Extracorporeal shock wave therapy combined with vascular endothelial growth factor-C hydrogel for lymphangiogenesis.
Identifieur interne : 001E27 ( PubMed/Curation ); précédent : 001E26; suivant : 001E28Extracorporeal shock wave therapy combined with vascular endothelial growth factor-C hydrogel for lymphangiogenesis.
Auteurs : In Gul Kim [Corée du Sud] ; Ji Youl Lee ; David S. Lee ; Jeong Yi Kwon ; Ji Hye HwangSource :
- Journal of vascular research [ 1423-0135 ] ; 2013.
Descripteurs français
- KwdFr :
- Activation des macrophages (), Animaux, Association thérapeutique, Cicatrisation de plaie (), Cicatrisation de plaie (physiologie), Facteur de croissance endothéliale vasculaire de type C (administration et posologie), Facteur de croissance endothéliale vasculaire de type C (biosynthèse), Facteur de croissance endothéliale vasculaire de type C (génétique), Facteur de croissance endothéliale vasculaire de type C (usage thérapeutique), Femelle, Gélatine, Hydrogels, Implant pharmaceutique, Lymphangiogenèse (), Lymphoedème (), Lymphoedème (traitement médicamenteux), Membre pelvien, Modèles animaux de maladie humaine, Ondes de choc de haute énergie (usage thérapeutique), Récepteur-3 au facteur croissance endothéliale vasculaire (biosynthèse), Récepteur-3 au facteur croissance endothéliale vasculaire (génétique), Régulation de l'expression des gènes (), Régénération (), Souris, Souris de lignée BALB C, Vaisseaux lymphatiques (), Vaisseaux lymphatiques (physiologie), Vaisseaux lymphatiques (traumatismes), Évaluation préclinique de médicament.
- MESH :
- administration et posologie : Facteur de croissance endothéliale vasculaire de type C.
- biosynthèse : Facteur de croissance endothéliale vasculaire de type C, Récepteur-3 au facteur croissance endothéliale vasculaire.
- génétique : Facteur de croissance endothéliale vasculaire de type C, Récepteur-3 au facteur croissance endothéliale vasculaire.
- physiologie : Cicatrisation de plaie, Vaisseaux lymphatiques.
- traitement médicamenteux : Lymphoedème.
- traumatismes : Vaisseaux lymphatiques.
- usage thérapeutique : Facteur de croissance endothéliale vasculaire de type C, Ondes de choc de haute énergie.
- Activation des macrophages, Animaux, Association thérapeutique, Cicatrisation de plaie, Femelle, Gélatine, Hydrogels, Implant pharmaceutique, Lymphangiogenèse, Lymphoedème, Membre pelvien, Modèles animaux de maladie humaine, Régulation de l'expression des gènes, Régénération, Souris, Souris de lignée BALB C, Vaisseaux lymphatiques, Évaluation préclinique de médicament.
English descriptors
- KwdEn :
- Animals, Combined Modality Therapy, Disease Models, Animal, Drug Evaluation, Preclinical, Drug Implants, Female, Gelatin, Gene Expression Regulation (drug effects), High-Energy Shock Waves (therapeutic use), Hindlimb, Hydrogels, Lymphangiogenesis (drug effects), Lymphatic Vessels (drug effects), Lymphatic Vessels (injuries), Lymphatic Vessels (physiology), Lymphedema (drug therapy), Lymphedema (therapy), Macrophage Activation (drug effects), Mice, Mice, Inbred BALB C, Regeneration (drug effects), Vascular Endothelial Growth Factor C (administration & dosage), Vascular Endothelial Growth Factor C (biosynthesis), Vascular Endothelial Growth Factor C (genetics), Vascular Endothelial Growth Factor C (therapeutic use), Vascular Endothelial Growth Factor Receptor-3 (biosynthesis), Vascular Endothelial Growth Factor Receptor-3 (genetics), Wound Healing (drug effects), Wound Healing (physiology).
- MESH :
- chemical , administration & dosage : Vascular Endothelial Growth Factor C.
- chemical , biosynthesis : Vascular Endothelial Growth Factor C, Vascular Endothelial Growth Factor Receptor-3.
- chemical , genetics : Vascular Endothelial Growth Factor C, Vascular Endothelial Growth Factor Receptor-3.
- chemical , therapeutic use : Vascular Endothelial Growth Factor C.
- chemical : Drug Implants, Gelatin, Hydrogels.
- drug effects : Gene Expression Regulation, Lymphangiogenesis, Lymphatic Vessels, Macrophage Activation, Regeneration, Wound Healing.
- drug therapy : Lymphedema.
- injuries : Lymphatic Vessels.
- physiology : Lymphatic Vessels, Wound Healing.
- therapeutic use : High-Energy Shock Waves.
- therapy : Lymphedema.
- Animals, Combined Modality Therapy, Disease Models, Animal, Drug Evaluation, Preclinical, Female, Hindlimb, Mice, Mice, Inbred BALB C.
Abstract
Lymphedema is a clinically incurable disease that occurs commonly after lymph node dissection and/or irradiation. Several studies have recently demonstrated that extracorporeal shock wave therapy (ESWT) could promote lymphangiogenesis associated with expression of vascular endothelial growth factor (VEGF)-C. This research concerned primarily the synergistic effect of ESWT combined with VEGF-C incorporated hydrogel (VEGF-C hydrogel) combination therapy for promoting lymphangiogenesis and ultimately alleviating lymphedema.
DOI: 10.1159/000343699
PubMed: 23208012
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pubmed:23208012Le document en format XML
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<term>Drug Evaluation, Preclinical</term>
<term>Drug Implants</term>
<term>Female</term>
<term>Gelatin</term>
<term>Gene Expression Regulation (drug effects)</term>
<term>High-Energy Shock Waves (therapeutic use)</term>
<term>Hindlimb</term>
<term>Hydrogels</term>
<term>Lymphangiogenesis (drug effects)</term>
<term>Lymphatic Vessels (drug effects)</term>
<term>Lymphatic Vessels (injuries)</term>
<term>Lymphatic Vessels (physiology)</term>
<term>Lymphedema (drug therapy)</term>
<term>Lymphedema (therapy)</term>
<term>Macrophage Activation (drug effects)</term>
<term>Mice</term>
<term>Mice, Inbred BALB C</term>
<term>Regeneration (drug effects)</term>
<term>Vascular Endothelial Growth Factor C (administration & dosage)</term>
<term>Vascular Endothelial Growth Factor C (biosynthesis)</term>
<term>Vascular Endothelial Growth Factor C (genetics)</term>
<term>Vascular Endothelial Growth Factor C (therapeutic use)</term>
<term>Vascular Endothelial Growth Factor Receptor-3 (biosynthesis)</term>
<term>Vascular Endothelial Growth Factor Receptor-3 (genetics)</term>
<term>Wound Healing (drug effects)</term>
<term>Wound Healing (physiology)</term>
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<keywords scheme="KwdFr" xml:lang="fr"><term>Activation des macrophages ()</term>
<term>Animaux</term>
<term>Association thérapeutique</term>
<term>Cicatrisation de plaie ()</term>
<term>Cicatrisation de plaie (physiologie)</term>
<term>Facteur de croissance endothéliale vasculaire de type C (administration et posologie)</term>
<term>Facteur de croissance endothéliale vasculaire de type C (biosynthèse)</term>
<term>Facteur de croissance endothéliale vasculaire de type C (génétique)</term>
<term>Facteur de croissance endothéliale vasculaire de type C (usage thérapeutique)</term>
<term>Femelle</term>
<term>Gélatine</term>
<term>Hydrogels</term>
<term>Implant pharmaceutique</term>
<term>Lymphangiogenèse ()</term>
<term>Lymphoedème ()</term>
<term>Lymphoedème (traitement médicamenteux)</term>
<term>Membre pelvien</term>
<term>Modèles animaux de maladie humaine</term>
<term>Ondes de choc de haute énergie (usage thérapeutique)</term>
<term>Récepteur-3 au facteur croissance endothéliale vasculaire (biosynthèse)</term>
<term>Récepteur-3 au facteur croissance endothéliale vasculaire (génétique)</term>
<term>Régulation de l'expression des gènes ()</term>
<term>Régénération ()</term>
<term>Souris</term>
<term>Souris de lignée BALB C</term>
<term>Vaisseaux lymphatiques ()</term>
<term>Vaisseaux lymphatiques (physiologie)</term>
<term>Vaisseaux lymphatiques (traumatismes)</term>
<term>Évaluation préclinique de médicament</term>
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<keywords scheme="MESH" type="chemical" qualifier="administration & dosage" xml:lang="en"><term>Vascular Endothelial Growth Factor C</term>
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<keywords scheme="MESH" type="chemical" qualifier="biosynthesis" xml:lang="en"><term>Vascular Endothelial Growth Factor C</term>
<term>Vascular Endothelial Growth Factor Receptor-3</term>
</keywords>
<keywords scheme="MESH" type="chemical" qualifier="genetics" xml:lang="en"><term>Vascular Endothelial Growth Factor C</term>
<term>Vascular Endothelial Growth Factor Receptor-3</term>
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<keywords scheme="MESH" type="chemical" qualifier="therapeutic use" xml:lang="en"><term>Vascular Endothelial Growth Factor C</term>
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<keywords scheme="MESH" type="chemical" xml:lang="en"><term>Drug Implants</term>
<term>Gelatin</term>
<term>Hydrogels</term>
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<term>Récepteur-3 au facteur croissance endothéliale vasculaire</term>
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<keywords scheme="MESH" qualifier="drug effects" xml:lang="en"><term>Gene Expression Regulation</term>
<term>Lymphangiogenesis</term>
<term>Lymphatic Vessels</term>
<term>Macrophage Activation</term>
<term>Regeneration</term>
<term>Wound Healing</term>
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<keywords scheme="MESH" qualifier="drug therapy" xml:lang="en"><term>Lymphedema</term>
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<keywords scheme="MESH" qualifier="génétique" xml:lang="fr"><term>Facteur de croissance endothéliale vasculaire de type C</term>
<term>Récepteur-3 au facteur croissance endothéliale vasculaire</term>
</keywords>
<keywords scheme="MESH" qualifier="injuries" xml:lang="en"><term>Lymphatic Vessels</term>
</keywords>
<keywords scheme="MESH" qualifier="physiologie" xml:lang="fr"><term>Cicatrisation de plaie</term>
<term>Vaisseaux lymphatiques</term>
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<keywords scheme="MESH" qualifier="physiology" xml:lang="en"><term>Lymphatic Vessels</term>
<term>Wound Healing</term>
</keywords>
<keywords scheme="MESH" qualifier="therapeutic use" xml:lang="en"><term>High-Energy Shock Waves</term>
</keywords>
<keywords scheme="MESH" qualifier="therapy" xml:lang="en"><term>Lymphedema</term>
</keywords>
<keywords scheme="MESH" qualifier="traitement médicamenteux" xml:lang="fr"><term>Lymphoedème</term>
</keywords>
<keywords scheme="MESH" qualifier="traumatismes" xml:lang="fr"><term>Vaisseaux lymphatiques</term>
</keywords>
<keywords scheme="MESH" qualifier="usage thérapeutique" xml:lang="fr"><term>Facteur de croissance endothéliale vasculaire de type C</term>
<term>Ondes de choc de haute énergie</term>
</keywords>
<keywords scheme="MESH" xml:lang="en"><term>Animals</term>
<term>Combined Modality Therapy</term>
<term>Disease Models, Animal</term>
<term>Drug Evaluation, Preclinical</term>
<term>Female</term>
<term>Hindlimb</term>
<term>Mice</term>
<term>Mice, Inbred BALB C</term>
</keywords>
<keywords scheme="MESH" xml:lang="fr"><term>Activation des macrophages</term>
<term>Animaux</term>
<term>Association thérapeutique</term>
<term>Cicatrisation de plaie</term>
<term>Femelle</term>
<term>Gélatine</term>
<term>Hydrogels</term>
<term>Implant pharmaceutique</term>
<term>Lymphangiogenèse</term>
<term>Lymphoedème</term>
<term>Membre pelvien</term>
<term>Modèles animaux de maladie humaine</term>
<term>Régulation de l'expression des gènes</term>
<term>Régénération</term>
<term>Souris</term>
<term>Souris de lignée BALB C</term>
<term>Vaisseaux lymphatiques</term>
<term>Évaluation préclinique de médicament</term>
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<front><div type="abstract" xml:lang="en">Lymphedema is a clinically incurable disease that occurs commonly after lymph node dissection and/or irradiation. Several studies have recently demonstrated that extracorporeal shock wave therapy (ESWT) could promote lymphangiogenesis associated with expression of vascular endothelial growth factor (VEGF)-C. This research concerned primarily the synergistic effect of ESWT combined with VEGF-C incorporated hydrogel (VEGF-C hydrogel) combination therapy for promoting lymphangiogenesis and ultimately alleviating lymphedema.</div>
</front>
</TEI>
<pubmed><MedlineCitation Status="MEDLINE" Owner="NLM"><PMID Version="1">23208012</PMID>
<DateCreated><Year>2013</Year>
<Month>02</Month>
<Day>22</Day>
</DateCreated>
<DateCompleted><Year>2013</Year>
<Month>04</Month>
<Day>23</Day>
</DateCompleted>
<DateRevised><Year>2013</Year>
<Month>02</Month>
<Day>22</Day>
</DateRevised>
<Article PubModel="Print-Electronic"><Journal><ISSN IssnType="Electronic">1423-0135</ISSN>
<JournalIssue CitedMedium="Internet"><Volume>50</Volume>
<Issue>2</Issue>
<PubDate><Year>2013</Year>
</PubDate>
</JournalIssue>
<Title>Journal of vascular research</Title>
<ISOAbbreviation>J. Vasc. Res.</ISOAbbreviation>
</Journal>
<ArticleTitle>Extracorporeal shock wave therapy combined with vascular endothelial growth factor-C hydrogel for lymphangiogenesis.</ArticleTitle>
<Pagination><MedlinePgn>124-33</MedlinePgn>
</Pagination>
<ELocationID EIdType="doi" ValidYN="Y">10.1159/000343699</ELocationID>
<Abstract><AbstractText Label="BACKGROUND/AIMS" NlmCategory="OBJECTIVE">Lymphedema is a clinically incurable disease that occurs commonly after lymph node dissection and/or irradiation. Several studies have recently demonstrated that extracorporeal shock wave therapy (ESWT) could promote lymphangiogenesis associated with expression of vascular endothelial growth factor (VEGF)-C. This research concerned primarily the synergistic effect of ESWT combined with VEGF-C incorporated hydrogel (VEGF-C hydrogel) combination therapy for promoting lymphangiogenesis and ultimately alleviating lymphedema.</AbstractText>
<AbstractText Label="METHODS" NlmCategory="METHODS">The VEGF-C hydrogel was applied to the injury site in a mouse model of lymphedema and then regularly underwent ESWT (0.05 mJ/mm(2), 500 shots) every 3 days for 4 weeks.</AbstractText>
<AbstractText Label="RESULTS" NlmCategory="RESULTS">Four weeks after the treatment, mice treated with VEGF-C hydrogel and ESWT showed signs of the greatest decrease in edema/collagenous deposits when compared with the other experimental group. LYVE-1-positive vessels also revealed that the VEGF-C/ESWT group had significantly induced the growth of new lymphatic vessels compared to the other groups. Western blot analysis showed that expression of VEGF-C (1.24-fold) and VEGF receptor-3 (1.41-fold) was significantly increased in the VEGF-C/ESWT group compared to the normal group.</AbstractText>
<AbstractText Label="CONCLUSION" NlmCategory="CONCLUSIONS">These results suggested that VEGF-C and ESWT had a synergistic effect and were very effective in alleviating the symptoms of lymphedema and promoting lymphangiogenesis.</AbstractText>
<CopyrightInformation>Copyright © 2012 S. Karger AG, Basel.</CopyrightInformation>
</Abstract>
<AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Kim</LastName>
<ForeName>In Gul</ForeName>
<Initials>IG</Initials>
<AffiliationInfo><Affiliation>Department of Urology, Seoul St. Mary's Hospital, The Catholic University of Korea, Seoul, Korea.</Affiliation>
</AffiliationInfo>
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<Author ValidYN="Y"><LastName>Lee</LastName>
<ForeName>Ji Youl</ForeName>
<Initials>JY</Initials>
</Author>
<Author ValidYN="Y"><LastName>Lee</LastName>
<ForeName>David S</ForeName>
<Initials>DS</Initials>
</Author>
<Author ValidYN="Y"><LastName>Kwon</LastName>
<ForeName>Jeong Yi</ForeName>
<Initials>JY</Initials>
</Author>
<Author ValidYN="Y"><LastName>Hwang</LastName>
<ForeName>Ji Hye</ForeName>
<Initials>JH</Initials>
</Author>
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<Language>eng</Language>
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<Month>11</Month>
<Day>27</Day>
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<MedlineJournalInfo><Country>Switzerland</Country>
<MedlineTA>J Vasc Res</MedlineTA>
<NlmUniqueID>9206092</NlmUniqueID>
<ISSNLinking>1018-1172</ISSNLinking>
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<ChemicalList><Chemical><RegistryNumber>0</RegistryNumber>
<NameOfSubstance UI="D004343">Drug Implants</NameOfSubstance>
</Chemical>
<Chemical><RegistryNumber>0</RegistryNumber>
<NameOfSubstance UI="D020100">Hydrogels</NameOfSubstance>
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<NameOfSubstance UI="C467483">vascular endothelial growth factor C, mouse</NameOfSubstance>
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<Chemical><RegistryNumber>9000-70-8</RegistryNumber>
<NameOfSubstance UI="D005780">Gelatin</NameOfSubstance>
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<Chemical><RegistryNumber>EC 2.7.10.1</RegistryNumber>
<NameOfSubstance UI="D040321">Vascular Endothelial Growth Factor Receptor-3</NameOfSubstance>
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<MeshHeadingList><MeshHeading><DescriptorName UI="D000818" MajorTopicYN="N">Animals</DescriptorName>
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<MeshHeading><DescriptorName UI="D005780" MajorTopicYN="N">Gelatin</DescriptorName>
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<MeshHeading><DescriptorName UI="D040321" MajorTopicYN="N">Vascular Endothelial Growth Factor Receptor-3</DescriptorName>
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