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Decongestion improves cell-mediated immunity in postmastectomy arm lymphoedema: a pilot study.

Identifieur interne : 004F64 ( Ncbi/Curation ); précédent : 004F63; suivant : 004F65

Decongestion improves cell-mediated immunity in postmastectomy arm lymphoedema: a pilot study.

Auteurs : G. Szolnoky [Hongrie] ; A. Dobozy ; L. Kemény

Source :

RBID : pubmed:22817274

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English descriptors

Abstract

Chronic lymphoedematous limbs have an increased propensity for infections and primary or secondary malignant tumours. It has been attributed to suppressed delayed-type hypersensitivity measured in lymphoedemas related to Stewart-Treves syndrome, Kaposi's sarcoma or breast cancer treatment. Cell-mediated immunity is an effective defence mechanism against bacteria, fungi, viruses and tumour cells.

DOI: 10.1111/j.1468-3083.2012.04647.x
PubMed: 22817274

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pubmed:22817274

Le document en format XML

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<nlm:affiliation>Department of Dermatology and Allergology, Szeged University, Szeged, Hungary, and Dermatological Research Group, Hungarian Academy of Sciences, Szeged, Hungary.</nlm:affiliation>
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<wicri:regionArea>Department of Dermatology and Allergology, Szeged University, Szeged, Hungary, and Dermatological Research Group, Hungarian Academy of Sciences, Szeged</wicri:regionArea>
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<term>Adulte d'âge moyen</term>
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<term>Immunité cellulaire</term>
<term>Lymphoedème ()</term>
<term>Lymphoedème (immunologie)</term>
<term>Lymphoedème (étiologie)</term>
<term>Mastectomie (effets indésirables)</term>
<term>Projets pilotes</term>
<term>Période postopératoire</term>
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<term>Mastectomy</term>
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<term>Mastectomie</term>
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<term>Lymphedema</term>
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<term>Lymphoedème</term>
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<term>Lymphoedème</term>
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<div type="abstract" xml:lang="en">Chronic lymphoedematous limbs have an increased propensity for infections and primary or secondary malignant tumours. It has been attributed to suppressed delayed-type hypersensitivity measured in lymphoedemas related to Stewart-Treves syndrome, Kaposi's sarcoma or breast cancer treatment. Cell-mediated immunity is an effective defence mechanism against bacteria, fungi, viruses and tumour cells.</div>
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