Camptocormia associated with focal myositis in multiple‐system atrophy
Identifieur interne : 002F98 ( Istex/Corpus ); précédent : 002F97; suivant : 002F99Camptocormia associated with focal myositis in multiple‐system atrophy
Auteurs : Nico J. Diederich ; Hans H. Goebel ; Georges Dooms ; Anja Bumb ; Frank Huber ; Katie Kompoliti ; Hans-Michael M. MeinckSource :
- Movement Disorders [ 0885-3185 ] ; 2006-03.
English descriptors
Abstract
Camptocormia (CC) or pronounced forward flexion of the trunk is a common symptom of Parkinson's disease. We describe 2 patients with probable, respectively possible multiple‐system atrophy and CC. Magnetic resonance imaging of the erector trunci showed focal patchy hyperintensities with gadolinium enhancement and muscle biopsy was indicative of variably pronounced focal myositis. CC was progressive and the major handicap for both patients after 1 and 1.5 years of follow‐up, respectively. The therapeutic response was poor. Similarities with the dropped‐head syndrome suggest that the muscle pathology may be either the primary cause of CC, a focal reaction to the CC posture, or a coincident syndrome of old age. © 2005 Movement Disorder Society
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DOI: 10.1002/mds.20686
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ISTEX:0FC9E1194FA11E0CF98B8D66AD884D8C44307EBFLe document en format XML
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Meinck</name><affiliation><mods:affiliation>Department of Neurology, University Hospitals, Heidelberg, Germany</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j">Movement Disorders</title><title level="j" type="abbrev">Mov. Disord.</title><idno type="ISSN">0885-3185</idno><idno type="eISSN">1531-8257</idno><imprint><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher><pubPlace>Hoboken</pubPlace><date type="published" when="2006-03">2006-03</date><biblScope unit="vol">21</biblScope><biblScope unit="issue">3</biblScope><biblScope unit="page" from="390">390</biblScope><biblScope unit="page" to="394">394</biblScope></imprint><idno type="ISSN">0885-3185</idno></series><idno type="istex">0FC9E1194FA11E0CF98B8D66AD884D8C44307EBF</idno><idno type="DOI">10.1002/mds.20686</idno><idno type="ArticleID">MDS20686</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0885-3185</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>bent‐spine syndrome</term><term>camptocormia, myositis</term><term>dropped‐head syndrome</term><term>multiple‐system atrophy</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Camptocormia (CC) or pronounced forward flexion of the trunk is a common symptom of Parkinson's disease. We describe 2 patients with probable, respectively possible multiple‐system atrophy and CC. Magnetic resonance imaging of the erector trunci showed focal patchy hyperintensities with gadolinium enhancement and muscle biopsy was indicative of variably pronounced focal myositis. CC was progressive and the major handicap for both patients after 1 and 1.5 years of follow‐up, respectively. The therapeutic response was poor. Similarities with the dropped‐head syndrome suggest that the muscle pathology may be either the primary cause of CC, a focal reaction to the CC posture, or a coincident syndrome of old age. © 2005 Movement Disorder Society</div></front></TEI><istex><corpusName>wiley</corpusName><author><json:item><name>Nico J. Diederich MD</name><affiliations><json:string>Department of Neuroscience, Centre Hospitalier de Luxembourg, Luxembourg, Luxembourg</json:string><json:string>Department of Neurological Sciences, Rush University Medical Center, Chicago, Illinois, USA</json:string></affiliations></json:item><json:item><name>Hans H. Goebel MD</name><affiliations><json:string>Department of Neuropathology, Johannes Gutenberg University Medical Center, Mainz, Germany</json:string></affiliations></json:item><json:item><name>Georges Dooms MD</name><affiliations><json:string>Department of Radiology, Centre Hospitalier de Luxembourg, Luxembourg, Luxembourg</json:string></affiliations></json:item><json:item><name>Anja Bumb MD</name><affiliations><json:string>Department of Neuroscience, Centre Hospitalier de Luxembourg, Luxembourg, Luxembourg</json:string></affiliations></json:item><json:item><name>Frank Huber MD</name><affiliations><json:string>Department of Neuroscience, Centre Hospitalier de Luxembourg, Luxembourg, Luxembourg</json:string></affiliations></json:item><json:item><name>Katie Kompoliti MD</name><affiliations><json:string>Department of Neurological Sciences, Rush University Medical Center, Chicago, Illinois, USA</json:string></affiliations></json:item><json:item><name>Hans‐Michael M. 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Magnetic resonance imaging of the erector trunci showed focal patchy hyperintensities with gadolinium enhancement and muscle biopsy was indicative of variably pronounced focal myositis. CC was progressive and the major handicap for both patients after 1 and 1.5 years of follow‐up, respectively. The therapeutic response was poor. 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Similarities with the dropped‐head syndrome suggest that the muscle pathology may be either the primary cause of CC, a focal reaction to the CC posture, or a coincident syndrome of old age. © 2005 Movement Disorder Society</p></abstract><textClass xml:lang="en"><keywords scheme="keyword"><list><head>Keywords</head><item><term>multiple‐system atrophy</term></item><item><term>camptocormia, myositis</term></item><item><term>bent‐spine syndrome</term></item><item><term>dropped‐head syndrome</term></item></list></keywords></textClass><textClass><keywords scheme="Journal Subject"><list><head>Article category</head><item><term>Brief Report</term></item></list></keywords></textClass></profileDesc><revisionDesc><change when="2004-09-02">Received</change><change when="2005-05-13">Registration</change><change when="2006-03">Published</change></revisionDesc></teiHeader></istex:fulltextTEI><json:item><original>false</original><mimetype>text/plain</mimetype><extension>txt</extension><uri>https://api.istex.fr/document/0FC9E1194FA11E0CF98B8D66AD884D8C44307EBF/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Wiley, elements deleted: body"><istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="yes"</istex:xmlDeclaration><istex:document><component version="2.0" type="serialArticle" xml:lang="en"><header><publicationMeta level="product"><publisherInfo><publisherName>Wiley Subscription Services, Inc., A Wiley Company</publisherName><publisherLoc>Hoboken</publisherLoc></publisherInfo><doi registered="yes">10.1002/(ISSN)1531-8257</doi><issn type="print">0885-3185</issn><issn type="electronic">1531-8257</issn><idGroup><id type="product" value="MDS"></id></idGroup><titleGroup><title type="main" xml:lang="en" sort="MOVEMENT DISORDERS">Movement Disorders</title><title type="short">Mov. 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type="organization"><unparsedAffiliation>Department of Neuroscience, Centre Hospitalier de Luxembourg, Luxembourg, Luxembourg</unparsedAffiliation></affiliation><affiliation xml:id="af2" countryCode="US" type="organization"><unparsedAffiliation>Department of Neurological Sciences, Rush University Medical Center, Chicago, Illinois, USA</unparsedAffiliation></affiliation><affiliation xml:id="af3" countryCode="DE" type="organization"><unparsedAffiliation>Department of Neuropathology, Johannes Gutenberg University Medical Center, Mainz, Germany</unparsedAffiliation></affiliation><affiliation xml:id="af4" countryCode="LU" type="organization"><unparsedAffiliation>Department of Radiology, Centre Hospitalier de Luxembourg, Luxembourg, Luxembourg</unparsedAffiliation></affiliation><affiliation xml:id="af5" countryCode="DE" type="organization"><unparsedAffiliation>Department of Neurology, University Hospitals, Heidelberg, Germany</unparsedAffiliation></affiliation></affiliationGroup><keywordGroup xml:lang="en" type="author"><keyword xml:id="kwd1">multiple‐system atrophy</keyword><keyword xml:id="kwd2">camptocormia, myositis</keyword><keyword xml:id="kwd3">bent‐spine syndrome</keyword><keyword xml:id="kwd4">dropped‐head syndrome</keyword></keywordGroup><abstractGroup><abstract type="main" xml:lang="en"><title type="main">Abstract</title><p>Camptocormia (CC) or pronounced forward flexion of the trunk is a common symptom of Parkinson's disease. We describe 2 patients with probable, respectively possible multiple‐system atrophy and CC. Magnetic resonance imaging of the erector trunci showed focal patchy hyperintensities with gadolinium enhancement and muscle biopsy was indicative of variably pronounced focal myositis. CC was progressive and the major handicap for both patients after 1 and 1.5 years of follow‐up, respectively. The therapeutic response was poor. Similarities with the dropped‐head syndrome suggest that the muscle pathology may be either the primary cause of CC, a focal reaction to the CC posture, or a coincident syndrome of old age. © 2005 Movement Disorder Society</p></abstract></abstractGroup></contentMeta></header></component></istex:document></istex:metadataXml><!--Version 0.6 générée le 3-12-2015--><mods version="3.6"><titleInfo lang="en"><title>Camptocormia associated with focal myositis in multiple‐system atrophy</title></titleInfo><titleInfo type="abbreviated" lang="en"><title>Camptocormia Associated With Focal Myositis</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>Camptocormia associated with focal myositis in multiple‐system atrophy</title></titleInfo><name type="personal"><namePart type="given">Nico J.</namePart><namePart type="family">Diederich</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Neuroscience, Centre Hospitalier de Luxembourg, Luxembourg, Luxembourg</affiliation><affiliation>Department of Neurological Sciences, Rush University Medical Center, Chicago, Illinois, USA</affiliation><description>Correspondence: Department of Neuroscience, Centre Hospitalier de Luxembourg, 5, rue Barblé, L‐1210 Luxembourg, Luxembourg</description><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Hans H.</namePart><namePart type="family">Goebel</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Neuropathology, Johannes Gutenberg University Medical Center, Mainz, Germany</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Georges</namePart><namePart type="family">Dooms</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Radiology, Centre Hospitalier de Luxembourg, Luxembourg, Luxembourg</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Anja</namePart><namePart type="family">Bumb</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Neuroscience, Centre Hospitalier de Luxembourg, Luxembourg, Luxembourg</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Frank</namePart><namePart type="family">Huber</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Neuroscience, Centre Hospitalier de Luxembourg, Luxembourg, Luxembourg</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Katie</namePart><namePart type="family">Kompoliti</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Neurological Sciences, Rush University Medical Center, Chicago, Illinois, USA</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Hans‐Michael M.</namePart><namePart type="family">Meinck</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Neurology, University Hospitals, Heidelberg, Germany</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre authority="originalCategForm">article</genre><originInfo><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher><place><placeTerm type="text">Hoboken</placeTerm></place><dateIssued encoding="w3cdtf">2006-03</dateIssued><dateCaptured encoding="w3cdtf">2004-09-02</dateCaptured><dateValid encoding="w3cdtf">2005-05-13</dateValid><copyrightDate encoding="w3cdtf">2006</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType><extent unit="figures">2</extent><extent unit="references">22</extent><extent unit="words">2573</extent></physicalDescription><abstract lang="en">Camptocormia (CC) or pronounced forward flexion of the trunk is a common symptom of Parkinson's disease. We describe 2 patients with probable, respectively possible multiple‐system atrophy and CC. Magnetic resonance imaging of the erector trunci showed focal patchy hyperintensities with gadolinium enhancement and muscle biopsy was indicative of variably pronounced focal myositis. CC was progressive and the major handicap for both patients after 1 and 1.5 years of follow‐up, respectively. The therapeutic response was poor. Similarities with the dropped‐head syndrome suggest that the muscle pathology may be either the primary cause of CC, a focal reaction to the CC posture, or a coincident syndrome of old age. © 2005 Movement Disorder Society</abstract><subject lang="en"><genre>Keywords</genre><topic>multiple‐system atrophy</topic><topic>camptocormia, myositis</topic><topic>bent‐spine syndrome</topic><topic>dropped‐head syndrome</topic></subject><relatedItem type="host"><titleInfo><title>Movement Disorders</title></titleInfo><titleInfo type="abbreviated"><title>Mov. Disord.</title></titleInfo><subject><genre>article category</genre><topic>Brief Report</topic></subject><identifier type="ISSN">0885-3185</identifier><identifier type="eISSN">1531-8257</identifier><identifier type="DOI">10.1002/(ISSN)1531-8257</identifier><identifier type="PublisherID">MDS</identifier><part><date>2006</date><detail type="volume"><caption>vol.</caption><number>21</number></detail><detail type="issue"><caption>no.</caption><number>3</number></detail><extent unit="pages"><start>390</start><end>394</end><total>5</total></extent></part></relatedItem><identifier type="istex">0FC9E1194FA11E0CF98B8D66AD884D8C44307EBF</identifier><identifier type="DOI">10.1002/mds.20686</identifier><identifier type="ArticleID">MDS20686</identifier><accessCondition type="use and reproduction" contentType="copyright">Copyright © 2005 Movement Disorder Society</accessCondition><recordInfo><recordOrigin>Wiley Subscription Services, Inc., A Wiley Company</recordOrigin><recordContentSource>WILEY</recordContentSource></recordInfo></mods></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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