Abnormalities of sensory processing and sensorimotor interactions in secondary dystonia: a neurophysiological study in two patients.
Identifieur interne : 001073 ( Ncbi/Curation ); précédent : 001072; suivant : 001074Abnormalities of sensory processing and sensorimotor interactions in secondary dystonia: a neurophysiological study in two patients.
Auteurs : Stefano Tamburin [Italie] ; Giampietro ZanetteSource :
- Movement disorders : official journal of the Movement Disorder Society [ 0885-3185 ] ; 2005.
English descriptors
- KwdEn :
- Adult, Brain (pathology), Dystonic Disorders (diagnosis), Dystonic Disorders (physiopathology), Electromyography (instrumentation), Evoked Potentials, Motor (physiology), Evoked Potentials, Somatosensory (physiology), Female, Humans, Magnetic Resonance Imaging, Magnetics (instrumentation), Male, Membrane Potentials (physiology), Perceptual Disorders (diagnosis), Skull.
- MESH :
- diagnosis : Dystonic Disorders, Perceptual Disorders.
- instrumentation : Electromyography, Magnetics.
- pathology : Brain.
- physiology : Evoked Potentials, Motor, Evoked Potentials, Somatosensory, Membrane Potentials.
- physiopathology : Dystonic Disorders.
- Adult, Female, Humans, Magnetic Resonance Imaging, Male, Skull.
Abstract
Experimental data suggest that abnormalities of sensory processing and sensorimotor integration may play a role in the genesis of symptoms in primary dystonia. We studied 2 patients with dystonia secondary to lesions in the somatosensory pathways. We documented sensorimotor alterations in these patients that strongly resemble those found in primary dystonia. Our data are consistent with the hypothesis that abnormalities in sensorimotor processing may contribute to the pathogenesis of dystonic conditions.
DOI: 10.1002/mds.20301
PubMed: 15487012
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pubmed:15487012Le document en format XML
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<sourceDesc><biblStruct><analytic><title xml:lang="en">Abnormalities of sensory processing and sensorimotor interactions in secondary dystonia: a neurophysiological study in two patients.</title>
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<front><div type="abstract" xml:lang="en">Experimental data suggest that abnormalities of sensory processing and sensorimotor integration may play a role in the genesis of symptoms in primary dystonia. We studied 2 patients with dystonia secondary to lesions in the somatosensory pathways. We documented sensorimotor alterations in these patients that strongly resemble those found in primary dystonia. Our data are consistent with the hypothesis that abnormalities in sensorimotor processing may contribute to the pathogenesis of dystonic conditions.</div>
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