MYC amplification in angiosarcomas arising in the setting of chronic lymphedema of morbid obesity.
Identifieur interne : 008854 ( Ncbi/Checkpoint ); précédent : 008853; suivant : 008855MYC amplification in angiosarcomas arising in the setting of chronic lymphedema of morbid obesity.
Auteurs : David Harker [États-Unis] ; Michael Jennings [États-Unis] ; Patrick Mcdonough [États-Unis] ; Melissa Mauskar [États-Unis] ; Stephanie Savory [États-Unis] ; Gregory A. Hosler [États-Unis] ; Travis Vandergriff [États-Unis]Source :
- Journal of cutaneous pathology [ 1600-0560 ] ; 2017.
Descripteurs français
- KwdFr :
- MESH :
English descriptors
- KwdEn :
- Chronic Disease, Female, Gene Amplification, Hemangiosarcoma (complications), Hemangiosarcoma (genetics), Humans, In Situ Hybridization, Fluorescence, Lymphedema (etiology), Middle Aged, Obesity, Morbid (complications), Proto-Oncogene Proteins c-myc (genetics), Skin Neoplasms (complications), Skin Neoplasms (genetics).
- MESH :
- chemical , genetics : Proto-Oncogene Proteins c-myc.
- complications : Hemangiosarcoma, Obesity, Morbid, Skin Neoplasms.
- etiology : Lymphedema.
- genetics : Hemangiosarcoma, Skin Neoplasms.
- Chronic Disease, Female, Gene Amplification, Humans, In Situ Hybridization, Fluorescence, Middle Aged.
Abstract
Angiosarcoma is a malignancy of vascular endothelial cells which may arise secondarily as a complication of lymphedema, including chronic lymphedema of morbid obesity. Amplifications in MYC are frequently present in secondary angiosarcoma (arising in irradiated sites and chronic lymphedema) and less frequently in primary cutaneous angiosarcoma.
DOI: 10.1111/cup.12827
PubMed: 27686553
Affiliations:
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pubmed:27686553Le document en format XML
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<term>Gene Amplification</term>
<term>Hemangiosarcoma (complications)</term>
<term>Hemangiosarcoma (genetics)</term>
<term>Humans</term>
<term>In Situ Hybridization, Fluorescence</term>
<term>Lymphedema (etiology)</term>
<term>Middle Aged</term>
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<term>Amplification de gène</term>
<term>Femelle</term>
<term>Humains</term>
<term>Hémangiosarcome ()</term>
<term>Hémangiosarcome (génétique)</term>
<term>Lymphoedème (étiologie)</term>
<term>Maladie chronique</term>
<term>Obésité morbide ()</term>
<term>Protéines proto-oncogènes c-myc (génétique)</term>
<term>Technique FISH</term>
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<term>Tumeurs cutanées (génétique)</term>
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<keywords scheme="MESH" qualifier="genetics" xml:lang="en"><term>Hemangiosarcoma</term>
<term>Skin Neoplasms</term>
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<term>Protéines proto-oncogènes c-myc</term>
<term>Tumeurs cutanées</term>
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<term>Female</term>
<term>Gene Amplification</term>
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<term>In Situ Hybridization, Fluorescence</term>
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<front><div type="abstract" xml:lang="en">Angiosarcoma is a malignancy of vascular endothelial cells which may arise secondarily as a complication of lymphedema, including chronic lymphedema of morbid obesity. Amplifications in MYC are frequently present in secondary angiosarcoma (arising in irradiated sites and chronic lymphedema) and less frequently in primary cutaneous angiosarcoma.</div>
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<region><li>Texas</li>
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<tree><country name="États-Unis"><region name="Texas"><name sortKey="Harker, David" sort="Harker, David" uniqKey="Harker D" first="David" last="Harker">David Harker</name>
</region>
<name sortKey="Hosler, Gregory A" sort="Hosler, Gregory A" uniqKey="Hosler G" first="Gregory A" last="Hosler">Gregory A. Hosler</name>
<name sortKey="Jennings, Michael" sort="Jennings, Michael" uniqKey="Jennings M" first="Michael" last="Jennings">Michael Jennings</name>
<name sortKey="Mauskar, Melissa" sort="Mauskar, Melissa" uniqKey="Mauskar M" first="Melissa" last="Mauskar">Melissa Mauskar</name>
<name sortKey="Mcdonough, Patrick" sort="Mcdonough, Patrick" uniqKey="Mcdonough P" first="Patrick" last="Mcdonough">Patrick Mcdonough</name>
<name sortKey="Savory, Stephanie" sort="Savory, Stephanie" uniqKey="Savory S" first="Stephanie" last="Savory">Stephanie Savory</name>
<name sortKey="Vandergriff, Travis" sort="Vandergriff, Travis" uniqKey="Vandergriff T" first="Travis" last="Vandergriff">Travis Vandergriff</name>
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