>Vascular disease</s0>
<s5>42</s5>
</fC07>
<fC07 i1="07" i2="X" l="SPA"><s0>Vaso sanguíneo patología</s0>
<s5>42</s5>
</fC07>
<fC07 i1="08" i2="X" l="FRE"><s0>Trouble neurologique</s0>
<s5>45</s5>
</fC07>
<fC07 i1="08" i2="X" l="ENG"><s0>Neurological disorder</s0>
<s5>45</s5>
</fC07>
<fC07 i1="08" i2="X" l="SPA"><s0>Trastorno neurológico</s0>
<s5>45</s5>
</fC07>
<fC07 i1="09" i2="X" l="FRE"><s0>Electrodiagnostic</s0>
<s5>53</s5>
</fC07>
<fC07 i1="09" i2="X" l="ENG"><s0>Electrodiagnosis</s0>
<s5>53</s5>
</fC07>
<fC07 i1="09" i2="X" l="SPA"><s0>Electrodiagnóstico</s0>
<s5>53</s5>
</fC07>
<fN21><s1>119</s1>
</fN21>
<fN82><s1>PSI</s1>
</fN82>
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<server><NO>PASCAL 02-0204641 INIST</NO>
<ET>Intracortical inhibition is reduced in a patient with a lesion in the posterolateral thalamus</ET>
<AU>MÜNCHAU (Alexander); ORTH (Michael); ROTHWELL (John C.); DI LAZZARO (Vincenzo); OLIVIERO (Antonio); PROFICE (Paolo); TONALI (Pietro); PRAMSTALLER (Peter P.); BHATIA (Kailash P.)</AU>
<AF>Institute of Neurology, The National Hospital for Neurology and Neurosurgery, Queen Square/London/Royaume-Uni (1 aut., 2 aut., 9 aut.); MRC Human Movement and Balance Unit, The National Hospital for Neurology and Neurosurgery, Queen Square/London/Royaume-Uni (3 aut.); Istitutio di Neurologia, Universita Cattolica/Rome/Italie (4 aut., 5 aut., 6 aut., 7 aut.); Divisione Neurologica Ospedale Regionale Bolzano/Italie (8 aut.)</AF>
<DT>Publication en série; Niveau analytique</DT>
<SO>Movement disorders; ISSN 0885-3185; Etats-Unis; Da. 2002; Vol. 17; No. 1; Pp. 208-212; Bibl. 33 ref.</SO>
<LA>Anglais</LA>
<EA>We describe a patient who developed a complex movement disorder following an ischemic lesion in the right posterolateral thalamus. Transcranial magnetic stimulation showed a shortening of the cortical silent period and deficient cortico-cortical inhibition using paired magnetic pulses on the affected side, indicating reduced effectiveness of intracortical inhibitory mechanisms.</EA>
<CC>002B16B</CC>
<FD>Ramollissement cérébral; Thalamus; Mouvement involontaire; Stimulus magnétique; Voie transcrânienne; Etude cas; Complication; Pathogénie; Adulte; Mâle</FD>
<FG>Homme; Système nerveux pathologie; Système nerveux central pathologie; Encéphale pathologie; Cérébrovasculaire pathologie; Appareil circulatoire pathologie; Vaisseau sanguin pathologie; Trouble neurologique; Electrodiagnostic</FG>
<ED>Cerebral infarction; Thalamus; Involuntary movement; Magnetic stimulus; Transcranial route; Case study; Complication; Pathogenesis; Adult; Male</ED>
<EG>Human; Nervous system diseases; Central nervous system disease; Cerebral disorder; Cerebrovascular disease; Cardiovascular disease; Vascular disease; Neurological disorder; Electrodiagnosis</EG>
<SD>Infarto cerebral; Tálamo; Movimiento involuntario; Estímulo magnético; Vía transcraneana; Estudio caso; Complicación; Patogenia; Adulto; Macho</SD>
<LO>INIST-20953.354000100147290390</LO>
<ID>02-0204641</ID>
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