Pathogenesis of angiodysplasias.
Identifieur interne : 00A929 ( Ncbi/Curation ); précédent : 00A928; suivant : 00A930Pathogenesis of angiodysplasias.
Auteurs : G. Tasnádi ; M. OsztovicsSource :
- Acta paediatrica Academiae Scientiarum Hungaricae [ 0001-6527 ] ; 1977.
Descripteurs français
- KwdFr :
- MESH :
- génétique : Hémangiome caverneux, Naevus, Varices.
- malformations : Vaisseaux sanguins.
- Angiomatose, Coarctation aortique, Femelle, Humains, Lymphoedème, Maladies intestinales, Mutation, Mâle, Nourrisson, Syndrome de Turner.
English descriptors
- KwdEn :
- MESH :
- abnormalities : Blood Vessels.
- complications : Angiomatosis, Aortic Coarctation, Lymphedema, Turner Syndrome.
- genetics : Hemangioma, Cavernous, Nevus, Varicose Veins.
- Female, Humans, Infant, Intestinal Diseases, Male, Mutation.
Abstract
The pathogenesis of angiodysplasias was studied in 382 patients. Of the primary and secondary morphogenetic factors importance is attached to fetal diseases, intrauterine reparation, hormonal effects in neonatal age and early walking. It is concluded that angiodysplasias develop under the joint effect of genetic and environmental factors. They are evolutional anomalies undergoing changes in the course of life.
PubMed: 613737
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pubmed:613737Le document en format XML
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<profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Angiomatosis (complications)</term>
<term>Aortic Coarctation (complications)</term>
<term>Blood Vessels (abnormalities)</term>
<term>Female</term>
<term>Hemangioma, Cavernous (genetics)</term>
<term>Humans</term>
<term>Infant</term>
<term>Intestinal Diseases</term>
<term>Lymphedema (complications)</term>
<term>Male</term>
<term>Mutation</term>
<term>Nevus (genetics)</term>
<term>Turner Syndrome (complications)</term>
<term>Varicose Veins (genetics)</term>
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<keywords scheme="KwdFr" xml:lang="fr"><term>Angiomatose ()</term>
<term>Coarctation aortique ()</term>
<term>Femelle</term>
<term>Humains</term>
<term>Hémangiome caverneux (génétique)</term>
<term>Lymphoedème ()</term>
<term>Maladies intestinales</term>
<term>Mutation</term>
<term>Mâle</term>
<term>Naevus (génétique)</term>
<term>Nourrisson</term>
<term>Syndrome de Turner ()</term>
<term>Vaisseaux sanguins (malformations)</term>
<term>Varices (génétique)</term>
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<keywords scheme="MESH" qualifier="abnormalities" xml:lang="en"><term>Blood Vessels</term>
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<keywords scheme="MESH" qualifier="complications" xml:lang="en"><term>Angiomatosis</term>
<term>Aortic Coarctation</term>
<term>Lymphedema</term>
<term>Turner Syndrome</term>
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<keywords scheme="MESH" qualifier="genetics" xml:lang="en"><term>Hemangioma, Cavernous</term>
<term>Nevus</term>
<term>Varicose Veins</term>
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<keywords scheme="MESH" qualifier="génétique" xml:lang="fr"><term>Hémangiome caverneux</term>
<term>Naevus</term>
<term>Varices</term>
</keywords>
<keywords scheme="MESH" qualifier="malformations" xml:lang="fr"><term>Vaisseaux sanguins</term>
</keywords>
<keywords scheme="MESH" xml:lang="en"><term>Female</term>
<term>Humans</term>
<term>Infant</term>
<term>Intestinal Diseases</term>
<term>Male</term>
<term>Mutation</term>
</keywords>
<keywords scheme="MESH" xml:lang="fr"><term>Angiomatose</term>
<term>Coarctation aortique</term>
<term>Femelle</term>
<term>Humains</term>
<term>Lymphoedème</term>
<term>Maladies intestinales</term>
<term>Mutation</term>
<term>Mâle</term>
<term>Nourrisson</term>
<term>Syndrome de Turner</term>
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<front><div type="abstract" xml:lang="en">The pathogenesis of angiodysplasias was studied in 382 patients. Of the primary and secondary morphogenetic factors importance is attached to fetal diseases, intrauterine reparation, hormonal effects in neonatal age and early walking. It is concluded that angiodysplasias develop under the joint effect of genetic and environmental factors. They are evolutional anomalies undergoing changes in the course of life.</div>
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