MovDisordV3, Ncbi, Curation, bibRecord, 000256

Early electrophysiological and histologic changes after global cerebral ischemia in rats.

Identifieur interne : 000256 ( Ncbi/Curation ); précédent : 000255; suivant : 000257

Early electrophysiological and histologic changes after global cerebral ischemia in rats.

Auteurs : R G Geocadin [États-Unis] ; J. Muthuswamy ; D L Sherman ; N V Thakor ; D F Hanley

Source :

Movement disorders : official journal of the Movement Disorder Society [ 0885-3185 ] ; 2000.

RBID : pubmed:10755267

English descriptors

KwdEn :
- Animals, Brain Ischemia (pathology), Brain Ischemia (physiopathology), Brain Mapping, Cerebral Cortex (pathology), Cerebral Cortex (physiopathology), Electroencephalography, Epilepsies, Myoclonic (pathology), Epilepsies, Myoclonic (physiopathology), Evoked Potentials, Somatosensory (physiology), Hypoxia, Brain (pathology), Hypoxia, Brain (physiopathology), Male, Myoclonus (pathology), Myoclonus (physiopathology), Neurons (pathology), Neurons (physiology), Rats, Rats, Wistar, Ventral Thalamic Nuclei (pathology), Ventral Thalamic Nuclei (physiopathology).
MESH :
- pathology : Brain Ischemia, Cerebral Cortex, Epilepsies, Myoclonic, Hypoxia, Brain, Myoclonus, Neurons, Ventral Thalamic Nuclei.
- physiology : Evoked Potentials, Somatosensory, Neurons.
- physiopathology : Brain Ischemia, Cerebral Cortex, Epilepsies, Myoclonic, Hypoxia, Brain, Myoclonus, Ventral Thalamic Nuclei.
- Animals, Brain Mapping, Electroencephalography, Male, Rats, Rats, Wistar.

Abstract

Cerebral anoxia is fundamental to morbidity and mortality after resuscitation from cardiac arrest. With no proven effective primary therapy for post-anoxic brain injury, the goal of neurologic care are supportive, to provide prognosis and prevention of further complications. With the multifaceted approach using electroencephalography (EEG), somatosensory evoked potentials (SEP), multiunit recordings, behavioral and histologic assessment, we investigated the hyperacute recovery period after resuscitation from cardiac arrest in a rat model to define the value of EEG and SEP in assessing neurologic injury.

PubMed: 10755267

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pubmed:10755267

Le document en format XML

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<author><name sortKey="Geocadin, R G" sort="Geocadin, R G" uniqKey="Geocadin R" first="R G" last="Geocadin">R G Geocadin</name>
<affiliation wicri:level="1"><nlm:affiliation>Department of Neurology, The Johns Hopkins University School of Medicine, Baltimore, Maryland 21287, USA.</nlm:affiliation>
<country xml:lang="fr">États-Unis</country>
<wicri:regionArea>Department of Neurology, The Johns Hopkins University School of Medicine, Baltimore, Maryland 21287</wicri:regionArea>
<wicri:noRegion>Maryland 21287</wicri:noRegion>
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<author><name sortKey="Muthuswamy, J" sort="Muthuswamy, J" uniqKey="Muthuswamy J" first="J" last="Muthuswamy">J. Muthuswamy</name>
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<author><name sortKey="Sherman, D L" sort="Sherman, D L" uniqKey="Sherman D" first="D L" last="Sherman">D L Sherman</name>
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<author><name sortKey="Hanley, D F" sort="Hanley, D F" uniqKey="Hanley D" first="D F" last="Hanley">D F Hanley</name>
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<affiliation wicri:level="1"><nlm:affiliation>Department of Neurology, The Johns Hopkins University School of Medicine, Baltimore, Maryland 21287, USA.</nlm:affiliation>
<country xml:lang="fr">États-Unis</country>
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<author><name sortKey="Sherman, D L" sort="Sherman, D L" uniqKey="Sherman D" first="D L" last="Sherman">D L Sherman</name>
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<author><name sortKey="Thakor, N V" sort="Thakor, N V" uniqKey="Thakor N" first="N V" last="Thakor">N V Thakor</name>
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<author><name sortKey="Hanley, D F" sort="Hanley, D F" uniqKey="Hanley D" first="D F" last="Hanley">D F Hanley</name>
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<series><title level="j">Movement disorders : official journal of the Movement Disorder Society</title>
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<term>Brain Mapping</term>
<term>Cerebral Cortex (pathology)</term>
<term>Cerebral Cortex (physiopathology)</term>
<term>Electroencephalography</term>
<term>Epilepsies, Myoclonic (pathology)</term>
<term>Epilepsies, Myoclonic (physiopathology)</term>
<term>Evoked Potentials, Somatosensory (physiology)</term>
<term>Hypoxia, Brain (pathology)</term>
<term>Hypoxia, Brain (physiopathology)</term>
<term>Male</term>
<term>Myoclonus (pathology)</term>
<term>Myoclonus (physiopathology)</term>
<term>Neurons (pathology)</term>
<term>Neurons (physiology)</term>
<term>Rats</term>
<term>Rats, Wistar</term>
<term>Ventral Thalamic Nuclei (pathology)</term>
<term>Ventral Thalamic Nuclei (physiopathology)</term>
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<keywords scheme="MESH" qualifier="pathology" xml:lang="en"><term>Brain Ischemia</term>
<term>Cerebral Cortex</term>
<term>Epilepsies, Myoclonic</term>
<term>Hypoxia, Brain</term>
<term>Myoclonus</term>
<term>Neurons</term>
<term>Ventral Thalamic Nuclei</term>
</keywords>
<keywords scheme="MESH" qualifier="physiology" xml:lang="en"><term>Evoked Potentials, Somatosensory</term>
<term>Neurons</term>
</keywords>
<keywords scheme="MESH" qualifier="physiopathology" xml:lang="en"><term>Brain Ischemia</term>
<term>Cerebral Cortex</term>
<term>Epilepsies, Myoclonic</term>
<term>Hypoxia, Brain</term>
<term>Myoclonus</term>
<term>Ventral Thalamic Nuclei</term>
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<keywords scheme="MESH" xml:lang="en"><term>Animals</term>
<term>Brain Mapping</term>
<term>Electroencephalography</term>
<term>Male</term>
<term>Rats</term>
<term>Rats, Wistar</term>
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<front><div type="abstract" xml:lang="en">Cerebral anoxia is fundamental to morbidity and mortality after resuscitation from cardiac arrest. With no proven effective primary therapy for post-anoxic brain injury, the goal of neurologic care are supportive, to provide prognosis and prevention of further complications. With the multifaceted approach using electroencephalography (EEG), somatosensory evoked potentials (SEP), multiunit recordings, behavioral and histologic assessment, we investigated the hyperacute recovery period after resuscitation from cardiac arrest in a rat model to define the value of EEG and SEP in assessing neurologic injury.</div>
</front>
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	Movement Disorders (revue)
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Movement Disorders (revue)

Early electrophysiological and histologic changes after global cerebral ischemia in rats.

Early electrophysiological and histologic changes after global cerebral ischemia in rats.

Source :

English descriptors

Abstract

Links toward previous steps (curation, corpus...)

Links to Exploration step

Le document en format XML

Pour manipuler ce document sous Unix (Dilib)

Pour mettre un lien sur cette page dans le réseau Wicri

Pour générer des pages wiki