Posttraumatic tremor without parkinsonism in a patient with complete contralateral loss of the nigrostriatal pathway.
Identifieur interne : 000915 ( Ncbi/Curation ); précédent : 000914; suivant : 000916Posttraumatic tremor without parkinsonism in a patient with complete contralateral loss of the nigrostriatal pathway.
Auteurs : Jan Zijlmans [Royaume-Uni] ; Jan Booij ; Jaap Valk ; Andrew Lees ; Martin HorstinkSource :
- Movement disorders : official journal of the Movement Disorder Society [ 0885-3185 ] ; 2002.
English descriptors
- KwdEn :
- Adult, Brain Injuries (complications), Brain Injuries (pathology), Corpus Striatum (blood supply), Corpus Striatum (pathology), Electromyography, Hand (physiopathology), Humans, Magnetic Resonance Imaging, Male, Muscle, Skeletal (physiopathology), Neural Pathways (blood supply), Neural Pathways (pathology), Substantia Nigra (blood supply), Substantia Nigra (pathology), Tomography, Emission-Computed, Single-Photon, Tremor (diagnosis), Tremor (etiology), Tremor (physiopathology).
- MESH :
- blood supply : Corpus Striatum, Neural Pathways, Substantia Nigra.
- complications : Brain Injuries.
- diagnosis : Tremor.
- etiology : Tremor.
- pathology : Brain Injuries, Corpus Striatum, Neural Pathways, Substantia Nigra.
- physiopathology : Hand, Muscle, Skeletal, Tremor.
- Adult, Electromyography, Humans, Magnetic Resonance Imaging, Male, Tomography, Emission-Computed, Single-Photon.
Abstract
We present a patient with posttraumatic tremor who did not show any [(123)I]FP-CIT uptake in the contralateral putamen and caudate. The absence of hypokinesia and rigidity is surprising in the presence of a striatal dopaminergic denervation that is even more severe than in Parkinson's disease. An explanation, therefore, could be that the lesion in the subthalamic nucleus in our patient prevented the onset of a Parkinson syndrome.
DOI: 10.1002/mds.10203
PubMed: 12360565
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pubmed:12360565Le document en format XML
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<affiliation wicri:level="3"><nlm:affiliation>Department of Neurology, The National Institute of Neurology and Neurosurgery, Queen Square, London, United Kingdom. j.zijlmans@vumc.nl</nlm:affiliation>
<country xml:lang="fr">Royaume-Uni</country>
<wicri:regionArea>Department of Neurology, The National Institute of Neurology and Neurosurgery, Queen Square, London</wicri:regionArea>
<placeName><settlement type="city">Londres</settlement>
<region type="country">Angleterre</region>
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<author><name sortKey="Booij, Jan" sort="Booij, Jan" uniqKey="Booij J" first="Jan" last="Booij">Jan Booij</name>
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<author><name sortKey="Valk, Jaap" sort="Valk, Jaap" uniqKey="Valk J" first="Jaap" last="Valk">Jaap Valk</name>
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<author><name sortKey="Lees, Andrew" sort="Lees, Andrew" uniqKey="Lees A" first="Andrew" last="Lees">Andrew Lees</name>
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<author><name sortKey="Horstink, Martin" sort="Horstink, Martin" uniqKey="Horstink M" first="Martin" last="Horstink">Martin Horstink</name>
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<author><name sortKey="Lees, Andrew" sort="Lees, Andrew" uniqKey="Lees A" first="Andrew" last="Lees">Andrew Lees</name>
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<series><title level="j">Movement disorders : official journal of the Movement Disorder Society</title>
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<term>Corpus Striatum (blood supply)</term>
<term>Corpus Striatum (pathology)</term>
<term>Electromyography</term>
<term>Hand (physiopathology)</term>
<term>Humans</term>
<term>Magnetic Resonance Imaging</term>
<term>Male</term>
<term>Muscle, Skeletal (physiopathology)</term>
<term>Neural Pathways (blood supply)</term>
<term>Neural Pathways (pathology)</term>
<term>Substantia Nigra (blood supply)</term>
<term>Substantia Nigra (pathology)</term>
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<term>Tremor (diagnosis)</term>
<term>Tremor (etiology)</term>
<term>Tremor (physiopathology)</term>
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<term>Neural Pathways</term>
<term>Substantia Nigra</term>
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<front><div type="abstract" xml:lang="en">We present a patient with posttraumatic tremor who did not show any [(123)I]FP-CIT uptake in the contralateral putamen and caudate. The absence of hypokinesia and rigidity is surprising in the presence of a striatal dopaminergic denervation that is even more severe than in Parkinson's disease. An explanation, therefore, could be that the lesion in the subthalamic nucleus in our patient prevented the onset of a Parkinson syndrome.</div>
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