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Pleuroperitoneal shunts in the management of neonatal chylothorax

Identifieur interne : 004F21 ( Istex/Curation ); précédent : 004F20; suivant : 004F22

Pleuroperitoneal shunts in the management of neonatal chylothorax

Auteurs : Richard G. Azizkhan [États-Unis] ; James Canfield [États-Unis] ; Bennett A. Alford [États-Unis] ; Bradley M. Rodgers [États-Unis]

Source :

RBID : ISTEX:A8A11634C688CE02BEB0FD01DD5C353B381AA4CA

English descriptors

Abstract

Pleuroperitoneal shunts have been placed in five ventilator-dependent newborns with persistent chylothorax. The etiology of the chylothorax appeared to be secondary to superior vena caval obstruction in three patients and was idiopathic in the remaining two. Despite traditional therapies these infants were on a progressively deteriorating clinical course. Hakim-Cordis low-pressure ventricular-peritoneal shunt catheter systems were used in each infant. Ultrasonography was used to follow the regression of pleural effusions and to determine the need for shunt compression. Shunt patency was confirmed with radionuclide studies. Four of five infants had a complete resolution of their chylothorax and pulmonary insufficiency. Three of these infants were extubated within 28 days following the placement of the shunt. Nutritional and metabolic stability was rapidly achieved. The shunts were removed several weeks later without recurrence of the chylothorax. A fifth infant failed to improve after the placement of the pleuroperitoneal shunt and died of progressive pulmonary insufficiency. The placement of pleuroperitoneal shunts in infants with refractory chylothroax is safe, technically easy to perform, and is associated with few complications.

Url:
DOI: 10.1016/S0022-3468(83)80034-7

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ISTEX:A8A11634C688CE02BEB0FD01DD5C353B381AA4CA

Le document en format XML

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<div type="abstract" xml:lang="en">Pleuroperitoneal shunts have been placed in five ventilator-dependent newborns with persistent chylothorax. The etiology of the chylothorax appeared to be secondary to superior vena caval obstruction in three patients and was idiopathic in the remaining two. Despite traditional therapies these infants were on a progressively deteriorating clinical course. Hakim-Cordis low-pressure ventricular-peritoneal shunt catheter systems were used in each infant. Ultrasonography was used to follow the regression of pleural effusions and to determine the need for shunt compression. Shunt patency was confirmed with radionuclide studies. Four of five infants had a complete resolution of their chylothorax and pulmonary insufficiency. Three of these infants were extubated within 28 days following the placement of the shunt. Nutritional and metabolic stability was rapidly achieved. The shunts were removed several weeks later without recurrence of the chylothorax. A fifth infant failed to improve after the placement of the pleuroperitoneal shunt and died of progressive pulmonary insufficiency. The placement of pleuroperitoneal shunts in infants with refractory chylothroax is safe, technically easy to perform, and is associated with few complications.</div>
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