[Causes of local edema in the infiltrative edematous form of breast cancer].
Identifieur interne : 006987 ( PubMed/Curation ); précédent : 006986; suivant : 006988[Causes of local edema in the infiltrative edematous form of breast cancer].
Auteurs : L A LiSource :
- Voprosy onkologii [ 0507-3758 ] ; 1984.
Descripteurs français
- KwdFr :
- MESH :
- physiologie : Exsudats et transsudats, Lymphe.
- physiopathologie : Lymphoedème, Tumeurs du sein.
- étiologie : Lymphoedème.
- Femelle, Humains, Hémodynamique, Maladie chronique, Métastase lymphatique, Perméabilité capillaire, Tumeurs du sein.
English descriptors
- KwdEn :
- MESH :
- complications : Breast Neoplasms.
- etiology : Lymphedema.
- physiology : Exudates and Transudates, Lymph.
- physiopathology : Breast Neoplasms, Lymphedema.
- Capillary Permeability, Chronic Disease, Female, Hemodynamics, Humans, Lymphatic Metastasis.
Abstract
The study was concerned with comparison of the characteristics of primary tumor in 80 cases of the infiltrative-edematous form of breast cancer. The control group included 192 patients with breast cancer unaggravated by edema. The involvement of regional lymph nodes proved to be the same in both groups. On the average tumors in the edematous breast cancer group were much larger and the rate of their growth much higher than in controls. The said pathogenetic differences are discussed. It is suggested that edema is primarily caused by disturbances in transcapillary metabolism at tumor site which are, in turn, induced by tumor metabolites.
PubMed: 6730414
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The control group included 192 patients with breast cancer unaggravated by edema. The involvement of regional lymph nodes proved to be the same in both groups. On the average tumors in the edematous breast cancer group were much larger and the rate of their growth much higher than in controls. The said pathogenetic differences are discussed. It is suggested that edema is primarily caused by disturbances in transcapillary metabolism at tumor site which are, in turn, induced by tumor metabolites.</div></front></TEI><pubmed><MedlineCitation Status="MEDLINE" Owner="NLM"><PMID Version="1">6730414</PMID><DateCreated><Year>1984</Year><Month>07</Month><Day>20</Day></DateCreated><DateCompleted><Year>1984</Year><Month>07</Month><Day>20</Day></DateCompleted><DateRevised><Year>2007</Year><Month>11</Month><Day>15</Day></DateRevised><Article PubModel="Print"><Journal><ISSN IssnType="Print">0507-3758</ISSN><JournalIssue CitedMedium="Print"><Volume>30</Volume><Issue>5</Issue><PubDate><Year>1984</Year></PubDate></JournalIssue><Title>Voprosy onkologii</Title><ISOAbbreviation>Vopr Onkol</ISOAbbreviation></Journal><ArticleTitle>[Causes of local edema in the infiltrative edematous form of breast cancer].</ArticleTitle><Pagination><MedlinePgn>35-8</MedlinePgn></Pagination><Abstract><AbstractText>The study was concerned with comparison of the characteristics of primary tumor in 80 cases of the infiltrative-edematous form of breast cancer. The control group included 192 patients with breast cancer unaggravated by edema. The involvement of regional lymph nodes proved to be the same in both groups. On the average tumors in the edematous breast cancer group were much larger and the rate of their growth much higher than in controls. The said pathogenetic differences are discussed. It is suggested that edema is primarily caused by disturbances in transcapillary metabolism at tumor site which are, in turn, induced by tumor metabolites.</AbstractText></Abstract><AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Li</LastName><ForeName>L A</ForeName><Initials>LA</Initials></Author></AuthorList><Language>rus</Language><PublicationTypeList><PublicationType UI="D003160">Comparative Study</PublicationType><PublicationType UI="D004740">English Abstract</PublicationType><PublicationType UI="D016428">Journal Article</PublicationType></PublicationTypeList><VernacularTitle>O prichinakh mestnogo oteka pri infil'trativno-otechnoĭ forme raka molochnoĭ zhelezy.</VernacularTitle></Article><MedlineJournalInfo><Country>Russia (Federation)</Country><MedlineTA>Vopr Onkol</MedlineTA><NlmUniqueID>0413775</NlmUniqueID><ISSNLinking>0507-3758</ISSNLinking></MedlineJournalInfo><CitationSubset>IM</CitationSubset><MeshHeadingList><MeshHeading><DescriptorName UI="D001943" MajorTopicYN="N">Breast Neoplasms</DescriptorName><QualifierName UI="Q000150" MajorTopicYN="Y">complications</QualifierName><QualifierName UI="Q000503" MajorTopicYN="N">physiopathology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D002199" MajorTopicYN="N">Capillary Permeability</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D002908" MajorTopicYN="N">Chronic Disease</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D005122" MajorTopicYN="N">Exudates and Transudates</DescriptorName><QualifierName UI="Q000502" MajorTopicYN="N">physiology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D005260" MajorTopicYN="N">Female</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D006439" MajorTopicYN="N">Hemodynamics</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D006801" MajorTopicYN="N">Humans</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D008196" MajorTopicYN="N">Lymph</DescriptorName><QualifierName UI="Q000502" MajorTopicYN="N">physiology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D008207" MajorTopicYN="N">Lymphatic Metastasis</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D008209" MajorTopicYN="N">Lymphedema</DescriptorName><QualifierName UI="Q000209" MajorTopicYN="Y">etiology</QualifierName><QualifierName UI="Q000503" MajorTopicYN="N">physiopathology</QualifierName></MeshHeading></MeshHeadingList></MedlineCitation><PubmedData><History><PubMedPubDate PubStatus="pubmed"><Year>1984</Year><Month>1</Month><Day>1</Day></PubMedPubDate><PubMedPubDate PubStatus="medline"><Year>1984</Year><Month>1</Month><Day>1</Day><Hour>0</Hour><Minute>1</Minute></PubMedPubDate><PubMedPubDate PubStatus="entrez"><Year>1984</Year><Month>1</Month><Day>1</Day><Hour>0</Hour><Minute>0</Minute></PubMedPubDate></History><PublicationStatus>ppublish</PublicationStatus><ArticleIdList><ArticleId IdType="pubmed">6730414</ArticleId></ArticleIdList></PubmedData></pubmed></record>Pour manipuler ce document sous Unix (Dilib)
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