Lymphatic endothelial cells, inflammatory lymphangiogenesis, and prospective players.
Identifieur interne : 002947 ( Ncbi/Curation ); précédent : 002946; suivant : 002948Lymphatic endothelial cells, inflammatory lymphangiogenesis, and prospective players.
Auteurs : R C Ji [Japon]Source :
- Current medicinal chemistry [ 0929-8673 ] ; 2007.
Descripteurs français
- KwdFr :
- Animaux, Cellules endothéliales (cytologie), Cellules endothéliales (immunologie), Cellules endothéliales (métabolisme), Chimiokines (métabolisme), Endothélium lymphatique (cytologie), Endothélium lymphatique (métabolisme), Humains, Inflammation (physiopathologie), Lymphangiogenèse, Vaisseaux lymphatiques (cytologie), Vaisseaux lymphatiques (immunologie), Vaisseaux lymphatiques (métabolisme).
- MESH :
- cytologie : Cellules endothéliales, Endothélium lymphatique, Vaisseaux lymphatiques.
- immunologie : Cellules endothéliales, Vaisseaux lymphatiques.
- métabolisme : Cellules endothéliales, Chimiokines, Endothélium lymphatique, Vaisseaux lymphatiques.
- physiopathologie : Inflammation.
- Animaux, Humains, Lymphangiogenèse.
English descriptors
- KwdEn :
- Animals, Chemokines (metabolism), Endothelial Cells (cytology), Endothelial Cells (immunology), Endothelial Cells (metabolism), Endothelium, Lymphatic (cytology), Endothelium, Lymphatic (metabolism), Humans, Inflammation (physiopathology), Lymphangiogenesis, Lymphatic Vessels (cytology), Lymphatic Vessels (immunology), Lymphatic Vessels (metabolism).
- MESH :
- chemical , metabolism : Chemokines.
- cytology : Endothelial Cells, Endothelium, Lymphatic, Lymphatic Vessels.
- immunology : Endothelial Cells, Lymphatic Vessels.
- metabolism : Endothelial Cells, Endothelium, Lymphatic, Lymphatic Vessels.
- physiopathology : Inflammation.
- Animals, Humans, Lymphangiogenesis.
Abstract
De novo lymphangiogenesis influences different pathological courses via modulating tissue fluid homeostasis, macromolecule absorption, and leukocyte transmigration. During the past decade, improved understanding of lymphatic biology, especially VEGF-C/-D/VEGFR-3-mediating lymphangiogenesis has substantially promoted clinical research in lymphatic insufficiency. The role of lymphangiogenesis in the setting of the inflammatory processes observed in transplants, inflamed cornea, wound healing, acquired lymphedema and tumor invasion, however, remains to be elucidated. The chemokine family of peptide chemoattractants and other mediators, e.g., CCL21-CCR7, D6, NF-kappaB and TNF-alpha may be important contributors to pathophysiological changes of lymphatic endothelial cells (LECs) and inflammatory lymphangiogenesis. Dendritic cells and macrophages may also involve in LEC proliferation and differentiation. Increased knowledge of LEC-specific modulators in inflammatory microenvironment is vital for prevention and treatment of lymphatic-associated diseases.
PubMed: 17896984
Links toward previous steps (curation, corpus...)
- to stream PubMed, to step Corpus: Pour aller vers cette notice dans l'étape Curation :003512
- to stream PubMed, to step Curation: Pour aller vers cette notice dans l'étape Curation :003512
- to stream PubMed, to step Checkpoint: Pour aller vers cette notice dans l'étape Curation :003512
- to stream Ncbi, to step Merge: Pour aller vers cette notice dans l'étape Curation :002947
Links to Exploration step
pubmed:17896984Le document en format XML
<record><TEI><teiHeader><fileDesc><titleStmt><title xml:lang="en">Lymphatic endothelial cells, inflammatory lymphangiogenesis, and prospective players.</title>
<author><name sortKey="Ji, R C" sort="Ji, R C" uniqKey="Ji R" first="R C" last="Ji">R C Ji</name>
<affiliation wicri:level="1"><nlm:affiliation>Department of Anatomy, Biology and Medicine, Oita University Faculty of Medicine, Oita 879-5593, Japan. JI@med.oita-u.ac.jp</nlm:affiliation>
<country xml:lang="fr">Japon</country>
<wicri:regionArea>Department of Anatomy, Biology and Medicine, Oita University Faculty of Medicine, Oita 879-5593</wicri:regionArea>
<wicri:noRegion>Oita 879-5593</wicri:noRegion>
</affiliation>
</author>
</titleStmt>
<publicationStmt><idno type="wicri:source">PubMed</idno>
<date when="2007">2007</date>
<idno type="RBID">pubmed:17896984</idno>
<idno type="pmid">17896984</idno>
<idno type="wicri:Area/PubMed/Corpus">003512</idno>
<idno type="wicri:explorRef" wicri:stream="PubMed" wicri:step="Corpus" wicri:corpus="PubMed">003512</idno>
<idno type="wicri:Area/PubMed/Curation">003512</idno>
<idno type="wicri:explorRef" wicri:stream="PubMed" wicri:step="Curation">003512</idno>
<idno type="wicri:Area/PubMed/Checkpoint">003512</idno>
<idno type="wicri:explorRef" wicri:stream="Checkpoint" wicri:step="PubMed">003512</idno>
<idno type="wicri:Area/Ncbi/Merge">002947</idno>
<idno type="wicri:Area/Ncbi/Curation">002947</idno>
</publicationStmt>
<sourceDesc><biblStruct><analytic><title xml:lang="en">Lymphatic endothelial cells, inflammatory lymphangiogenesis, and prospective players.</title>
<author><name sortKey="Ji, R C" sort="Ji, R C" uniqKey="Ji R" first="R C" last="Ji">R C Ji</name>
<affiliation wicri:level="1"><nlm:affiliation>Department of Anatomy, Biology and Medicine, Oita University Faculty of Medicine, Oita 879-5593, Japan. JI@med.oita-u.ac.jp</nlm:affiliation>
<country xml:lang="fr">Japon</country>
<wicri:regionArea>Department of Anatomy, Biology and Medicine, Oita University Faculty of Medicine, Oita 879-5593</wicri:regionArea>
<wicri:noRegion>Oita 879-5593</wicri:noRegion>
</affiliation>
</author>
</analytic>
<series><title level="j">Current medicinal chemistry</title>
<idno type="ISSN">0929-8673</idno>
<imprint><date when="2007" type="published">2007</date>
</imprint>
</series>
</biblStruct>
</sourceDesc>
</fileDesc>
<profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Animals</term>
<term>Chemokines (metabolism)</term>
<term>Endothelial Cells (cytology)</term>
<term>Endothelial Cells (immunology)</term>
<term>Endothelial Cells (metabolism)</term>
<term>Endothelium, Lymphatic (cytology)</term>
<term>Endothelium, Lymphatic (metabolism)</term>
<term>Humans</term>
<term>Inflammation (physiopathology)</term>
<term>Lymphangiogenesis</term>
<term>Lymphatic Vessels (cytology)</term>
<term>Lymphatic Vessels (immunology)</term>
<term>Lymphatic Vessels (metabolism)</term>
</keywords>
<keywords scheme="KwdFr" xml:lang="fr"><term>Animaux</term>
<term>Cellules endothéliales (cytologie)</term>
<term>Cellules endothéliales (immunologie)</term>
<term>Cellules endothéliales (métabolisme)</term>
<term>Chimiokines (métabolisme)</term>
<term>Endothélium lymphatique (cytologie)</term>
<term>Endothélium lymphatique (métabolisme)</term>
<term>Humains</term>
<term>Inflammation (physiopathologie)</term>
<term>Lymphangiogenèse</term>
<term>Vaisseaux lymphatiques (cytologie)</term>
<term>Vaisseaux lymphatiques (immunologie)</term>
<term>Vaisseaux lymphatiques (métabolisme)</term>
</keywords>
<keywords scheme="MESH" type="chemical" qualifier="metabolism" xml:lang="en"><term>Chemokines</term>
</keywords>
<keywords scheme="MESH" qualifier="cytologie" xml:lang="fr"><term>Cellules endothéliales</term>
<term>Endothélium lymphatique</term>
<term>Vaisseaux lymphatiques</term>
</keywords>
<keywords scheme="MESH" qualifier="cytology" xml:lang="en"><term>Endothelial Cells</term>
<term>Endothelium, Lymphatic</term>
<term>Lymphatic Vessels</term>
</keywords>
<keywords scheme="MESH" qualifier="immunologie" xml:lang="fr"><term>Cellules endothéliales</term>
<term>Vaisseaux lymphatiques</term>
</keywords>
<keywords scheme="MESH" qualifier="immunology" xml:lang="en"><term>Endothelial Cells</term>
<term>Lymphatic Vessels</term>
</keywords>
<keywords scheme="MESH" qualifier="metabolism" xml:lang="en"><term>Endothelial Cells</term>
<term>Endothelium, Lymphatic</term>
<term>Lymphatic Vessels</term>
</keywords>
<keywords scheme="MESH" qualifier="métabolisme" xml:lang="fr"><term>Cellules endothéliales</term>
<term>Chimiokines</term>
<term>Endothélium lymphatique</term>
<term>Vaisseaux lymphatiques</term>
</keywords>
<keywords scheme="MESH" qualifier="physiopathologie" xml:lang="fr"><term>Inflammation</term>
</keywords>
<keywords scheme="MESH" qualifier="physiopathology" xml:lang="en"><term>Inflammation</term>
</keywords>
<keywords scheme="MESH" xml:lang="en"><term>Animals</term>
<term>Humans</term>
<term>Lymphangiogenesis</term>
</keywords>
<keywords scheme="MESH" xml:lang="fr"><term>Animaux</term>
<term>Humains</term>
<term>Lymphangiogenèse</term>
</keywords>
</textClass>
</profileDesc>
</teiHeader>
<front><div type="abstract" xml:lang="en">De novo lymphangiogenesis influences different pathological courses via modulating tissue fluid homeostasis, macromolecule absorption, and leukocyte transmigration. During the past decade, improved understanding of lymphatic biology, especially VEGF-C/-D/VEGFR-3-mediating lymphangiogenesis has substantially promoted clinical research in lymphatic insufficiency. The role of lymphangiogenesis in the setting of the inflammatory processes observed in transplants, inflamed cornea, wound healing, acquired lymphedema and tumor invasion, however, remains to be elucidated. The chemokine family of peptide chemoattractants and other mediators, e.g., CCL21-CCR7, D6, NF-kappaB and TNF-alpha may be important contributors to pathophysiological changes of lymphatic endothelial cells (LECs) and inflammatory lymphangiogenesis. Dendritic cells and macrophages may also involve in LEC proliferation and differentiation. Increased knowledge of LEC-specific modulators in inflammatory microenvironment is vital for prevention and treatment of lymphatic-associated diseases.</div>
</front>
</TEI>
</record>
Pour manipuler ce document sous Unix (Dilib)
EXPLOR_STEP=$WICRI_ROOT/Wicri/Sante/explor/LymphedemaV1/Data/Ncbi/Curation
HfdSelect -h $EXPLOR_STEP/biblio.hfd -nk 002947 | SxmlIndent | more
Ou
HfdSelect -h $EXPLOR_AREA/Data/Ncbi/Curation/biblio.hfd -nk 002947 | SxmlIndent | more
Pour mettre un lien sur cette page dans le réseau Wicri
{{Explor lien |wiki= Wicri/Sante |area= LymphedemaV1 |flux= Ncbi |étape= Curation |type= RBID |clé= pubmed:17896984 |texte= Lymphatic endothelial cells, inflammatory lymphangiogenesis, and prospective players. }}
Pour générer des pages wiki
HfdIndexSelect -h $EXPLOR_AREA/Data/Ncbi/Curation/RBID.i -Sk "pubmed:17896984" \ | HfdSelect -Kh $EXPLOR_AREA/Data/Ncbi/Curation/biblio.hfd \ | NlmPubMed2Wicri -a LymphedemaV1
This area was generated with Dilib version V0.6.31. |