Suppression of cortical myoclonus by levetiracetam.
Identifieur interne : 000742 ( Ncbi/Merge ); précédent : 000741; suivant : 000743Suppression of cortical myoclonus by levetiracetam.
Auteurs : Robert Schauer [Autriche] ; Markus Singer ; Leopold Saltuari ; Markus KoflerSource :
- Movement disorders : official journal of the Movement Disorder Society [ 0885-3185 ] ; 2002.
English descriptors
- KwdEn :
- Adolescent, Anticonvulsants (adverse effects), Anticonvulsants (therapeutic use), Cerebral Cortex (drug effects), Cerebral Cortex (injuries), Dominance, Cerebral (physiology), Drug Therapy, Combination, Epilepsy, Post-Traumatic (diagnosis), Epilepsy, Post-Traumatic (drug therapy), Humans, Male, Myoclonic Epilepsy, Juvenile (diagnosis), Myoclonic Epilepsy, Juvenile (drug therapy), Piracetam (adverse effects), Piracetam (analogs & derivatives), Piracetam (therapeutic use).
- MESH :
- chemical , adverse effects : Anticonvulsants, Piracetam.
- chemical , analogs & derivatives : Piracetam.
- chemical , therapeutic use : Anticonvulsants, Piracetam.
- diagnosis : Epilepsy, Post-Traumatic, Myoclonic Epilepsy, Juvenile.
- drug effects : Cerebral Cortex.
- drug therapy : Epilepsy, Post-Traumatic, Myoclonic Epilepsy, Juvenile.
- injuries : Cerebral Cortex.
- physiology : Dominance, Cerebral.
- Adolescent, Drug Therapy, Combination, Humans, Male.
Abstract
A 16-year-old boy suffered severely disabling posthypoxic myoclonus. Neurophysiological investigation showed cortical but not reticular reflex myoclonus. Add-on therapy with levetiracetam significantly improved the patient's clinical condition, suppressed cortical myoclonus-associated spikes, and enabled further neurorehabilitation.
PubMed: 11921136
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pubmed:11921136Le document en format XML
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<author><name sortKey="Schauer, Robert" sort="Schauer, Robert" uniqKey="Schauer R" first="Robert" last="Schauer">Robert Schauer</name>
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<wicri:regionArea>Department of Neurology, Hospital Hochzirl, Zirl</wicri:regionArea>
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<author><name sortKey="Singer, Markus" sort="Singer, Markus" uniqKey="Singer M" first="Markus" last="Singer">Markus Singer</name>
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<author><name sortKey="Saltuari, Leopold" sort="Saltuari, Leopold" uniqKey="Saltuari L" first="Leopold" last="Saltuari">Leopold Saltuari</name>
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<author><name sortKey="Kofler, Markus" sort="Kofler, Markus" uniqKey="Kofler M" first="Markus" last="Kofler">Markus Kofler</name>
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<affiliation wicri:level="1"><nlm:affiliation>Department of Neurology, Hospital Hochzirl, Zirl, Austria.</nlm:affiliation>
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<term>Cerebral Cortex (drug effects)</term>
<term>Cerebral Cortex (injuries)</term>
<term>Dominance, Cerebral (physiology)</term>
<term>Drug Therapy, Combination</term>
<term>Epilepsy, Post-Traumatic (diagnosis)</term>
<term>Epilepsy, Post-Traumatic (drug therapy)</term>
<term>Humans</term>
<term>Male</term>
<term>Myoclonic Epilepsy, Juvenile (diagnosis)</term>
<term>Myoclonic Epilepsy, Juvenile (drug therapy)</term>
<term>Piracetam (adverse effects)</term>
<term>Piracetam (analogs & derivatives)</term>
<term>Piracetam (therapeutic use)</term>
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<front><div type="abstract" xml:lang="en">A 16-year-old boy suffered severely disabling posthypoxic myoclonus. Neurophysiological investigation showed cortical but not reticular reflex myoclonus. Add-on therapy with levetiracetam significantly improved the patient's clinical condition, suppressed cortical myoclonus-associated spikes, and enabled further neurorehabilitation.</div>
</front>
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<Month>03</Month>
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<DateCompleted><Year>2002</Year>
<Month>05</Month>
<Day>17</Day>
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<DateRevised><Year>2013</Year>
<Month>11</Month>
<Day>21</Day>
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<Article PubModel="Print"><Journal><ISSN IssnType="Print">0885-3185</ISSN>
<JournalIssue CitedMedium="Print"><Volume>17</Volume>
<Issue>2</Issue>
<PubDate><Year>2002</Year>
<Month>Mar</Month>
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<Title>Movement disorders : official journal of the Movement Disorder Society</Title>
<ISOAbbreviation>Mov. Disord.</ISOAbbreviation>
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<ArticleTitle>Suppression of cortical myoclonus by levetiracetam.</ArticleTitle>
<Pagination><MedlinePgn>411-5</MedlinePgn>
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<Abstract><AbstractText>A 16-year-old boy suffered severely disabling posthypoxic myoclonus. Neurophysiological investigation showed cortical but not reticular reflex myoclonus. Add-on therapy with levetiracetam significantly improved the patient's clinical condition, suppressed cortical myoclonus-associated spikes, and enabled further neurorehabilitation.</AbstractText>
<CopyrightInformation>Copyright 2002 Movement Disorder Society.</CopyrightInformation>
</Abstract>
<AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Schauer</LastName>
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