Middle East respiratory syndrome coronavirus: transmission, virology and therapeutic targeting to aid in outbreak control.
Identifieur interne : 001488 ( PubMed/Curation ); précédent : 001487; suivant : 001489Middle East respiratory syndrome coronavirus: transmission, virology and therapeutic targeting to aid in outbreak control.
Auteurs : Prasannavenkatesh Durai [Corée du Sud] ; Maria Batool [Corée du Sud] ; Masaud Shah [Corée du Sud] ; Sangdun Choi [Corée du Sud]Source :
- Experimental & molecular medicine [ 2092-6413 ] ; 2015.
Descripteurs français
- KwdFr :
- Animaux, Antiviraux (pharmacologie), Coronavirus du syndrome respiratoire du Moyen-Orient (), Coronavirus du syndrome respiratoire du Moyen-Orient (physiologie), Dipeptidyl peptidase 4 (métabolisme), Découverte de médicament, Flambées de maladies, Glycoprotéine de spicule des coronavirus (métabolisme), Humains, Infections à coronavirus (métabolisme), Infections à coronavirus (traitement médicamenteux), Infections à coronavirus (transmission), Infections à coronavirus (épidémiologie), Interactions hôte-pathogène (), Lignée cellulaire, Thérapie moléculaire ciblée.
- MESH :
- métabolisme : Dipeptidyl peptidase 4, Glycoprotéine de spicule des coronavirus, Infections à coronavirus.
- pharmacologie : Antiviraux.
- physiologie : Coronavirus du syndrome respiratoire du Moyen-Orient.
- traitement médicamenteux : Infections à coronavirus.
- épidémiologie : Infections à coronavirus.
- Animaux, Coronavirus du syndrome respiratoire du Moyen-Orient, Découverte de médicament, Flambées de maladies, Humains, Interactions hôte-pathogène, Lignée cellulaire, Thérapie moléculaire ciblée.
English descriptors
- KwdEn :
- Animals, Antiviral Agents (pharmacology), Cell Line, Coronavirus Infections (drug therapy), Coronavirus Infections (epidemiology), Coronavirus Infections (metabolism), Coronavirus Infections (transmission), Dipeptidyl Peptidase 4 (metabolism), Disease Outbreaks, Drug Discovery, Host-Pathogen Interactions (drug effects), Humans, Middle East Respiratory Syndrome Coronavirus (drug effects), Middle East Respiratory Syndrome Coronavirus (physiology), Molecular Targeted Therapy, Spike Glycoprotein, Coronavirus (metabolism).
- MESH :
- chemical , metabolism : Dipeptidyl Peptidase 4, Spike Glycoprotein, Coronavirus.
- chemical , pharmacology : Antiviral Agents.
- drug effects : Host-Pathogen Interactions, Middle East Respiratory Syndrome Coronavirus.
- drug therapy : Coronavirus Infections.
- epidemiology : Coronavirus Infections.
- metabolism : Coronavirus Infections.
- physiology : Middle East Respiratory Syndrome Coronavirus.
- transmission : Coronavirus Infections.
- Animals, Cell Line, Disease Outbreaks, Drug Discovery, Humans, Molecular Targeted Therapy.
Abstract
Middle East respiratory syndrome coronavirus (MERS-CoV) causes high fever, cough, acute respiratory tract infection and multiorgan dysfunction that may eventually lead to the death of the infected individuals. MERS-CoV is thought to be transmitted to humans through dromedary camels. The occurrence of the virus was first reported in the Middle East and it subsequently spread to several parts of the world. Since 2012, about 1368 infections, including ~487 deaths, have been reported worldwide. Notably, the recent human-to-human 'superspreading' of MERS-CoV in hospitals in South Korea has raised a major global health concern. The fatality rate in MERS-CoV infection is four times higher compared with that of the closely related severe acute respiratory syndrome coronavirus infection. Currently, no drug has been clinically approved to control MERS-CoV infection. In this study, we highlight the potential drug targets that can be used to develop anti-MERS-CoV therapeutics.
DOI: 10.1038/emm.2015.76
PubMed: 26315600
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qualifier="épidémiologie" xml:lang="fr"><term>Infections à coronavirus</term></keywords><keywords scheme="MESH" xml:lang="en"><term>Animals</term><term>Cell Line</term><term>Disease Outbreaks</term><term>Drug Discovery</term><term>Humans</term><term>Molecular Targeted Therapy</term></keywords><keywords scheme="MESH" xml:lang="fr"><term>Animaux</term><term>Coronavirus du syndrome respiratoire du Moyen-Orient</term><term>Découverte de médicament</term><term>Flambées de maladies</term><term>Humains</term><term>Interactions hôte-pathogène</term><term>Lignée cellulaire</term><term>Thérapie moléculaire ciblée</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Middle East respiratory syndrome coronavirus (MERS-CoV) causes high fever, cough, acute respiratory tract infection and multiorgan dysfunction that may eventually lead to the death of the infected individuals. MERS-CoV is thought to be transmitted to humans through dromedary camels. The occurrence of the virus was first reported in the Middle East and it subsequently spread to several parts of the world. Since 2012, about 1368 infections, including ~487 deaths, have been reported worldwide. Notably, the recent human-to-human 'superspreading' of MERS-CoV in hospitals in South Korea has raised a major global health concern. The fatality rate in MERS-CoV infection is four times higher compared with that of the closely related severe acute respiratory syndrome coronavirus infection. Currently, no drug has been clinically approved to control MERS-CoV infection. In this study, we highlight the potential drug targets that can be used to develop anti-MERS-CoV therapeutics. </div></front></TEI><pubmed><MedlineCitation Status="MEDLINE" Owner="NLM"><PMID Version="1">26315600</PMID><DateCompleted><Year>2016</Year><Month>01</Month><Day>26</Day></DateCompleted><DateRevised><Year>2020</Year><Month>03</Month><Day>25</Day></DateRevised><Article PubModel="Electronic"><Journal><ISSN IssnType="Electronic">2092-6413</ISSN><JournalIssue CitedMedium="Internet"><Volume>47</Volume><PubDate><Year>2015</Year><Month>Aug</Month><Day>28</Day></PubDate></JournalIssue><Title>Experimental & molecular medicine</Title><ISOAbbreviation>Exp. Mol. Med.</ISOAbbreviation></Journal><ArticleTitle>Middle East respiratory syndrome coronavirus: transmission, virology and therapeutic targeting to aid in outbreak control.</ArticleTitle><Pagination><MedlinePgn>e181</MedlinePgn></Pagination><ELocationID EIdType="doi" ValidYN="Y">10.1038/emm.2015.76</ELocationID><Abstract><AbstractText>Middle East respiratory syndrome coronavirus (MERS-CoV) causes high fever, cough, acute respiratory tract infection and multiorgan dysfunction that may eventually lead to the death of the infected individuals. MERS-CoV is thought to be transmitted to humans through dromedary camels. The occurrence of the virus was first reported in the Middle East and it subsequently spread to several parts of the world. Since 2012, about 1368 infections, including ~487 deaths, have been reported worldwide. Notably, the recent human-to-human 'superspreading' of MERS-CoV in hospitals in South Korea has raised a major global health concern. The fatality rate in MERS-CoV infection is four times higher compared with that of the closely related severe acute respiratory syndrome coronavirus infection. Currently, no drug has been clinically approved to control MERS-CoV infection. In this study, we highlight the potential drug targets that can be used to develop anti-MERS-CoV therapeutics. </AbstractText></Abstract><AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Durai</LastName><ForeName>Prasannavenkatesh</ForeName><Initials>P</Initials><AffiliationInfo><Affiliation>Department of Molecular Science and Technology, Ajou University, Suwon, Korea.</Affiliation></AffiliationInfo></Author><Author ValidYN="Y"><LastName>Batool</LastName><ForeName>Maria</ForeName><Initials>M</Initials><AffiliationInfo><Affiliation>Department of Molecular Science and Technology, Ajou University, Suwon, Korea.</Affiliation></AffiliationInfo></Author><Author ValidYN="Y"><LastName>Shah</LastName><ForeName>Masaud</ForeName><Initials>M</Initials><AffiliationInfo><Affiliation>Department of Molecular Science and Technology, Ajou University, Suwon, Korea.</Affiliation></AffiliationInfo></Author><Author ValidYN="Y"><LastName>Choi</LastName><ForeName>Sangdun</ForeName><Initials>S</Initials><AffiliationInfo><Affiliation>Department of Molecular Science and Technology, Ajou University, Suwon, Korea.</Affiliation></AffiliationInfo></Author></AuthorList><Language>eng</Language><PublicationTypeList><PublicationType UI="D016428">Journal Article</PublicationType><PublicationType UI="D013485">Research Support, Non-U.S. Gov't</PublicationType><PublicationType UI="D016454">Review</PublicationType></PublicationTypeList><ArticleDate DateType="Electronic"><Year>2015</Year><Month>08</Month><Day>28</Day></ArticleDate></Article><MedlineJournalInfo><Country>United States</Country><MedlineTA>Exp Mol Med</MedlineTA><NlmUniqueID>9607880</NlmUniqueID><ISSNLinking>1226-3613</ISSNLinking></MedlineJournalInfo><ChemicalList><Chemical><RegistryNumber>0</RegistryNumber><NameOfSubstance UI="D000998">Antiviral Agents</NameOfSubstance></Chemical><Chemical><RegistryNumber>0</RegistryNumber><NameOfSubstance 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