Involvement of GABAA receptors in myoclonus
Identifieur interne : 002B85 ( PascalFrancis/Checkpoint ); précédent : 002B84; suivant : 002B86Involvement of GABAA receptors in myoclonus
Auteurs : R. R. Matsumoto [États-Unis] ; D. D. Truong [États-Unis] ; K. D. Nguyen [États-Unis] ; A. T. Dang [États-Unis] ; T. T. Hoang [États-Unis] ; P. Q. Vo [États-Unis] ; P. Sandroni [États-Unis]Source :
- Movement disorders [ 0885-3185 ] ; 2000.
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Abstract
Alterations in multiple neurochemical systems have been reported in animal and human studies of posthypoxic myoclonus. It is impossible, however, to establish causative relationships between the observed changes and the myoclonic movements from these studies. Therefore, to establish causative links between neurochemical changes and myoclonus, ligands that target neurotransmitter systems that are altered in posthypoxic myoclonus were microinjected into the lateral ventricles of normal rats to identify the changes that can produce myoclonus. Of the ligands that were tested, only the GABAA antagonists produced myoclonus after intracerebroventricular administration, suggesting the importance of disinhibition of GABAergic systems in myoclonus, To further examine the role of GABA in myoclonus, GABAergic antagonists were microinjected into the nucleus reticularis of the thalamus (NRT), an area of the brain in which extensive pathologic changes are seen in posthypoxic animals. GABAA, but not GABAB, antagonists produced myoclonus after microinjection into the NRT. Earlier investigators have further reported the ability of GABAA antagonists to produce myoclonus after microinjection into the caudate. The data therefore suggest that disruption of activity at GABAA receptors at any one of a number of levels in the neural axis can produce myoclonus.
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Sandroni</name><affiliation wicri:level="2"><inist:fA14 i1="01"><s1>University of California Irvine, Department of Neurology</s1><s2>Irvine, California</s2><s3>USA</s3><sZ>1 aut.</sZ><sZ>2 aut.</sZ><sZ>3 aut.</sZ><sZ>4 aut.</sZ><sZ>5 aut.</sZ><sZ>6 aut.</sZ><sZ>7 aut.</sZ></inist:fA14><country>États-Unis</country><placeName><region type="state">Californie</region></placeName></affiliation><affiliation wicri:level="2"><inist:fA14 i1="04"><s1>Mayo Clinic, Department of Neurology</s1><s2>Rochester, Minnesota</s2><s3>USA</s3><sZ>7 aut.</sZ></inist:fA14><country>États-Unis</country><placeName><region type="state">Minnesota</region></placeName></affiliation></author></titleStmt><publicationStmt><idno type="wicri:source">INIST</idno><idno type="inist">00-0253242</idno><date when="2000">2000</date><idno type="stanalyst">PASCAL 00-0253242 INIST</idno><idno type="RBID">Pascal:00-0253242</idno><idno type="wicri:Area/PascalFrancis/Corpus">002C18</idno><idno type="wicri:Area/PascalFrancis/Curation">000103</idno><idno type="wicri:Area/PascalFrancis/Checkpoint">002B85</idno></publicationStmt><sourceDesc><biblStruct><analytic><title xml:lang="en" level="a">Involvement of GABA<sub>A</sub> receptors in myoclonus</title><author><name sortKey="Matsumoto, R R" sort="Matsumoto, R R" uniqKey="Matsumoto R" first="R. R." last="Matsumoto">R. R. Matsumoto</name><affiliation wicri:level="2"><inist:fA14 i1="01"><s1>University of California Irvine, Department of Neurology</s1><s2>Irvine, California</s2><s3>USA</s3><sZ>1 aut.</sZ><sZ>2 aut.</sZ><sZ>3 aut.</sZ><sZ>4 aut.</sZ><sZ>5 aut.</sZ><sZ>6 aut.</sZ><sZ>7 aut.</sZ></inist:fA14><country>États-Unis</country><placeName><region type="state">Californie</region></placeName></affiliation><affiliation wicri:level="2"><inist:fA14 i1="02"><s1>University of Oklahoma Health Sciences Center, Department of Pharmacology & Toxicology</s1><s2>Oklahoma City, Oklahoma</s2><s3>USA</s3><sZ>1 aut.</sZ></inist:fA14><country>États-Unis</country><placeName><region type="state">Oklahoma</region></placeName></affiliation></author><author><name sortKey="Truong, D D" sort="Truong, D D" uniqKey="Truong D" first="D. D." last="Truong">D. D. Truong</name><affiliation wicri:level="2"><inist:fA14 i1="01"><s1>University of California Irvine, Department of Neurology</s1><s2>Irvine, California</s2><s3>USA</s3><sZ>1 aut.</sZ><sZ>2 aut.</sZ><sZ>3 aut.</sZ><sZ>4 aut.</sZ><sZ>5 aut.</sZ><sZ>6 aut.</sZ><sZ>7 aut.</sZ></inist:fA14><country>États-Unis</country><placeName><region type="state">Californie</region></placeName></affiliation><affiliation wicri:level="2"><inist:fA14 i1="03"><s1>Long Beach Memorial Medical Center</s1><s2>Long Beach, California</s2><s3>USA</s3><sZ>2 aut.</sZ></inist:fA14><country>États-Unis</country><placeName><region type="state">Californie</region></placeName></affiliation></author><author><name sortKey="Nguyen, K D" sort="Nguyen, K D" uniqKey="Nguyen K" first="K. D." last="Nguyen">K. D. Nguyen</name><affiliation wicri:level="2"><inist:fA14 i1="01"><s1>University of California Irvine, Department of Neurology</s1><s2>Irvine, California</s2><s3>USA</s3><sZ>1 aut.</sZ><sZ>2 aut.</sZ><sZ>3 aut.</sZ><sZ>4 aut.</sZ><sZ>5 aut.</sZ><sZ>6 aut.</sZ><sZ>7 aut.</sZ></inist:fA14><country>États-Unis</country><placeName><region type="state">Californie</region></placeName></affiliation></author><author><name sortKey="Dang, A T" sort="Dang, A T" uniqKey="Dang A" first="A. T." last="Dang">A. T. Dang</name><affiliation wicri:level="2"><inist:fA14 i1="01"><s1>University of California Irvine, Department of Neurology</s1><s2>Irvine, California</s2><s3>USA</s3><sZ>1 aut.</sZ><sZ>2 aut.</sZ><sZ>3 aut.</sZ><sZ>4 aut.</sZ><sZ>5 aut.</sZ><sZ>6 aut.</sZ><sZ>7 aut.</sZ></inist:fA14><country>États-Unis</country><placeName><region type="state">Californie</region></placeName></affiliation></author><author><name sortKey="Hoang, T T" sort="Hoang, T T" uniqKey="Hoang T" first="T. T." last="Hoang">T. T. Hoang</name><affiliation wicri:level="2"><inist:fA14 i1="01"><s1>University of California Irvine, Department of Neurology</s1><s2>Irvine, California</s2><s3>USA</s3><sZ>1 aut.</sZ><sZ>2 aut.</sZ><sZ>3 aut.</sZ><sZ>4 aut.</sZ><sZ>5 aut.</sZ><sZ>6 aut.</sZ><sZ>7 aut.</sZ></inist:fA14><country>États-Unis</country><placeName><region type="state">Californie</region></placeName></affiliation></author><author><name sortKey="Vo, P Q" sort="Vo, P Q" uniqKey="Vo P" first="P. Q." last="Vo">P. Q. Vo</name><affiliation wicri:level="2"><inist:fA14 i1="01"><s1>University of California Irvine, Department of Neurology</s1><s2>Irvine, California</s2><s3>USA</s3><sZ>1 aut.</sZ><sZ>2 aut.</sZ><sZ>3 aut.</sZ><sZ>4 aut.</sZ><sZ>5 aut.</sZ><sZ>6 aut.</sZ><sZ>7 aut.</sZ></inist:fA14><country>États-Unis</country><placeName><region type="state">Californie</region></placeName></affiliation></author><author><name sortKey="Sandroni, P" sort="Sandroni, P" uniqKey="Sandroni P" first="P." last="Sandroni">P. Sandroni</name><affiliation wicri:level="2"><inist:fA14 i1="01"><s1>University of California Irvine, Department of Neurology</s1><s2>Irvine, California</s2><s3>USA</s3><sZ>1 aut.</sZ><sZ>2 aut.</sZ><sZ>3 aut.</sZ><sZ>4 aut.</sZ><sZ>5 aut.</sZ><sZ>6 aut.</sZ><sZ>7 aut.</sZ></inist:fA14><country>États-Unis</country><placeName><region type="state">Californie</region></placeName></affiliation><affiliation wicri:level="2"><inist:fA14 i1="04"><s1>Mayo Clinic, Department of Neurology</s1><s2>Rochester, Minnesota</s2><s3>USA</s3><sZ>7 aut.</sZ></inist:fA14><country>États-Unis</country><placeName><region type="state">Minnesota</region></placeName></affiliation></author></analytic><series><title level="j" type="main">Movement disorders</title><title level="j" type="abbreviated">Mov. disord.</title><idno type="ISSN">0885-3185</idno><imprint><date when="2000">2000</date></imprint></series></biblStruct></sourceDesc><seriesStmt><title level="j" type="main">Movement disorders</title><title level="j" type="abbreviated">Mov. disord.</title><idno type="ISSN">0885-3185</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Animal</term><term>Experimental study</term><term>Gabaergic receptor A</term><term>Myoclonus</term><term>Pathophysiology</term><term>Rat</term></keywords><keywords scheme="Pascal" xml:lang="fr"><term>Myoclonie</term><term>Récepteur gabaergique A</term><term>Etude expérimentale</term><term>Physiopathologie</term><term>Animal</term><term>Rat</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Alterations in multiple neurochemical systems have been reported in animal and human studies of posthypoxic myoclonus. It is impossible, however, to establish causative relationships between the observed changes and the myoclonic movements from these studies. Therefore, to establish causative links between neurochemical changes and myoclonus, ligands that target neurotransmitter systems that are altered in posthypoxic myoclonus were microinjected into the lateral ventricles of normal rats to identify the changes that can produce myoclonus. Of the ligands that were tested, only the GABA<sub>A</sub> antagonists produced myoclonus after intracerebroventricular administration, suggesting the importance of disinhibition of GABAergic systems in myoclonus, To further examine the role of GABA in myoclonus, GABAergic antagonists were microinjected into the nucleus reticularis of the thalamus (NRT), an area of the brain in which extensive pathologic changes are seen in posthypoxic animals. GABA<sub>A</sub>, but not GABA<sub>B</sub>, antagonists produced myoclonus after microinjection into the NRT. Earlier investigators have further reported the ability of GABA<sub>A</sub> antagonists to produce myoclonus after microinjection into the caudate. The data therefore suggest that disruption of activity at GABA<sub>A</sub> receptors at any one of a number of levels in the neural axis can produce myoclonus.</div></front></TEI><inist><standard h6="B"><pA><fA01 i1="01" i2="1"><s0>0885-3185</s0></fA01><fA03 i2="1"><s0>Mov. disord.</s0></fA03><fA05><s2>15</s2></fA05><fA06><s3>SUP1</s3></fA06><fA08 i1="01" i2="1" l="ENG"><s1>Involvement of GABA<sub>A</sub> receptors in myoclonus</s1></fA08><fA09 i1="01" i2="1" l="ENG"><s1>Posthypoxic Myoclonus</s1></fA09><fA11 i1="01" i2="1"><s1>MATSUMOTO (R. R.)</s1></fA11><fA11 i1="02" i2="1"><s1>TRUONG (D. D.)</s1></fA11><fA11 i1="03" i2="1"><s1>NGUYEN (K. D.)</s1></fA11><fA11 i1="04" i2="1"><s1>DANG (A. T.)</s1></fA11><fA11 i1="05" i2="1"><s1>HOANG (T. T.)</s1></fA11><fA11 i1="06" i2="1"><s1>VO (P. Q.)</s1></fA11><fA11 i1="07" i2="1"><s1>SANDRONI (P.)</s1></fA11><fA12 i1="01" i2="1"><s1>FRUCHT (Steven)</s1><s9>présent.</s9></fA12><fA12 i1="02" i2="1"><s1>FAHN (Stanley)</s1><s9>présent.</s9></fA12><fA12 i1="03" i2="1"><s1>HALLETT (Mark)</s1><s9>présent.</s9></fA12><fA14 i1="01"><s1>University of California Irvine, Department of Neurology</s1><s2>Irvine, California</s2><s3>USA</s3><sZ>1 aut.</sZ><sZ>2 aut.</sZ><sZ>3 aut.</sZ><sZ>4 aut.</sZ><sZ>5 aut.</sZ><sZ>6 aut.</sZ><sZ>7 aut.</sZ></fA14><fA14 i1="02"><s1>University of Oklahoma Health Sciences Center, Department of Pharmacology & Toxicology</s1><s2>Oklahoma City, Oklahoma</s2><s3>USA</s3><sZ>1 aut.</sZ></fA14><fA14 i1="03"><s1>Long Beach Memorial Medical Center</s1><s2>Long Beach, California</s2><s3>USA</s3><sZ>2 aut.</sZ></fA14><fA14 i1="04"><s1>Mayo Clinic, Department of Neurology</s1><s2>Rochester, Minnesota</s2><s3>USA</s3><sZ>7 aut.</sZ></fA14><fA15 i1="01"><s1>Columbia-Presbyterian Medical Center</s1><s2>New York, NY</s2><s3>USA</s3><sZ>1 aut.</sZ><sZ>2 aut.</sZ></fA15><fA15 i1="02"><s1>Human Motor Control Section, NINDS, NIH</s1><s2>Bethesda, Maryland</s2><s3>USA</s3><sZ>3 aut.</sZ></fA15><fA20><s1>47-52</s1></fA20><fA21><s1>2000</s1></fA21><fA23 i1="01"><s0>ENG</s0></fA23><fA43 i1="01"><s1>INIST</s1><s2>20953</s2><s5>354000082104040080</s5></fA43><fA44><s0>0000</s0><s1>© 2000 INIST-CNRS. All rights reserved.</s1></fA44><fA45><s0>27 ref.</s0></fA45><fA47 i1="01" i2="1"><s0>00-0253242</s0></fA47><fA60><s1>P</s1></fA60><fA61><s0>A</s0></fA61><fA64 i1="01" i2="1"><s0>Movement disorders</s0></fA64><fA66 i1="01"><s0>USA</s0></fA66><fC01 i1="01" l="ENG"><s0>Alterations in multiple neurochemical systems have been reported in animal and human studies of posthypoxic myoclonus. It is impossible, however, to establish causative relationships between the observed changes and the myoclonic movements from these studies. Therefore, to establish causative links between neurochemical changes and myoclonus, ligands that target neurotransmitter systems that are altered in posthypoxic myoclonus were microinjected into the lateral ventricles of normal rats to identify the changes that can produce myoclonus. Of the ligands that were tested, only the GABA<sub>A</sub> antagonists produced myoclonus after intracerebroventricular administration, suggesting the importance of disinhibition of GABAergic systems in myoclonus, To further examine the role of GABA in myoclonus, GABAergic antagonists were microinjected into the nucleus reticularis of the thalamus (NRT), an area of the brain in which extensive pathologic changes are seen in posthypoxic animals. GABA<sub>A</sub>, but not GABA<sub>B</sub>, antagonists produced myoclonus after microinjection into the NRT. Earlier investigators have further reported the ability of GABA<sub>A</sub> antagonists to produce myoclonus after microinjection into the caudate. The data therefore suggest that disruption of activity at GABA<sub>A</sub> receptors at any one of a number of levels in the neural axis can produce myoclonus.</s0></fC01><fC02 i1="01" i2="X"><s0>002B17A01</s0></fC02><fC03 i1="01" i2="X" l="FRE"><s0>Myoclonie</s0><s5>01</s5></fC03><fC03 i1="01" i2="X" l="ENG"><s0>Myoclonus</s0><s5>01</s5></fC03><fC03 i1="01" i2="X" l="SPA"><s0>Mioclonia</s0><s5>01</s5></fC03><fC03 i1="02" i2="X" l="FRE"><s0>Récepteur gabaergique A</s0><s5>04</s5></fC03><fC03 i1="02" i2="X" l="ENG"><s0>Gabaergic receptor A</s0><s5>04</s5></fC03><fC03 i1="02" i2="X" l="SPA"><s0>Receptor gabaminérgico A</s0><s5>04</s5></fC03><fC03 i1="03" i2="X" l="FRE"><s0>Etude expérimentale</s0><s5>16</s5></fC03><fC03 i1="03" i2="X" l="ENG"><s0>Experimental study</s0><s5>16</s5></fC03><fC03 i1="03" i2="X" l="SPA"><s0>Estudio experimental</s0><s5>16</s5></fC03><fC03 i1="04" i2="X" l="FRE"><s0>Physiopathologie</s0><s5>17</s5></fC03><fC03 i1="04" i2="X" l="ENG"><s0>Pathophysiology</s0><s5>17</s5></fC03><fC03 i1="04" i2="X" l="SPA"><s0>Fisiopatología</s0><s5>17</s5></fC03><fC03 i1="05" i2="X" l="FRE"><s0>Animal</s0><s5>20</s5></fC03><fC03 i1="05" i2="X" l="ENG"><s0>Animal</s0><s5>20</s5></fC03><fC03 i1="05" i2="X" l="SPA"><s0>Animal</s0><s5>20</s5></fC03><fC03 i1="06" i2="X" l="FRE"><s0>Rat</s0><s5>21</s5></fC03><fC03 i1="06" i2="X" l="ENG"><s0>Rat</s0><s5>21</s5></fC03><fC03 i1="06" i2="X" l="SPA"><s0>Rata</s0><s5>21</s5></fC03><fC07 i1="01" i2="X" l="FRE"><s0>Rodentia</s0><s2>NS</s2></fC07><fC07 i1="01" i2="X" l="ENG"><s0>Rodentia</s0><s2>NS</s2></fC07><fC07 i1="01" i2="X" l="SPA"><s0>Rodentia</s0><s2>NS</s2></fC07><fC07 i1="02" i2="X" l="FRE"><s0>Mammalia</s0><s2>NS</s2></fC07><fC07 i1="02" i2="X" l="ENG"><s0>Mammalia</s0><s2>NS</s2></fC07><fC07 i1="02" i2="X" l="SPA"><s0>Mammalia</s0><s2>NS</s2></fC07><fC07 i1="03" i2="X" l="FRE"><s0>Vertebrata</s0><s2>NS</s2></fC07><fC07 i1="03" i2="X" l="ENG"><s0>Vertebrata</s0><s2>NS</s2></fC07><fC07 i1="03" i2="X" l="SPA"><s0>Vertebrata</s0><s2>NS</s2></fC07><fC07 i1="04" i2="X" l="FRE"><s0>Système nerveux pathologie</s0><s5>37</s5></fC07><fC07 i1="04" i2="X" l="ENG"><s0>Nervous system diseases</s0><s5>37</s5></fC07><fC07 i1="04" i2="X" l="SPA"><s0>Sistema nervioso patología</s0><s5>37</s5></fC07><fC07 i1="05" i2="X" l="FRE"><s0>Trouble neurologique</s0><s5>38</s5></fC07><fC07 i1="05" i2="X" l="ENG"><s0>Neurological disorder</s0><s5>38</s5></fC07><fC07 i1="05" i2="X" l="SPA"><s0>Trastorno neurológico</s0><s5>38</s5></fC07><fC07 i1="06" i2="X" l="FRE"><s0>Mouvement involontaire</s0><s5>39</s5></fC07><fC07 i1="06" i2="X" l="ENG"><s0>Involuntary movement</s0><s5>39</s5></fC07><fC07 i1="06" i2="X" l="SPA"><s0>Movimiento involuntario</s0><s5>39</s5></fC07><fN21><s1>171</s1></fN21></pA></standard></inist><affiliations><list><country><li>États-Unis</li></country><region><li>Californie</li><li>Minnesota</li><li>Oklahoma</li></region></list><tree><country name="États-Unis"><region name="Californie"><name sortKey="Matsumoto, R R" sort="Matsumoto, R R" uniqKey="Matsumoto R" first="R. R." last="Matsumoto">R. R. Matsumoto</name></region><name sortKey="Dang, A T" sort="Dang, A T" uniqKey="Dang A" first="A. T." last="Dang">A. T. Dang</name><name sortKey="Hoang, T T" sort="Hoang, T T" uniqKey="Hoang T" first="T. T." last="Hoang">T. T. Hoang</name><name sortKey="Matsumoto, R R" sort="Matsumoto, R R" uniqKey="Matsumoto R" first="R. R." last="Matsumoto">R. R. Matsumoto</name><name sortKey="Nguyen, K D" sort="Nguyen, K D" uniqKey="Nguyen K" first="K. D." last="Nguyen">K. D. Nguyen</name><name sortKey="Sandroni, P" sort="Sandroni, P" uniqKey="Sandroni P" first="P." last="Sandroni">P. Sandroni</name><name sortKey="Sandroni, P" sort="Sandroni, P" uniqKey="Sandroni P" first="P." last="Sandroni">P. Sandroni</name><name sortKey="Truong, D D" sort="Truong, D D" uniqKey="Truong D" first="D. D." last="Truong">D. D. Truong</name><name sortKey="Truong, D D" sort="Truong, D D" uniqKey="Truong D" first="D. D." last="Truong">D. D. 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