MovDisordV3, PascalFrancis, Corpus, bibRecord, 000C76

Imaging Amyloid in Parkinson's Disease Dementia and Dementia with Lewy Bodies with Positron Emission Tomography

Identifieur interne : 000C76 ( PascalFrancis/Corpus ); précédent : 000C75; suivant : 000C77

Imaging Amyloid in Parkinson's Disease Dementia and Dementia with Lewy Bodies with Positron Emission Tomography

Auteurs : David J. Brooks

Source :

Movement disorders [ 0885-3185 ] ; 2009.

RBID : Pascal:10-0038132

Descripteurs français

Pascal (Inist)
- Maladie de Parkinson, Démence à corps de Lewy, Pathologie du système nerveux, Amyloïde, Tomoscintigraphie, Tomographie par émission de positons, Corps Lewy.

English descriptors

KwdEn :
- Amyloid, Emission tomography, Lewy body, Lewy body dementia, Nervous system diseases, Parkinson disease, Positron emission tomography.

Abstract

Although Parkinson's disease with later dementia (PDD) and dementia with Lewy bodies (DLB) are pathologically characterized by the presence of intraneuronal Lewy inclusion bodies, amyloid deposition is also associated to varying degrees with both these disorders. Fibrillar amyloid load can now be quantitated in vivo with positron emission tomography (PET) using imaging biomarkers. Here the reported findings of ¹¹C-PIB PET studies concerning the amyloid load associated with PD and its influence on dementia are reviewed. It is concluded that the presence of amyloid acts to accelerate the dementia process in Lewy body disorders, though has little influence on its nature. Anti-amyloid strategies could be a relevant approach for slowing dementia in a number of DLB and PDD cases.

Notice en format standard (ISO 2709)

Pour connaître la documentation sur le format Inist Standard.

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A03		`1`		`@0 Mov. disord.`
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A08	`01`	`1`	`ENG`	`@1 Imaging Amyloid in Parkinson's Disease Dementia and Dementia with Lewy Bodies with Positron Emission Tomography`
A09	`01`	`1`	`ENG`	`@1 Imaging in Parkinson's Disease`
A11	`01`	`1`		`@1 BROOKS (David J.)`
A12	`01`	`1`		`@1 POEWE (Werner) @9 ed.`
A12	`02`	`1`		`@1 SCHERFLER (Christopher) @9 ed.`
A14	`01`			`@1 MRC Clinical Sciences Centre and Division of Neuroscience, Imperial College @2 London @3 GBR @Z 1 aut.`
A15	`01`			`@1 Department of Neurology, Innsbruck Medical University @2 Innsbruck @3 AUT @Z 1 aut. @Z 2 aut.`
A20				`@2 S742-S747`
A21				`@1 2009`
A23	`01`			`@0 ENG`
A43	`01`			`@1 INIST @2 20953 @5 354000171427130130`
A44				`@0 0000 @1 © 2010 INIST-CNRS. All rights reserved.`
A45				`@0 38 ref.`
A47	`01`	`1`		`@0 10-0038132`
A60				`@1 P`
A61				`@0 A`
A64	`01`	`1`		`@0 Movement disorders`
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C01	`01`		`ENG`	@0 Although Parkinson's disease with later dementia (PDD) and dementia with Lewy bodies (DLB) are pathologically characterized by the presence of intraneuronal Lewy inclusion bodies, amyloid deposition is also associated to varying degrees with both these disorders. Fibrillar amyloid load can now be quantitated in vivo with positron emission tomography (PET) using imaging biomarkers. Here the reported findings of ¹¹C-PIB PET studies concerning the amyloid load associated with PD and its influence on dementia are reviewed. It is concluded that the presence of amyloid acts to accelerate the dementia process in Lewy body disorders, though has little influence on its nature. Anti-amyloid strategies could be a relevant approach for slowing dementia in a number of DLB and PDD cases.
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C03	`01`	`X`	`FRE`	`@0 Maladie de Parkinson @2 NM @5 01`
C03	`01`	`X`	`ENG`	`@0 Parkinson disease @2 NM @5 01`
C03	`01`	`X`	`SPA`	`@0 Parkinson enfermedad @2 NM @5 01`
C03	`02`	`X`	`FRE`	`@0 Démence à corps de Lewy @2 NM @5 02`
C03	`02`	`X`	`ENG`	`@0 Lewy body dementia @2 NM @5 02`
C03	`02`	`X`	`SPA`	`@0 Demencia cuerpos Lewy @2 NM @5 02`
C03	`03`	`X`	`FRE`	`@0 Pathologie du système nerveux @5 03`
C03	`03`	`X`	`ENG`	`@0 Nervous system diseases @5 03`
C03	`03`	`X`	`SPA`	`@0 Sistema nervioso patología @5 03`
C03	`04`	`X`	`FRE`	`@0 Amyloïde @5 09`
C03	`04`	`X`	`ENG`	`@0 Amyloid @5 09`
C03	`04`	`X`	`SPA`	`@0 Amiloide @5 09`
C03	`05`	`X`	`FRE`	`@0 Tomoscintigraphie @5 10`
C03	`05`	`X`	`ENG`	`@0 Emission tomography @5 10`
C03	`05`	`X`	`SPA`	`@0 Tomocentelleografía @5 10`
C03	`06`	`X`	`FRE`	`@0 Tomographie par émission de positons @5 11`
C03	`06`	`X`	`ENG`	`@0 Positron emission tomography @5 11`
C03	`06`	`X`	`SPA`	`@0 Tomografía emisión positrones @5 11`
C03	`07`	`X`	`FRE`	`@0 Corps Lewy @5 12`
C03	`07`	`X`	`ENG`	`@0 Lewy body @5 12`
C03	`07`	`X`	`SPA`	`@0 Cuerpo Lewy @5 12`
C07	`01`	`X`	`FRE`	`@0 Pathologie de l'encéphale @5 37`
C07	`01`	`X`	`ENG`	`@0 Cerebral disorder @5 37`
C07	`01`	`X`	`SPA`	`@0 Encéfalo patología @5 37`
C07	`02`	`X`	`FRE`	`@0 Syndrome extrapyramidal @5 38`
C07	`02`	`X`	`ENG`	`@0 Extrapyramidal syndrome @5 38`
C07	`02`	`X`	`SPA`	`@0 Extrapiramidal síndrome @5 38`
C07	`03`	`X`	`FRE`	`@0 Maladie dégénérative @5 39`
C07	`03`	`X`	`ENG`	`@0 Degenerative disease @5 39`
C07	`03`	`X`	`SPA`	`@0 Enfermedad degenerativa @5 39`
C07	`04`	`X`	`FRE`	`@0 Pathologie du système nerveux central @5 40`
C07	`04`	`X`	`ENG`	`@0 Central nervous system disease @5 40`
C07	`04`	`X`	`SPA`	`@0 Sistema nervosio central patología @5 40`
N21				`@1 025`
N44	`01`			`@1 OTO`
N82				`@1 OTO`

Format Inist (serveur)

NO :	PASCAL 10-0038132 INIST
ET :	Imaging Amyloid in Parkinson's Disease Dementia and Dementia with Lewy Bodies with Positron Emission Tomography
AU :	BROOKS (David J.); POEWE (Werner); SCHERFLER (Christopher)
AF :	MRC Clinical Sciences Centre and Division of Neuroscience, Imperial College/London/Royaume-Uni (1 aut.); Department of Neurology, Innsbruck Medical University/Innsbruck/Autriche (1 aut., 2 aut.)
DT :	Publication en série; Niveau analytique
SO :	Movement disorders; ISSN 0885-3185; Etats-Unis; Da. 2009; Vol. 24; No. 14 SUP; S742-S747; Bibl. 38 ref.
LA :	Anglais
EA :	Although Parkinson's disease with later dementia (PDD) and dementia with Lewy bodies (DLB) are pathologically characterized by the presence of intraneuronal Lewy inclusion bodies, amyloid deposition is also associated to varying degrees with both these disorders. Fibrillar amyloid load can now be quantitated in vivo with positron emission tomography (PET) using imaging biomarkers. Here the reported findings of ¹¹C-PIB PET studies concerning the amyloid load associated with PD and its influence on dementia are reviewed. It is concluded that the presence of amyloid acts to accelerate the dementia process in Lewy body disorders, though has little influence on its nature. Anti-amyloid strategies could be a relevant approach for slowing dementia in a number of DLB and PDD cases.
CC :	002B17; 002B17G
FD :	Maladie de Parkinson; Démence à corps de Lewy; Pathologie du système nerveux; Amyloïde; Tomoscintigraphie; Tomographie par émission de positons; Corps Lewy
FG :	Pathologie de l'encéphale; Syndrome extrapyramidal; Maladie dégénérative; Pathologie du système nerveux central
ED :	Parkinson disease; Lewy body dementia; Nervous system diseases; Amyloid; Emission tomography; Positron emission tomography; Lewy body
EG :	Cerebral disorder; Extrapyramidal syndrome; Degenerative disease; Central nervous system disease
SD :	Parkinson enfermedad; Demencia cuerpos Lewy; Sistema nervioso patología; Amiloide; Tomocentelleografía; Tomografía emisión positrones; Cuerpo Lewy
LO :	INIST-20953.354000171427130130
ID :	10-0038132

Links to Exploration step

Pascal:10-0038132

Le document en format XML

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<front><div type="abstract" xml:lang="en">Although Parkinson's disease with later dementia (PDD) and dementia with Lewy bodies (DLB) are pathologically characterized by the presence of intraneuronal Lewy inclusion bodies, amyloid deposition is also associated to varying degrees with both these disorders. Fibrillar amyloid load can now be quantitated in vivo with positron emission tomography (PET) using imaging biomarkers. Here the reported findings of <sup>11</sup>
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<ET>Imaging Amyloid in Parkinson's Disease Dementia and Dementia with Lewy Bodies with Positron Emission Tomography</ET>
<AU>BROOKS (David J.); POEWE (Werner); SCHERFLER (Christopher)</AU>
<AF>MRC Clinical Sciences Centre and Division of Neuroscience, Imperial College/London/Royaume-Uni (1 aut.); Department of Neurology, Innsbruck Medical University/Innsbruck/Autriche (1 aut., 2 aut.)</AF>
<DT>Publication en série; Niveau analytique</DT>
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<EA>Although Parkinson's disease with later dementia (PDD) and dementia with Lewy bodies (DLB) are pathologically characterized by the presence of intraneuronal Lewy inclusion bodies, amyloid deposition is also associated to varying degrees with both these disorders. Fibrillar amyloid load can now be quantitated in vivo with positron emission tomography (PET) using imaging biomarkers. Here the reported findings of <sup>11</sup>
C-PIB PET studies concerning the amyloid load associated with PD and its influence on dementia are reviewed. It is concluded that the presence of amyloid acts to accelerate the dementia process in Lewy body disorders, though has little influence on its nature. Anti-amyloid strategies could be a relevant approach for slowing dementia in a number of DLB and PDD cases.</EA>
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	Movement Disorders (revue)
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Movement Disorders (revue)

Imaging Amyloid in Parkinson's Disease Dementia and Dementia with Lewy Bodies with Positron Emission Tomography

Imaging Amyloid in Parkinson's Disease Dementia and Dementia with Lewy Bodies with Positron Emission Tomography

Source :

Descripteurs français

English descriptors

Abstract

Notice en format standard (ISO 2709)

Format Inist (serveur)

Links to Exploration step

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