MovDisordV3, PascalFrancis, Checkpoint, bibRecord, 002820

Contribution of the interleukin-1β gene polymorphism in multiple system atrophy

Identifieur interne : 002820 ( PascalFrancis/Checkpoint ); précédent : 002819; suivant : 002821

Contribution of the interleukin-1β gene polymorphism in multiple system atrophy

Auteurs : Masataka Nishimura [Japon] ; Hideshi Kawakami [Japon] ; Osamu Komure [Japon] ; Hirofumi Maruyama [Japon] ; Hiroyuki Morino [Japon] ; Yuishin Izumi [Japon] ; Shigenobu Nakamura [Japon] ; Ryuji Kaji [Japon] ; Sadako Kuno [Japon]

Source :

Movement disorders [ 0885-3185 ] ; 2002.

RBID : Pascal:02-0456877

Descripteurs français

Pascal (Inist)
- Atrophie multisystématisée, Polymorphisme, Gène, Promoteur, Interleukine 1β, Interleukine 1α, Déterminisme génétique, Adulte.
Wicri :
- topic : Adulte.

English descriptors

KwdEn :
- Adult, Gene, Genetic determinism, Interleukin 1α, Interleukin 1β, Multiple system atrophy, Polymorphism, Promoter.

Abstract

We studied genetic polymorphisms in the promoter region at position -511 of the interleukin (IL) -1β gene (IL-1B-511) and at position -889 of the IL-1α gene (IL-1A-889), in 111 Japanese patients with multiple system atrophy (MSA) and 160 controls. The distribution of IL-1B-511 was significantly different between MSA patients and controls, because of the under-representation of patients with homozygotes for allele 2 (IL-1B-511*2), a high producer of IL-1β. The frequency of IL-1A-889*2, a high secretor of IL-1α, was also decreased in MSA patients. Our findings suggest that abnormal cytokine expression may be implicated in the pathogenesis of MSA.

Affiliations:

Japon

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Le document en format XML

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<author><name sortKey="Morino, Hiroyuki" sort="Morino, Hiroyuki" uniqKey="Morino H" first="Hiroyuki" last="Morino">Hiroyuki Morino</name>
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<author><name sortKey="Izumi, Yuishin" sort="Izumi, Yuishin" uniqKey="Izumi Y" first="Yuishin" last="Izumi">Yuishin Izumi</name>
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<author><name sortKey="Nakamura, Shigenobu" sort="Nakamura, Shigenobu" uniqKey="Nakamura S" first="Shigenobu" last="Nakamura">Shigenobu Nakamura</name>
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<author><name sortKey="Kaji, Ryuji" sort="Kaji, Ryuji" uniqKey="Kaji R" first="Ryuji" last="Kaji">Ryuji Kaji</name>
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<author><name sortKey="Kuno, Sadako" sort="Kuno, Sadako" uniqKey="Kuno S" first="Sadako" last="Kuno">Sadako Kuno</name>
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<s2>Kyoto</s2>
<s3>JPN</s3>
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<sourceDesc><biblStruct><analytic><title xml:lang="en" level="a">Contribution of the interleukin-1β gene polymorphism in multiple system atrophy</title>
<author><name sortKey="Nishimura, Masataka" sort="Nishimura, Masataka" uniqKey="Nishimura M" first="Masataka" last="Nishimura">Masataka Nishimura</name>
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<s2>Tokushima</s2>
<s3>JPN</s3>
<sZ>1 aut.</sZ>
<sZ>6 aut.</sZ>
<sZ>8 aut.</sZ>
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<author><name sortKey="Kawakami, Hideshi" sort="Kawakami, Hideshi" uniqKey="Kawakami H" first="Hideshi" last="Kawakami">Hideshi Kawakami</name>
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<s2>Hiroshima</s2>
<s3>JPN</s3>
<sZ>2 aut.</sZ>
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<author><name sortKey="Komure, Osamu" sort="Komure, Osamu" uniqKey="Komure O" first="Osamu" last="Komure">Osamu Komure</name>
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<s2>Hiroshima</s2>
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<s2>Hiroshima</s2>
<s3>JPN</s3>
<sZ>2 aut.</sZ>
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<sZ>5 aut.</sZ>
<sZ>7 aut.</sZ>
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<author><name sortKey="Izumi, Yuishin" sort="Izumi, Yuishin" uniqKey="Izumi Y" first="Yuishin" last="Izumi">Yuishin Izumi</name>
<affiliation wicri:level="1"><inist:fA14 i1="01"><s1>Department of Clinical Neuroscience, Tokushima University Hospital</s1>
<s2>Tokushima</s2>
<s3>JPN</s3>
<sZ>1 aut.</sZ>
<sZ>6 aut.</sZ>
<sZ>8 aut.</sZ>
</inist:fA14>
<country>Japon</country>
<wicri:noRegion>Tokushima</wicri:noRegion>
</affiliation>
</author>
<author><name sortKey="Nakamura, Shigenobu" sort="Nakamura, Shigenobu" uniqKey="Nakamura S" first="Shigenobu" last="Nakamura">Shigenobu Nakamura</name>
<affiliation wicri:level="1"><inist:fA14 i1="02"><s1>Third Department of Internal Medicine, Hiroshima University School of Medicine</s1>
<s2>Hiroshima</s2>
<s3>JPN</s3>
<sZ>2 aut.</sZ>
<sZ>4 aut.</sZ>
<sZ>5 aut.</sZ>
<sZ>7 aut.</sZ>
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</affiliation>
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<author><name sortKey="Kaji, Ryuji" sort="Kaji, Ryuji" uniqKey="Kaji R" first="Ryuji" last="Kaji">Ryuji Kaji</name>
<affiliation wicri:level="1"><inist:fA14 i1="01"><s1>Department of Clinical Neuroscience, Tokushima University Hospital</s1>
<s2>Tokushima</s2>
<s3>JPN</s3>
<sZ>1 aut.</sZ>
<sZ>6 aut.</sZ>
<sZ>8 aut.</sZ>
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<country>Japon</country>
<wicri:noRegion>Tokushima</wicri:noRegion>
</affiliation>
</author>
<author><name sortKey="Kuno, Sadako" sort="Kuno, Sadako" uniqKey="Kuno S" first="Sadako" last="Kuno">Sadako Kuno</name>
<affiliation wicri:level="1"><inist:fA14 i1="03"><s1>Department of Neurology and Clinical Research Center, Utano National Hospital</s1>
<s2>Kyoto</s2>
<s3>JPN</s3>
<sZ>3 aut.</sZ>
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<series><title level="j" type="main">Movement disorders</title>
<title level="j" type="abbreviated">Mov. disord.</title>
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<term>Gene</term>
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<front><div type="abstract" xml:lang="en">We studied genetic polymorphisms in the promoter region at position -511 of the interleukin (IL) -1β gene (IL-1B-511) and at position -889 of the IL-1α gene (IL-1A-889), in 111 Japanese patients with multiple system atrophy (MSA) and 160 controls. The distribution of IL-1B-511 was significantly different between MSA patients and controls, because of the under-representation of patients with homozygotes for allele 2 (IL-1B-511*2), a high producer of IL-1β. The frequency of IL-1A-889*2, a high secretor of IL-1α, was also decreased in MSA patients. Our findings suggest that abnormal cytokine expression may be implicated in the pathogenesis of MSA.</div>
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<fA14 i1="03"><s1>Department of Neurology and Clinical Research Center, Utano National Hospital</s1>
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<fC01 i1="01" l="ENG"><s0>We studied genetic polymorphisms in the promoter region at position -511 of the interleukin (IL) -1β gene (IL-1B-511) and at position -889 of the IL-1α gene (IL-1A-889), in 111 Japanese patients with multiple system atrophy (MSA) and 160 controls. The distribution of IL-1B-511 was significantly different between MSA patients and controls, because of the under-representation of patients with homozygotes for allele 2 (IL-1B-511*2), a high producer of IL-1β. The frequency of IL-1A-889*2, a high secretor of IL-1α, was also decreased in MSA patients. Our findings suggest that abnormal cytokine expression may be implicated in the pathogenesis of MSA.</s0>
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<s5>37</s5>
</fC07>
<fC07 i1="02" i2="X" l="SPA"><s0>Sistema nervioso patología</s0>
<s5>37</s5>
</fC07>
<fC07 i1="03" i2="X" l="FRE"><s0>Système nerveux central pathologie</s0>
<s5>38</s5>
</fC07>
<fC07 i1="03" i2="X" l="ENG"><s0>Central nervous system disease</s0>
<s5>38</s5>
</fC07>
<fC07 i1="03" i2="X" l="SPA"><s0>Sistema nervosio central patología</s0>
<s5>38</s5>
</fC07>
<fC07 i1="04" i2="X" l="FRE"><s0>Encéphale pathologie</s0>
<s5>39</s5>
</fC07>
<fC07 i1="04" i2="X" l="ENG"><s0>Cerebral disorder</s0>
<s5>39</s5>
</fC07>
<fC07 i1="04" i2="X" l="SPA"><s0>Encéfalo patología</s0>
<s5>39</s5>
</fC07>
<fC07 i1="05" i2="X" l="FRE"><s0>Maladie dégénérative</s0>
<s5>40</s5>
</fC07>
<fC07 i1="05" i2="X" l="ENG"><s0>Degenerative disease</s0>
<s5>40</s5>
</fC07>
<fC07 i1="05" i2="X" l="SPA"><s0>Enfermedad degenerativa</s0>
<s5>40</s5>
</fC07>
<fC07 i1="06" i2="X" l="FRE"><s0>Cytokine</s0>
<s5>53</s5>
</fC07>
<fC07 i1="06" i2="X" l="ENG"><s0>Cytokine</s0>
<s5>53</s5>
</fC07>
<fC07 i1="06" i2="X" l="SPA"><s0>Citoquina</s0>
<s5>53</s5>
</fC07>
<fN21><s1>266</s1>
</fN21>
<fN82><s1>PSI</s1>
</fN82>
</pA>
</standard>
</inist>
<affiliations><list><country><li>Japon</li>
</country>
</list>
<tree><country name="Japon"><noRegion><name sortKey="Nishimura, Masataka" sort="Nishimura, Masataka" uniqKey="Nishimura M" first="Masataka" last="Nishimura">Masataka Nishimura</name>
</noRegion>
<name sortKey="Izumi, Yuishin" sort="Izumi, Yuishin" uniqKey="Izumi Y" first="Yuishin" last="Izumi">Yuishin Izumi</name>
<name sortKey="Kaji, Ryuji" sort="Kaji, Ryuji" uniqKey="Kaji R" first="Ryuji" last="Kaji">Ryuji Kaji</name>
<name sortKey="Kawakami, Hideshi" sort="Kawakami, Hideshi" uniqKey="Kawakami H" first="Hideshi" last="Kawakami">Hideshi Kawakami</name>
<name sortKey="Komure, Osamu" sort="Komure, Osamu" uniqKey="Komure O" first="Osamu" last="Komure">Osamu Komure</name>
<name sortKey="Kuno, Sadako" sort="Kuno, Sadako" uniqKey="Kuno S" first="Sadako" last="Kuno">Sadako Kuno</name>
<name sortKey="Maruyama, Hirofumi" sort="Maruyama, Hirofumi" uniqKey="Maruyama H" first="Hirofumi" last="Maruyama">Hirofumi Maruyama</name>
<name sortKey="Morino, Hiroyuki" sort="Morino, Hiroyuki" uniqKey="Morino H" first="Hiroyuki" last="Morino">Hiroyuki Morino</name>
<name sortKey="Nakamura, Shigenobu" sort="Nakamura, Shigenobu" uniqKey="Nakamura S" first="Shigenobu" last="Nakamura">Shigenobu Nakamura</name>
</country>
</tree>
</affiliations>
</record>

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	Movement Disorders (revue)
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Movement Disorders (revue)

Contribution of the interleukin-1β gene polymorphism in multiple system atrophy

Contribution of the interleukin-1β gene polymorphism in multiple system atrophy

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