Serum from Sydenham's chorea patients modifies intracellular calcium levels in PC12 cells by a complement-independent mechanism
Identifieur interne : 001E40 ( PascalFrancis/Corpus ); précédent : 001E39; suivant : 001E41Serum from Sydenham's chorea patients modifies intracellular calcium levels in PC12 cells by a complement-independent mechanism
Auteurs : Antonio L. Jr Teixeira ; Melissa M. Guimaraes ; Marco Aurélio Romano-Silva ; Francisco CardosoSource :
- Movement disorders [ 0885-3185 ] ; 2005.
Descripteurs français
- Pascal (Inist)
English descriptors
- KwdEn :
Abstract
The proposed pathogenesis of Sydenham's chorea (SC) is an autoantibody-mediated basal ganglia dysfunction. Our study has shown that incubation of PC12 cells with complement-inactivated serum from SC patients was associated with a significant increase in Ca2+ levels evoked by KCl stimulus (mean ± SEM, 341.0 ± 8.7% of fluorescence intensity, arbitrary units) when compared with incubation with control serum (313.8 ± 8.7% of fluorescence intensity, arbitrary units; P = 0.01). The increase in Ca2+ levels determined by SC patients sera correlated directly with the enzyme-linked immunosorbent assay optical density values for anti-basal ganglia antibodies. Our study supports the hypothesis that antibodies against basal ganglia in SC may cause their dysfunction.
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Format Inist (serveur)
NO : | PASCAL 05-0387178 INIST |
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ET : | Serum from Sydenham's chorea patients modifies intracellular calcium levels in PC12 cells by a complement-independent mechanism |
AU : | TEIXEIRA (Antonio L. JR); GUIMARAES (Melissa M.); ROMANO-SILVA (Marco Aurélio); CARDOSO (Francisco) |
AF : | Movement Disorders Clinic, Hospital das Clinicas, Universidade Federal de Minas Gerais/Belo Horizonte/Brésil (1 aut., 4 aut.); Laboratory of Neuropharmacology, Institute of Biological Sciences, Universidade Federal de Minas Gerais/Belo Horizonte/Brésil (2 aut., 3 aut.) |
DT : | Publication en série; Niveau analytique |
SO : | Movement disorders; ISSN 0885-3185; Etats-Unis; Da. 2005; Vol. 20; No. 7; Pp. 843-845; Bibl. 14 ref. |
LA : | Anglais |
EA : | The proposed pathogenesis of Sydenham's chorea (SC) is an autoantibody-mediated basal ganglia dysfunction. Our study has shown that incubation of PC12 cells with complement-inactivated serum from SC patients was associated with a significant increase in Ca2+ levels evoked by KCl stimulus (mean ± SEM, 341.0 ± 8.7% of fluorescence intensity, arbitrary units) when compared with incubation with control serum (313.8 ± 8.7% of fluorescence intensity, arbitrary units; P = 0.01). The increase in Ca2+ levels determined by SC patients sera correlated directly with the enzyme-linked immunosorbent assay optical density values for anti-basal ganglia antibodies. Our study supports the hypothesis that antibodies against basal ganglia in SC may cause their dysfunction. |
CC : | 002B17; 002B17F; 002A25L |
FD : | Système nerveux pathologie; Chorée Sydenham; Streptococcie; Homme; Intracellulaire; Calcium; Complément; Noyau gris central; Streptococcus |
FG : | Bactériose; Infection; Streptococcaceae; Micrococcales; Bactérie; Extrapyramidal syndrome; Maladie inflammatoire; Système nerveux central pathologie; Encéphale pathologie; Système nerveux central |
ED : | Nervous system diseases; Sydenham chorea; Streptococcal infection; Human; Intracellular; Calcium; Complement; Basal ganglion; Streptococcus |
EG : | Bacteriosis; Infection; Streptococcaceae; Micrococcales; Bacteria; Extrapyramidal syndrome; Inflammatory disease; Central nervous system disease; Cerebral disorder; Central nervous system |
SD : | Sistema nervioso patología; Corea Sydenham; Estreptococia; Hombre; Intracelular; Calcio; Complemento; Núcleo basal; Streptococcus |
LO : | INIST-20953.354000132363110100 |
ID : | 05-0387178 |
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Pascal:05-0387178Le document en format XML
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<front><div type="abstract" xml:lang="en">The proposed pathogenesis of Sydenham's chorea (SC) is an autoantibody-mediated basal ganglia dysfunction. Our study has shown that incubation of PC12 cells with complement-inactivated serum from SC patients was associated with a significant increase in Ca<sup>2+</sup>
levels evoked by KCl stimulus (mean ± SEM, 341.0 ± 8.7% of fluorescence intensity, arbitrary units) when compared with incubation with control serum (313.8 ± 8.7% of fluorescence intensity, arbitrary units; P = 0.01). The increase in Ca<sup>2+</sup>
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<server><NO>PASCAL 05-0387178 INIST</NO>
<ET>Serum from Sydenham's chorea patients modifies intracellular calcium levels in PC12 cells by a complement-independent mechanism</ET>
<AU>TEIXEIRA (Antonio L. JR); GUIMARAES (Melissa M.); ROMANO-SILVA (Marco Aurélio); CARDOSO (Francisco)</AU>
<AF>Movement Disorders Clinic, Hospital das Clinicas, Universidade Federal de Minas Gerais/Belo Horizonte/Brésil (1 aut., 4 aut.); Laboratory of Neuropharmacology, Institute of Biological Sciences, Universidade Federal de Minas Gerais/Belo Horizonte/Brésil (2 aut., 3 aut.)</AF>
<DT>Publication en série; Niveau analytique</DT>
<SO>Movement disorders; ISSN 0885-3185; Etats-Unis; Da. 2005; Vol. 20; No. 7; Pp. 843-845; Bibl. 14 ref.</SO>
<LA>Anglais</LA>
<EA>The proposed pathogenesis of Sydenham's chorea (SC) is an autoantibody-mediated basal ganglia dysfunction. Our study has shown that incubation of PC12 cells with complement-inactivated serum from SC patients was associated with a significant increase in Ca<sup>2+</sup>
levels evoked by KCl stimulus (mean ± SEM, 341.0 ± 8.7% of fluorescence intensity, arbitrary units) when compared with incubation with control serum (313.8 ± 8.7% of fluorescence intensity, arbitrary units; P = 0.01). The increase in Ca<sup>2+</sup>
levels determined by SC patients sera correlated directly with the enzyme-linked immunosorbent assay optical density values for anti-basal ganglia antibodies. Our study supports the hypothesis that antibodies against basal ganglia in SC may cause their dysfunction.</EA>
<CC>002B17; 002B17F; 002A25L</CC>
<FD>Système nerveux pathologie; Chorée Sydenham; Streptococcie; Homme; Intracellulaire; Calcium; Complément; Noyau gris central; Streptococcus</FD>
<FG>Bactériose; Infection; Streptococcaceae; Micrococcales; Bactérie; Extrapyramidal syndrome; Maladie inflammatoire; Système nerveux central pathologie; Encéphale pathologie; Système nerveux central</FG>
<ED>Nervous system diseases; Sydenham chorea; Streptococcal infection; Human; Intracellular; Calcium; Complement; Basal ganglion; Streptococcus</ED>
<EG>Bacteriosis; Infection; Streptococcaceae; Micrococcales; Bacteria; Extrapyramidal syndrome; Inflammatory disease; Central nervous system disease; Cerebral disorder; Central nervous system</EG>
<SD>Sistema nervioso patología; Corea Sydenham; Estreptococia; Hombre; Intracelular; Calcio; Complemento; Núcleo basal; Streptococcus</SD>
<LO>INIST-20953.354000132363110100</LO>
<ID>05-0387178</ID>
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