Reiterated DNA Fragments in Defective Genomes of Autographa californica Nuclear Polyhedrosis Virus Are Competent for AcMNPV-Dependent DNA Replication
Identifieur interne : 002666 ( Istex/Corpus ); précédent : 002665; suivant : 002667Reiterated DNA Fragments in Defective Genomes of Autographa californica Nuclear Polyhedrosis Virus Are Competent for AcMNPV-Dependent DNA Replication
Auteurs : Hoyun Lee ; Peter J. KrellSource :
- Virology [ 0042-6822 ] ; 1994.
Abstract
Abstract: We previously reported on the generation of ≈50-kb size defective genomes (DGs) which appeared to retain less than 2.2% of the standard Autographa californica nuclear polyhedrosis virus (AcMNPV) DNA between 85.0 and 87.2 MU while the rest of the virus DNA had been largely deleted (Lee and Krell, J. Virol., 66:4339-4347, 1992). To investigate these presumably repeated sequences further, we cloned and analyzed the most abundant hypermolar 1.80-kb Xhol DNA fragment as well as a minor but also supermolar 1.74-kb Xhol fragment of the DGs. These two DNA segments collectively covered 2371-bp of the standard AcMNPV DNA with a 1174-bp overlap around the Xhol site at 85.9 MU. Analysis of DGs by two-dimensional gel electrophoresis indicated that the 1.80- and 1.74-kb Xhol fragments (and most other novel Xhol fragments of the DG) were organized as tandem repeats in the DGs. We identified, in the DG population, small supercoiled DNA molecules approximately 1.0- to 8.2-kb (and possibly up to around 50 kb, the size of the major defective DNA species) in size and which contained the same DNA sequence as that of the major 1.80-kb repeat in the DGs. Furthermore, these cloned repeat sequences, represented by pLK1.80 and pLK1.74 showed AcMNPV infection-dependent autonomous replication, suggesting that an origin of DNA replication might reside within the HindIII to EcoRI segment (85.1 to 86.6 MU) of the HindIII-K fragment.
Url:
DOI: 10.1006/viro.1994.1358
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Krell</name><affiliation><mods:affiliation>Department of Microbiology, University of Guelph, Guelph, Ontario, Canada N1G 2W1</mods:affiliation></affiliation></author></titleStmt><publicationStmt><idno type="wicri:source">ISTEX</idno><idno type="RBID">ISTEX:E3EF3CB5550630D10E60B78CC8F47ED9CD6B2B6B</idno><date when="1994" year="1994">1994</date><idno type="doi">10.1006/viro.1994.1358</idno><idno type="url">https://api.istex.fr/ark:/67375/6H6-T18S198W-F/fulltext.pdf</idno><idno type="wicri:Area/Istex/Corpus">002666</idno><idno type="wicri:explorRef" wicri:stream="Istex" wicri:step="Corpus" wicri:corpus="ISTEX">002666</idno></publicationStmt><sourceDesc><biblStruct><analytic><title level="a" type="main" xml:lang="en">Reiterated DNA Fragments in Defective Genomes of Autographa californica Nuclear Polyhedrosis Virus Are Competent for AcMNPV-Dependent DNA Replication</title><author><name sortKey="Lee, Hoyun" sort="Lee, Hoyun" uniqKey="Lee H" first="Hoyun" last="Lee">Hoyun Lee</name><affiliation><mods:affiliation>Department of Microbiology, University of Guelph, Guelph, Ontario, Canada N1G 2W1</mods:affiliation></affiliation></author><author><name sortKey="Krell, Peter J" sort="Krell, Peter J" uniqKey="Krell P" first="Peter J." last="Krell">Peter J. Krell</name><affiliation><mods:affiliation>Department of Microbiology, University of Guelph, Guelph, Ontario, Canada N1G 2W1</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j">Virology</title><title level="j" type="abbrev">YVIRO</title><idno type="ISSN">0042-6822</idno><imprint><publisher>ELSEVIER</publisher><date type="published" when="1994">1994</date><biblScope unit="volume">202</biblScope><biblScope unit="issue">1</biblScope><biblScope unit="page" from="418">418</biblScope><biblScope unit="page" to="429">429</biblScope></imprint><idno type="ISSN">0042-6822</idno></series></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0042-6822</idno></seriesStmt></fileDesc><profileDesc><textClass></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Abstract: We previously reported on the generation of ≈50-kb size defective genomes (DGs) which appeared to retain less than 2.2% of the standard Autographa californica nuclear polyhedrosis virus (AcMNPV) DNA between 85.0 and 87.2 MU while the rest of the virus DNA had been largely deleted (Lee and Krell, J. Virol., 66:4339-4347, 1992). To investigate these presumably repeated sequences further, we cloned and analyzed the most abundant hypermolar 1.80-kb Xhol DNA fragment as well as a minor but also supermolar 1.74-kb Xhol fragment of the DGs. These two DNA segments collectively covered 2371-bp of the standard AcMNPV DNA with a 1174-bp overlap around the Xhol site at 85.9 MU. Analysis of DGs by two-dimensional gel electrophoresis indicated that the 1.80- and 1.74-kb Xhol fragments (and most other novel Xhol fragments of the DG) were organized as tandem repeats in the DGs. We identified, in the DG population, small supercoiled DNA molecules approximately 1.0- to 8.2-kb (and possibly up to around 50 kb, the size of the major defective DNA species) in size and which contained the same DNA sequence as that of the major 1.80-kb repeat in the DGs. Furthermore, these cloned repeat sequences, represented by pLK1.80 and pLK1.74 showed AcMNPV infection-dependent autonomous replication, suggesting that an origin of DNA replication might reside within the HindIII to EcoRI segment (85.1 to 86.6 MU) of the HindIII-K fragment.</div></front></TEI><istex><corpusName>elsevier</corpusName><author><json:item><name>Hoyun Lee</name><affiliations><json:string>Department of Microbiology, University of Guelph, Guelph, Ontario, Canada N1G 2W1</json:string></affiliations></json:item><json:item><name>Peter J. 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These two DNA segments collectively covered 2371-bp of the standard AcMNPV DNA with a 1174-bp overlap around the Xhol site at 85.9 MU. Analysis of DGs by two-dimensional gel electrophoresis indicated that the 1.80- and 1.74-kb Xhol fragments (and most other novel Xhol fragments of the DG) were organized as tandem repeats in the DGs. We identified, in the DG population, small supercoiled DNA molecules approximately 1.0- to 8.2-kb (and possibly up to around 50 kb, the size of the major defective DNA species) in size and which contained the same DNA sequence as that of the major 1.80-kb repeat in the DGs. 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Virol., 66:4339-4347, 1992). To investigate these presumably repeated sequences further, we cloned and analyzed the most abundant hypermolar 1.80-kb Xhol DNA fragment as well as a minor but also supermolar 1.74-kb Xhol fragment of the DGs. These two DNA segments collectively covered 2371-bp of the standard AcMNPV DNA with a 1174-bp overlap around the Xhol site at 85.9 MU. Analysis of DGs by two-dimensional gel electrophoresis indicated that the 1.80- and 1.74-kb Xhol fragments (and most other novel Xhol fragments of the DG) were organized as tandem repeats in the DGs. We identified, in the DG population, small supercoiled DNA molecules approximately 1.0- to 8.2-kb (and possibly up to around 50 kb, the size of the major defective DNA species) in size and which contained the same DNA sequence as that of the major 1.80-kb repeat in the DGs. Furthermore, these cloned repeat sequences, represented by pLK1.80 and pLK1.74 showed AcMNPV infection-dependent autonomous replication, suggesting that an origin of DNA replication might reside within the HindIII to EcoRI segment (85.1 to 86.6 MU) of the HindIII-K fragment.</p></abstract></profileDesc><revisionDesc><change when="1994">Published</change></revisionDesc></teiHeader></istex:fulltextTEI><json:item><extension>txt</extension><original>false</original><mimetype>text/plain</mimetype><uri>https://api.istex.fr/ark:/67375/6H6-T18S198W-F/fulltext.txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Elsevier converted-article found"><istex:xmlDeclaration>version="1.0" encoding="utf-8"</istex:xmlDeclaration><istex:docType PUBLIC="-//ES//DTD journal article DTD version 4.5.2//EN//XML" URI="art452.dtd" name="istex:docType"></istex:docType><istex:document><converted-article version="4.5.2" docsubtype="fla" xml:lang="en"><item-info><jid>YVIRO</jid><aid>71358</aid><ce:pii>S0042-6822(84)71358-4</ce:pii><ce:doi>10.1006/viro.1994.1358</ce:doi><ce:copyright type="full-transfer" year="1994">Academic Press</ce:copyright></item-info><head><ce:dochead><ce:textfn>Regular Article</ce:textfn></ce:dochead><ce:title>Reiterated DNA Fragments in Defective Genomes of <ce:italic>Autographa californica</ce:italic> Nuclear Polyhedrosis Virus Are Competent for AcMNPV-Dependent DNA Replication</ce:title><ce:author-group><ce:author><ce:given-name>Hoyun</ce:given-name><ce:surname>Lee</ce:surname></ce:author><ce:author><ce:given-name>Peter J.</ce:given-name><ce:surname>Krell</ce:surname></ce:author><ce:affiliation><ce:textfn>Department of Microbiology, University of Guelph, Guelph, Ontario, Canada N1G 2W1</ce:textfn></ce:affiliation></ce:author-group><ce:abstract><ce:section-title>Abstract</ce:section-title><ce:abstract-sec><ce:simple-para>We previously reported on the generation of ≈50-kb size defective genomes (DGs) which appeared to retain less than 2.2% of the standard <ce:italic>Autographa californica</ce:italic> nuclear polyhedrosis virus (AcMNPV) DNA between 85.0 and 87.2 MU while the rest of the virus DNA had been largely deleted (Lee and Krell, <ce:italic>J. Virol.,</ce:italic> 66:4339-4347, 1992). To investigate these presumably repeated sequences further, we cloned and analyzed the most abundant hypermolar 1.80-kb <ce:italic>Xhol</ce:italic> DNA fragment as well as a minor but also supermolar 1.74-kb <ce:italic>Xhol</ce:italic> fragment of the DGs. These two DNA segments collectively covered 2371-bp of the standard AcMNPV DNA with a 1174-bp overlap around the <ce:italic>Xhol</ce:italic> site at 85.9 MU. Analysis of DGs by two-dimensional gel electrophoresis indicated that the 1.80- and 1.74-kb <ce:italic>Xhol</ce:italic> fragments (and most other novel <ce:italic>Xhol</ce:italic> fragments of the DG) were organized as tandem repeats in the DGs. We identified, in the DG population, small supercoiled DNA molecules approximately 1.0- to 8.2-kb (and possibly up to around 50 kb, the size of the major defective DNA species) in size and which contained the same DNA sequence as that of the major 1.80-kb repeat in the DGs. Furthermore, these cloned repeat sequences, represented by pLK1.80 and pLK1.74 showed AcMNPV infection-dependent autonomous replication, suggesting that an origin of DNA replication might reside within the <ce:italic>Hin</ce:italic>dIII to <ce:italic>Eco</ce:italic>RI segment (85.1 to 86.6 MU) of the <ce:italic>Hin</ce:italic>dIII-K fragment.</ce:simple-para></ce:abstract-sec></ce:abstract></head></converted-article></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Reiterated DNA Fragments in Defective Genomes of Autographa californica Nuclear Polyhedrosis Virus Are Competent for AcMNPV-Dependent DNA Replication</title></titleInfo><titleInfo type="alternative" lang="en" contentType="CDATA"><title>Reiterated DNA Fragments in Defective Genomes of</title></titleInfo><name type="personal"><namePart type="given">Hoyun</namePart><namePart type="family">Lee</namePart><affiliation>Department of Microbiology, University of Guelph, Guelph, Ontario, Canada N1G 2W1</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Peter J.</namePart><namePart type="family">Krell</namePart><affiliation>Department of Microbiology, University of Guelph, Guelph, Ontario, Canada N1G 2W1</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="research-article" displayLabel="Full-length article" authority="ISTEX" authorityURI="https://content-type.data.istex.fr" valueURI="https://content-type.data.istex.fr/ark:/67375/XTP-1JC4F85T-7">research-article</genre><originInfo><publisher>ELSEVIER</publisher><dateIssued encoding="w3cdtf">1994</dateIssued><copyrightDate encoding="w3cdtf">1994</copyrightDate></originInfo><language><languageTerm type="code" authority="iso639-2b">eng</languageTerm><languageTerm type="code" authority="rfc3066">en</languageTerm></language><abstract lang="en">Abstract: We previously reported on the generation of ≈50-kb size defective genomes (DGs) which appeared to retain less than 2.2% of the standard Autographa californica nuclear polyhedrosis virus (AcMNPV) DNA between 85.0 and 87.2 MU while the rest of the virus DNA had been largely deleted (Lee and Krell, J. Virol., 66:4339-4347, 1992). To investigate these presumably repeated sequences further, we cloned and analyzed the most abundant hypermolar 1.80-kb Xhol DNA fragment as well as a minor but also supermolar 1.74-kb Xhol fragment of the DGs. These two DNA segments collectively covered 2371-bp of the standard AcMNPV DNA with a 1174-bp overlap around the Xhol site at 85.9 MU. Analysis of DGs by two-dimensional gel electrophoresis indicated that the 1.80- and 1.74-kb Xhol fragments (and most other novel Xhol fragments of the DG) were organized as tandem repeats in the DGs. We identified, in the DG population, small supercoiled DNA molecules approximately 1.0- to 8.2-kb (and possibly up to around 50 kb, the size of the major defective DNA species) in size and which contained the same DNA sequence as that of the major 1.80-kb repeat in the DGs. Furthermore, these cloned repeat sequences, represented by pLK1.80 and pLK1.74 showed AcMNPV infection-dependent autonomous replication, suggesting that an origin of DNA replication might reside within the HindIII to EcoRI segment (85.1 to 86.6 MU) of the HindIII-K fragment.</abstract><note type="content">Section title: Regular Article</note><relatedItem type="host"><titleInfo><title>Virology</title></titleInfo><titleInfo type="abbreviated"><title>YVIRO</title></titleInfo><genre type="journal" authority="ISTEX" authorityURI="https://publication-type.data.istex.fr" valueURI="https://publication-type.data.istex.fr/ark:/67375/JMC-0GLKJH51-B">journal</genre><originInfo><publisher>ELSEVIER</publisher><dateIssued encoding="w3cdtf">1994</dateIssued></originInfo><identifier type="ISSN">0042-6822</identifier><identifier type="PII">S0042-6822(00)X0163-8</identifier><part><date>1994</date><detail type="volume"><number>202</number><caption>vol.</caption></detail><detail type="issue"><number>1</number><caption>no.</caption></detail><extent unit="issue-pages"><start>1</start><end>520</end></extent><extent unit="pages"><start>418</start><end>429</end></extent></part></relatedItem><identifier type="istex">E3EF3CB5550630D10E60B78CC8F47ED9CD6B2B6B</identifier><identifier type="ark">ark:/67375/6H6-T18S198W-F</identifier><identifier type="DOI">10.1006/viro.1994.1358</identifier><identifier type="PII">S0042-6822(84)71358-4</identifier><accessCondition type="use and reproduction" contentType="copyright">©1994 Academic Press</accessCondition><recordInfo><recordContentSource authority="ISTEX" authorityURI="https://loaded-corpus.data.istex.fr" valueURI="https://loaded-corpus.data.istex.fr/ark:/67375/XBH-HKKZVM7B-M">elsevier</recordContentSource><recordOrigin>Academic Press, ©1994</recordOrigin></recordInfo></mods><json:item><extension>json</extension><original>false</original><mimetype>application/json</mimetype><uri>https://api.istex.fr/ark:/67375/6H6-T18S198W-F/record.json</uri></json:item></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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