Electrophysiology of the corticomotoneurone pathways in patients with movement disorders
Identifieur interne : 003695 ( Istex/Corpus ); précédent : 003694; suivant : 003696Electrophysiology of the corticomotoneurone pathways in patients with movement disorders
Auteurs : P. D. Thompson ; J. P. R. Dick ; B. L. Day ; J. C. Rothwell ; A. Berardelli ; T. Kachi ; C. D. MarsdenSource :
- Movement Disorders [ 0885-3185 ] ; 1986.
English descriptors
Abstract
The corticomotoneurone pathways were examined in 21 patients with movement disorders, using the technique of percutaneous electrical stimulation of the motor cortex. Conduction in these pathways was assessed by measuring the latency to onset of electromyographic activity in the muscles of the upper limb after cortical stimulation. In all patients [five with primary (idiopathic) torsion dystonia and two with secondary (symptomatic) hemidystonia, seven with Huntington's disease, four with essential tremor, and three with Parkinson's disease] central motor conduction was normal. This and other evidence suggests that the origin of the disorder of movement in these conditions lies in the delivery of abnormal motor commands to a normal corticomotoneuronal system.
Url:
DOI: 10.1002/mds.870010205
Links to Exploration step
ISTEX:F0ACE88E36EAD95399D3F98FE03C5F9891B7B1ECLe document en format XML
<record><TEI wicri:istexFullTextTei="biblStruct"><teiHeader><fileDesc><titleStmt><title xml:lang="en">Electrophysiology of the corticomotoneurone pathways in patients with movement disorders</title>
<author><name sortKey="Thompson, P D" sort="Thompson, P D" uniqKey="Thompson P" first="P. D." last="Thompson">P. D. Thompson</name>
<affiliation><mods:affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</mods:affiliation>
</affiliation>
</author>
<author><name sortKey="Dick, J P R" sort="Dick, J P R" uniqKey="Dick J" first="J. P. R." last="Dick">J. P. R. Dick</name>
<affiliation><mods:affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</mods:affiliation>
</affiliation>
</author>
<author><name sortKey="Day, B L" sort="Day, B L" uniqKey="Day B" first="B. L." last="Day">B. L. Day</name>
<affiliation><mods:affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</mods:affiliation>
</affiliation>
</author>
<author><name sortKey="Rothwell, J C" sort="Rothwell, J C" uniqKey="Rothwell J" first="J. C." last="Rothwell">J. C. Rothwell</name>
<affiliation><mods:affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</mods:affiliation>
</affiliation>
</author>
<author><name sortKey="Berardelli, A" sort="Berardelli, A" uniqKey="Berardelli A" first="A." last="Berardelli">A. Berardelli</name>
<affiliation><mods:affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</mods:affiliation>
</affiliation>
</author>
<author><name sortKey="Kachi, T" sort="Kachi, T" uniqKey="Kachi T" first="T." last="Kachi">T. Kachi</name>
<affiliation><mods:affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</mods:affiliation>
</affiliation>
</author>
<author><name sortKey="Marsden, C D" sort="Marsden, C D" uniqKey="Marsden C" first="C. D." last="Marsden">C. D. Marsden</name>
<affiliation><mods:affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</mods:affiliation>
</affiliation>
</author>
</titleStmt>
<publicationStmt><idno type="wicri:source">ISTEX</idno>
<idno type="RBID">ISTEX:F0ACE88E36EAD95399D3F98FE03C5F9891B7B1EC</idno>
<date when="1986" year="1986">1986</date>
<idno type="doi">10.1002/mds.870010205</idno>
<idno type="url">https://api.istex.fr/document/F0ACE88E36EAD95399D3F98FE03C5F9891B7B1EC/fulltext/pdf</idno>
<idno type="wicri:Area/Istex/Corpus">003695</idno>
</publicationStmt>
<sourceDesc><biblStruct><analytic><title level="a" type="main" xml:lang="en">Electrophysiology of the corticomotoneurone pathways in patients with movement disorders</title>
<author><name sortKey="Thompson, P D" sort="Thompson, P D" uniqKey="Thompson P" first="P. D." last="Thompson">P. D. Thompson</name>
<affiliation><mods:affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</mods:affiliation>
</affiliation>
</author>
<author><name sortKey="Dick, J P R" sort="Dick, J P R" uniqKey="Dick J" first="J. P. R." last="Dick">J. P. R. Dick</name>
<affiliation><mods:affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</mods:affiliation>
</affiliation>
</author>
<author><name sortKey="Day, B L" sort="Day, B L" uniqKey="Day B" first="B. L." last="Day">B. L. Day</name>
<affiliation><mods:affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</mods:affiliation>
</affiliation>
</author>
<author><name sortKey="Rothwell, J C" sort="Rothwell, J C" uniqKey="Rothwell J" first="J. C." last="Rothwell">J. C. Rothwell</name>
<affiliation><mods:affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</mods:affiliation>
</affiliation>
</author>
<author><name sortKey="Berardelli, A" sort="Berardelli, A" uniqKey="Berardelli A" first="A." last="Berardelli">A. Berardelli</name>
<affiliation><mods:affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</mods:affiliation>
</affiliation>
</author>
<author><name sortKey="Kachi, T" sort="Kachi, T" uniqKey="Kachi T" first="T." last="Kachi">T. Kachi</name>
<affiliation><mods:affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</mods:affiliation>
</affiliation>
</author>
<author><name sortKey="Marsden, C D" sort="Marsden, C D" uniqKey="Marsden C" first="C. D." last="Marsden">C. D. Marsden</name>
<affiliation><mods:affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</mods:affiliation>
</affiliation>
</author>
</analytic>
<monogr></monogr>
<series><title level="j">Movement Disorders</title>
<title level="j" type="sub">Official Journal of the Movement Disorder Society</title>
<title level="j" type="abbrev">Mov. Disord.</title>
<idno type="ISSN">0885-3185</idno>
<idno type="eISSN">1531-8257</idno>
<imprint><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher>
<pubPlace>Hoboken</pubPlace>
<date type="published" when="1986">1986</date>
<biblScope unit="vol">1</biblScope>
<biblScope unit="issue">2</biblScope>
<biblScope unit="page" from="113">113</biblScope>
<biblScope unit="page" to="117">117</biblScope>
</imprint>
<idno type="ISSN">0885-3185</idno>
</series>
<idno type="istex">F0ACE88E36EAD95399D3F98FE03C5F9891B7B1EC</idno>
<idno type="DOI">10.1002/mds.870010205</idno>
<idno type="ArticleID">MDS870010205</idno>
</biblStruct>
</sourceDesc>
<seriesStmt><idno type="ISSN">0885-3185</idno>
</seriesStmt>
</fileDesc>
<profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Central motor conduction</term>
<term>Corticomotoneurone pathways</term>
<term>Movement disorders</term>
</keywords>
</textClass>
<langUsage><language ident="en">en</language>
</langUsage>
</profileDesc>
</teiHeader>
<front><div type="abstract" xml:lang="en">The corticomotoneurone pathways were examined in 21 patients with movement disorders, using the technique of percutaneous electrical stimulation of the motor cortex. Conduction in these pathways was assessed by measuring the latency to onset of electromyographic activity in the muscles of the upper limb after cortical stimulation. In all patients [five with primary (idiopathic) torsion dystonia and two with secondary (symptomatic) hemidystonia, seven with Huntington's disease, four with essential tremor, and three with Parkinson's disease] central motor conduction was normal. This and other evidence suggests that the origin of the disorder of movement in these conditions lies in the delivery of abnormal motor commands to a normal corticomotoneuronal system.</div>
</front>
</TEI>
<istex><corpusName>wiley</corpusName>
<author><json:item><name>P. D. Thompson</name>
<affiliations><json:string>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</json:string>
</affiliations>
</json:item>
<json:item><name>J. P. R. Dick</name>
<affiliations><json:string>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</json:string>
</affiliations>
</json:item>
<json:item><name>B. L. Day</name>
<affiliations><json:string>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</json:string>
</affiliations>
</json:item>
<json:item><name>J. C. Rothwell</name>
<affiliations><json:string>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</json:string>
</affiliations>
</json:item>
<json:item><name>A. Berardelli</name>
<affiliations><json:string>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</json:string>
</affiliations>
</json:item>
<json:item><name>T. Kachi</name>
<affiliations><json:string>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</json:string>
</affiliations>
</json:item>
<json:item><name>C. D. Marsden</name>
<affiliations><json:string>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</json:string>
</affiliations>
</json:item>
</author>
<subject><json:item><lang><json:string>eng</json:string>
</lang>
<value>Corticomotoneurone pathways</value>
</json:item>
<json:item><lang><json:string>eng</json:string>
</lang>
<value>Movement disorders</value>
</json:item>
<json:item><lang><json:string>eng</json:string>
</lang>
<value>Central motor conduction</value>
</json:item>
</subject>
<language><json:string>eng</json:string>
</language>
<abstract>The corticomotoneurone pathways were examined in 21 patients with movement disorders, using the technique of percutaneous electrical stimulation of the motor cortex. Conduction in these pathways was assessed by measuring the latency to onset of electromyographic activity in the muscles of the upper limb after cortical stimulation. In all patients [five with primary (idiopathic) torsion dystonia and two with secondary (symptomatic) hemidystonia, seven with Huntington's disease, four with essential tremor, and three with Parkinson's disease] central motor conduction was normal. This and other evidence suggests that the origin of the disorder of movement in these conditions lies in the delivery of abnormal motor commands to a normal corticomotoneuronal system.</abstract>
<qualityIndicators><score>2.89</score>
<pdfVersion>1.3</pdfVersion>
<pdfPageSize>468 x 720 pts</pdfPageSize>
<refBibsNative>true</refBibsNative>
<abstractCharCount>767</abstractCharCount>
<pdfWordCount>1582</pdfWordCount>
<pdfCharCount>9681</pdfCharCount>
<pdfPageCount>5</pdfPageCount>
<abstractWordCount>109</abstractWordCount>
</qualityIndicators>
<title>Electrophysiology of the corticomotoneurone pathways in patients with movement disorders</title>
<genre><json:string>Serial article</json:string>
</genre>
<host><volume>1</volume>
<pages><total>5</total>
<last>117</last>
<first>113</first>
</pages>
<issn><json:string>0885-3185</json:string>
</issn>
<issue>2</issue>
<subject><json:item><value>Original Article</value>
</json:item>
</subject>
<genre></genre>
<language><json:string>unknown</json:string>
</language>
<title>Movement Disorders</title>
<doi><json:string>10.1002/(ISSN)1531-8257</json:string>
</doi>
</host>
<publicationDate>1986</publicationDate>
<copyrightDate>1986</copyrightDate>
<doi><json:string>10.1002/mds.870010205</json:string>
</doi>
<id>F0ACE88E36EAD95399D3F98FE03C5F9891B7B1EC</id>
<fulltext><json:item><original>true</original>
<mimetype>application/pdf</mimetype>
<extension>pdf</extension>
<uri>https://api.istex.fr/document/F0ACE88E36EAD95399D3F98FE03C5F9891B7B1EC/fulltext/pdf</uri>
</json:item>
<json:item><original>false</original>
<mimetype>application/zip</mimetype>
<extension>zip</extension>
<uri>https://api.istex.fr/document/F0ACE88E36EAD95399D3F98FE03C5F9891B7B1EC/fulltext/zip</uri>
</json:item>
<istex:fulltextTEI uri="https://api.istex.fr/document/F0ACE88E36EAD95399D3F98FE03C5F9891B7B1EC/fulltext/tei"><teiHeader type="text"><fileDesc><titleStmt><title level="a" type="main" xml:lang="en">Electrophysiology of the corticomotoneurone pathways in patients with movement disorders</title>
</titleStmt>
<publicationStmt><authority>ISTEX</authority>
<publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher>
<pubPlace>Hoboken</pubPlace>
<availability><p>Wiley Subscription Services, Inc., A Wiley Company</p>
</availability>
<date>1986</date>
</publicationStmt>
<sourceDesc><biblStruct type="inbook"><analytic><title level="a" type="main" xml:lang="en">Electrophysiology of the corticomotoneurone pathways in patients with movement disorders</title>
<author><persName><forename type="first">P. D.</forename>
<surname>Thompson</surname>
</persName>
<affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</affiliation>
</author>
<author><persName><forename type="first">J. P. R.</forename>
<surname>Dick</surname>
</persName>
<affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</affiliation>
</author>
<author><persName><forename type="first">B. L.</forename>
<surname>Day</surname>
</persName>
<affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</affiliation>
</author>
<author><persName><forename type="first">J. C.</forename>
<surname>Rothwell</surname>
</persName>
<affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</affiliation>
</author>
<author><persName><forename type="first">A.</forename>
<surname>Berardelli</surname>
</persName>
<affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</affiliation>
</author>
<author><persName><forename type="first">T.</forename>
<surname>Kachi</surname>
</persName>
<affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</affiliation>
</author>
<author><persName><forename type="first">C. D.</forename>
<surname>Marsden</surname>
</persName>
<note type="correspondence"><p>Correspondence: University Department of Neurology, London SE5 8AF, England</p>
</note>
<affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</affiliation>
</author>
</analytic>
<monogr><title level="j">Movement Disorders</title>
<title level="j" type="sub">Official Journal of the Movement Disorder Society</title>
<title level="j" type="abbrev">Mov. Disord.</title>
<idno type="pISSN">0885-3185</idno>
<idno type="eISSN">1531-8257</idno>
<idno type="DOI">10.1002/(ISSN)1531-8257</idno>
<imprint><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher>
<pubPlace>Hoboken</pubPlace>
<date type="published" when="1986"></date>
<biblScope unit="vol">1</biblScope>
<biblScope unit="issue">2</biblScope>
<biblScope unit="page" from="113">113</biblScope>
<biblScope unit="page" to="117">117</biblScope>
</imprint>
</monogr>
<idno type="istex">F0ACE88E36EAD95399D3F98FE03C5F9891B7B1EC</idno>
<idno type="DOI">10.1002/mds.870010205</idno>
<idno type="ArticleID">MDS870010205</idno>
</biblStruct>
</sourceDesc>
</fileDesc>
<profileDesc><creation><date>1986</date>
</creation>
<langUsage><language ident="en">en</language>
</langUsage>
<abstract xml:lang="en"><p>The corticomotoneurone pathways were examined in 21 patients with movement disorders, using the technique of percutaneous electrical stimulation of the motor cortex. Conduction in these pathways was assessed by measuring the latency to onset of electromyographic activity in the muscles of the upper limb after cortical stimulation. In all patients [five with primary (idiopathic) torsion dystonia and two with secondary (symptomatic) hemidystonia, seven with Huntington's disease, four with essential tremor, and three with Parkinson's disease] central motor conduction was normal. This and other evidence suggests that the origin of the disorder of movement in these conditions lies in the delivery of abnormal motor commands to a normal corticomotoneuronal system.</p>
</abstract>
<textClass xml:lang="en"><keywords scheme="keyword"><list><head>Keywords</head>
<item><term>Corticomotoneurone pathways</term>
</item>
<item><term>Movement disorders</term>
</item>
<item><term>Central motor conduction</term>
</item>
</list>
</keywords>
</textClass>
<textClass><keywords scheme="Journal Subject"><list><head>Article category</head>
<item><term>Original Article</term>
</item>
</list>
</keywords>
</textClass>
</profileDesc>
<revisionDesc><change when="1986">Published</change>
</revisionDesc>
</teiHeader>
</istex:fulltextTEI>
<json:item><original>false</original>
<mimetype>text/plain</mimetype>
<extension>txt</extension>
<uri>https://api.istex.fr/document/F0ACE88E36EAD95399D3F98FE03C5F9891B7B1EC/fulltext/txt</uri>
</json:item>
</fulltext>
<metadata><istex:metadataXml wicri:clean="Wiley, elements deleted: body"><istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="yes"</istex:xmlDeclaration>
<istex:document><component version="2.0" type="serialArticle" xml:lang="en"><header><publicationMeta level="product"><publisherInfo><publisherName>Wiley Subscription Services, Inc., A Wiley Company</publisherName>
<publisherLoc>Hoboken</publisherLoc>
</publisherInfo>
<doi registered="yes">10.1002/(ISSN)1531-8257</doi>
<issn type="print">0885-3185</issn>
<issn type="electronic">1531-8257</issn>
<idGroup><id type="product" value="MDS"></id>
</idGroup>
<titleGroup><title type="main" xml:lang="en" sort="MOVEMENT DISORDERS">Movement Disorders</title>
<title type="subtitle">Official Journal of the Movement Disorder Society</title>
<title type="short">Mov. Disord.</title>
</titleGroup>
</publicationMeta>
<publicationMeta level="part" position="20"><doi origin="wiley" registered="yes">10.1002/mds.v1:2</doi>
<numberingGroup><numbering type="journalVolume" number="1">1</numbering>
<numbering type="journalIssue">2</numbering>
</numberingGroup>
<coverDate startDate="1986">1986</coverDate>
</publicationMeta>
<publicationMeta level="unit" type="article" position="5" status="forIssue"><doi origin="wiley" registered="yes">10.1002/mds.870010205</doi>
<idGroup><id type="unit" value="MDS870010205"></id>
</idGroup>
<countGroup><count type="pageTotal" number="5"></count>
</countGroup>
<titleGroup><title type="articleCategory">Original Article</title>
<title type="tocHeading1">Original Articles</title>
</titleGroup>
<copyright ownership="thirdParty">Copyright © 1986 Movement Disorder Society</copyright>
<eventGroup><event type="firstOnline" date="2005-05-23"></event>
<event type="publishedOnlineFinalForm" date="2005-05-23"></event>
<event type="xmlConverted" agent="Converter:JWSART34_TO_WML3G version:2.3.2 mode:FullText source:HeaderRef result:HeaderRef" date="2010-03-09"></event>
<event type="xmlConverted" agent="Converter:WILEY_ML3G_TO_WILEY_ML3GV2 version:3.8.8" date="2014-02-02"></event>
<event type="xmlConverted" agent="Converter:WML3G_To_WML3G version:4.1.7 mode:FullText,remove_FC" date="2014-10-31"></event>
</eventGroup>
<numberingGroup><numbering type="pageFirst">113</numbering>
<numbering type="pageLast">117</numbering>
</numberingGroup>
<correspondenceTo>University Department of Neurology, London SE5 8AF, England</correspondenceTo>
<linkGroup><link type="toTypesetVersion" href="file:MDS.MDS870010205.pdf"></link>
</linkGroup>
</publicationMeta>
<contentMeta><countGroup><count type="figureTotal" number="1"></count>
<count type="tableTotal" number="1"></count>
<count type="referenceTotal" number="5"></count>
</countGroup>
<titleGroup><title type="main" xml:lang="en">Electrophysiology of the corticomotoneurone pathways in patients with movement disorders</title>
<title type="short" xml:lang="en">CORTICOMOTONEURONE PATHWAYS</title>
</titleGroup>
<creators><creator xml:id="au1" creatorRole="author" affiliationRef="#af1"><personName><givenNames>P. D.</givenNames>
<familyName>Thompson</familyName>
</personName>
</creator>
<creator xml:id="au2" creatorRole="author" affiliationRef="#af1"><personName><givenNames>J. P. R.</givenNames>
<familyName>Dick</familyName>
</personName>
</creator>
<creator xml:id="au3" creatorRole="author" affiliationRef="#af1"><personName><givenNames>B. L.</givenNames>
<familyName>Day</familyName>
</personName>
</creator>
<creator xml:id="au4" creatorRole="author" affiliationRef="#af1"><personName><givenNames>J. C.</givenNames>
<familyName>Rothwell</familyName>
</personName>
</creator>
<creator xml:id="au5" creatorRole="author" affiliationRef="#af1"><personName><givenNames>A.</givenNames>
<familyName>Berardelli</familyName>
</personName>
</creator>
<creator xml:id="au6" creatorRole="author" affiliationRef="#af1"><personName><givenNames>T.</givenNames>
<familyName>Kachi</familyName>
</personName>
</creator>
<creator xml:id="au7" creatorRole="author" affiliationRef="#af1" corresponding="yes"><personName><givenNames>C. D.</givenNames>
<familyName>Marsden</familyName>
</personName>
</creator>
</creators>
<affiliationGroup><affiliation xml:id="af1" countryCode="GB" type="organization"><unparsedAffiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</unparsedAffiliation>
</affiliation>
</affiliationGroup>
<keywordGroup xml:lang="en" type="author"><keyword xml:id="kwd1">Corticomotoneurone pathways</keyword>
<keyword xml:id="kwd2">Movement disorders</keyword>
<keyword xml:id="kwd3">Central motor conduction</keyword>
</keywordGroup>
<abstractGroup><abstract type="main" xml:lang="en"><title type="main">Abstract</title>
<p>The corticomotoneurone pathways were examined in 21 patients with movement disorders, using the technique of percutaneous electrical stimulation of the motor cortex. Conduction in these pathways was assessed by measuring the latency to onset of electromyographic activity in the muscles of the upper limb after cortical stimulation. In all patients [five with primary (idiopathic) torsion dystonia and two with secondary (symptomatic) hemidystonia, seven with Huntington's disease, four with essential tremor, and three with Parkinson's disease] central motor conduction was normal. This and other evidence suggests that the origin of the disorder of movement in these conditions lies in the delivery of abnormal motor commands to a normal corticomotoneuronal system.</p>
</abstract>
</abstractGroup>
</contentMeta>
</header>
</component>
</istex:document>
</istex:metadataXml>
<!--Version 0.6 générée le 4-12-2015--><mods version="3.6"><titleInfo lang="en"><title>Electrophysiology of the corticomotoneurone pathways in patients with movement disorders</title>
</titleInfo>
<titleInfo type="abbreviated" lang="en"><title>CORTICOMOTONEURONE PATHWAYS</title>
</titleInfo>
<titleInfo type="alternative" contentType="CDATA" lang="en"><title>Electrophysiology of the corticomotoneurone pathways in patients with movement disorders</title>
</titleInfo>
<name type="personal"><namePart type="given">P. D.</namePart>
<namePart type="family">Thompson</namePart>
<affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</affiliation>
<role><roleTerm type="text">author</roleTerm>
</role>
</name>
<name type="personal"><namePart type="given">J. P. R.</namePart>
<namePart type="family">Dick</namePart>
<affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</affiliation>
<role><roleTerm type="text">author</roleTerm>
</role>
</name>
<name type="personal"><namePart type="given">B. L.</namePart>
<namePart type="family">Day</namePart>
<affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</affiliation>
<role><roleTerm type="text">author</roleTerm>
</role>
</name>
<name type="personal"><namePart type="given">J. C.</namePart>
<namePart type="family">Rothwell</namePart>
<affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</affiliation>
<role><roleTerm type="text">author</roleTerm>
</role>
</name>
<name type="personal"><namePart type="given">A.</namePart>
<namePart type="family">Berardelli</namePart>
<affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</affiliation>
<role><roleTerm type="text">author</roleTerm>
</role>
</name>
<name type="personal"><namePart type="given">T.</namePart>
<namePart type="family">Kachi</namePart>
<affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</affiliation>
<role><roleTerm type="text">author</roleTerm>
</role>
</name>
<name type="personal"><namePart type="given">C. D.</namePart>
<namePart type="family">Marsden</namePart>
<affiliation>University Department of Neurology, King's College Hospital, and the Institute of the Psychiatry, London, England</affiliation>
<description>Correspondence: University Department of Neurology, London SE5 8AF, England</description>
<role><roleTerm type="text">author</roleTerm>
</role>
</name>
<typeOfResource>text</typeOfResource>
<genre authority="originalCategForm">article</genre>
<originInfo><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher>
<place><placeTerm type="text">Hoboken</placeTerm>
</place>
<dateIssued encoding="w3cdtf">1986</dateIssued>
<copyrightDate encoding="w3cdtf">1986</copyrightDate>
</originInfo>
<language><languageTerm type="code" authority="rfc3066">en</languageTerm>
<languageTerm type="code" authority="iso639-2b">eng</languageTerm>
</language>
<physicalDescription><internetMediaType>text/html</internetMediaType>
<extent unit="figures">1</extent>
<extent unit="tables">1</extent>
<extent unit="references">5</extent>
</physicalDescription>
<abstract lang="en">The corticomotoneurone pathways were examined in 21 patients with movement disorders, using the technique of percutaneous electrical stimulation of the motor cortex. Conduction in these pathways was assessed by measuring the latency to onset of electromyographic activity in the muscles of the upper limb after cortical stimulation. In all patients [five with primary (idiopathic) torsion dystonia and two with secondary (symptomatic) hemidystonia, seven with Huntington's disease, four with essential tremor, and three with Parkinson's disease] central motor conduction was normal. This and other evidence suggests that the origin of the disorder of movement in these conditions lies in the delivery of abnormal motor commands to a normal corticomotoneuronal system.</abstract>
<subject lang="en"><genre>Keywords</genre>
<topic>Corticomotoneurone pathways</topic>
<topic>Movement disorders</topic>
<topic>Central motor conduction</topic>
</subject>
<relatedItem type="host"><titleInfo><title>Movement Disorders</title>
<subTitle>Official Journal of the Movement Disorder Society</subTitle>
</titleInfo>
<titleInfo type="abbreviated"><title>Mov. Disord.</title>
</titleInfo>
<subject><genre>article category</genre>
<topic>Original Article</topic>
</subject>
<identifier type="ISSN">0885-3185</identifier>
<identifier type="eISSN">1531-8257</identifier>
<identifier type="DOI">10.1002/(ISSN)1531-8257</identifier>
<identifier type="PublisherID">MDS</identifier>
<part><date>1986</date>
<detail type="volume"><caption>vol.</caption>
<number>1</number>
</detail>
<detail type="issue"><caption>no.</caption>
<number>2</number>
</detail>
<extent unit="pages"><start>113</start>
<end>117</end>
<total>5</total>
</extent>
</part>
</relatedItem>
<identifier type="istex">F0ACE88E36EAD95399D3F98FE03C5F9891B7B1EC</identifier>
<identifier type="DOI">10.1002/mds.870010205</identifier>
<identifier type="ArticleID">MDS870010205</identifier>
<accessCondition type="use and reproduction" contentType="copyright">Copyright © 1986 Movement Disorder Society</accessCondition>
<recordInfo><recordOrigin>Wiley Subscription Services, Inc., A Wiley Company</recordOrigin>
<recordContentSource>WILEY</recordContentSource>
</recordInfo>
</mods>
</metadata>
<serie></serie>
</istex>
</record>
Pour manipuler ce document sous Unix (Dilib)
EXPLOR_STEP=$WICRI_ROOT/Wicri/Santé/explor/MovDisordV3/Data/Istex/Corpus
HfdSelect -h $EXPLOR_STEP/biblio.hfd -nk 003695 | SxmlIndent | more
Ou
HfdSelect -h $EXPLOR_AREA/Data/Istex/Corpus/biblio.hfd -nk 003695 | SxmlIndent | more
Pour mettre un lien sur cette page dans le réseau Wicri
{{Explor lien |wiki= Wicri/Santé |area= MovDisordV3 |flux= Istex |étape= Corpus |type= RBID |clé= ISTEX:F0ACE88E36EAD95399D3F98FE03C5F9891B7B1EC |texte= Electrophysiology of the corticomotoneurone pathways in patients with movement disorders }}
![]() | This area was generated with Dilib version V0.6.23. | ![]() |