[Venous and lymphatic reasons for edema--the swollen leg from the angiologist's point of view].
Identifieur interne : 003959 ( PubMed/Checkpoint ); précédent : 003958; suivant : 003960[Venous and lymphatic reasons for edema--the swollen leg from the angiologist's point of view].
Auteurs : S. Friedli [Suisse] ; F. MahlerSource :
- Therapeutische Umschau. Revue therapeutique [ 0040-5930 ] ; 2004.
Descripteurs français
- KwdFr :
- Diagnostic différentiel, Humains, Insuffisance veineuse (), Insuffisance veineuse (diagnostic), Insuffisance veineuse (physiopathologie), Jambe (), Lymphoedème (), Lymphoedème (physiopathologie), Lymphoedème (étiologie), Oedème (physiopathologie), Oedème (étiologie), Perméabilité capillaire (physiologie), Pression veineuse (physiologie), Syndrome post-phlébitique (), Syndrome post-phlébitique (diagnostic), Syndrome post-phlébitique (physiopathologie), Thrombophlébite (), Thrombophlébite (diagnostic), Thrombophlébite (physiopathologie), Vaisseaux lymphatiques (physiopathologie).
- MESH :
- diagnostic : Insuffisance veineuse, Syndrome post-phlébitique, Thrombophlébite.
- physiologie : Perméabilité capillaire, Pression veineuse.
- physiopathologie : Insuffisance veineuse, Lymphoedème, Oedème, Syndrome post-phlébitique, Thrombophlébite, Vaisseaux lymphatiques.
- étiologie : Lymphoedème, Oedème.
- Diagnostic différentiel, Humains, Insuffisance veineuse, Jambe, Lymphoedème, Syndrome post-phlébitique, Thrombophlébite.
English descriptors
- KwdEn :
- Capillary Permeability (physiology), Diagnosis, Differential, Edema (etiology), Edema (physiopathology), Humans, Leg (blood supply), Lymphatic Vessels (physiopathology), Lymphedema (etiology), Lymphedema (physiopathology), Lymphedema (therapy), Postphlebitic Syndrome (diagnosis), Postphlebitic Syndrome (physiopathology), Postphlebitic Syndrome (therapy), Thrombophlebitis (diagnosis), Thrombophlebitis (physiopathology), Thrombophlebitis (therapy), Venous Insufficiency (diagnosis), Venous Insufficiency (physiopathology), Venous Insufficiency (therapy), Venous Pressure (physiology).
- MESH :
- blood supply : Leg.
- diagnosis : Postphlebitic Syndrome, Thrombophlebitis, Venous Insufficiency.
- etiology : Edema, Lymphedema.
- physiology : Capillary Permeability, Venous Pressure.
- physiopathology : Edema, Lymphatic Vessels, Lymphedema, Postphlebitic Syndrome, Thrombophlebitis, Venous Insufficiency.
- therapy : Lymphedema, Postphlebitic Syndrome, Thrombophlebitis, Venous Insufficiency.
- Diagnosis, Differential, Humans.
Abstract
Peripheral edema develop as a consequence of imbalance in the processes of filtration, resorption and lymphatic transport in the capillary bed. Venous hypertension and impaired lymphatic function belong to the most important underlying pathomechanisms. Chronic venous insufficiency as a result of valve degeneration as well as venous obstruction in acute deep thrombosis lead to venous hypertension and to an increase of filtration pressure. As venous diseases are frequent, they are one of the most reasons for a swollen leg in clinical everyday life. Primary and secondary disturbances of the lymphatic system are another important reason for interstitial liquid retention. Although there are about 140 millions of people suffering from lymphedema worldwide, the disease is still underdiagnosed.
DOI: 10.1024/0040-5930.61.11.643
PubMed: 15605455
Affiliations:
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Friedli</name><affiliation wicri:level="3"><nlm:affiliation>Abteilung für Angiologie, Inselspital, Bern. sandra.friedli@insel.ch</nlm:affiliation><country>Suisse</country><placeName><settlement type="city">Berne</settlement><region type="région" nuts="3">Canton de Berne</region></placeName><wicri:orgArea>Abteilung für Angiologie, Inselspital</wicri:orgArea></affiliation></author><author><name sortKey="Mahler, F" sort="Mahler, F" uniqKey="Mahler F" first="F" last="Mahler">F. Mahler</name></author></analytic><series><title level="j">Therapeutische Umschau. 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Venous hypertension and impaired lymphatic function belong to the most important underlying pathomechanisms. Chronic venous insufficiency as a result of valve degeneration as well as venous obstruction in acute deep thrombosis lead to venous hypertension and to an increase of filtration pressure. As venous diseases are frequent, they are one of the most reasons for a swollen leg in clinical everyday life. Primary and secondary disturbances of the lymphatic system are another important reason for interstitial liquid retention. Although there are about 140 millions of people suffering from lymphedema worldwide, the disease is still underdiagnosed.</div></front></TEI><pubmed><MedlineCitation Status="MEDLINE" Owner="NLM"><PMID Version="1">15605455</PMID><DateCreated><Year>2004</Year><Month>12</Month><Day>20</Day></DateCreated><DateCompleted><Year>2005</Year><Month>03</Month><Day>23</Day></DateCompleted><DateRevised><Year>2008</Year><Month>02</Month><Day>12</Day></DateRevised><Article PubModel="Print"><Journal><ISSN IssnType="Print">0040-5930</ISSN><JournalIssue CitedMedium="Print"><Volume>61</Volume><Issue>11</Issue><PubDate><Year>2004</Year><Month>Nov</Month></PubDate></JournalIssue><Title>Therapeutische Umschau. Revue therapeutique</Title><ISOAbbreviation>Ther Umsch</ISOAbbreviation></Journal><ArticleTitle>[Venous and lymphatic reasons for edema--the swollen leg from the angiologist's point of view].</ArticleTitle><Pagination><MedlinePgn>643-7</MedlinePgn></Pagination><Abstract><AbstractText>Peripheral edema develop as a consequence of imbalance in the processes of filtration, resorption and lymphatic transport in the capillary bed. Venous hypertension and impaired lymphatic function belong to the most important underlying pathomechanisms. Chronic venous insufficiency as a result of valve degeneration as well as venous obstruction in acute deep thrombosis lead to venous hypertension and to an increase of filtration pressure. As venous diseases are frequent, they are one of the most reasons for a swollen leg in clinical everyday life. Primary and secondary disturbances of the lymphatic system are another important reason for interstitial liquid retention. Although there are about 140 millions of people suffering from lymphedema worldwide, the disease is still underdiagnosed.</AbstractText></Abstract><AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Friedli</LastName><ForeName>S</ForeName><Initials>S</Initials><AffiliationInfo><Affiliation>Abteilung für Angiologie, Inselspital, Bern. sandra.friedli@insel.ch</Affiliation></AffiliationInfo></Author><Author ValidYN="Y"><LastName>Mahler</LastName><ForeName>F</ForeName><Initials>F</Initials></Author></AuthorList><Language>ger</Language><PublicationTypeList><PublicationType UI="D004740">English Abstract</PublicationType><PublicationType UI="D016428">Journal Article</PublicationType><PublicationType UI="D016454">Review</PublicationType></PublicationTypeList><VernacularTitle>Phlebödem und Lymphödem--das dicke Bein aus angiologischer Sicht.</VernacularTitle></Article><MedlineJournalInfo><Country>Switzerland</Country><MedlineTA>Ther Umsch</MedlineTA><NlmUniqueID>0407224</NlmUniqueID><ISSNLinking>0040-5930</ISSNLinking></MedlineJournalInfo><CitationSubset>IM</CitationSubset><MeshHeadingList><MeshHeading><DescriptorName UI="D002199" MajorTopicYN="N">Capillary Permeability</DescriptorName><QualifierName UI="Q000502" MajorTopicYN="N">physiology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D003937" MajorTopicYN="N">Diagnosis, Differential</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D004487" MajorTopicYN="N">Edema</DescriptorName><QualifierName UI="Q000209" MajorTopicYN="Y">etiology</QualifierName><QualifierName UI="Q000503" MajorTopicYN="N">physiopathology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D006801" MajorTopicYN="N">Humans</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D007866" MajorTopicYN="Y">Leg</DescriptorName><QualifierName UI="Q000098" MajorTopicYN="N">blood supply</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D042601" MajorTopicYN="N">Lymphatic Vessels</DescriptorName><QualifierName UI="Q000503" MajorTopicYN="N">physiopathology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D008209" MajorTopicYN="N">Lymphedema</DescriptorName><QualifierName UI="Q000209" MajorTopicYN="Y">etiology</QualifierName><QualifierName UI="Q000503" MajorTopicYN="N">physiopathology</QualifierName><QualifierName UI="Q000628" MajorTopicYN="N">therapy</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D011186" MajorTopicYN="N">Postphlebitic Syndrome</DescriptorName><QualifierName UI="Q000175" MajorTopicYN="Y">diagnosis</QualifierName><QualifierName UI="Q000503" MajorTopicYN="N">physiopathology</QualifierName><QualifierName UI="Q000628" MajorTopicYN="N">therapy</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D013924" MajorTopicYN="N">Thrombophlebitis</DescriptorName><QualifierName UI="Q000175" MajorTopicYN="Y">diagnosis</QualifierName><QualifierName UI="Q000503" MajorTopicYN="N">physiopathology</QualifierName><QualifierName UI="Q000628" MajorTopicYN="N">therapy</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D014689" MajorTopicYN="N">Venous Insufficiency</DescriptorName><QualifierName UI="Q000175" MajorTopicYN="N">diagnosis</QualifierName><QualifierName UI="Q000503" MajorTopicYN="N">physiopathology</QualifierName><QualifierName UI="Q000628" MajorTopicYN="N">therapy</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D014690" MajorTopicYN="N">Venous Pressure</DescriptorName><QualifierName UI="Q000502" MajorTopicYN="N">physiology</QualifierName></MeshHeading></MeshHeadingList><NumberOfReferences>43</NumberOfReferences></MedlineCitation><PubmedData><History><PubMedPubDate PubStatus="pubmed"><Year>2004</Year><Month>12</Month><Day>21</Day><Hour>9</Hour><Minute>0</Minute></PubMedPubDate><PubMedPubDate PubStatus="medline"><Year>2005</Year><Month>3</Month><Day>24</Day><Hour>9</Hour><Minute>0</Minute></PubMedPubDate><PubMedPubDate PubStatus="entrez"><Year>2004</Year><Month>12</Month><Day>21</Day><Hour>9</Hour><Minute>0</Minute></PubMedPubDate></History><PublicationStatus>ppublish</PublicationStatus><ArticleIdList><ArticleId IdType="pubmed">15605455</ArticleId><ArticleId IdType="doi">10.1024/0040-5930.61.11.643</ArticleId></ArticleIdList></PubmedData></pubmed><affiliations><list><country><li>Suisse</li></country><region><li>Canton de Berne</li></region><settlement><li>Berne</li></settlement></list><tree><noCountry><name sortKey="Mahler, F" sort="Mahler, F" uniqKey="Mahler F" first="F" last="Mahler">F. Mahler</name></noCountry><country name="Suisse"><region name="Canton de Berne"><name sortKey="Friedli, S" sort="Friedli, S" uniqKey="Friedli S" first="S" last="Friedli">S. Friedli</name></region></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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