MovDisordV3, Main, Merge, bibRecord, 003843

Group I Nonreciprocal Inhibition in Primary Restless Legs Syndrome

Identifieur interne : 003843 ( Main/Merge ); précédent : 003842; suivant : 003844

Group I Nonreciprocal Inhibition in Primary Restless Legs Syndrome

Auteurs : Cesa Scaglione [Italie] ; Roberto Vetrugno [Italie] ; Giuseppe Plazzi [Italie] ; Giovanni Rizzo [Italie] ; Federica Provini [Italie] ; Pasquale Montagna [Italie] ; Paolo Martinelli [Italie]

Source :

Movement disorders [ 0885-3185 ] ; 2008.

RBID : Pascal:08-0115902

Descripteurs français

Pascal (Inist)
- Syndrome des jambes sans repos, Pathologie du système nerveux, Réflexe, Formation réticulée.

English descriptors

KwdEn :
- Nervous system diseases, Reflex, Restless legs syndrome, Reticular formation.

Abstract

Electrophysiological investigations of restless legs syndrome (RLS) have found spinal circuits impinging on motoneurones. We evaluated the H reflex threshold, latency, the Hmax/Mmax ratio, and the short latency autogenic inhibition in 7 patients with RLS and 10 age-matched controls by testing the excitability changes in soleus H reflex Ib interneuron function. A significant reduction in Ib inhibition at 4 (P = 0.043), 5 (P = 0.007), and 6 ms (P = 0.001) of H reflex conditioning inter-stimulus interval was found in RLS patients. Data support the hypothesis that altered group I nonreciprocal inhibition is implicated in enhancing the spinal circuitry excitability of RLS, and are consistent with the view of an abnormal supraspinal drive to spinal interneurons in RLS.

Links toward previous steps (curation, corpus...)

to stream PascalFrancis, to step Corpus: 001411
to stream PascalFrancis, to step Curation: 001908
to stream PascalFrancis, to step Checkpoint: 001235

Links to Exploration step

Pascal:08-0115902

Le document en format XML

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<profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Nervous system diseases</term>
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<front><div type="abstract" xml:lang="en">Electrophysiological investigations of restless legs syndrome (RLS) have found spinal circuits impinging on motoneurones. We evaluated the H reflex threshold, latency, the Hmax/Mmax ratio, and the short latency autogenic inhibition in 7 patients with RLS and 10 age-matched controls by testing the excitability changes in soleus H reflex Ib interneuron function. A significant reduction in Ib inhibition at 4 (P = 0.043), 5 (P = 0.007), and 6 ms (P = 0.001) of H reflex conditioning inter-stimulus interval was found in RLS patients. Data support the hypothesis that altered group I nonreciprocal inhibition is implicated in enhancing the spinal circuitry excitability of RLS, and are consistent with the view of an abnormal supraspinal drive to spinal interneurons in RLS.</div>
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	Movement Disorders (revue)
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Movement Disorders (revue)

Group I Nonreciprocal Inhibition in Primary Restless Legs Syndrome

Group I Nonreciprocal Inhibition in Primary Restless Legs Syndrome

Source :

Descripteurs français

English descriptors

Abstract

Links toward previous steps (curation, corpus...)

Links to Exploration step

Le document en format XML

Pour manipuler ce document sous Unix (Dilib)

Pour mettre un lien sur cette page dans le réseau Wicri