Electrophysiology of the corticomotoneurone pathways in patients with movement disorders
Identifieur interne : 003695 ( Istex/Corpus ); précédent : 003694; suivant : 003696Electrophysiology of the corticomotoneurone pathways in patients with movement disorders
Auteurs : P. D. Thompson ; J. P. R. Dick ; B. L. Day ; J. C. Rothwell ; A. Berardelli ; T. Kachi ; C. D. MarsdenSource :
- Movement Disorders [ 0885-3185 ] ; 1986.
English descriptors
Abstract
The corticomotoneurone pathways were examined in 21 patients with movement disorders, using the technique of percutaneous electrical stimulation of the motor cortex. Conduction in these pathways was assessed by measuring the latency to onset of electromyographic activity in the muscles of the upper limb after cortical stimulation. In all patients [five with primary (idiopathic) torsion dystonia and two with secondary (symptomatic) hemidystonia, seven with Huntington's disease, four with essential tremor, and three with Parkinson's disease] central motor conduction was normal. This and other evidence suggests that the origin of the disorder of movement in these conditions lies in the delivery of abnormal motor commands to a normal corticomotoneuronal system.
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DOI: 10.1002/mds.870010205
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D.</namePart><namePart type="family">Marsden</namePart><affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</affiliation><description>Correspondence: University Department of Neurology, London SE5 8AF, England</description><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre authority="originalCategForm">article</genre><originInfo><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher><place><placeTerm type="text">Hoboken</placeTerm></place><dateIssued encoding="w3cdtf">1986</dateIssued><copyrightDate encoding="w3cdtf">1986</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType><extent unit="figures">1</extent><extent unit="tables">1</extent><extent unit="references">5</extent></physicalDescription><abstract lang="en">The corticomotoneurone pathways were examined in 21 patients with movement disorders, using the technique of percutaneous electrical stimulation of the motor cortex. Conduction in these pathways was assessed by measuring the latency to onset of electromyographic activity in the muscles of the upper limb after cortical stimulation. In all patients [five with primary (idiopathic) torsion dystonia and two with secondary (symptomatic) hemidystonia, seven with Huntington's disease, four with essential tremor, and three with Parkinson's disease] central motor conduction was normal. This and other evidence suggests that the origin of the disorder of movement in these conditions lies in the delivery of abnormal motor commands to a normal corticomotoneuronal system.</abstract><subject lang="en"><genre>Keywords</genre><topic>Corticomotoneurone pathways</topic><topic>Movement disorders</topic><topic>Central motor conduction</topic></subject><relatedItem type="host"><titleInfo><title>Movement Disorders</title><subTitle>Official Journal of the Movement Disorder Society</subTitle></titleInfo><titleInfo type="abbreviated"><title>Mov. 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