Edema after lower limb arterial reconstruction. Influence of background factors, surgical technique and potentially prophylactic methods.
Identifieur interne : 003A04 ( Ncbi/Merge ); précédent : 003A03; suivant : 003A05Edema after lower limb arterial reconstruction. Influence of background factors, surgical technique and potentially prophylactic methods.
Auteurs : N H Persson ; R. Takolander ; D. BergovistSource :
- VASA. Zeitschrift fur Gefasskrankheiten [ 0301-1526 ] ; 1991.
Descripteurs français
- KwdFr :
- MESH :
English descriptors
- KwdEn :
- MESH :
- blood supply : Leg.
- prevention & control : Lymphedema, Postoperative Complications.
- surgery : Arterial Occlusive Diseases, Diabetic Angiopathies, Ischemia.
- transplantation : Saphenous Vein.
- Aged, Blood Vessel Prosthesis, Female, Humans, Male, Risk Factors.
Abstract
The pathogenesis of lower leg edema regularly following a distal arterial reconstruction is unclear and generally no treatment is used. In 85 patients background factors such as diabetes, age, sex and side of operation were analysed and found not to influence the degree of edema. A significantly less pronounced edema was found when a prosthetic graft was used as compared to a vein graft as well as if the distal anastomosis was located above as compared to below the knee. Both these findings might be explained by differences in incisions. In a pilot series the prophylactic effect of various pharmacological regimens was studied and in another 22 patients leg elevation was prescribed. None of the drugs (furosemide, mannitol, terbutaline and corticosteroids) appeared to prevent the development of edema. Within the leg elevation group there was, like in patients without special treatment, less swelling if the distal anastomosis was performed above the knee and if a prosthetic graft was used. In these cases leg elevation seemed to prevent swelling with significantly less edema than corresponding patients treated without leg elevation.
PubMed: 2031402
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pubmed:2031402Le document en format XML
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Influence of background factors, surgical technique and potentially prophylactic methods.</title><author><name sortKey="Persson, N H" sort="Persson, N H" uniqKey="Persson N" first="N H" last="Persson">N H Persson</name><affiliation><nlm:affiliation>Department of Surgery, University of Lund, Malmö General Hospital.</nlm:affiliation><wicri:noCountry code="subField">Malmö General Hospital</wicri:noCountry></affiliation></author><author><name sortKey="Takolander, R" sort="Takolander, R" uniqKey="Takolander R" first="R" last="Takolander">R. Takolander</name></author><author><name sortKey="Bergovist, D" sort="Bergovist, D" uniqKey="Bergovist D" first="D" last="Bergovist">D. Bergovist</name></author></analytic><series><title level="j">VASA. 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In 85 patients background factors such as diabetes, age, sex and side of operation were analysed and found not to influence the degree of edema. A significantly less pronounced edema was found when a prosthetic graft was used as compared to a vein graft as well as if the distal anastomosis was located above as compared to below the knee. Both these findings might be explained by differences in incisions. In a pilot series the prophylactic effect of various pharmacological regimens was studied and in another 22 patients leg elevation was prescribed. None of the drugs (furosemide, mannitol, terbutaline and corticosteroids) appeared to prevent the development of edema. Within the leg elevation group there was, like in patients without special treatment, less swelling if the distal anastomosis was performed above the knee and if a prosthetic graft was used. In these cases leg elevation seemed to prevent swelling with significantly less edema than corresponding patients treated without leg elevation.</div></front></TEI><pubmed><MedlineCitation Status="MEDLINE" Owner="NLM"><PMID Version="1">2031402</PMID><DateCreated><Year>1991</Year><Month>06</Month><Day>19</Day></DateCreated><DateCompleted><Year>1991</Year><Month>06</Month><Day>19</Day></DateCompleted><DateRevised><Year>2012</Year><Month>10</Month><Day>19</Day></DateRevised><Article PubModel="Print"><Journal><ISSN IssnType="Print">0301-1526</ISSN><JournalIssue CitedMedium="Print"><Volume>20</Volume><Issue>1</Issue><PubDate><Year>1991</Year></PubDate></JournalIssue><Title>VASA. Zeitschrift fur Gefasskrankheiten</Title><ISOAbbreviation>VASA</ISOAbbreviation></Journal><ArticleTitle>Edema after lower limb arterial reconstruction. Influence of background factors, surgical technique and potentially prophylactic methods.</ArticleTitle><Pagination><MedlinePgn>57-62</MedlinePgn></Pagination><Abstract><AbstractText>The pathogenesis of lower leg edema regularly following a distal arterial reconstruction is unclear and generally no treatment is used. In 85 patients background factors such as diabetes, age, sex and side of operation were analysed and found not to influence the degree of edema. A significantly less pronounced edema was found when a prosthetic graft was used as compared to a vein graft as well as if the distal anastomosis was located above as compared to below the knee. Both these findings might be explained by differences in incisions. In a pilot series the prophylactic effect of various pharmacological regimens was studied and in another 22 patients leg elevation was prescribed. None of the drugs (furosemide, mannitol, terbutaline and corticosteroids) appeared to prevent the development of edema. Within the leg elevation group there was, like in patients without special treatment, less swelling if the distal anastomosis was performed above the knee and if a prosthetic graft was used. In these cases leg elevation seemed to prevent swelling with significantly less edema than corresponding patients treated without leg elevation.</AbstractText></Abstract><AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Persson</LastName><ForeName>N H</ForeName><Initials>NH</Initials><AffiliationInfo><Affiliation>Department of Surgery, University of Lund, Malmö General Hospital.</Affiliation></AffiliationInfo></Author><Author ValidYN="Y"><LastName>Takolander</LastName><ForeName>R</ForeName><Initials>R</Initials></Author><Author ValidYN="Y"><LastName>Bergovist</LastName><ForeName>D</ForeName><Initials>D</Initials></Author></AuthorList><Language>eng</Language><PublicationTypeList><PublicationType UI="D016428">Journal Article</PublicationType><PublicationType UI="D013485">Research Support, Non-U.S. Gov't</PublicationType></PublicationTypeList></Article><MedlineJournalInfo><Country>Switzerland</Country><MedlineTA>Vasa</MedlineTA><NlmUniqueID>0317051</NlmUniqueID><ISSNLinking>0301-1526</ISSNLinking></MedlineJournalInfo><CitationSubset>IM</CitationSubset><MeshHeadingList><MeshHeading><DescriptorName UI="D000368" MajorTopicYN="N">Aged</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D001157" MajorTopicYN="N">Arterial Occlusive Diseases</DescriptorName><QualifierName UI="Q000601" MajorTopicYN="Y">surgery</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D001807" MajorTopicYN="N">Blood Vessel Prosthesis</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D003925" MajorTopicYN="N">Diabetic Angiopathies</DescriptorName><QualifierName UI="Q000601" MajorTopicYN="N">surgery</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D005260" MajorTopicYN="N">Female</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D006801" MajorTopicYN="N">Humans</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D007511" MajorTopicYN="N">Ischemia</DescriptorName><QualifierName UI="Q000601" MajorTopicYN="Y">surgery</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D007866" MajorTopicYN="N">Leg</DescriptorName><QualifierName UI="Q000098" MajorTopicYN="Y">blood supply</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D008209" MajorTopicYN="N">Lymphedema</DescriptorName><QualifierName UI="Q000517" MajorTopicYN="Y">prevention & control</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D008297" MajorTopicYN="N">Male</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D011183" MajorTopicYN="N">Postoperative Complications</DescriptorName><QualifierName UI="Q000517" MajorTopicYN="Y">prevention & control</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D012307" MajorTopicYN="N">Risk Factors</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D012501" MajorTopicYN="N">Saphenous Vein</DescriptorName><QualifierName UI="Q000637" MajorTopicYN="N">transplantation</QualifierName></MeshHeading></MeshHeadingList></MedlineCitation><PubmedData><History><PubMedPubDate PubStatus="pubmed"><Year>1991</Year><Month>1</Month><Day>1</Day></PubMedPubDate><PubMedPubDate PubStatus="medline"><Year>1991</Year><Month>1</Month><Day>1</Day><Hour>0</Hour><Minute>1</Minute></PubMedPubDate><PubMedPubDate PubStatus="entrez"><Year>1991</Year><Month>1</Month><Day>1</Day><Hour>0</Hour><Minute>0</Minute></PubMedPubDate></History><PublicationStatus>ppublish</PublicationStatus><ArticleIdList><ArticleId IdType="pubmed">2031402</ArticleId></ArticleIdList></PubmedData></pubmed><affiliations><list></list><tree><noCountry><name sortKey="Bergovist, D" sort="Bergovist, D" uniqKey="Bergovist D" first="D" last="Bergovist">D. Bergovist</name><name sortKey="Persson, N H" sort="Persson, N H" uniqKey="Persson N" first="N H" last="Persson">N H Persson</name><name sortKey="Takolander, R" sort="Takolander, R" uniqKey="Takolander R" first="R" last="Takolander">R. Takolander</name></noCountry></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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