Comparison of acute‐ and delayed‐onset posttraumatic cervical dystonia
Identifieur interne : 002C00 ( Istex/Corpus ); précédent : 002B99; suivant : 002C01Comparison of acute‐ and delayed‐onset posttraumatic cervical dystonia
Auteurs : Daniel TarsySource :
- Movement Disorders [ 0885-3185 ] ; 1998-05.
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- KwdEn :
Abstract
Head, neck, or shoulder trauma is an occasional antecedent event before the appearance of cervical dystonia. A clinically distinctive syndrome of acute‐onset posttraumatic cervical dystonia characterized by markedly restricted range of neck motion, absence of phasic involuntary movements, and poor response to treatment has previously been described. Patients with cervical dystonia attending a movement disorder clinic were reviewed for history of trauma before onset of symptoms. Patients with symptom onset within 4 weeks of trauma were compared with patients who developed symptoms between 3 months and 1 year after trauma. Acute‐onset cervical dystonia was characterized by markedly reduced cervical mobility; prominent shoulder elevation with trapezius hypertrophy in most patients, absence of involuntary movements, sensory tricks, or activation maneuvers; and poor response to botulinum toxin injection. By contrast, delayed‐onset cervical dystonia was clinically indistinguishable from nontraumatic idiopathic cervical dystonia. Acute‐onset posttraumatic cervical dystonia is similar to limb dystonia after peripheral trauma and may represent a form of nondystonic muscle spasm similar to torticollis associated with musculoskeletal injuries of the cervical spine and craniocervical junction.
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DOI: 10.1002/mds.870130318
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ISTEX:6B8578DEE12606D6760B48CE118EDB422A0B1EC4Le document en format XML
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A clinically distinctive syndrome of acute‐onset posttraumatic cervical dystonia characterized by markedly restricted range of neck motion, absence of phasic involuntary movements, and poor response to treatment has previously been described. Patients with cervical dystonia attending a movement disorder clinic were reviewed for history of trauma before onset of symptoms. Patients with symptom onset within 4 weeks of trauma were compared with patients who developed symptoms between 3 months and 1 year after trauma. Acute‐onset cervical dystonia was characterized by markedly reduced cervical mobility; prominent shoulder elevation with trapezius hypertrophy in most patients, absence of involuntary movements, sensory tricks, or activation maneuvers; and poor response to botulinum toxin injection. By contrast, delayed‐onset cervical dystonia was clinically indistinguishable from nontraumatic idiopathic cervical dystonia. Acute‐onset posttraumatic cervical dystonia is similar to limb dystonia after peripheral trauma and may represent a form of nondystonic muscle spasm similar to torticollis associated with musculoskeletal injuries of the cervical spine and craniocervical junction.</div></front></TEI><istex><corpusName>wiley</corpusName><author><json:item><name>Daniel Tarsy MD</name><affiliations><json:string>Department of Neurology, Beth Israel Deaconess Medical Center, and Department of Neurology, Harvard Medical School, Boston, Massachusetts, U.S.A.</json:string></affiliations></json:item></author><subject><json:item><lang><json:string>eng</json:string></lang><value>Cervical dystonia</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Cervical trauma</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Focal dystonia</value></json:item></subject><language><json:string>eng</json:string></language><abstract>Head, neck, or shoulder trauma is an occasional antecedent event before the appearance of cervical dystonia. 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Acute‐onset posttraumatic cervical dystonia is similar to limb dystonia after peripheral trauma and may represent a form of nondystonic muscle spasm similar to torticollis associated with musculoskeletal injuries of the cervical spine and craniocervical junction.</abstract><qualityIndicators><score>4.567</score><pdfVersion>1.3</pdfVersion><pdfPageSize>612 x 792 pts (letter)</pdfPageSize><refBibsNative>true</refBibsNative><abstractCharCount>1302</abstractCharCount><pdfWordCount>2491</pdfWordCount><pdfCharCount>17877</pdfCharCount><pdfPageCount>5</pdfPageCount><abstractWordCount>173</abstractWordCount></qualityIndicators><title>Comparison of acute‐ and delayed‐onset posttraumatic cervical dystonia</title><genre><json:string>Serial article</json:string></genre><host><volume>13</volume><pages><total>5</total><last>485</last><first>481</first></pages><issn><json:string>0885-3185</json:string></issn><issue>3</issue><subject><json:item><value>Article</value></json:item></subject><genre></genre><language><json:string>unknown</json:string></language><title>Movement Disorders</title><doi><json:string>10.1002/(ISSN)1531-8257</json:string></doi></host><publicationDate>1998</publicationDate><copyrightDate>1998</copyrightDate><doi><json:string>10.1002/mds.870130318</json:string></doi><id>6B8578DEE12606D6760B48CE118EDB422A0B1EC4</id><fulltext><json:item><original>true</original><mimetype>application/pdf</mimetype><extension>pdf</extension><uri>https://api.istex.fr/document/6B8578DEE12606D6760B48CE118EDB422A0B1EC4/fulltext/pdf</uri></json:item><json:item><original>false</original><mimetype>application/zip</mimetype><extension>zip</extension><uri>https://api.istex.fr/document/6B8578DEE12606D6760B48CE118EDB422A0B1EC4/fulltext/zip</uri></json:item><istex:fulltextTEI uri="https://api.istex.fr/document/6B8578DEE12606D6760B48CE118EDB422A0B1EC4/fulltext/tei"><teiHeader type="text"><fileDesc><titleStmt><title level="a" type="main" xml:lang="en">Comparison of acute‐ and delayed‐onset posttraumatic cervical dystonia</title></titleStmt><publicationStmt><authority>ISTEX</authority><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher><pubPlace>Hoboken</pubPlace><availability><p>Wiley Subscription Services, Inc., A Wiley Company</p></availability><date>1998</date></publicationStmt><notesStmt><note type="content">*Presented at the Tenth Annual Symposium on Etiology, Pathogenesis, and Treatment of Parkinson's Disease and Hyperkinetic Movement Disorders; Miami, Florida; October 1996.</note></notesStmt><sourceDesc><biblStruct type="inbook"><analytic><title level="a" type="main" xml:lang="en">Comparison of acute‐ and delayed‐onset posttraumatic cervical dystonia</title><author><persName><forename type="first">Daniel</forename><surname>Tarsy</surname><roleName type="degree">MD</roleName></persName><note type="correspondence"><p>Correspondence: Beth Israel Deaconess Medical Center, 330 Brookline Ave., Boston, MA 02215, U.S.A.===</p></note><affiliation>Department of Neurology, Beth Israel Deaconess Medical Center, and Department of Neurology, Harvard Medical School, Boston, Massachusetts, U.S.A.</affiliation></author></analytic><monogr><title level="j">Movement Disorders</title><title level="j" type="abbrev">Mov. 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A clinically distinctive syndrome of acute‐onset posttraumatic cervical dystonia characterized by markedly restricted range of neck motion, absence of phasic involuntary movements, and poor response to treatment has previously been described. Patients with cervical dystonia attending a movement disorder clinic were reviewed for history of trauma before onset of symptoms. Patients with symptom onset within 4 weeks of trauma were compared with patients who developed symptoms between 3 months and 1 year after trauma. Acute‐onset cervical dystonia was characterized by markedly reduced cervical mobility; prominent shoulder elevation with trapezius hypertrophy in most patients, absence of involuntary movements, sensory tricks, or activation maneuvers; and poor response to botulinum toxin injection. By contrast, delayed‐onset cervical dystonia was clinically indistinguishable from nontraumatic idiopathic cervical dystonia. 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A clinically distinctive syndrome of acute‐onset posttraumatic cervical dystonia characterized by markedly restricted range of neck motion, absence of phasic involuntary movements, and poor response to treatment has previously been described. Patients with cervical dystonia attending a movement disorder clinic were reviewed for history of trauma before onset of symptoms. Patients with symptom onset within 4 weeks of trauma were compared with patients who developed symptoms between 3 months and 1 year after trauma. Acute‐onset cervical dystonia was characterized by markedly reduced cervical mobility; prominent shoulder elevation with trapezius hypertrophy in most patients, absence of involuntary movements, sensory tricks, or activation maneuvers; and poor response to botulinum toxin injection. By contrast, delayed‐onset cervical dystonia was clinically indistinguishable from nontraumatic idiopathic cervical dystonia. Acute‐onset posttraumatic cervical dystonia is similar to limb dystonia after peripheral trauma and may represent a form of nondystonic muscle spasm similar to torticollis associated with musculoskeletal injuries of the cervical spine and craniocervical junction.</p></abstract></abstractGroup></contentMeta><noteGroup><note xml:id="fn1"><p>Presented at the Tenth Annual Symposium on Etiology, Pathogenesis, and Treatment of Parkinson's Disease and Hyperkinetic Movement Disorders; Miami, Florida; October 1996.</p></note></noteGroup></header></component></istex:document></istex:metadataXml><!--Version 0.6 générée le 3-12-2015--><mods version="3.6"><titleInfo lang="en"><title>Comparison of acute‐ and delayed‐onset posttraumatic cervical dystonia</title></titleInfo><titleInfo type="abbreviated" lang="en"><title>ACUTE‐ AND DELAYED‐ONSET CERVICAL DYSTONIA</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>Comparison of acute‐ and delayed‐onset posttraumatic cervical dystonia</title></titleInfo><name type="personal"><namePart type="given">Daniel</namePart><namePart type="family">Tarsy</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Neurology, Beth Israel Deaconess Medical Center, and Department of Neurology, Harvard Medical School, Boston, Massachusetts, U.S.A.</affiliation><description>Correspondence: Beth Israel Deaconess Medical Center, 330 Brookline Ave., Boston, MA 02215, U.S.A.===</description><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre authority="originalCategForm">article</genre><originInfo><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher><place><placeTerm type="text">Hoboken</placeTerm></place><dateIssued encoding="w3cdtf">1998-05</dateIssued><dateCaptured encoding="w3cdtf">1997-08-05</dateCaptured><dateValid encoding="w3cdtf">1997-11-03</dateValid><copyrightDate encoding="w3cdtf">1998</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType><extent unit="figures">1</extent><extent unit="tables">2</extent><extent unit="references">16</extent></physicalDescription><abstract lang="en">Head, neck, or shoulder trauma is an occasional antecedent event before the appearance of cervical dystonia. A clinically distinctive syndrome of acute‐onset posttraumatic cervical dystonia characterized by markedly restricted range of neck motion, absence of phasic involuntary movements, and poor response to treatment has previously been described. Patients with cervical dystonia attending a movement disorder clinic were reviewed for history of trauma before onset of symptoms. Patients with symptom onset within 4 weeks of trauma were compared with patients who developed symptoms between 3 months and 1 year after trauma. Acute‐onset cervical dystonia was characterized by markedly reduced cervical mobility; prominent shoulder elevation with trapezius hypertrophy in most patients, absence of involuntary movements, sensory tricks, or activation maneuvers; and poor response to botulinum toxin injection. By contrast, delayed‐onset cervical dystonia was clinically indistinguishable from nontraumatic idiopathic cervical dystonia. Acute‐onset posttraumatic cervical dystonia is similar to limb dystonia after peripheral trauma and may represent a form of nondystonic muscle spasm similar to torticollis associated with musculoskeletal injuries of the cervical spine and craniocervical junction.</abstract><note type="content">*Presented at the Tenth Annual Symposium on Etiology, Pathogenesis, and Treatment of Parkinson's Disease and Hyperkinetic Movement Disorders; Miami, Florida; October 1996.</note><subject lang="en"><genre>Keywords</genre><topic>Cervical dystonia</topic><topic>Cervical trauma</topic><topic>Focal dystonia</topic></subject><relatedItem type="host"><titleInfo><title>Movement Disorders</title></titleInfo><titleInfo type="abbreviated"><title>Mov. 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