LymphedemaV1, PubMed, Corpus, bibRecord, 001211

GATA2 deficiency.

Identifieur interne : 001211 ( PubMed/Corpus ); précédent : 001210; suivant : 001212

GATA2 deficiency.

Auteurs : Amy P. Hsu ; Lisa J. Mcreynolds ; Steven M. Holland

Source :

Current opinion in allergy and clinical immunology [ 1473-6322 ] ; 2015.

RBID : pubmed:25397911

English descriptors

KwdEn :
- Animals, Bone Marrow Transplantation, GATA2 Transcription Factor (genetics), Genetic Predisposition to Disease, Hematologic Neoplasms (genetics), Hematologic Neoplasms (therapy), Hematopoiesis (genetics), Humans, Infection (genetics), Infection (therapy), Mutation (genetics), Polymorphism, Genetic.
MESH :
- chemical , genetics : GATA2 Transcription Factor.
- genetics : Hematologic Neoplasms, Hematopoiesis, Infection, Mutation.
- therapy : Hematologic Neoplasms, Infection.
- Animals, Bone Marrow Transplantation, Genetic Predisposition to Disease, Humans, Polymorphism, Genetic.

Abstract

GATA2 deficiency is a germline disease that causes a wide spectrum of phenotypes including viral and bacterial infections, cytopenias, myelodysplasia, myeloid leukemias, pulmonary alveolar proteinosis and lymphedema. The age of clinical presentation ranges from early childhood to late adulthood, with most occurring in adolescence to early adulthood. We review the expanding GATA2-deficient phenotype, molecular genetics of disease and developments in treatment.

DOI: 10.1097/ACI.0000000000000126
PubMed: 25397911

Links to Exploration step

pubmed:25397911

Le document en format XML

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<author><name sortKey="Hsu, Amy P" sort="Hsu, Amy P" uniqKey="Hsu A" first="Amy P" last="Hsu">Amy P. Hsu</name>
<affiliation><nlm:affiliation>aLaboratory of Clinical Infectious Diseases, National Institute of Allergy and Infectious Diseases bPediatric Oncology Branch, National Cancer Institute, National Institutes of Health, Bethesda, Maryland, USA.</nlm:affiliation>
</affiliation>
</author>
<author><name sortKey="Mcreynolds, Lisa J" sort="Mcreynolds, Lisa J" uniqKey="Mcreynolds L" first="Lisa J" last="Mcreynolds">Lisa J. Mcreynolds</name>
</author>
<author><name sortKey="Holland, Steven M" sort="Holland, Steven M" uniqKey="Holland S" first="Steven M" last="Holland">Steven M. Holland</name>
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<sourceDesc><biblStruct><analytic><title xml:lang="en">GATA2 deficiency.</title>
<author><name sortKey="Hsu, Amy P" sort="Hsu, Amy P" uniqKey="Hsu A" first="Amy P" last="Hsu">Amy P. Hsu</name>
<affiliation><nlm:affiliation>aLaboratory of Clinical Infectious Diseases, National Institute of Allergy and Infectious Diseases bPediatric Oncology Branch, National Cancer Institute, National Institutes of Health, Bethesda, Maryland, USA.</nlm:affiliation>
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<author><name sortKey="Mcreynolds, Lisa J" sort="Mcreynolds, Lisa J" uniqKey="Mcreynolds L" first="Lisa J" last="Mcreynolds">Lisa J. Mcreynolds</name>
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<author><name sortKey="Holland, Steven M" sort="Holland, Steven M" uniqKey="Holland S" first="Steven M" last="Holland">Steven M. Holland</name>
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<series><title level="j">Current opinion in allergy and clinical immunology</title>
<idno type="eISSN">1473-6322</idno>
<imprint><date when="2015" type="published">2015</date>
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<profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Animals</term>
<term>Bone Marrow Transplantation</term>
<term>GATA2 Transcription Factor (genetics)</term>
<term>Genetic Predisposition to Disease</term>
<term>Hematologic Neoplasms (genetics)</term>
<term>Hematologic Neoplasms (therapy)</term>
<term>Hematopoiesis (genetics)</term>
<term>Humans</term>
<term>Infection (genetics)</term>
<term>Infection (therapy)</term>
<term>Mutation (genetics)</term>
<term>Polymorphism, Genetic</term>
</keywords>
<keywords scheme="MESH" type="chemical" qualifier="genetics" xml:lang="en"><term>GATA2 Transcription Factor</term>
</keywords>
<keywords scheme="MESH" qualifier="genetics" xml:lang="en"><term>Hematologic Neoplasms</term>
<term>Hematopoiesis</term>
<term>Infection</term>
<term>Mutation</term>
</keywords>
<keywords scheme="MESH" qualifier="therapy" xml:lang="en"><term>Hematologic Neoplasms</term>
<term>Infection</term>
</keywords>
<keywords scheme="MESH" xml:lang="en"><term>Animals</term>
<term>Bone Marrow Transplantation</term>
<term>Genetic Predisposition to Disease</term>
<term>Humans</term>
<term>Polymorphism, Genetic</term>
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<front><div type="abstract" xml:lang="en">GATA2 deficiency is a germline disease that causes a wide spectrum of phenotypes including viral and bacterial infections, cytopenias, myelodysplasia, myeloid leukemias, pulmonary alveolar proteinosis and lymphedema. The age of clinical presentation ranges from early childhood to late adulthood, with most occurring in adolescence to early adulthood. We review the expanding GATA2-deficient phenotype, molecular genetics of disease and developments in treatment.</div>
</front>
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<DateCreated><Year>2014</Year>
<Month>12</Month>
<Day>25</Day>
</DateCreated>
<DateCompleted><Year>2015</Year>
<Month>09</Month>
<Day>08</Day>
</DateCompleted>
<DateRevised><Year>2016</Year>
<Month>10</Month>
<Day>19</Day>
</DateRevised>
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<Issue>1</Issue>
<PubDate><Year>2015</Year>
<Month>Feb</Month>
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<Title>Current opinion in allergy and clinical immunology</Title>
<ISOAbbreviation>Curr Opin Allergy Clin Immunol</ISOAbbreviation>
</Journal>
<ArticleTitle>GATA2 deficiency.</ArticleTitle>
<Pagination><MedlinePgn>104-9</MedlinePgn>
</Pagination>
<ELocationID EIdType="doi" ValidYN="Y">10.1097/ACI.0000000000000126</ELocationID>
<Abstract><AbstractText Label="PURPOSE OF REVIEW" NlmCategory="OBJECTIVE">GATA2 deficiency is a germline disease that causes a wide spectrum of phenotypes including viral and bacterial infections, cytopenias, myelodysplasia, myeloid leukemias, pulmonary alveolar proteinosis and lymphedema. The age of clinical presentation ranges from early childhood to late adulthood, with most occurring in adolescence to early adulthood. We review the expanding GATA2-deficient phenotype, molecular genetics of disease and developments in treatment.</AbstractText>
<AbstractText Label="RECENT FINDINGS" NlmCategory="RESULTS">GATA2 mutations have been found in up to 10% of those with congenital neutropenia and/or aplastic anemia. Heterozygous mutations appear to cause haploinsufficiency due to either protein dysfunction or uniallelic reduced transcription. Disease-associated mutations in intronic regulatory elements or variations within the 5' leader exons indicate that regulation of GATA2 is critical. Those with GATA2 mutations are at high risk for myelodysplasia, cytogenetic abnormalities, acute myeloid leukemia or chronic myelomonocytic leukemia. Bone marrow transplantation has been successful for both hematopoietic and pulmonary alveolar proteinosis repair.</AbstractText>
<AbstractText Label="SUMMARY" NlmCategory="CONCLUSIONS">GATA2 is a zinc finger transcription factor essential for embryonic and definitive hematopoiesis as well as lymphatic angiogenesis. GATA2 deficiency is caused by a variety of mutations in the GATA2 gene and can have variable presentation, onset and outcome. Patients are susceptible to mycobacterial, viral and fungal infections and can develop myelodysplasia, acute or chronic leukemias, lymphedema and pulmonary alveolar proteinosis. Hematopoietic stem cell transplantation reverses most of the clinical phenotype with good long-term outcomes.</AbstractText>
</Abstract>
<AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Hsu</LastName>
<ForeName>Amy P</ForeName>
<Initials>AP</Initials>
<AffiliationInfo><Affiliation>aLaboratory of Clinical Infectious Diseases, National Institute of Allergy and Infectious Diseases bPediatric Oncology Branch, National Cancer Institute, National Institutes of Health, Bethesda, Maryland, USA.</Affiliation>
</AffiliationInfo>
</Author>
<Author ValidYN="Y"><LastName>McReynolds</LastName>
<ForeName>Lisa J</ForeName>
<Initials>LJ</Initials>
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<Author ValidYN="Y"><LastName>Holland</LastName>
<ForeName>Steven M</ForeName>
<Initials>SM</Initials>
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<Agency>NCI NIH HHS</Agency>
<Country>United States</Country>
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<Agency>Intramural NIH HHS</Agency>
<Country>United States</Country>
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<Agency>NCI NIH HHS</Agency>
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<NameOfSubstance UI="C494711">GATA2 protein, human</NameOfSubstance>
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GATA2 deficiency.

GATA2 deficiency.

Source :

English descriptors

Abstract

Links to Exploration step

Le document en format XML

Pour manipuler ce document sous Unix (Dilib)

Pour mettre un lien sur cette page dans le réseau Wicri

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