Anatamopathological spectrum of tauopathies
Identifieur interne :
002386 ( PascalFrancis/Corpus );
précédent :
002385;
suivant :
002387
Anatamopathological spectrum of tauopathies
Auteurs : Tamas Revesz ;
Janice L. HoltonSource :
-
Movement disorders [ 0885-3185 ] ; 2003.
RBID : Pascal:04-0080429
Descripteurs français
English descriptors
Abstract
The presence of tau-positive intraneuronal filamentous inclusions with or without additional inclusions in glial cells has been recognised as a major neuropathological feature in a significant group of neurodegenerative diseases, which are described as tauopathies. In one category of such diseases, the neuronal inclusions occur in association with extracellular deposition of a second aggregated protein (secondary tauopathies), whereas in another, the filamentous inclusions composed of tau are the sole neuropathological abnormality (primary tauopathies). Genetic studies of tauopathies in general, and in frontotemporal dementia with parkinsonism linked to chromosome 17 in particular, have significantly contributed to our knowledge about the pathogenesis not only of rare hereditary conditions but also of other more common diseases such as Alzheimer's disease and progressive supranuclear palsy.
Notice en format standard (ISO 2709)
Pour connaître la documentation sur le format Inist Standard.
| pA |
| A01 | 01 | 1 | | @0 0885-3185 |
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| A03 | | 1 | | @0 Mov. disord. |
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| A05 | | | | @2 18 |
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| A06 | | | | @2 6 @3 SUP |
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| A08 | 01 | 1 | ENG | @1 Anatamopathological spectrum of tauopathies |
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| A09 | 01 | 1 | ENG | @1 Parkinsonism and Dementia: Synucleinopathies, Tauopathies, and Beyond. Proceedings of the "Movement" Disorder Society-European Division-Sponsored Workshop, Istanbul, Turkey, 9-11 May 2002 |
|---|
| A11 | 01 | 1 | | @1 REVESZ (Tamas) |
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| A11 | 02 | 1 | | @1 HOLTON (Janice L.) |
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| A14 | 01 | | | @1 Queen Square Brain Bank, Department of Molecular Neuroscience and Division of Neuropathology, Institute of Neurology, University College London @2 London @3 GBR @Z 1 aut. @Z 2 aut. |
|---|
| A18 | 01 | 1 | | @1 "Movement" Disorder Society. European Division @2 Milwaukee, Wisconsin @3 USA @9 patr. |
|---|
| A20 | | | | @2 S13-S20 |
|---|
| A21 | | | | @1 2003 |
|---|
| A23 | 01 | | | @0 ENG |
|---|
| A43 | 01 | | | @1 INIST @2 20953 @5 354000113058290020 |
|---|
| A44 | | | | @0 0000 @1 © 2004 INIST-CNRS. All rights reserved. |
|---|
| A45 | | | | @0 71 ref. |
|---|
| A47 | 01 | 1 | | @0 04-0080429 |
|---|
| A60 | | | | @1 P @2 C |
|---|
| A61 | | | | @0 A |
|---|
| A64 | 01 | 1 | | @0 Movement disorders |
|---|
| A66 | 01 | | | @0 USA |
|---|
| C01 | 01 | | ENG | @0 The presence of tau-positive intraneuronal filamentous inclusions with or without additional inclusions in glial cells has been recognised as a major neuropathological feature in a significant group of neurodegenerative diseases, which are described as tauopathies. In one category of such diseases, the neuronal inclusions occur in association with extracellular deposition of a second aggregated protein (secondary tauopathies), whereas in another, the filamentous inclusions composed of tau are the sole neuropathological abnormality (primary tauopathies). Genetic studies of tauopathies in general, and in frontotemporal dementia with parkinsonism linked to chromosome 17 in particular, have significantly contributed to our knowledge about the pathogenesis not only of rare hereditary conditions but also of other more common diseases such as Alzheimer's disease and progressive supranuclear palsy. |
|---|
| C02 | 01 | X | | @0 002B17G |
|---|
| C03 | 01 | X | FRE | @0 Démence Alzheimer @5 01 |
|---|
| C03 | 01 | X | ENG | @0 Alzheimer disease @5 01 |
|---|
| C03 | 01 | X | SPA | @0 Demencia Alzheimer @5 01 |
|---|
| C03 | 02 | X | FRE | @0 Gerstmann Sträussler Scheinker syndrome @2 NM @5 04 |
|---|
| C03 | 02 | X | ENG | @0 Gerstmann Sträussler Scheinker syndrome @2 NM @5 04 |
|---|
| C03 | 02 | X | SPA | @0 Gerstmann Sträussler Scheinker síndrome @2 NM @5 04 |
|---|
| C03 | 03 | X | FRE | @0 Ophtalmoplégie supranucléaire @5 07 |
|---|
| C03 | 03 | X | ENG | @0 Supranuclear ophthalmoplegia @5 07 |
|---|
| C03 | 03 | X | SPA | @0 Oftalmoplejía supranuclear @5 07 |
|---|
| C03 | 04 | X | FRE | @0 Progressif @5 08 |
|---|
| C03 | 04 | X | ENG | @0 Progressive @5 08 |
|---|
| C03 | 04 | X | SPA | @0 Progresivo @5 08 |
|---|
| C03 | 05 | X | FRE | @0 Démence Pick @5 10 |
|---|
| C03 | 05 | X | ENG | @0 Pick disease @5 10 |
|---|
| C03 | 05 | X | SPA | @0 Demencia Pick @5 10 |
|---|
| C03 | 06 | X | FRE | @0 Protéine tau @5 13 |
|---|
| C03 | 06 | X | ENG | @0 Tau protein @5 13 |
|---|
| C03 | 06 | X | SPA | @0 Proteína tau @5 13 |
|---|
| C03 | 07 | X | FRE | @0 Exploration @5 17 |
|---|
| C03 | 07 | X | ENG | @0 Exploration @5 17 |
|---|
| C03 | 07 | X | SPA | @0 Exploración @5 17 |
|---|
| C03 | 08 | X | FRE | @0 Anatomopathologie @5 18 |
|---|
| C03 | 08 | X | ENG | @0 Anatomic pathology @5 18 |
|---|
| C03 | 08 | X | SPA | @0 Anatomía patológica @5 18 |
|---|
| C03 | 09 | X | FRE | @0 Homme @5 20 |
|---|
| C03 | 09 | X | ENG | @0 Human @5 20 |
|---|
| C03 | 09 | X | SPA | @0 Hombre @5 20 |
|---|
| C03 | 10 | X | FRE | @0 Prion @5 27 |
|---|
| C03 | 10 | X | ENG | @0 Prion @5 27 |
|---|
| C03 | 10 | X | SPA | @0 Prion @5 27 |
|---|
| C07 | 01 | X | FRE | @0 Infection @2 NM |
|---|
| C07 | 01 | X | ENG | @0 Infection @2 NM |
|---|
| C07 | 01 | X | SPA | @0 Infección @2 NM |
|---|
| C07 | 02 | X | FRE | @0 Système nerveux pathologie @5 37 |
|---|
| C07 | 02 | X | ENG | @0 Nervous system diseases @5 37 |
|---|
| C07 | 02 | X | SPA | @0 Sistema nervioso patología @5 37 |
|---|
| C07 | 03 | X | FRE | @0 Système nerveux central pathologie @5 38 |
|---|
| C07 | 03 | X | ENG | @0 Central nervous system disease @5 38 |
|---|
| C07 | 03 | X | SPA | @0 Sistema nervosio central patología @5 38 |
|---|
| C07 | 04 | X | FRE | @0 Encéphale pathologie @5 39 |
|---|
| C07 | 04 | X | ENG | @0 Cerebral disorder @5 39 |
|---|
| C07 | 04 | X | SPA | @0 Encéfalo patología @5 39 |
|---|
| C07 | 05 | X | FRE | @0 Maladie dégénérative @5 40 |
|---|
| C07 | 05 | X | ENG | @0 Degenerative disease @5 40 |
|---|
| C07 | 05 | X | SPA | @0 Enfermedad degenerativa @5 40 |
|---|
| C07 | 06 | X | FRE | @0 Oeil pathologie @5 53 |
|---|
| C07 | 06 | X | ENG | @0 Eye disease @5 53 |
|---|
| C07 | 06 | X | SPA | @0 Ojo patología @5 53 |
|---|
| C07 | 07 | X | FRE | @0 Oculomotricité syndrome @5 54 |
|---|
| C07 | 07 | X | ENG | @0 Oculomotor syndrome @5 54 |
|---|
| C07 | 07 | X | SPA | @0 Oculomotricidad síndrome @5 54 |
|---|
| C07 | 08 | X | FRE | @0 Tronc cérébral syndrome @5 57 |
|---|
| C07 | 08 | X | ENG | @0 Brain stem syndrome @5 57 |
|---|
| C07 | 08 | X | SPA | @0 Tallo encefalico sindrome @5 57 |
|---|
| N21 | | | | @1 054 |
|---|
| N82 | | | | @1 PSI |
|---|
|
|---|
| pR |
| A30 | 01 | 1 | ENG | @1 Parkinsonism and Dementia: Sinucleinopathies, Tauopathies, and Beyond. Workshop @3 Istanbul TUR @4 2002-05-09 |
|---|
|
|---|
Format Inist (serveur)
| NO : | PASCAL 04-0080429 INIST |
|---|
| ET : | Anatamopathological spectrum of tauopathies |
|---|
| AU : | REVESZ (Tamas); HOLTON (Janice L.) |
|---|
| AF : | Queen Square Brain Bank, Department of Molecular Neuroscience and Division of Neuropathology, Institute of Neurology, University College London/London/Royaume-Uni (1 aut., 2 aut.) |
|---|
| DT : | Publication en série; Congrès; Niveau analytique |
|---|
| SO : | Movement disorders; ISSN 0885-3185; Etats-Unis; Da. 2003; Vol. 18; No. 6 SUP; S13-S20; Bibl. 71 ref. |
|---|
| LA : | Anglais |
|---|
| EA : | The presence of tau-positive intraneuronal filamentous inclusions with or without additional inclusions in glial cells has been recognised as a major neuropathological feature in a significant group of neurodegenerative diseases, which are described as tauopathies. In one category of such diseases, the neuronal inclusions occur in association with extracellular deposition of a second aggregated protein (secondary tauopathies), whereas in another, the filamentous inclusions composed of tau are the sole neuropathological abnormality (primary tauopathies). Genetic studies of tauopathies in general, and in frontotemporal dementia with parkinsonism linked to chromosome 17 in particular, have significantly contributed to our knowledge about the pathogenesis not only of rare hereditary conditions but also of other more common diseases such as Alzheimer's disease and progressive supranuclear palsy. |
|---|
| CC : | 002B17G |
|---|
| FD : | Démence Alzheimer; Gerstmann Sträussler Scheinker syndrome; Ophtalmoplégie supranucléaire; Progressif; Démence Pick; Protéine tau; Exploration; Anatomopathologie; Homme; Prion |
|---|
| FG : | Infection; Système nerveux pathologie; Système nerveux central pathologie; Encéphale pathologie; Maladie dégénérative; Oeil pathologie; Oculomotricité syndrome; Tronc cérébral syndrome |
|---|
| ED : | Alzheimer disease; Gerstmann Sträussler Scheinker syndrome; Supranuclear ophthalmoplegia; Progressive; Pick disease; Tau protein; Exploration; Anatomic pathology; Human; Prion |
|---|
| EG : | Infection; Nervous system diseases; Central nervous system disease; Cerebral disorder; Degenerative disease; Eye disease; Oculomotor syndrome; Brain stem syndrome |
|---|
| SD : | Demencia Alzheimer; Gerstmann Sträussler Scheinker síndrome; Oftalmoplejía supranuclear; Progresivo; Demencia Pick; Proteína tau; Exploración; Anatomía patológica; Hombre; Prion |
|---|
| LO : | INIST-20953.354000113058290020 |
|---|
| ID : | 04-0080429 |
|---|
Links to Exploration step
Pascal:04-0080429
Le document en format XML
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<term>Démence Pick</term>
<term>Protéine tau</term>
<term>Exploration</term>
<term>Anatomopathologie</term>
<term>Homme</term>
<term>Prion</term>
</keywords><front><div type="abstract" xml:lang="en">The presence of tau-positive intraneuronal filamentous inclusions with or without additional inclusions in glial cells has been recognised as a major neuropathological feature in a significant group of neurodegenerative diseases, which are described as tauopathies. In one category of such diseases, the neuronal inclusions occur in association with extracellular deposition of a second aggregated protein (secondary tauopathies), whereas in another, the filamentous inclusions composed of tau are the sole neuropathological abnormality (primary tauopathies). Genetic studies of tauopathies in general, and in frontotemporal dementia with parkinsonism linked to chromosome 17 in particular, have significantly contributed to our knowledge about the pathogenesis not only of rare hereditary conditions but also of other more common diseases such as Alzheimer's disease and progressive supranuclear palsy.</div>
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</fA03><fA06><s2>6</s2>
<s3>SUP</s3>
</fA06><fA08 i1="01" i2="1" l="ENG"><s1>Anatamopathological spectrum of tauopathies</s1>
</fA08><fA09 i1="01" i2="1" l="ENG"><s1>Parkinsonism and Dementia: Synucleinopathies, Tauopathies, and Beyond. Proceedings of the "Movement" Disorder Society-European Division-Sponsored Workshop, Istanbul, Turkey, 9-11 May 2002</s1>
</fA09><fA11 i1="01" i2="1"><s1>REVESZ (Tamas)</s1>
</fA11><fA11 i1="02" i2="1"><s1>HOLTON (Janice L.)</s1>
</fA11><fA14 i1="01"><s1>Queen Square Brain Bank, Department of Molecular Neuroscience and Division of Neuropathology, Institute of Neurology, University College London</s1>
<s2>London</s2>
<s3>GBR</s3>
<sZ>1 aut.</sZ>
<sZ>2 aut.</sZ>
</fA14><fA18 i1="01" i2="1"><s1>"Movement" Disorder Society. European Division</s1>
<s2>Milwaukee, Wisconsin</s2>
<s3>USA</s3>
<s9>patr.</s9>
</fA18><fA20><s2>S13-S20</s2>
</fA20><fA21><s1>2003</s1>
</fA21><fA23 i1="01"><s0>ENG</s0>
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<s2>20953</s2>
<s5>354000113058290020</s5>
</fA43><fA44><s0>0000</s0>
<s1>© 2004 INIST-CNRS. All rights reserved.</s1>
</fA44><fA45><s0>71 ref.</s0>
</fA45><fA47 i1="01" i2="1"><s0>04-0080429</s0>
</fA47><fA60><s1>P</s1>
<s2>C</s2>
</fA60><fA64 i1="01" i2="1"><s0>Movement disorders</s0>
</fA64><fA66 i1="01"><s0>USA</s0>
</fA66><fC01 i1="01" l="ENG"><s0>The presence of tau-positive intraneuronal filamentous inclusions with or without additional inclusions in glial cells has been recognised as a major neuropathological feature in a significant group of neurodegenerative diseases, which are described as tauopathies. In one category of such diseases, the neuronal inclusions occur in association with extracellular deposition of a second aggregated protein (secondary tauopathies), whereas in another, the filamentous inclusions composed of tau are the sole neuropathological abnormality (primary tauopathies). Genetic studies of tauopathies in general, and in frontotemporal dementia with parkinsonism linked to chromosome 17 in particular, have significantly contributed to our knowledge about the pathogenesis not only of rare hereditary conditions but also of other more common diseases such as Alzheimer's disease and progressive supranuclear palsy.</s0>
</fC01><fC02 i1="01" i2="X"><s0>002B17G</s0>
</fC02><fC03 i1="01" i2="X" l="FRE"><s0>Démence Alzheimer</s0>
<s5>01</s5>
</fC03><fC03 i1="01" i2="X" l="ENG"><s0>Alzheimer disease</s0>
<s5>01</s5>
</fC03><fC03 i1="01" i2="X" l="SPA"><s0>Demencia Alzheimer</s0>
<s5>01</s5>
</fC03><fC03 i1="02" i2="X" l="FRE"><s0>Gerstmann Sträussler Scheinker syndrome</s0>
<s2>NM</s2>
<s5>04</s5>
</fC03><fC03 i1="02" i2="X" l="ENG"><s0>Gerstmann Sträussler Scheinker syndrome</s0>
<s2>NM</s2>
<s5>04</s5>
</fC03><fC03 i1="02" i2="X" l="SPA"><s0>Gerstmann Sträussler Scheinker síndrome</s0>
<s2>NM</s2>
<s5>04</s5>
</fC03><fC03 i1="03" i2="X" l="FRE"><s0>Ophtalmoplégie supranucléaire</s0>
<s5>07</s5>
</fC03><fC03 i1="03" i2="X" l="ENG"><s0>Supranuclear ophthalmoplegia</s0>
<s5>07</s5>
</fC03><fC03 i1="03" i2="X" l="SPA"><s0>Oftalmoplejía supranuclear</s0>
<s5>07</s5>
</fC03><fC03 i1="04" i2="X" l="FRE"><s0>Progressif</s0>
<s5>08</s5>
</fC03><fC03 i1="04" i2="X" l="ENG"><s0>Progressive</s0>
<s5>08</s5>
</fC03><fC03 i1="04" i2="X" l="SPA"><s0>Progresivo</s0>
<s5>08</s5>
</fC03><fC03 i1="05" i2="X" l="FRE"><s0>Démence Pick</s0>
<s5>10</s5>
</fC03><fC03 i1="05" i2="X" l="ENG"><s0>Pick disease</s0>
<s5>10</s5>
</fC03><fC03 i1="05" i2="X" l="SPA"><s0>Demencia Pick</s0>
<s5>10</s5>
</fC03><fC03 i1="06" i2="X" l="FRE"><s0>Protéine tau</s0>
<s5>13</s5>
</fC03><fC03 i1="06" i2="X" l="ENG"><s0>Tau protein</s0>
<s5>13</s5>
</fC03><fC03 i1="06" i2="X" l="SPA"><s0>Proteína tau</s0>
<s5>13</s5>
</fC03><fC03 i1="07" i2="X" l="FRE"><s0>Exploration</s0>
<s5>17</s5>
</fC03><fC03 i1="07" i2="X" l="ENG"><s0>Exploration</s0>
<s5>17</s5>
</fC03><fC03 i1="07" i2="X" l="SPA"><s0>Exploración</s0>
<s5>17</s5>
</fC03><fC03 i1="08" i2="X" l="FRE"><s0>Anatomopathologie</s0>
<s5>18</s5>
</fC03><fC03 i1="08" i2="X" l="ENG"><s0>Anatomic pathology</s0>
<s5>18</s5>
</fC03><fC03 i1="08" i2="X" l="SPA"><s0>Anatomía patológica</s0>
<s5>18</s5>
</fC03><fC03 i1="09" i2="X" l="FRE"><s0>Homme</s0>
<s5>20</s5>
</fC03><fC03 i1="09" i2="X" l="ENG"><s0>Human</s0>
<s5>20</s5>
</fC03><fC03 i1="09" i2="X" l="SPA"><s0>Hombre</s0>
<s5>20</s5>
</fC03><fC03 i1="10" i2="X" l="FRE"><s0>Prion</s0>
<s5>27</s5>
</fC03><fC03 i1="10" i2="X" l="ENG"><s0>Prion</s0>
<s5>27</s5>
</fC03><fC03 i1="10" i2="X" l="SPA"><s0>Prion</s0>
<s5>27</s5>
</fC03><fC07 i1="01" i2="X" l="FRE"><s0>Infection</s0>
<s2>NM</s2>
</fC07><fC07 i1="01" i2="X" l="ENG"><s0>Infection</s0>
<s2>NM</s2>
</fC07><fC07 i1="01" i2="X" l="SPA"><s0>Infección</s0>
<s2>NM</s2>
</fC07><fC07 i1="02" i2="X" l="FRE"><s0>Système nerveux pathologie</s0>
<s5>37</s5>
</fC07><fC07 i1="02" i2="X" l="ENG"><s0>Nervous system diseases</s0>
<s5>37</s5>
</fC07><fC07 i1="02" i2="X" l="SPA"><s0>Sistema nervioso patología</s0>
<s5>37</s5>
</fC07><fC07 i1="03" i2="X" l="FRE"><s0>Système nerveux central pathologie</s0>
<s5>38</s5>
</fC07><fC07 i1="03" i2="X" l="ENG"><s0>Central nervous system disease</s0>
<s5>38</s5>
</fC07><fC07 i1="03" i2="X" l="SPA"><s0>Sistema nervosio central patología</s0>
<s5>38</s5>
</fC07><fC07 i1="04" i2="X" l="FRE"><s0>Encéphale pathologie</s0>
<s5>39</s5>
</fC07><fC07 i1="04" i2="X" l="ENG"><s0>Cerebral disorder</s0>
<s5>39</s5>
</fC07><fC07 i1="04" i2="X" l="SPA"><s0>Encéfalo patología</s0>
<s5>39</s5>
</fC07><fC07 i1="05" i2="X" l="FRE"><s0>Maladie dégénérative</s0>
<s5>40</s5>
</fC07><fC07 i1="05" i2="X" l="ENG"><s0>Degenerative disease</s0>
<s5>40</s5>
</fC07><fC07 i1="05" i2="X" l="SPA"><s0>Enfermedad degenerativa</s0>
<s5>40</s5>
</fC07><fC07 i1="06" i2="X" l="FRE"><s0>Oeil pathologie</s0>
<s5>53</s5>
</fC07><fC07 i1="06" i2="X" l="ENG"><s0>Eye disease</s0>
<s5>53</s5>
</fC07><fC07 i1="06" i2="X" l="SPA"><s0>Ojo patología</s0>
<s5>53</s5>
</fC07><fC07 i1="07" i2="X" l="FRE"><s0>Oculomotricité syndrome</s0>
<s5>54</s5>
</fC07><fC07 i1="07" i2="X" l="ENG"><s0>Oculomotor syndrome</s0>
<s5>54</s5>
</fC07><fC07 i1="07" i2="X" l="SPA"><s0>Oculomotricidad síndrome</s0>
<s5>54</s5>
</fC07><fC07 i1="08" i2="X" l="FRE"><s0>Tronc cérébral syndrome</s0>
<s5>57</s5>
</fC07><fC07 i1="08" i2="X" l="ENG"><s0>Brain stem syndrome</s0>
<s5>57</s5>
</fC07><fC07 i1="08" i2="X" l="SPA"><s0>Tallo encefalico sindrome</s0>
<s5>57</s5>
</fC07><fN21><s1>054</s1>
</fN21><fN82><s1>PSI</s1>
</fN82><pR><fA30 i1="01" i2="1" l="ENG"><s1>Parkinsonism and Dementia: Sinucleinopathies, Tauopathies, and Beyond. Workshop</s1>
<s3>Istanbul TUR</s3>
<s4>2002-05-09</s4>
</fA30><server><NO>PASCAL 04-0080429 INIST</NO>
<ET>Anatamopathological spectrum of tauopathies</ET>
<AU>REVESZ (Tamas); HOLTON (Janice L.)</AU>
<AF>Queen Square Brain Bank, Department of Molecular Neuroscience and Division of Neuropathology, Institute of Neurology, University College London/London/Royaume-Uni (1 aut., 2 aut.)</AF>
<DT>Publication en série; Congrès; Niveau analytique</DT>
<SO>Movement disorders; ISSN 0885-3185; Etats-Unis; Da. 2003; Vol. 18; No. 6 SUP; S13-S20; Bibl. 71 ref.</SO>
<LA>Anglais</LA>
<EA>The presence of tau-positive intraneuronal filamentous inclusions with or without additional inclusions in glial cells has been recognised as a major neuropathological feature in a significant group of neurodegenerative diseases, which are described as tauopathies. In one category of such diseases, the neuronal inclusions occur in association with extracellular deposition of a second aggregated protein (secondary tauopathies), whereas in another, the filamentous inclusions composed of tau are the sole neuropathological abnormality (primary tauopathies). Genetic studies of tauopathies in general, and in frontotemporal dementia with parkinsonism linked to chromosome 17 in particular, have significantly contributed to our knowledge about the pathogenesis not only of rare hereditary conditions but also of other more common diseases such as Alzheimer's disease and progressive supranuclear palsy.</EA>
<CC>002B17G</CC>
<FD>Démence Alzheimer; Gerstmann Sträussler Scheinker syndrome; Ophtalmoplégie supranucléaire; Progressif; Démence Pick; Protéine tau; Exploration; Anatomopathologie; Homme; Prion</FD>
<FG>Infection; Système nerveux pathologie; Système nerveux central pathologie; Encéphale pathologie; Maladie dégénérative; Oeil pathologie; Oculomotricité syndrome; Tronc cérébral syndrome</FG>
<ED>Alzheimer disease; Gerstmann Sträussler Scheinker syndrome; Supranuclear ophthalmoplegia; Progressive; Pick disease; Tau protein; Exploration; Anatomic pathology; Human; Prion</ED>
<EG>Infection; Nervous system diseases; Central nervous system disease; Cerebral disorder; Degenerative disease; Eye disease; Oculomotor syndrome; Brain stem syndrome</EG>
<SD>Demencia Alzheimer; Gerstmann Sträussler Scheinker síndrome; Oftalmoplejía supranuclear; Progresivo; Demencia Pick; Proteína tau; Exploración; Anatomía patológica; Hombre; Prion</SD>
<LO>INIST-20953.354000113058290020</LO>
<ID>04-0080429</ID>
</server>
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