Alterations of striatal neurons in benign hereditary chorea
Identifieur interne : 003126 ( Istex/Curation ); précédent : 003125; suivant : 003127Alterations of striatal neurons in benign hereditary chorea
Auteurs : Galit Kleiner-Fisman [États-Unis] ; Noel Y. Calingasan [États-Unis] ; Mary Putt [États-Unis] ; June Chen [États-Unis] ; M. Flint Beal [États-Unis] ; Anthony E. Lang [Canada]Source :
- Movement Disorders [ 0885-3185 ] ; 2005-10.
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Abstract
Benign hereditary chorea (BHC) recently has been associated with mutations in TITF‐1 gene, although a pathological study of an individual with BHC and a TITF‐1 mutation revealed no significant gross or microscopic abnormalities using standard methods. Immunohistochemical staining of striatal tissue from a BHC‐affected postmortem brain was performed using antibodies against neurotransmitters of interneurons whose tangential migration is mediated by TITF‐1. There was a loss of most TITF‐1–mediated striatal interneurons in the BHC specimen compared to four matched control brains. © 2005 Movement Disorder Society
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DOI: 10.1002/mds.20577
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<front><div type="abstract" xml:lang="en">Benign hereditary chorea (BHC) recently has been associated with mutations in TITF‐1 gene, although a pathological study of an individual with BHC and a TITF‐1 mutation revealed no significant gross or microscopic abnormalities using standard methods. Immunohistochemical staining of striatal tissue from a BHC‐affected postmortem brain was performed using antibodies against neurotransmitters of interneurons whose tangential migration is mediated by TITF‐1. There was a loss of most TITF‐1–mediated striatal interneurons in the BHC specimen compared to four matched control brains. © 2005 Movement Disorder Society</div>
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