Reflex blepharospasm associated with bilateral basal ganglia lesion
Identifieur interne : 003A03 ( Istex/Corpus ); précédent : 003A02; suivant : 003A04Reflex blepharospasm associated with bilateral basal ganglia lesion
Auteurs : R. Larumbe ; J. Vaamonde ; J. Artieda ; J. L. Zubieta ; ObesoSource :
- Movement Disorders [ 0885-3185 ] ; 1993.
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Abstract
A patient with a bilateral striatal lesion secondary to anoxia presented reflex blepharospasm associated with parkinsonism and dystonia in the limbs. The blink reflex excitability curve was enhanced and the R‐2 response prolonged as in patients with essential blepharospasm. The findings in this patient support the notion that blepharospasm may be secondary to basal ganglia dysfunction through abnormal facilitation of reticular formation neurons controlling facial nucleus motoneuron excitability.
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DOI: 10.1002/mds.870080215
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ISTEX:7105A63D544E0592F95C09006AF69D4AEC4A3432Le document en format XML
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The blink reflex excitability curve was enhanced and the R‐2 response prolonged as in patients with essential blepharospasm. The findings in this patient support the notion that blepharospasm may be secondary to basal ganglia dysfunction through abnormal facilitation of reticular formation neurons controlling facial nucleus motoneuron excitability.</div></front></TEI><istex><corpusName>wiley</corpusName><author><json:item><name>R. Larumbe</name><affiliations><json:string>Movement Disorders Unit and Clinical Neurophysiology Service, Department of Neurology and Neuroradiology Unit, Department of Radiology, Clinica Universitaria, University of Navarra Medical School, Pamplona, Spain</json:string></affiliations></json:item><json:item><name>J. Vaamonde</name><affiliations><json:string>Movement Disorders Unit and Clinical Neurophysiology Service, Department of Neurology and Neuroradiology Unit, Department of Radiology, Clinica Universitaria, University of Navarra Medical School, Pamplona, Spain</json:string></affiliations></json:item><json:item><name>J. Artieda</name><affiliations><json:string>Movement Disorders Unit and Clinical Neurophysiology Service, Department of Neurology and Neuroradiology Unit, Department of Radiology, Clinica Universitaria, University of Navarra Medical School, Pamplona, Spain</json:string></affiliations></json:item><json:item><name>J. L. 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The blink reflex excitability curve was enhanced and the R‐2 response prolonged as in patients with essential blepharospasm. 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L.</namePart><namePart type="family">Zubieta</namePart><affiliation>Movement Disorders Unit and Clinical Neurophysiology Service, Department of Neurology and Neuroradiology Unit, Department of Radiology, Clinica Universitaria, University of Navarra Medical School, Pamplona, Spain</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="termsOfAddress">Dr.</namePart><namePart type="family">Obeso</namePart><affiliation>Movement Disorders Unit and Clinical Neurophysiology Service, Department of Neurology and Neuroradiology Unit, Department of Radiology, Clinica Universitaria, University of Navarra Medical School, Pamplona, Spain</affiliation><description>Correspondence: Department of Neurology, Clinica Universitaria, Apdo. 192, 31080 Pamplona, Spain</description><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre authority="originalCategForm">article</genre><originInfo><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher><place><placeTerm type="text">Hoboken</placeTerm></place><dateIssued encoding="w3cdtf">1993</dateIssued><copyrightDate encoding="w3cdtf">1993</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType><extent unit="figures">3</extent><extent unit="references">22</extent></physicalDescription><abstract lang="en">A patient with a bilateral striatal lesion secondary to anoxia presented reflex blepharospasm associated with parkinsonism and dystonia in the limbs. The blink reflex excitability curve was enhanced and the R‐2 response prolonged as in patients with essential blepharospasm. The findings in this patient support the notion that blepharospasm may be secondary to basal ganglia dysfunction through abnormal facilitation of reticular formation neurons controlling facial nucleus motoneuron excitability.</abstract><subject lang="en"><genre>Keywords</genre><topic>Blepharospasm</topic><topic>Blink reflex</topic><topic>Striatal lesion</topic></subject><relatedItem type="host"><titleInfo><title>Movement Disorders</title><subTitle>Official Journal of the Movement Disorder Society</subTitle></titleInfo><titleInfo type="abbreviated"><title>Mov. 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