Atypical and typical cranial dystonia following dental procedures
Identifieur interne : 001961 ( Istex/Corpus ); précédent : 001960; suivant : 001962Atypical and typical cranial dystonia following dental procedures
Auteurs : Anette Schrag ; Kailash P. Bhatia ; Niall P. Quinn ; C. David MarsdenSource :
- Movement Disorders [ 0885-3185 ] ; 1999-05.
English descriptors
- KwdEn :
Abstract
It is generally recognized that focal dystonia of the limbs or cervical region and blepharospasm sometimes follow, and in these cases may be caused or triggered by, peripheral injury. However, the association between peripheral injury and lower cranial dystonia is rare. We report eight cases who developed cranial dystonia within hours to months following a dental procedure. One group of five cases, all women, developed atypical dystonia associated with painful paresthesias at the site of dystonia. Two of these five cases had fixed jaw‐deviating dystonia, whereas the remaining three had additional tremor and spread of their dystonia to involve the tongue in all three, and the lips and neck in two cases. These five patients are reminiscent of cases of limb causalgia–dystonia syndrome, which occurs after minor peripheral trauma and can spread. The remaining three cases developed more typical cranial dystonia following the dental procedure. There was no family history of dystonia or prior use of neuroleptics in any of the patients. The close association in time and location of the procedure and onset of symptoms suggests that the onset of the dystonia may have been caused by the dental intervention, but whether there is a causal relationship between the dental intervention and the development of the dyskinesias requires further epidemiologic studies.
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DOI: 10.1002/1531-8257(199905)14:3<492::AID-MDS1018>3.0.CO;2-4
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ISTEX:6C4F21298841363E74A78B8CD1954D699ED1162DLe document en format XML
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However, the association between peripheral injury and lower cranial dystonia is rare. We report eight cases who developed cranial dystonia within hours to months following a dental procedure. One group of five cases, all women, developed atypical dystonia associated with painful paresthesias at the site of dystonia. Two of these five cases had fixed jaw‐deviating dystonia, whereas the remaining three had additional tremor and spread of their dystonia to involve the tongue in all three, and the lips and neck in two cases. These five patients are reminiscent of cases of limb causalgia–dystonia syndrome, which occurs after minor peripheral trauma and can spread. The remaining three cases developed more typical cranial dystonia following the dental procedure. There was no family history of dystonia or prior use of neuroleptics in any of the patients. The close association in time and location of the procedure and onset of symptoms suggests that the onset of the dystonia may have been caused by the dental intervention, but whether there is a causal relationship between the dental intervention and the development of the dyskinesias requires further epidemiologic studies.</div></front></TEI><istex><corpusName>wiley</corpusName><author><json:item><name>Anette Schrag MD</name><affiliations><json:string>Department of Clinical Neurology, Institute of Neurology, Queen Square, University College, London, U.K.</json:string></affiliations></json:item><json:item><name>Kailash P. Bhatia MD</name><affiliations><json:string>Department of Clinical Neurology, Institute of Neurology, Queen Square, University College, London, U.K.</json:string></affiliations></json:item><json:item><name>Niall P. 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David Marsden DSc</name><affiliations><json:string>Department of Clinical Neurology, Institute of Neurology, Queen Square, University College, London, U.K.</json:string></affiliations></json:item></author><subject><json:item><lang><json:string>eng</json:string></lang><value>Cranial dystonia</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Atypical</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Dental</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Trauma</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Peripheral</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Pain</value></json:item></subject><language><json:string>eng</json:string></language><abstract>It is generally recognized that focal dystonia of the limbs or cervical region and blepharospasm sometimes follow, and in these cases may be caused or triggered by, peripheral injury. 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David</forename><surname>Marsden</surname><roleName type="degree">DSc</roleName></persName><note type="biography">Deceased September 29, 1998.</note><affiliation>Deceased September 29, 1998.</affiliation><affiliation>Department of Clinical Neurology, Institute of Neurology, Queen Square, University College, London, U.K.</affiliation></author></analytic><monogr><title level="j">Movement Disorders</title><title level="j" type="sub">Official Journal of the Movement Disorder Society</title><title level="j" type="abbrev">Mov. 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The close association in time and location of the procedure and onset of symptoms suggests that the onset of the dystonia may have been caused by the dental intervention, but whether there is a causal relationship between the dental intervention and the development of the dyskinesias requires further epidemiologic studies.</p></abstract><textClass xml:lang="en"><keywords scheme="keyword"><list><head>Keywords</head><item><term>Cranial dystonia</term></item><item><term>Atypical</term></item><item><term>Dental</term></item><item><term>Trauma</term></item><item><term>Peripheral</term></item><item><term>Pain</term></item></list></keywords></textClass><textClass><keywords scheme="Journal Subject"><list><head>Article category</head><item><term>Brief Report</term></item></list></keywords></textClass></profileDesc><revisionDesc><change when="1998-06-01">Received</change><change when="1999-01-26">Registration</change><change when="1999-05">Published</change></revisionDesc></teiHeader></istex:fulltextTEI><json:item><original>false</original><mimetype>text/plain</mimetype><extension>txt</extension><uri>https://api.istex.fr/document/6C4F21298841363E74A78B8CD1954D699ED1162D/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Wiley, elements deleted: body"><istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="yes"</istex:xmlDeclaration><istex:document><component version="2.0" type="serialArticle" xml:lang="en"><header><publicationMeta level="product"><publisherInfo><publisherName>John Wiley & Sons, Inc.</publisherName><publisherLoc>New York</publisherLoc></publisherInfo><doi registered="yes">10.1002/(ISSN)1531-8257</doi><issn type="print">0885-3185</issn><issn type="electronic">1531-8257</issn><idGroup><id type="product" value="MDS"></id></idGroup><titleGroup><title type="main" xml:lang="en" sort="MOVEMENT DISORDERS">Movement Disorders</title><title type="subtitle">Official Journal of the Movement Disorder Society</title><title type="short">Mov. 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David</givenNames><familyName>Marsden</familyName><degrees>DSc</degrees></personName></creator></creators><affiliationGroup><affiliation xml:id="af1" countryCode="US" type="organization"><unparsedAffiliation>Department of Clinical Neurology, Institute of Neurology, Queen Square, University College, London, U.K.</unparsedAffiliation></affiliation></affiliationGroup><keywordGroup xml:lang="en" type="author"><keyword xml:id="kwd1">Cranial dystonia</keyword><keyword xml:id="kwd2">Atypical</keyword><keyword xml:id="kwd3">Dental</keyword><keyword xml:id="kwd4">Trauma</keyword><keyword xml:id="kwd5">Peripheral</keyword><keyword xml:id="kwd6">Pain</keyword></keywordGroup><abstractGroup><abstract type="main" xml:lang="en"><title type="main">Abstract</title><p>It is generally recognized that focal dystonia of the limbs or cervical region and blepharospasm sometimes follow, and in these cases may be caused or triggered by, peripheral injury. However, the association between peripheral injury and lower cranial dystonia is rare. We report eight cases who developed cranial dystonia within hours to months following a dental procedure. One group of five cases, all women, developed atypical dystonia associated with painful paresthesias at the site of dystonia. Two of these five cases had fixed jaw‐deviating dystonia, whereas the remaining three had additional tremor and spread of their dystonia to involve the tongue in all three, and the lips and neck in two cases. These five patients are reminiscent of cases of limb causalgia–dystonia syndrome, which occurs after minor peripheral trauma and can spread. The remaining three cases developed more typical cranial dystonia following the dental procedure. There was no family history of dystonia or prior use of neuroleptics in any of the patients. The close association in time and location of the procedure and onset of symptoms suggests that the onset of the dystonia may have been caused by the dental intervention, but whether there is a causal relationship between the dental intervention and the development of the dyskinesias requires further epidemiologic studies.</p></abstract></abstractGroup></contentMeta><noteGroup><note xml:id="fn1"><p>Deceased September 29, 1998.</p></note></noteGroup></header></component></istex:document></istex:metadataXml><!--Version 0.6 générée le 4-12-2015--><mods version="3.6"><titleInfo lang="en"><title>Atypical and typical cranial dystonia following dental procedures</title></titleInfo><titleInfo type="abbreviated" lang="en"><title>Cranial Dystonia Following Dental Procedures</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>Atypical and typical cranial dystonia following dental procedures</title></titleInfo><name type="personal"><namePart type="given">Anette</namePart><namePart type="family">Schrag</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Clinical Neurology, Institute of Neurology, Queen Square, University College, London, U.K.</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Kailash P.</namePart><namePart type="family">Bhatia</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Clinical Neurology, Institute of Neurology, Queen Square, University College, London, U.K.</affiliation><description>Correspondence: Department of Clinical Neurology, Institute of Neurology, Queen Square, London WC1N 3BG, U.K.</description><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Niall P.</namePart><namePart type="family">Quinn</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Clinical Neurology, Institute of Neurology, Queen Square, University College, London, U.K.</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">C. David</namePart><namePart type="family">Marsden</namePart><namePart type="termsOfAddress">DSc</namePart><affiliation>Department of Clinical Neurology, Institute of Neurology, Queen Square, University College, London, U.K.</affiliation><description>Deceased September 29, 1998.</description><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre authority="originalCategForm">shortCommunication</genre><originInfo><publisher>John Wiley & Sons, Inc.</publisher><place><placeTerm type="text">New York</placeTerm></place><dateIssued encoding="w3cdtf">1999-05</dateIssued><dateCaptured encoding="w3cdtf">1998-06-01</dateCaptured><dateValid encoding="w3cdtf">1999-01-26</dateValid><copyrightDate encoding="w3cdtf">1999</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType><extent unit="tables">1</extent><extent unit="references">18</extent><extent unit="words">3303</extent></physicalDescription><abstract lang="en">It is generally recognized that focal dystonia of the limbs or cervical region and blepharospasm sometimes follow, and in these cases may be caused or triggered by, peripheral injury. However, the association between peripheral injury and lower cranial dystonia is rare. We report eight cases who developed cranial dystonia within hours to months following a dental procedure. One group of five cases, all women, developed atypical dystonia associated with painful paresthesias at the site of dystonia. Two of these five cases had fixed jaw‐deviating dystonia, whereas the remaining three had additional tremor and spread of their dystonia to involve the tongue in all three, and the lips and neck in two cases. These five patients are reminiscent of cases of limb causalgia–dystonia syndrome, which occurs after minor peripheral trauma and can spread. The remaining three cases developed more typical cranial dystonia following the dental procedure. There was no family history of dystonia or prior use of neuroleptics in any of the patients. The close association in time and location of the procedure and onset of symptoms suggests that the onset of the dystonia may have been caused by the dental intervention, but whether there is a causal relationship between the dental intervention and the development of the dyskinesias requires further epidemiologic studies.</abstract><subject lang="en"><genre>Keywords</genre><topic>Cranial dystonia</topic><topic>Atypical</topic><topic>Dental</topic><topic>Trauma</topic><topic>Peripheral</topic><topic>Pain</topic></subject><relatedItem type="host"><titleInfo><title>Movement Disorders</title><subTitle>Official Journal of the Movement Disorder Society</subTitle></titleInfo><titleInfo type="abbreviated"><title>Mov. Disord.</title></titleInfo><subject><genre>article category</genre><topic>Brief Report</topic></subject><identifier type="ISSN">0885-3185</identifier><identifier type="eISSN">1531-8257</identifier><identifier type="DOI">10.1002/(ISSN)1531-8257</identifier><identifier type="PublisherID">MDS</identifier><part><date>1999</date><detail type="volume"><caption>vol.</caption><number>14</number></detail><detail type="issue"><caption>no.</caption><number>3</number></detail><extent unit="pages"><start>492</start><end>496</end><total>5</total></extent></part></relatedItem><identifier type="istex">6C4F21298841363E74A78B8CD1954D699ED1162D</identifier><identifier type="DOI">10.1002/1531-8257(199905)14:3<492::AID-MDS1018>3.0.CO;2-4</identifier><identifier type="ArticleID">MDS1018</identifier><accessCondition type="use and reproduction" contentType="copyright">Copyright © 1999 Movement Disorder Society</accessCondition><recordInfo><recordOrigin>John Wiley & Sons, Inc.</recordOrigin><recordContentSource>WILEY</recordContentSource></recordInfo></mods></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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