A model of the factors affecting interstitial volume in oedema. Part II: Their effects at various abnormal steady-states.
Identifieur interne : 005811 ( PubMed/Curation ); précédent : 005810; suivant : 005812A model of the factors affecting interstitial volume in oedema. Part II: Their effects at various abnormal steady-states.
Auteurs : J R Casley-Smith [Australie]Source :
- Biorheology [ 0006-355X ]
Descripteurs français
- KwdFr :
- MESH :
- métabolisme : Espace extracellulaire, Lymphoedème, Oedème, Protéines.
- physiologie : Espace extracellulaire.
- physiopathologie : Oedème.
- Fibrose, Humains, Hydrolyse, Mathématiques, Modèles biologiques.
English descriptors
- KwdEn :
- MESH :
- chemical , metabolism : Proteins.
- metabolism : Edema, Extracellular Space, Lymphedema.
- physiology : Extracellular Space.
- physiopathology : Edema.
- Fibrosis, Humans, Hydrolysis, Mathematics, Models, Biological.
Abstract
A mathematical model has been used to investigate the alterations, in amount and importance, of the Factors controlling interstitial fluid volume in a number of steady-state oedemas. Steady-states were used because it is much easier to evaluate the effect of a specific Factor on oedema if all the others are constant. They are best compared if expressed in the same units: ml/min/100 g of tissue. The effects of lymphatic drainage on trauma are very important, as is the effect of tissue proteolysis on lymphoedema. Tissue proteolysis has minimal effects on the normal state and trauma; yet modelling its increase with benzo-pyrones markedly reduces oedema in both trauma and lymphoedema. Excess fibrosis greatly reduces lymphoedema. The effects of the individual Factors vary considerably between the different steady-states.
PubMed: 8374104
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pubmed:8374104Le document en format XML
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<term>Edema (physiopathology)</term>
<term>Extracellular Space (metabolism)</term>
<term>Extracellular Space (physiology)</term>
<term>Fibrosis</term>
<term>Humans</term>
<term>Hydrolysis</term>
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<keywords scheme="MESH" qualifier="métabolisme" xml:lang="fr"><term>Espace extracellulaire</term>
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<term>Protéines</term>
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<term>Humans</term>
<term>Hydrolysis</term>
<term>Mathematics</term>
<term>Models, Biological</term>
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<keywords scheme="MESH" xml:lang="fr"><term>Fibrose</term>
<term>Humains</term>
<term>Hydrolyse</term>
<term>Mathématiques</term>
<term>Modèles biologiques</term>
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<front><div type="abstract" xml:lang="en">A mathematical model has been used to investigate the alterations, in amount and importance, of the Factors controlling interstitial fluid volume in a number of steady-state oedemas. Steady-states were used because it is much easier to evaluate the effect of a specific Factor on oedema if all the others are constant. They are best compared if expressed in the same units: ml/min/100 g of tissue. The effects of lymphatic drainage on trauma are very important, as is the effect of tissue proteolysis on lymphoedema. Tissue proteolysis has minimal effects on the normal state and trauma; yet modelling its increase with benzo-pyrones markedly reduces oedema in both trauma and lymphoedema. Excess fibrosis greatly reduces lymphoedema. The effects of the individual Factors vary considerably between the different steady-states.</div>
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<Abstract><AbstractText>A mathematical model has been used to investigate the alterations, in amount and importance, of the Factors controlling interstitial fluid volume in a number of steady-state oedemas. Steady-states were used because it is much easier to evaluate the effect of a specific Factor on oedema if all the others are constant. They are best compared if expressed in the same units: ml/min/100 g of tissue. The effects of lymphatic drainage on trauma are very important, as is the effect of tissue proteolysis on lymphoedema. Tissue proteolysis has minimal effects on the normal state and trauma; yet modelling its increase with benzo-pyrones markedly reduces oedema in both trauma and lymphoedema. Excess fibrosis greatly reduces lymphoedema. The effects of the individual Factors vary considerably between the different steady-states.</AbstractText>
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