Movement Disorders (revue) - Exploration (Accueil)

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Receptors, Dopamine D1 (ultrastructure) < Receptors, Dopamine D2 < Receptors, Dopamine D2 (agonists)  Facettes :

List of bibliographic references indexed by Receptors, Dopamine D2

Number of relevant bibliographic references: 7.
Ident.Authors (with country if any)Title
005F93 (1992) Schwarz [Allemagne] ; K. Tatsch [Allemagne] ; T. Vogl [Allemagne] ; C. M. Kirsch [Allemagne] ; C. Trenkwalder [Allemagne] ; G. Arnold [Allemagne] ; T. Gasser [Allemagne] ; W. H. Oertel [Allemagne]Marked reduction of striatal dopamine D2 receptors as detected by 123IBZM‐SPECT in a Wilson's disease patient with generalized dystonia
006271 (1990) Cedarbaum [États-Unis] ; Mary Clark [États-Unis] ; Linda H. Toy [États-Unis] ; Alison Green-Parsons [États-Unis]Sustained‐release (+)‐PHNO [MK‐458 (HPMC)] in the treatment of Parkinson's disease: Evidence for tolerance to a selective D2‐receptor agonist administered as a long‐acting formulation
006272 (1990) Lucinda G. Miller [États-Unis] ; Jankovic [États-Unis]Sulpiride‐induced tardive dystonia
006286 (1990) Rinne [Finlande] ; A. Laihinen [Finlande] ; J. O. Rinne [Finlande] ; K. Na Gren [Finlande] ; J. Bergman [Finlande] ; U. Ruotsalainen [Finlande]Positron emission tomography demonstrates dopamine D2 receptor supersensitivity in the striatum of patients with early Parkinson's disease
006287 (1990) K. D. Sethi [États-Unis] ; Ana Hitri [États-Unis] ; Bruce I. Diamond [États-Unis]Phenytoin potentiation of neuroleptic‐induced dyskinesias
006681 (1986) Leenders [Royaume-Uni] ; R. S. J. Frackowiak [Royaume-Uni] ; N. Quinn [Royaume-Uni] ; D. Brooks [Royaume-Uni] ; D. Sumner [Royaume-Uni] ; C. D. Marsden [Royaume-Uni]Ipsilateral blepharospasm and contralateral hemidystonia and parkinsonism in a patient with a unilateral rostral brainstem– thalamic lesion: Structural and functional abnormalities studied with CT, MRI, and PET scanning
006697 (1986) Leenders [Royaume-Uni] ; R. S. J. Frackowiak [Royaume-Uni] ; N. Quinn [Royaume-Uni] ; C. D. Marsden [Royaume-Uni]Brain energy metabolism and dopaminergic function in Huntington's disease measured in vivo using positron emission tomography

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