List of bibliographic references indexed by Pathogenesis
Number of relevant bibliographic references: 124.
[0-20] [
0 - 20][
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20-40]
Ident. | Authors (with country if any) | Title |
---|
001671 (2011) |
Anthony H. Schapira [Royaume-Uni] ; Peter Jenner [Royaume-Uni] | Etiology and pathogenesis of Parkinson's disease |
001C23 (2010) |
Serge Przedborski [États-Unis] | Inflammation and Parkinson's disease pathogenesis |
001F71 (2010) |
| Annual Symposium on Etiology, Pathogenesis, and treatment of Parkinson's disease and other movement disorders |
002269 (2009) |
Nagaendran Kandiah [Singapour] ; Kevin Tan [Singapour] ; C. C. Tchoyoson Lim [Singapour] ; Narayanaswamy Venketasubramanian [Singapour] | Hyperglycemic choreoathetosis: Role of the putamen in pathogenesis |
002530 (2009) |
| Annual symposium on etiology, pathogenesis, and treatment of Parkinson's disease and other movement disorders |
002827 (2008) |
Meg E. Morris [Australie] ; Robert Iansek [Australie] ; Brook Galna [Australie] | Gait festination and freezing in Parkinson's disease: Pathogenesis and rehabilitation |
002A64 (2008) |
Alicja Lemer [États-Unis] ; Anto Bagic [États-Unis] | Olfactory Pathogenesis of Idiopathic Parkinson Disease Revisited |
002B32 (2007) |
C. Warren Olanow [États-Unis] | The pathogenesis of cell death in Parkinson's disease – 2007 |
003109 (2007) |
| Annual Symposium on Etiology, Pathogenesis, and Treatment of Parkinson's Disese and Other Movement Disorders |
003219 (2006) |
Alex J. Sinclair [Royaume-Uni] ; Lucy Barling [Royaume-Uni] ; Simon Nightingale [Royaume-Uni] | Recurrent dystonia in homocystinuria: A metabolic pathogenesis |
003578 (2006) |
Martin Wilhelm Kurz [Allemagne, Norvège] ; Jan Petter Larsen [Norvège] ; Jan Terje Kvaloy [Norvège] ; Dag Aarsland [Norvège] | Associations between family history of Parkinson's disease and dementia and risk of dementia in Parkinson's disease: A community‐based, longitudinal study |
003618 (2006) |
| Annual Symposia on Etiology, Pathogenesis, and Treatment of Parkinson's Disease and Other Movement Disorders |
003827 (2005) |
C. Warren Olanow [États-Unis] ; Joseph Jankovic [États-Unis] | Neuroprotective therapy in Parkinson's disease and motor complications: A search for a pathogenesis‐targeted, disease‐modifying strategy |
003E43 (2004) |
H. A. Jinnah [États-Unis] ; Ellen J. Hess [États-Unis] ; Mark S. Ledoux [États-Unis] ; Nutan Sharma [États-Unis] ; Mark G. Baxter [Royaume-Uni] ; Mahlon R. Delong [États-Unis] | Rodent models for dystonia research : Characteristics, evaluation, and utility |
003F00 (2003) |
Alberto J. Espay [Canada] ; Peter Ashby [Canada] ; Ritsuko Hanajima [Canada] ; Mandar S. Jog [Canada] ; Anthony E. Lang [Canada] | Unique form of propriospinal myoclonus as a possible complication of an enteropathogenic toxin |
003F45 (2003) |
Giovanni Abbruzzese [Italie] ; Alfredo Berardelli [Italie] | Sensorimotor integration in movement disorders |
003F47 (2003) |
Mark S. Ledoux [États-Unis] ; Kimberly A. Brady [États-Unis] | Secondary cervical dystonia associated with structural lesions of the central nervous system |
004007 (2003) |
Peter Teismann [États-Unis] ; Kim Tieu [États-Unis] ; Oren Cohen [États-Unis] ; Dong-Kug Choi [États-Unis] ; Du Chu Wu [États-Unis] ; Daniel Marks [États-Unis] ; Miquel Vila [États-Unis] ; Vernice Jackson-Lewis [États-Unis] ; Serge Przedborski [États-Unis] | Pathogenic role of glial cells in Parkinson's disease |
004026 (2003) |
Paul S. Fitzmaurice [Canada] ; Lee Ang [Canada] ; Mark Guttman [Canada] ; Ali H. Rajput [Canada] ; Yoshiaki Furukawa [Canada] ; Stephen J. Kish [Canada] | Nigral glutathione deficiency is not specific for idiopathic Parkinson's disease |
004051 (2003) |
Ram N C. Leiguarda [Argentine] ; Marcelo Merello [Argentine] ; María Inés Nouzeilles [Argentine] ; Jorge Balej [Argentine] ; Alberto Rivero [Argentine] ; Martín Nogués [Argentine] | Limb‐kinetic apraxia in corticobasal degeneration: Clinical and kinematic features |
004092 (2003) |
Jay S. Schneider [États-Unis] ; Timothy V. Wade [États-Unis] | Experimental parkinsonism is associated with increased pallidal GAD gene expression and is reversed by site‐directed antisense gene therapy |
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