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<record><TEI><teiHeader><fileDesc><titleStmt><title xml:lang="en">Mendelian Genetics of Human Susceptibility to Fungal Infection</title><author><name sortKey="Lionakis, Michail S" sort="Lionakis, Michail S" uniqKey="Lionakis M" first="Michail S." last="Lionakis">Michail S. Lionakis</name><affiliation><nlm:aff id="af1">Fungal Pathogenesis Unit, Laboratory of Clinical Infectious Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland 20892</nlm:aff></affiliation></author><author><name sortKey="Netea, Mihai G" sort="Netea, Mihai G" uniqKey="Netea M" first="Mihai G." last="Netea">Mihai G. Netea</name><affiliation><nlm:aff id="af2">Department of Internal Medicine, and Nijmegen Institute for Infection, Inflammation and Immunity (N4i), Radboud University Nijmegen Medical Center, Nijmegen, The Netherlands</nlm:aff></affiliation></author><author><name sortKey="Holland, Steven M" sort="Holland, Steven M" uniqKey="Holland S" first="Steven M." last="Holland">Steven M. Holland</name><affiliation><nlm:aff id="af3">Immunopathogenesis Section, Laboratory of Clinical Infectious Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland 20892</nlm:aff></affiliation></author></titleStmt><publicationStmt><idno type="wicri:source">PMC</idno><idno type="pmid">24890837</idno><idno type="pmc">4031953</idno><idno type="url">http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4031953</idno><idno type="RBID">PMC:4031953</idno><idno type="doi">10.1101/cshperspect.a019638</idno><date when="2014">2014</date><idno type="wicri:Area/Pmc/Corpus">004814</idno><idno type="wicri:explorRef" wicri:stream="Pmc" wicri:step="Corpus" wicri:corpus="PMC">004814</idno></publicationStmt><sourceDesc><biblStruct><analytic><title xml:lang="en" level="a" type="main">Mendelian Genetics of Human Susceptibility to Fungal Infection</title><author><name sortKey="Lionakis, Michail S" sort="Lionakis, Michail S" uniqKey="Lionakis M" first="Michail S." last="Lionakis">Michail S. Lionakis</name><affiliation><nlm:aff id="af1">Fungal Pathogenesis Unit, Laboratory of Clinical Infectious Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland 20892</nlm:aff></affiliation></author><author><name sortKey="Netea, Mihai G" sort="Netea, Mihai G" uniqKey="Netea M" first="Mihai G." last="Netea">Mihai G. Netea</name><affiliation><nlm:aff id="af2">Department of Internal Medicine, and Nijmegen Institute for Infection, Inflammation and Immunity (N4i), Radboud University Nijmegen Medical Center, Nijmegen, The Netherlands</nlm:aff></affiliation></author><author><name sortKey="Holland, Steven M" sort="Holland, Steven M" uniqKey="Holland S" first="Steven M." last="Holland">Steven M. Holland</name><affiliation><nlm:aff id="af3">Immunopathogenesis Section, Laboratory of Clinical Infectious Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland 20892</nlm:aff></affiliation></author></analytic><series><title level="j">Cold Spring Harbor Perspectives in Medicine</title><idno type="eISSN">2157-1422</idno><imprint><date when="2014">2014</date></imprint></series></biblStruct></sourceDesc></fileDesc><profileDesc><textClass></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en"><p>A recent surge in newly described inborn errors of immune function-related genes that result in susceptibility to fungal disease has greatly enhanced our understanding of the cellular and molecular basis of antifungal immune responses. Characterization of single-gene defects that predispose to various combinations of superficial and deep-seated infections caused by yeasts, molds, and dimorphic fungi has unmasked the critical role of novel molecules and signaling pathways in mucosal and systemic antifungal host defense. These experiments of nature offer a unique opportunity for developing new knowledge in immunological research and form the foundation for devising immune-based therapeutic approaches for patients infected with fungal pathogens.</p></div></front></TEI><pmc article-type="review-article"><pmc-comment>The publisher of this article does not allow downloading of the full text in XML form.</pmc-comment>
<front><journal-meta><journal-id journal-id-type="nlm-ta">Cold Spring Harb Perspect Med</journal-id><journal-id journal-id-type="iso-abbrev">Cold Spring Harb Perspect Med</journal-id><journal-id journal-id-type="publisher-id">cshperspectmed</journal-id><journal-id journal-id-type="hwp">cshperspectmed</journal-id><journal-title-group><journal-title>Cold Spring Harbor Perspectives in Medicine</journal-title></journal-title-group><issn pub-type="epub">2157-1422</issn><publisher><publisher-name>Cold Spring Harbor Laboratory Press</publisher-name></publisher></journal-meta><article-meta><article-id pub-id-type="pmid">24890837</article-id><article-id pub-id-type="pmc">4031953</article-id><article-id pub-id-type="doi">10.1101/cshperspect.a019638</article-id><article-id pub-id-type="publisher-id">a019638</article-id><article-categories><subj-group subj-group-type="hwp-journal-coll"><subject>094</subject></subj-group><subj-group subj-group-type="heading"><subject>Perspectives</subject></subj-group></article-categories><title-group><article-title>Mendelian Genetics of Human Susceptibility to Fungal Infection</article-title><alt-title alt-title-type="left-running">M.S. Lionakis et al.</alt-title><alt-title alt-title-type="right-running">Genetics of Susceptibility to Fungal Infection</alt-title></title-group><contrib-group><contrib contrib-type="author"><name><surname>Lionakis</surname><given-names>Michail S.</given-names></name><xref ref-type="aff" rid="af1">1</xref></contrib><contrib contrib-type="author"><name><surname>Netea</surname><given-names>Mihai G.</given-names></name><xref ref-type="aff" rid="af2">2</xref></contrib><contrib contrib-type="author"><name><surname>Holland</surname><given-names>Steven M.</given-names></name><xref ref-type="aff" rid="af3">3</xref></contrib></contrib-group><aff id="af1"><label>1</label>Fungal Pathogenesis Unit, Laboratory of Clinical Infectious Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland 20892</aff><aff id="af2"><label>2</label>Department of Internal Medicine, and Nijmegen Institute for Infection, Inflammation and Immunity (N4i), Radboud University Nijmegen Medical Center, Nijmegen, The Netherlands</aff><aff id="af3"><label>3</label>Immunopathogenesis Section, Laboratory of Clinical Infectious Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland 20892</aff><author-notes><corresp><italic>Correspondence:</italic><email>lionakism@niaid.nih.gov</email></corresp></author-notes><pub-date pub-type="ppub"><month>6</month><year>2014</year></pub-date><volume>4</volume><issue>6</issue><elocation-id>a019638</elocation-id><permissions><copyright-statement>Copyright © 2014 Cold Spring Harbor Laboratory Press; all rights reserved</copyright-statement><copyright-year>2014</copyright-year></permissions><self-uri content-type="pdf" xlink:type="simple" xlink:href="cshperspectmed-HFP-a019638.pdf"></self-uri><abstract><p>A recent surge in newly described inborn errors of immune function-related genes that result in susceptibility to fungal disease has greatly enhanced our understanding of the cellular and molecular basis of antifungal immune responses. Characterization of single-gene defects that predispose to various combinations of superficial and deep-seated infections caused by yeasts, molds, and dimorphic fungi has unmasked the critical role of novel molecules and signaling pathways in mucosal and systemic antifungal host defense. These experiments of nature offer a unique opportunity for developing new knowledge in immunological research and form the foundation for devising immune-based therapeutic approaches for patients infected with fungal pathogens.</p></abstract><abstract abstract-type="precis"><p>Several dozen genetic defects that predispose to various fungal infections have been identified. Their characterization has improved our understanding of the cellular and molecular basis of antifungal immune responses.</p></abstract><counts><page-count count="21"></page-count></counts></article-meta></front></pmc></record>Pour manipuler ce document sous Unix (Dilib)
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