Holmes tremor : Application of modern neuroimaging techniques
Identifieur interne : 001843 ( PascalFrancis/Corpus ); précédent : 001842; suivant : 001844Holmes tremor : Application of modern neuroimaging techniques
Auteurs : Dominic C. Paviour ; H. Rolf J Ger ; Leonora Wilkinson ; Marjan Jahanshahi ; Andrew J. LeesSource :
- Movement disorders [ 0885-3185 ] ; 2006.
Descripteurs français
- Pascal (Inist)
English descriptors
- KwdEn :
Abstract
Holmes tremor has a characteristic rest, intention, and postural component. The syndrome arises as a consequence of a lesion in the upper brainstem and cerebral peduncles, which, it is postulated, interrupts the cerebello-rubrothalamic pathway. Ataxia, ophthalmoplegia, and bradykinesia are associated features. We present a case of Holmes tremor secondary to a midbrain cavernoma. Modern neuroimaging techniques in this case confirm that a combination of damage to the cerebello-rubrothalamic pathway and the nigrostriatal pathway is required for the full Holmes tremor syndrome to occur.
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NO : | PASCAL 07-0090896 INIST |
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ET : | Holmes tremor : Application of modern neuroimaging techniques |
AU : | PAVIOUR (Dominic C.); ROLF JÄGER (H.); WILKINSON (Leonora); JAHANSHAHI (Marjan); LEES (Andrew J.) |
AF : | The Sara Koe PSP Research Centre, The Institute of Neurology, UCL/London/Royaume-Uni (1 aut., 5 aut.); The Lysholm Department of Neuroradiology, National Hospital for Neurology and Neurosurgery, UCLH NHS Foundation Trust/London/Royaume-Uni (2 aut.); The Cognitive-Motor Neuroscience Group, Sobell Department of Motor Neuroscience and Movement Disorders, Institute of Neurology, Queen Square/London/Royaume-Uni (3 aut., 4 aut.); Department of Clinical Neurology, National Hospital for Neurology and Neurosurgery, Queen Square/London/Royaume-Uni (5 aut.) |
DT : | Publication en série; Courte communication, note brève; Niveau analytique |
SO : | Movement disorders; ISSN 0885-3185; Etats-Unis; Da. 2006; Vol. 21; No. 12; Pp. 2260-2262; Bibl. 7 ref. |
LA : | Anglais |
EA : | Holmes tremor has a characteristic rest, intention, and postural component. The syndrome arises as a consequence of a lesion in the upper brainstem and cerebral peduncles, which, it is postulated, interrupts the cerebello-rubrothalamic pathway. Ataxia, ophthalmoplegia, and bradykinesia are associated features. We present a case of Holmes tremor secondary to a midbrain cavernoma. Modern neuroimaging techniques in this case confirm that a combination of damage to the cerebello-rubrothalamic pathway and the nigrostriatal pathway is required for the full Holmes tremor syndrome to occur. |
CC : | 002B17; 002B17A01; 002B24A06 |
FD : | Système nerveux pathologie; Tremblement; Technique |
FG : | Mouvement involontaire; Trouble neurologique |
ED : | Nervous system diseases; Tremor; Technique |
EG : | Involuntary movement; Neurological disorder |
SD : | Sistema nervioso patología; Temblor; Técnica |
LO : | INIST-20953.354000145356790460 |
ID : | 07-0090896 |
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Pascal:07-0090896Le document en format XML
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<front><div type="abstract" xml:lang="en">Holmes tremor has a characteristic rest, intention, and postural component. The syndrome arises as a consequence of a lesion in the upper brainstem and cerebral peduncles, which, it is postulated, interrupts the cerebello-rubrothalamic pathway. Ataxia, ophthalmoplegia, and bradykinesia are associated features. We present a case of Holmes tremor secondary to a midbrain cavernoma. Modern neuroimaging techniques in this case confirm that a combination of damage to the cerebello-rubrothalamic pathway and the nigrostriatal pathway is required for the full Holmes tremor syndrome to occur.</div>
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<AF>The Sara Koe PSP Research Centre, The Institute of Neurology, UCL/London/Royaume-Uni (1 aut., 5 aut.); The Lysholm Department of Neuroradiology, National Hospital for Neurology and Neurosurgery, UCLH NHS Foundation Trust/London/Royaume-Uni (2 aut.); The Cognitive-Motor Neuroscience Group, Sobell Department of Motor Neuroscience and Movement Disorders, Institute of Neurology, Queen Square/London/Royaume-Uni (3 aut., 4 aut.); Department of Clinical Neurology, National Hospital for Neurology and Neurosurgery, Queen Square/London/Royaume-Uni (5 aut.)</AF>
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