Use of botulinum toxin in pediatric spasticity (cerebral palsy).
Identifieur interne : 000D87 ( Ncbi/Checkpoint ); précédent : 000D86; suivant : 000D88Use of botulinum toxin in pediatric spasticity (cerebral palsy).
Auteurs : Steffen Berweck [Allemagne] ; Florian HeinenSource :
- Movement disorders : official journal of the Movement Disorder Society [ 0885-3185 ] ; 2004.
English descriptors
- KwdEn :
- Botulinum Toxins (therapeutic use), Botulinum Toxins, Type A (therapeutic use), Brain (drug effects), Brain (pathology), Cerebral Palsy (drug therapy), Cerebral Palsy (pathology), Humans, Injections, Intramuscular, Magnetic Resonance Imaging (methods), Neuromuscular Agents (therapeutic use), Pediatrics (methods), Time Factors, Ultrasonography (methods).
- MESH :
- chemical , therapeutic use : Botulinum Toxins, Botulinum Toxins, Type A, Neuromuscular Agents.
- drug effects : Brain.
- drug therapy : Cerebral Palsy.
- methods : Magnetic Resonance Imaging, Pediatrics, Ultrasonography.
- pathology : Brain, Cerebral Palsy.
- Humans, Injections, Intramuscular, Time Factors.
Abstract
Local injection of botulinum toxin (BT) is a well-established treatment option for spastic movement disorders in children. BT blocks the release of acetylcholine from the axon terminal into the synaptic cleft of the motor endplate resulting in paresis of the injected musculature. Such localised, temporary chemodenervation of affected muscles can lead to functional gains and may improve the child's daily routine and rehabilitative care. We summarise state-of-the-art treatment of spasticity in children with BT type A, addressing critical issues and introducing recent advances, such as sonography-guided injection of BT and the distal injection of the psoas muscle without the need for general anaesthesia. First-hand experience with BT type B in children is presented.
DOI: 10.1002/mds.20088
PubMed: 15027070
Affiliations:
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pubmed:15027070Le document en format XML
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BT blocks the release of acetylcholine from the axon terminal into the synaptic cleft of the motor endplate resulting in paresis of the injected musculature. Such localised, temporary chemodenervation of affected muscles can lead to functional gains and may improve the child's daily routine and rehabilitative care. We summarise state-of-the-art treatment of spasticity in children with BT type A, addressing critical issues and introducing recent advances, such as sonography-guided injection of BT and the distal injection of the psoas muscle without the need for general anaesthesia. First-hand experience with BT type B in children is presented.</div></front></TEI><affiliations><list><country><li>Allemagne</li></country><region><li>Bavière</li><li>District de Haute-Bavière</li></region><settlement><li>Munich</li></settlement></list><tree><noCountry><name sortKey="Heinen, Florian" sort="Heinen, Florian" uniqKey="Heinen F" first="Florian" last="Heinen">Florian Heinen</name></noCountry><country name="Allemagne"><region name="Bavière"><name sortKey="Berweck, Steffen" sort="Berweck, Steffen" uniqKey="Berweck S" first="Steffen" last="Berweck">Steffen Berweck</name></region></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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