Cerebellar hypermetria: reduction in the early component of the antagonist electromyogram.
Identifieur interne : 004D53 ( Ncbi/Merge ); précédent : 004D52; suivant : 004D54Cerebellar hypermetria: reduction in the early component of the antagonist electromyogram.
Auteurs : B. Wild [Allemagne] ; D M CorcosSource :
- Movement disorders : official journal of the Movement Disorder Society [ 0885-3185 ] ; 1997.
English descriptors
- KwdEn :
- MESH :
- methods : Electromyography.
- physiopathology : Cerebellar Diseases, Movement Disorders, Muscle, Skeletal.
- statistics & numerical data : Electromyography.
- Extremities, Humans, Reaction Time.
Abstract
Several articles have described the electromyographic (EMG) patterns underlying cerebellar hypermetria. However, the description of the EMG associated with hypermetria is not consistent between studies. It is hypothesized that the reason for this concerns the criteria used to define and describe the antagonist latency. Several studies on neurologically normal individuals have shown that there are two components to the antagonist EMG: an early component and a late component. However, many studies have identified only one component and have not defined whether it was the early or late component. A reanalysis of one published data set that had previously identified only one antagonist component was performed. The reanalysis suggests that hypermetria can be caused by an absence of an early antagonist EMG component, a delayed late component, or both. In addition, a new measure is suggested that allows this hypothesis to be further evaluated.
DOI: 10.1002/mds.870120420
PubMed: 9251082
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Wild</name><affiliation wicri:level="1"><nlm:affiliation>Department of Psychiatry, University of Tübingen, Germany.</nlm:affiliation><country xml:lang="fr">Allemagne</country><wicri:regionArea>Department of Psychiatry, University of Tübingen</wicri:regionArea><wicri:noRegion>University of Tübingen</wicri:noRegion><wicri:noRegion>University of Tübingen</wicri:noRegion><wicri:noRegion>University of Tübingen</wicri:noRegion></affiliation></author><author><name sortKey="Corcos, D M" sort="Corcos, D M" uniqKey="Corcos D" first="D M" last="Corcos">D M Corcos</name></author></titleStmt><publicationStmt><idno type="wicri:source">PubMed</idno><date when="1997">1997</date><idno type="RBID">pubmed:9251082</idno><idno type="pmid">9251082</idno><idno type="doi">10.1002/mds.870120420</idno><idno type="wicri:Area/PubMed/Corpus">004630</idno><idno type="wicri:Area/PubMed/Curation">004630</idno><idno type="wicri:Area/PubMed/Checkpoint">004705</idno><idno type="wicri:Area/Ncbi/Merge">004D53</idno></publicationStmt><sourceDesc><biblStruct><analytic><title xml:lang="en">Cerebellar hypermetria: reduction in the early component of the antagonist electromyogram.</title><author><name sortKey="Wild, B" sort="Wild, B" uniqKey="Wild B" first="B" last="Wild">B. Wild</name><affiliation wicri:level="1"><nlm:affiliation>Department of Psychiatry, University of Tübingen, Germany.</nlm:affiliation><country xml:lang="fr">Allemagne</country><wicri:regionArea>Department of Psychiatry, University of Tübingen</wicri:regionArea><wicri:noRegion>University of Tübingen</wicri:noRegion><wicri:noRegion>University of Tübingen</wicri:noRegion><wicri:noRegion>University of Tübingen</wicri:noRegion></affiliation></author><author><name sortKey="Corcos, D M" sort="Corcos, D M" uniqKey="Corcos D" first="D M" last="Corcos">D M Corcos</name></author></analytic><series><title level="j">Movement disorders : official journal of the Movement Disorder Society</title><idno type="ISSN">0885-3185</idno><imprint><date when="1997" type="published">1997</date></imprint></series></biblStruct></sourceDesc></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Cerebellar Diseases (physiopathology)</term><term>Electromyography (methods)</term><term>Electromyography (statistics & numerical data)</term><term>Extremities</term><term>Humans</term><term>Movement Disorders (physiopathology)</term><term>Muscle, Skeletal (physiopathology)</term><term>Reaction Time</term></keywords><keywords scheme="MESH" qualifier="methods" xml:lang="en"><term>Electromyography</term></keywords><keywords scheme="MESH" qualifier="physiopathology" xml:lang="en"><term>Cerebellar Diseases</term><term>Movement Disorders</term><term>Muscle, Skeletal</term></keywords><keywords scheme="MESH" qualifier="statistics & numerical data" xml:lang="en"><term>Electromyography</term></keywords><keywords scheme="MESH" xml:lang="en"><term>Extremities</term><term>Humans</term><term>Reaction Time</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Several articles have described the electromyographic (EMG) patterns underlying cerebellar hypermetria. However, the description of the EMG associated with hypermetria is not consistent between studies. It is hypothesized that the reason for this concerns the criteria used to define and describe the antagonist latency. Several studies on neurologically normal individuals have shown that there are two components to the antagonist EMG: an early component and a late component. However, many studies have identified only one component and have not defined whether it was the early or late component. A reanalysis of one published data set that had previously identified only one antagonist component was performed. The reanalysis suggests that hypermetria can be caused by an absence of an early antagonist EMG component, a delayed late component, or both. In addition, a new measure is suggested that allows this hypothesis to be further evaluated.</div></front></TEI><pubmed><MedlineCitation Owner="NLM" Status="MEDLINE"><PMID Version="1">9251082</PMID><DateCreated><Year>1997</Year><Month>10</Month><Day>14</Day></DateCreated><DateCompleted><Year>1997</Year><Month>10</Month><Day>14</Day></DateCompleted><DateRevised><Year>2007</Year><Month>11</Month><Day>14</Day></DateRevised><Article PubModel="Print"><Journal><ISSN IssnType="Print">0885-3185</ISSN><JournalIssue CitedMedium="Print"><Volume>12</Volume><Issue>4</Issue><PubDate><Year>1997</Year><Month>Jul</Month></PubDate></JournalIssue><Title>Movement disorders : official journal of the Movement Disorder Society</Title><ISOAbbreviation>Mov. Disord.</ISOAbbreviation></Journal><ArticleTitle>Cerebellar hypermetria: reduction in the early component of the antagonist electromyogram.</ArticleTitle><Pagination><MedlinePgn>604-7</MedlinePgn></Pagination><Abstract><AbstractText>Several articles have described the electromyographic (EMG) patterns underlying cerebellar hypermetria. However, the description of the EMG associated with hypermetria is not consistent between studies. It is hypothesized that the reason for this concerns the criteria used to define and describe the antagonist latency. Several studies on neurologically normal individuals have shown that there are two components to the antagonist EMG: an early component and a late component. However, many studies have identified only one component and have not defined whether it was the early or late component. A reanalysis of one published data set that had previously identified only one antagonist component was performed. The reanalysis suggests that hypermetria can be caused by an absence of an early antagonist EMG component, a delayed late component, or both. In addition, a new measure is suggested that allows this hypothesis to be further evaluated.</AbstractText></Abstract><AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Wild</LastName><ForeName>B</ForeName><Initials>B</Initials><AffiliationInfo><Affiliation>Department of Psychiatry, University of Tübingen, Germany.</Affiliation></AffiliationInfo></Author><Author ValidYN="Y"><LastName>Corcos</LastName><ForeName>D M</ForeName><Initials>DM</Initials></Author></AuthorList><Language>eng</Language><GrantList CompleteYN="Y"><Grant><GrantID>KO4-NS01508</GrantID><Acronym>NS</Acronym><Agency>NINDS NIH HHS</Agency><Country>United States</Country></Grant><Grant><GrantID>R01-AR33189</GrantID><Acronym>AR</Acronym><Agency>NIAMS NIH HHS</Agency><Country>United States</Country></Grant><Grant><GrantID>R01-NS28127</GrantID><Acronym>NS</Acronym><Agency>NINDS NIH HHS</Agency><Country>United States</Country></Grant></GrantList><PublicationTypeList><PublicationType UI="D016428">Journal Article</PublicationType><PublicationType UI="D013487">Research Support, U.S. Gov't, P.H.S.</PublicationType><PublicationType UI="D016454">Review</PublicationType></PublicationTypeList></Article><MedlineJournalInfo><Country>UNITED STATES</Country><MedlineTA>Mov Disord</MedlineTA><NlmUniqueID>8610688</NlmUniqueID><ISSNLinking>0885-3185</ISSNLinking></MedlineJournalInfo><CitationSubset>IM</CitationSubset><MeshHeadingList><MeshHeading><DescriptorName MajorTopicYN="N" UI="D002526">Cerebellar Diseases</DescriptorName><QualifierName MajorTopicYN="Y" UI="Q000503">physiopathology</QualifierName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D004576">Electromyography</DescriptorName><QualifierName MajorTopicYN="N" UI="Q000379">methods</QualifierName><QualifierName MajorTopicYN="N" UI="Q000706">statistics & numerical data</QualifierName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D005121">Extremities</DescriptorName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D006801">Humans</DescriptorName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D009069">Movement Disorders</DescriptorName><QualifierName MajorTopicYN="Y" UI="Q000503">physiopathology</QualifierName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D018482">Muscle, Skeletal</DescriptorName><QualifierName MajorTopicYN="N" UI="Q000503">physiopathology</QualifierName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D011930">Reaction Time</DescriptorName></MeshHeading></MeshHeadingList><NumberOfReferences>10</NumberOfReferences></MedlineCitation><PubmedData><History><PubMedPubDate PubStatus="pubmed"><Year>1997</Year><Month>7</Month><Day>1</Day></PubMedPubDate><PubMedPubDate PubStatus="medline"><Year>1997</Year><Month>7</Month><Day>1</Day><Hour>0</Hour><Minute>1</Minute></PubMedPubDate><PubMedPubDate PubStatus="entrez"><Year>1997</Year><Month>7</Month><Day>1</Day><Hour>0</Hour><Minute>0</Minute></PubMedPubDate></History><PublicationStatus>ppublish</PublicationStatus><ArticleIdList><ArticleId IdType="pubmed">9251082</ArticleId><ArticleId IdType="doi">10.1002/mds.870120420</ArticleId></ArticleIdList></PubmedData></pubmed><affiliations><list><country><li>Allemagne</li></country></list><tree><noCountry><name sortKey="Corcos, D M" sort="Corcos, D M" uniqKey="Corcos D" first="D M" last="Corcos">D M Corcos</name></noCountry><country name="Allemagne"><noRegion><name sortKey="Wild, B" sort="Wild, B" uniqKey="Wild B" first="B" last="Wild">B. 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