Distinct basal ganglia hyperechogenicity in idiopathic basal ganglia calcification
Identifieur interne : 002179 ( Main/Merge ); précédent : 002178; suivant : 002180Distinct basal ganglia hyperechogenicity in idiopathic basal ganglia calcification
Auteurs : Norbert Brüggemann [Allemagne] ; Susanne A. Schneider [Allemagne] ; Thurid Sander [Allemagne] ; Christine Klein [Allemagne] ; Johann Hagenah [Allemagne]Source :
- Movement Disorders [ 0885-3185 ] ; 2010-11-15.
English descriptors
- KwdEn :
- Aged, Basal Ganglia (pathology), Basal Ganglia (ultrasonography), Basal Ganglia Diseases (pathology), Basal Ganglia Diseases (ultrasonography), Calcinosis (pathology), Calcinosis (ultrasonography), Cognition Disorders (pathology), Cognition Disorders (ultrasonography), Humans, Male, Parkinsonian Disorders (pathology), Parkinsonian Disorders (ultrasonography), Ultrasonography, Doppler, Transcranial, idiopathic basal ganglia calcification, lenticular nucleus, parkinsonism, transcranial sonography.
- MESH :
- pathology : Basal Ganglia, Basal Ganglia Diseases, Calcinosis, Cognition Disorders, Parkinsonian Disorders.
- ultrasonography : Basal Ganglia, Basal Ganglia Diseases, Calcinosis, Cognition Disorders, Parkinsonian Disorders.
- Aged, Humans, Male, Ultrasonography, Doppler, Transcranial.
Abstract
We report a 67‐year‐old patient with idiopathic basal ganglia calcification (IBGC). He presented with progressive cognitive impairment, frontal lobe dysfunction, mild leg spasticity, and levodopa (L‐dopa)‐responsive parkinsonism. Transcranial sonography (TCS) revealed marked hyperechogenicity of the basal ganglia and periventricular spaces bilaterally. The detected signal alterations showed a fairly symmetric distribution and corresponded to the hyperintense calcifications depicted on the computer tomography brain scan. The combination of symmetric hyperechogenic areas adjacent to the lateral ventricles and of the basal ganglia may serve as an imaging marker characteristic of IBGC. Hyperechogenicity due to extended basal ganglia calcification as presented here is distinct from the pattern of hyperechogenicity caused by heavy metal accumulation, which is described to be less striking. In addition to atypical parkinsonian syndromes such as progressive supranuclear palsy and multiple system atrophy, IBGC is thus another differential diagnosis of parkinsonism with basal ganglia hyperechogenicity. © 2010 Movement Disorder Society.
Url:
DOI: 10.1002/mds.23264
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<front><div type="abstract" xml:lang="en">We report a 67‐year‐old patient with idiopathic basal ganglia calcification (IBGC). He presented with progressive cognitive impairment, frontal lobe dysfunction, mild leg spasticity, and levodopa (L‐dopa)‐responsive parkinsonism. Transcranial sonography (TCS) revealed marked hyperechogenicity of the basal ganglia and periventricular spaces bilaterally. The detected signal alterations showed a fairly symmetric distribution and corresponded to the hyperintense calcifications depicted on the computer tomography brain scan. The combination of symmetric hyperechogenic areas adjacent to the lateral ventricles and of the basal ganglia may serve as an imaging marker characteristic of IBGC. Hyperechogenicity due to extended basal ganglia calcification as presented here is distinct from the pattern of hyperechogenicity caused by heavy metal accumulation, which is described to be less striking. In addition to atypical parkinsonian syndromes such as progressive supranuclear palsy and multiple system atrophy, IBGC is thus another differential diagnosis of parkinsonism with basal ganglia hyperechogenicity. © 2010 Movement Disorder Society.</div>
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<term>Calcinosis (ultrasonography)</term>
<term>Cognition Disorders (pathology)</term>
<term>Cognition Disorders (ultrasonography)</term>
<term>Humans</term>
<term>Male</term>
<term>Parkinsonian Disorders (pathology)</term>
<term>Parkinsonian Disorders (ultrasonography)</term>
<term>Ultrasonography, Doppler, Transcranial</term>
</keywords>
<keywords scheme="MESH" qualifier="pathology" xml:lang="en"><term>Basal Ganglia</term>
<term>Basal Ganglia Diseases</term>
<term>Calcinosis</term>
<term>Cognition Disorders</term>
<term>Parkinsonian Disorders</term>
</keywords>
<keywords scheme="MESH" qualifier="ultrasonography" xml:lang="en"><term>Basal Ganglia</term>
<term>Basal Ganglia Diseases</term>
<term>Calcinosis</term>
<term>Cognition Disorders</term>
<term>Parkinsonian Disorders</term>
</keywords>
<keywords scheme="MESH" xml:lang="en"><term>Aged</term>
<term>Humans</term>
<term>Male</term>
<term>Ultrasonography, Doppler, Transcranial</term>
</keywords>
</textClass>
</profileDesc>
</teiHeader>
<front><div type="abstract" xml:lang="en">We report a 67-year-old patient with idiopathic basal ganglia calcification (IBGC). He presented with progressive cognitive impairment, frontal lobe dysfunction, mild leg spasticity, and levodopa (L-dopa)-responsive parkinsonism. Transcranial sonography (TCS) revealed marked hyperechogenicity of the basal ganglia and periventricular spaces bilaterally. The detected signal alterations showed a fairly symmetric distribution and corresponded to the hyperintense calcifications depicted on the computer tomography brain scan. The combination of symmetric hyperechogenic areas adjacent to the lateral ventricles and of the basal ganglia may serve as an imaging marker characteristic of IBGC. Hyperechogenicity due to extended basal ganglia calcification as presented here is distinct from the pattern of hyperechogenicity caused by heavy metal accumulation, which is described to be less striking. In addition to atypical parkinsonian syndromes such as progressive supranuclear palsy and multiple system atrophy, IBGC is thus another differential diagnosis of parkinsonism with basal ganglia hyperechogenicity.</div>
</front>
</TEI>
</PubMed>
</double>
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