The syndrome of gait ignition failure: A report of six cases
Identifieur interne : 001605 ( Istex/Corpus ); précédent : 001604; suivant : 001606The syndrome of gait ignition failure: A report of six cases
Auteurs : P. R. Atchison ; P. D. Thompson ; R. S. J. Frackowiak ; MarsdenSource :
- Movement Disorders [ 0885-3185 ] ; 1993.
English descriptors
- KwdEn :
Abstract
The syndrome of gait ignition failure is described in six patients in whom difficulty initiating walking was the major symptom. The gait had elements of parkinsonism with start and turn hesitation, shuffling, and freezing. Unlike parkinsonism, however, the gait was relatively normal once entrained; the posture was upright, and good arm swing, a normal stride length, and no festination were seen. Equilibrium was normal or near normal, and when seated or lying, rhythmic leg movements were generated normally. Facial expression, upper limb mobility, and whole body movements were wellpreserved. This gait disorder differed from that seen in Parkinson's disease and the so‐called “frontal” or “senile” disorders of gait and gait “apraxia”. The causes of this gait syndrome are not clear but it may be due to frontal lobe vascular disease and/or focal degeneration of the frontal lobes.
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DOI: 10.1002/mds.870080306
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ISTEX:4BA7A4A389DEC205DD5AA193714B2AC782D0AE5BLe document en format XML
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The gait had elements of parkinsonism with start and turn hesitation, shuffling, and freezing. Unlike parkinsonism, however, the gait was relatively normal once entrained; the posture was upright, and good arm swing, a normal stride length, and no festination were seen. Equilibrium was normal or near normal, and when seated or lying, rhythmic leg movements were generated normally. Facial expression, upper limb mobility, and whole body movements were wellpreserved. This gait disorder differed from that seen in Parkinson's disease and the so‐called “frontal” or “senile” disorders of gait and gait “apraxia”. The causes of this gait syndrome are not clear but it may be due to frontal lobe vascular disease and/or focal degeneration of the frontal lobes.</div></front></TEI><istex><corpusName>wiley</corpusName><author><json:item><name>P. R. 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The gait had elements of parkinsonism with start and turn hesitation, shuffling, and freezing. Unlike parkinsonism, however, the gait was relatively normal once entrained; the posture was upright, and good arm swing, a normal stride length, and no festination were seen. Equilibrium was normal or near normal, and when seated or lying, rhythmic leg movements were generated normally. Facial expression, upper limb mobility, and whole body movements were wellpreserved. This gait disorder differed from that seen in Parkinson's disease and the so‐called “frontal” or “senile” disorders of gait and gait “apraxia”. The causes of this gait syndrome are not clear but it may be due to frontal lobe vascular disease and/or focal degeneration of the frontal lobes.</p></abstract></abstractGroup></contentMeta></header></component></istex:document></istex:metadataXml><!--Version 0.6 générée le 4-12-2015--><mods version="3.6"><titleInfo lang="en"><title>The syndrome of gait ignition failure: A report of six cases</title></titleInfo><titleInfo type="abbreviated" lang="en"><title>GAIT IGNITION FAILURE</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>The syndrome of gait ignition failure: A report of six cases</title></titleInfo><name type="personal"><namePart type="given">P. R.</namePart><namePart type="family">Atchison</namePart><affiliation>MRC Human Movement and Balance Unit, Hammersmith Hospital, London, England</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">P. 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The gait had elements of parkinsonism with start and turn hesitation, shuffling, and freezing. Unlike parkinsonism, however, the gait was relatively normal once entrained; the posture was upright, and good arm swing, a normal stride length, and no festination were seen. Equilibrium was normal or near normal, and when seated or lying, rhythmic leg movements were generated normally. Facial expression, upper limb mobility, and whole body movements were wellpreserved. This gait disorder differed from that seen in Parkinson's disease and the so‐called “frontal” or “senile” disorders of gait and gait “apraxia”. The causes of this gait syndrome are not clear but it may be due to frontal lobe vascular disease and/or focal degeneration of the frontal lobes.</abstract><subject lang="en"><genre>Keywords</genre><topic>Gait ignition failure</topic></subject><relatedItem type="host"><titleInfo><title>Movement Disorders</title><subTitle>Official Journal of the Movement Disorder Society</subTitle></titleInfo><titleInfo type="abbreviated"><title>Mov. 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