Dopamine D2 receptor TaqIA and TaqIB polymorphisms in Parkinson's disease
Identifieur interne : 001B60 ( Istex/Corpus ); précédent : 001B59; suivant : 001B61Dopamine D2 receptor TaqIA and TaqIB polymorphisms in Parkinson's disease
Auteurs : Eng-King Tan ; Yanni Tan ; Anthea Chai ; Christopher Tan ; Hui Shen ; Sau-Ying Lum ; Stephanie M. C. Fook-Cheong ; Mei-Ling Teoh ; Yuan Yih ; Meng-Cheong Wong ; Yi ZhaoSource :
- Movement Disorders [ 0885-3185 ] ; 2003-05.
English descriptors
Abstract
In a case control study, we examined the association of DRD2 Taq1A and Taq1B polymorphisms and risk of PD, and evaluated the strength of linkage disequilibrium of the polymorphisms. The Taq1A and Taq1B polymorphisms were in strong linkage disequilibrium. There was, however, no significant association of the two polymorphisms with PD. © 2003 Movement Disorder Society
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DOI: 10.1002/mds.10406
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ISTEX:0C1E01E57BAB424D6CB7054B76BA502BEEF09DB8Le document en format XML
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The Taq1A and Taq1B polymorphisms were in strong linkage disequilibrium. There was, however, no significant association of the two polymorphisms with PD. © 2003 Movement Disorder Society</div></front></TEI><istex><corpusName>wiley</corpusName><author><json:item><name>Eng‐King Tan MD</name><affiliations><json:string>Department of Neurology, Singapore General Hospital, Singapore</json:string><json:string>National Neuroscience Institute, SingHealth, Singapore</json:string></affiliations></json:item><json:item><name>Yanni Tan MS</name><affiliations><json:string>Department of Neurology, Singapore General Hospital, Singapore</json:string></affiliations></json:item><json:item><name>Anthea Chai BSc</name><affiliations><json:string>Department of Neurology, Singapore General Hospital, Singapore</json:string></affiliations></json:item><json:item><name>Christopher Tan BSc</name><affiliations><json:string>Department of Neurology, Singapore General Hospital, Singapore</json:string></affiliations></json:item><json:item><name>Hui Shen PhD</name><affiliations><json:string>Department of Neurology, Singapore General Hospital, Singapore</json:string></affiliations></json:item><json:item><name>Sau‐Ying Lum BSc</name><affiliations><json:string>Department of Neurology, Singapore General Hospital, Singapore</json:string></affiliations></json:item><json:item><name>Stephanie M.C. Fook‐Cheong MSc</name><affiliations><json:string>Department of Clinical Research, Singapore General Hospital, Singapore</json:string></affiliations></json:item><json:item><name>Mei‐Ling Teoh MD</name><affiliations><json:string>Department of Health Screening, Singapore General Hospital, Singapore</json:string></affiliations></json:item><json:item><name>Yuan Yih MD</name><affiliations><json:string>Department of Health Screening, Singapore General Hospital, Singapore</json:string></affiliations></json:item><json:item><name>Meng‐Cheong Wong MD</name><affiliations><json:string>Department of Neurology, Singapore General Hospital, Singapore</json:string></affiliations></json:item><json:item><name>Yi Zhao MD, PhD</name><affiliations><json:string>Department of Clinical Research, Singapore General Hospital, Singapore</json:string></affiliations></json:item></author><subject><json:item><lang><json:string>eng</json:string></lang><value>dopamine receptor D2</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>polymorphism</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>smoking</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Parkinson's disease</value></json:item></subject><language><json:string>eng</json:string></language><abstract>In a case control study, we examined the association of DRD2 Taq1A and Taq1B polymorphisms and risk of PD, and evaluated the strength of linkage disequilibrium of the polymorphisms. 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The Taq1A and Taq1B polymorphisms were in strong linkage disequilibrium. There was, however, no significant association of the two polymorphisms with PD. © 2003 Movement Disorder Society</p></abstract></abstractGroup></contentMeta></header></component></istex:document></istex:metadataXml><!--Version 0.6 générée le 4-12-2015--><mods version="3.6"><titleInfo lang="en"><title>Dopamine D2 receptor TaqIA and TaqIB polymorphisms in Parkinson's disease</title></titleInfo><titleInfo type="abbreviated" lang="en"><title>Clinical/Scientific Notes</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>Dopamine D2 receptor TaqIA and TaqIB polymorphisms in Parkinson's disease</title></titleInfo><name type="personal"><namePart type="given">Eng‐King</namePart><namePart type="family">Tan</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Neurology, Singapore General Hospital, Singapore</affiliation><affiliation>National Neuroscience Institute, SingHealth, Singapore</affiliation><description>Correspondence: Department of Neurology, Singapore General Hospital, Outram Road, 169608 Singapore</description><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Yanni</namePart><namePart type="family">Tan</namePart><namePart type="termsOfAddress">MS</namePart><affiliation>Department of Neurology, Singapore General Hospital, Singapore</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Anthea</namePart><namePart type="family">Chai</namePart><namePart type="termsOfAddress">BSc</namePart><affiliation>Department of Neurology, Singapore General Hospital, Singapore</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Christopher</namePart><namePart type="family">Tan</namePart><namePart type="termsOfAddress">BSc</namePart><affiliation>Department of Neurology, Singapore General Hospital, Singapore</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Hui</namePart><namePart type="family">Shen</namePart><namePart type="termsOfAddress">PhD</namePart><affiliation>Department of Neurology, Singapore General Hospital, Singapore</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Sau‐Ying</namePart><namePart type="family">Lum</namePart><namePart type="termsOfAddress">BSc</namePart><affiliation>Department of Neurology, Singapore General Hospital, Singapore</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Stephanie M.C.</namePart><namePart type="family">Fook‐Cheong</namePart><namePart type="termsOfAddress">MSc</namePart><affiliation>Department of Clinical Research, Singapore General Hospital, Singapore</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Mei‐Ling</namePart><namePart type="family">Teoh</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Health Screening, Singapore General Hospital, Singapore</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Yuan</namePart><namePart type="family">Yih</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Health Screening, Singapore General Hospital, Singapore</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Meng‐Cheong</namePart><namePart type="family">Wong</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Neurology, Singapore General Hospital, Singapore</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Yi</namePart><namePart type="family">Zhao</namePart><namePart type="termsOfAddress">MD, PhD</namePart><affiliation>Department of Clinical Research, Singapore General Hospital, Singapore</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre authority="originalCategForm">shortCommunication</genre><originInfo><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher><place><placeTerm type="text">New York</placeTerm></place><dateIssued encoding="w3cdtf">2003-05</dateIssued><dateCaptured encoding="w3cdtf">2002-07-03</dateCaptured><dateValid encoding="w3cdtf">2002-10-18</dateValid><copyrightDate encoding="w3cdtf">2003</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType><extent unit="tables">2</extent><extent unit="references">24</extent><extent unit="words">1937</extent></physicalDescription><abstract lang="en">In a case control study, we examined the association of DRD2 Taq1A and Taq1B polymorphisms and risk of PD, and evaluated the strength of linkage disequilibrium of the polymorphisms. The Taq1A and Taq1B polymorphisms were in strong linkage disequilibrium. There was, however, no significant association of the two polymorphisms with PD. © 2003 Movement Disorder Society</abstract><subject lang="en"><genre>Keywords</genre><topic>dopamine receptor D2</topic><topic>polymorphism</topic><topic>smoking</topic><topic>Parkinson's disease</topic></subject><relatedItem type="host"><titleInfo><title>Movement Disorders</title></titleInfo><titleInfo type="abbreviated"><title>Mov. Disord.</title></titleInfo><subject><genre>article category</genre><topic>Clinical/Scientific Note</topic></subject><identifier type="ISSN">0885-3185</identifier><identifier type="eISSN">1531-8257</identifier><identifier type="DOI">10.1002/(ISSN)1531-8257</identifier><identifier type="PublisherID">MDS</identifier><part><date>2003</date><detail type="volume"><caption>vol.</caption><number>18</number></detail><detail type="issue"><caption>no.</caption><number>5</number></detail><extent unit="pages"><start>593</start><end>595</end><total>3</total></extent></part></relatedItem><identifier type="istex">0C1E01E57BAB424D6CB7054B76BA502BEEF09DB8</identifier><identifier type="DOI">10.1002/mds.10406</identifier><identifier type="ArticleID">MDS10406</identifier><accessCondition type="use and reproduction" contentType="copyright">Copyright © 2003 Movement Disorders Society</accessCondition><recordInfo><recordOrigin>Wiley Subscription Services, Inc., A Wiley Company</recordOrigin><recordContentSource>WILEY</recordContentSource></recordInfo></mods></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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