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Angiotensin mediated interaction of fetal kidney and placenta in the control of fetal arterial pressure and its role in hydrops fetalis

Identifieur interne : 000F63 ( PascalFrancis/Curation ); précédent : 000F62; suivant : 000F64

Angiotensin mediated interaction of fetal kidney and placenta in the control of fetal arterial pressure and its role in hydrops fetalis

Auteurs : J. J. Faber [États-Unis] ; D. F. Anderson [États-Unis]

Source :

RBID : Pascal:97-0330119

Descripteurs français

English descriptors

Abstract

Fetal cardiovascular control is effected by an interaction of the fetal somatic and placental circulations. Three primary regulatory mechanisms are involved: transplacental transfer of extracellular fluid, driven by a difference in hydrostatic and oncotic pressures; modulation of fetal placental and somatic vascular resistances by means of blood pressure controlled production of angiotensin; and somatic autoregulation of flow. A systems analysis incorporates these and other fetal cardiovascular functions and this analysis was modelled for computer simulation. Given physiologically plausible values for known cardiovascular parameters in the fetal sheep, the model reproduced in detail a variety of experimental protocols with known outcomes; these included the normal fetus, the fetus after bilateral nephrectomy, the nephrectomized fetus infused with angiotensin, the intact fetus infused with NaCl solutions, the fetus with lymphatic obstruction and the severely anaemic fetus. The systems analysis demonstrated that fetal cardiac failure constituted the strongest stimulus for the formation of fetal oedema of any tested pathological intervention.
pA  
A01 01  1    @0 0143-4004
A02 01      @0 PLACDF
A03   1    @0 Placenta : (Eastbourne)
A05       @2 18
A06       @2 4
A08 01  1  ENG  @1 Angiotensin mediated interaction of fetal kidney and placenta in the control of fetal arterial pressure and its role in hydrops fetalis
A11 01  1    @1 FABER (J. J.)
A11 02  1    @1 ANDERSON (D. F.)
A14 01      @1 Department of Physiology and Pharmacology, School of Medicine, L334, Oregon Health Sciences University @2 Portland, OR 97201-3098 @3 USA @Z 1 aut. @Z 2 aut.
A20       @1 313-326
A21       @1 1997
A23 01      @0 ENG
A43 01      @1 INIST @2 18818 @5 354000061462790110
A44       @0 0000 @1 © 1997 INIST-CNRS. All rights reserved.
A45       @0 43 ref.
A47 01  1    @0 97-0330119
A60       @1 P
A61       @0 A
A64 01  1    @0 Placenta : (Eastbourne)
A66 01      @0 GBR
C01 01    ENG  @0 Fetal cardiovascular control is effected by an interaction of the fetal somatic and placental circulations. Three primary regulatory mechanisms are involved: transplacental transfer of extracellular fluid, driven by a difference in hydrostatic and oncotic pressures; modulation of fetal placental and somatic vascular resistances by means of blood pressure controlled production of angiotensin; and somatic autoregulation of flow. A systems analysis incorporates these and other fetal cardiovascular functions and this analysis was modelled for computer simulation. Given physiologically plausible values for known cardiovascular parameters in the fetal sheep, the model reproduced in detail a variety of experimental protocols with known outcomes; these included the normal fetus, the fetus after bilateral nephrectomy, the nephrectomized fetus infused with angiotensin, the intact fetus infused with NaCl solutions, the fetus with lymphatic obstruction and the severely anaemic fetus. The systems analysis demonstrated that fetal cardiac failure constituted the strongest stimulus for the formation of fetal oedema of any tested pathological intervention.
C02 01  X    @0 002B20F02
C03 01  X  FRE  @0 Foetus @5 01
C03 01  X  ENG  @0 Fetus @5 01
C03 01  X  SPA  @0 Feto @5 01
C03 02  X  FRE  @0 Angiotensine @2 NK @2 FR @5 02
C03 02  X  ENG  @0 Angiotensin @2 NK @2 FR @5 02
C03 02  X  SPA  @0 Angiotensina @2 NK @2 FR @5 02
C03 03  X  FRE  @0 Contrôle cardiovasculaire @5 03
C03 03  X  ENG  @0 Cardiovascular control @5 03
C03 03  X  SPA  @0 Control cardiovascular @5 03
C03 04  X  FRE  @0 Rein @5 04
C03 04  X  ENG  @0 Kidney @5 04
C03 04  X  SPA  @0 Riñón @5 04
C03 05  X  FRE  @0 Placenta @5 05
C03 05  X  ENG  @0 Placenta @5 05
C03 05  X  SPA  @0 Placenta @5 05
C03 06  X  FRE  @0 Hémodynamique @5 07
C03 06  X  ENG  @0 Hemodynamics @5 07
C03 06  X  SPA  @0 Hemodinámica @5 07
C03 07  X  FRE  @0 Analyse assistée @5 08
C03 07  X  ENG  @0 Computer aided analysis @5 08
C03 07  X  SPA  @0 Análisis asistido @5 08
C03 08  X  FRE  @0 Néphrectomie @5 09
C03 08  X  ENG  @0 Nephrectomy @5 09
C03 08  X  SPA  @0 Nefrectomía @5 09
C03 09  X  FRE  @0 Foetus pathologie @5 10
C03 09  X  ENG  @0 Fetal diseases @5 10
C03 09  X  SPA  @0 Feto patología @5 10
C03 10  X  FRE  @0 Lymphoedème @5 11
C03 10  X  ENG  @0 Lymphedema @5 11
C03 10  X  SPA  @0 Linfedema @5 11
C03 11  X  FRE  @0 Anasarque foetoplacentaire @5 12
C03 11  X  ENG  @0 Hydrops fetalis @5 12
C03 11  X  SPA  @0 Anasarca fetoplacentaria @5 12
C03 12  X  FRE  @0 Anémie @5 13
C03 12  X  ENG  @0 Anemia @5 13
C03 12  X  SPA  @0 Anemia @5 13
C03 13  X  FRE  @0 Appareil circulatoire pathologie @5 14
C03 13  X  ENG  @0 Cardiovascular disease @5 14
C03 13  X  SPA  @0 Aparato circulatorio patología @5 14
C03 14  X  FRE  @0 Mouton @5 54
C03 14  X  ENG  @0 Sheep @5 54
C03 14  X  SPA  @0 Carnero @5 54
C03 15  X  FRE  @0 Modèle animal @5 55
C03 15  X  ENG  @0 Animal model @5 55
C03 15  X  SPA  @0 Modelo animal @5 55
C07 01  X  FRE  @0 Exploration @5 20
C07 01  X  ENG  @0 Exploration @5 20
C07 01  X  SPA  @0 Exploración @5 20
C07 02  X  FRE  @0 Système renin angiotensine @5 23
C07 02  X  ENG  @0 Renin angiotensin system @5 23
C07 02  X  SPA  @0 Sistema renin angiotensina @5 23
C07 03  X  FRE  @0 Appareil urinaire @5 29
C07 03  X  ENG  @0 Urinary system @5 29
C07 03  X  SPA  @0 Aparato urinario @5 29
C07 04  X  FRE  @0 Annexe embryonnaire @5 32
C07 04  X  ENG  @0 Fetal membrane @5 32
C07 04  X  SPA  @0 Membrana fetal @5 32
C07 05  X  FRE  @0 Lymphatique pathologie @5 38
C07 05  X  ENG  @0 Lymphatic vessel disease @5 38
C07 05  X  SPA  @0 Linfático patología @5 38
C07 06  X  FRE  @0 Hémopathie @5 39
C07 06  X  ENG  @0 Hemopathy @5 39
C07 06  X  SPA  @0 Hemopatía @5 39
C07 07  X  FRE  @0 Gestation pathologie @5 45
C07 07  X  ENG  @0 Pregnancy disorders @5 45
C07 07  X  SPA  @0 Gestación patología @5 45
C07 08  X  FRE  @0 Artiodactyla @2 NS
C07 08  X  ENG  @0 Artiodactyla @2 NS
C07 08  X  SPA  @0 Artiodactyla @2 NS
C07 09  X  FRE  @0 Ungulata @2 NS
C07 09  X  ENG  @0 Ungulata @2 NS
C07 09  X  SPA  @0 Ungulata @2 NS
C07 10  X  FRE  @0 Mammalia @2 NS
C07 10  X  ENG  @0 Mammalia @2 NS
C07 10  X  SPA  @0 Mammalia @2 NS
C07 11  X  FRE  @0 Vertebrata @2 NS
C07 11  X  ENG  @0 Vertebrata @2 NS
C07 11  X  SPA  @0 Vertebrata @2 NS
N21       @1 188

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Pascal:97-0330119

Le document en format XML

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<term>Cardiovascular disease</term>
<term>Computer aided analysis</term>
<term>Fetal diseases</term>
<term>Fetus</term>
<term>Hemodynamics</term>
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<term>Kidney</term>
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<term>Sheep</term>
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<term>Hémodynamique</term>
<term>Analyse assistée</term>
<term>Néphrectomie</term>
<term>Foetus pathologie</term>
<term>Lymphoedème</term>
<term>Anasarque foetoplacentaire</term>
<term>Anémie</term>
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<term>Mouton</term>
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<div type="abstract" xml:lang="en">Fetal cardiovascular control is effected by an interaction of the fetal somatic and placental circulations. Three primary regulatory mechanisms are involved: transplacental transfer of extracellular fluid, driven by a difference in hydrostatic and oncotic pressures; modulation of fetal placental and somatic vascular resistances by means of blood pressure controlled production of angiotensin; and somatic autoregulation of flow. A systems analysis incorporates these and other fetal cardiovascular functions and this analysis was modelled for computer simulation. Given physiologically plausible values for known cardiovascular parameters in the fetal sheep, the model reproduced in detail a variety of experimental protocols with known outcomes; these included the normal fetus, the fetus after bilateral nephrectomy, the nephrectomized fetus infused with angiotensin, the intact fetus infused with NaCl solutions, the fetus with lymphatic obstruction and the severely anaemic fetus. The systems analysis demonstrated that fetal cardiac failure constituted the strongest stimulus for the formation of fetal oedema of any tested pathological intervention.</div>
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<s0>Fetal cardiovascular control is effected by an interaction of the fetal somatic and placental circulations. Three primary regulatory mechanisms are involved: transplacental transfer of extracellular fluid, driven by a difference in hydrostatic and oncotic pressures; modulation of fetal placental and somatic vascular resistances by means of blood pressure controlled production of angiotensin; and somatic autoregulation of flow. A systems analysis incorporates these and other fetal cardiovascular functions and this analysis was modelled for computer simulation. Given physiologically plausible values for known cardiovascular parameters in the fetal sheep, the model reproduced in detail a variety of experimental protocols with known outcomes; these included the normal fetus, the fetus after bilateral nephrectomy, the nephrectomized fetus infused with angiotensin, the intact fetus infused with NaCl solutions, the fetus with lymphatic obstruction and the severely anaemic fetus. The systems analysis demonstrated that fetal cardiac failure constituted the strongest stimulus for the formation of fetal oedema of any tested pathological intervention.</s0>
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<s5>02</s5>
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<s5>02</s5>
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<s5>02</s5>
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<s5>03</s5>
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<s5>03</s5>
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<fC03 i1="03" i2="X" l="SPA">
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<s5>03</s5>
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<s5>04</s5>
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<s5>05</s5>
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<s0>Placenta</s0>
<s5>05</s5>
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<s5>05</s5>
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<s5>07</s5>
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<s5>07</s5>
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<s5>07</s5>
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<s5>08</s5>
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<s5>09</s5>
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<s5>09</s5>
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<s5>10</s5>
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<s5>10</s5>
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<s5>11</s5>
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<s5>11</s5>
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<s5>11</s5>
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<s0>Anasarque foetoplacentaire</s0>
<s5>12</s5>
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<s0>Hydrops fetalis</s0>
<s5>12</s5>
</fC03>
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<s0>Anasarca fetoplacentaria</s0>
<s5>12</s5>
</fC03>
<fC03 i1="12" i2="X" l="FRE">
<s0>Anémie</s0>
<s5>13</s5>
</fC03>
<fC03 i1="12" i2="X" l="ENG">
<s0>Anemia</s0>
<s5>13</s5>
</fC03>
<fC03 i1="12" i2="X" l="SPA">
<s0>Anemia</s0>
<s5>13</s5>
</fC03>
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<s0>Appareil circulatoire pathologie</s0>
<s5>14</s5>
</fC03>
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<s0>Cardiovascular disease</s0>
<s5>14</s5>
</fC03>
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<s5>14</s5>
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<fC03 i1="14" i2="X" l="FRE">
<s0>Mouton</s0>
<s5>54</s5>
</fC03>
<fC03 i1="14" i2="X" l="ENG">
<s0>Sheep</s0>
<s5>54</s5>
</fC03>
<fC03 i1="14" i2="X" l="SPA">
<s0>Carnero</s0>
<s5>54</s5>
</fC03>
<fC03 i1="15" i2="X" l="FRE">
<s0>Modèle animal</s0>
<s5>55</s5>
</fC03>
<fC03 i1="15" i2="X" l="ENG">
<s0>Animal model</s0>
<s5>55</s5>
</fC03>
<fC03 i1="15" i2="X" l="SPA">
<s0>Modelo animal</s0>
<s5>55</s5>
</fC03>
<fC07 i1="01" i2="X" l="FRE">
<s0>Exploration</s0>
<s5>20</s5>
</fC07>
<fC07 i1="01" i2="X" l="ENG">
<s0>Exploration</s0>
<s5>20</s5>
</fC07>
<fC07 i1="01" i2="X" l="SPA">
<s0>Exploración</s0>
<s5>20</s5>
</fC07>
<fC07 i1="02" i2="X" l="FRE">
<s0>Système renin angiotensine</s0>
<s5>23</s5>
</fC07>
<fC07 i1="02" i2="X" l="ENG">
<s0>Renin angiotensin system</s0>
<s5>23</s5>
</fC07>
<fC07 i1="02" i2="X" l="SPA">
<s0>Sistema renin angiotensina</s0>
<s5>23</s5>
</fC07>
<fC07 i1="03" i2="X" l="FRE">
<s0>Appareil urinaire</s0>
<s5>29</s5>
</fC07>
<fC07 i1="03" i2="X" l="ENG">
<s0>Urinary system</s0>
<s5>29</s5>
</fC07>
<fC07 i1="03" i2="X" l="SPA">
<s0>Aparato urinario</s0>
<s5>29</s5>
</fC07>
<fC07 i1="04" i2="X" l="FRE">
<s0>Annexe embryonnaire</s0>
<s5>32</s5>
</fC07>
<fC07 i1="04" i2="X" l="ENG">
<s0>Fetal membrane</s0>
<s5>32</s5>
</fC07>
<fC07 i1="04" i2="X" l="SPA">
<s0>Membrana fetal</s0>
<s5>32</s5>
</fC07>
<fC07 i1="05" i2="X" l="FRE">
<s0>Lymphatique pathologie</s0>
<s5>38</s5>
</fC07>
<fC07 i1="05" i2="X" l="ENG">
<s0>Lymphatic vessel disease</s0>
<s5>38</s5>
</fC07>
<fC07 i1="05" i2="X" l="SPA">
<s0>Linfático patología</s0>
<s5>38</s5>
</fC07>
<fC07 i1="06" i2="X" l="FRE">
<s0>Hémopathie</s0>
<s5>39</s5>
</fC07>
<fC07 i1="06" i2="X" l="ENG">
<s0>Hemopathy</s0>
<s5>39</s5>
</fC07>
<fC07 i1="06" i2="X" l="SPA">
<s0>Hemopatía</s0>
<s5>39</s5>
</fC07>
<fC07 i1="07" i2="X" l="FRE">
<s0>Gestation pathologie</s0>
<s5>45</s5>
</fC07>
<fC07 i1="07" i2="X" l="ENG">
<s0>Pregnancy disorders</s0>
<s5>45</s5>
</fC07>
<fC07 i1="07" i2="X" l="SPA">
<s0>Gestación patología</s0>
<s5>45</s5>
</fC07>
<fC07 i1="08" i2="X" l="FRE">
<s0>Artiodactyla</s0>
<s2>NS</s2>
</fC07>
<fC07 i1="08" i2="X" l="ENG">
<s0>Artiodactyla</s0>
<s2>NS</s2>
</fC07>
<fC07 i1="08" i2="X" l="SPA">
<s0>Artiodactyla</s0>
<s2>NS</s2>
</fC07>
<fC07 i1="09" i2="X" l="FRE">
<s0>Ungulata</s0>
<s2>NS</s2>
</fC07>
<fC07 i1="09" i2="X" l="ENG">
<s0>Ungulata</s0>
<s2>NS</s2>
</fC07>
<fC07 i1="09" i2="X" l="SPA">
<s0>Ungulata</s0>
<s2>NS</s2>
</fC07>
<fC07 i1="10" i2="X" l="FRE">
<s0>Mammalia</s0>
<s2>NS</s2>
</fC07>
<fC07 i1="10" i2="X" l="ENG">
<s0>Mammalia</s0>
<s2>NS</s2>
</fC07>
<fC07 i1="10" i2="X" l="SPA">
<s0>Mammalia</s0>
<s2>NS</s2>
</fC07>
<fC07 i1="11" i2="X" l="FRE">
<s0>Vertebrata</s0>
<s2>NS</s2>
</fC07>
<fC07 i1="11" i2="X" l="ENG">
<s0>Vertebrata</s0>
<s2>NS</s2>
</fC07>
<fC07 i1="11" i2="X" l="SPA">
<s0>Vertebrata</s0>
<s2>NS</s2>
</fC07>
<fN21>
<s1>188</s1>
</fN21>
</pA>
</standard>
</inist>
</record>

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   |texte=   Angiotensin mediated interaction of fetal kidney and placenta in the control of fetal arterial pressure and its role in hydrops fetalis
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