Primärer Achselvenenstau (Paget-von Schroetter-Syndrom)
Identifieur interne : 004320 ( Istex/Corpus ); précédent : 004319; suivant : 004321Primärer Achselvenenstau (Paget-von Schroetter-Syndrom)
Auteurs : H. R. Hölzl ; G. Flora ; M. MarbergerSource :
- Langenbecks Archiv für Chirurgie [ 0023-8236 ] ; 1972-12-01.
Abstract
Summary: The primary stop of backflow of the axillary vein marked by swelling, pains, cyanosis and increasing superficial vein-prominence of the arm and of the neighboured shoulder-chest region. This pathological symptom has been wellknown under many synonyma since a 100 years, mostly induced by thrombotic closing of the axillary vein, as a result of irritation on locus minoris resistentiae (Costoclavicular region). The etiology is manyfold. Therefore in literature this syndrom is not always strictly separable from many other illnesses with the same or similar clinical symptoms (secundary disturbance of the backflow of the axillary vein, lymphedema). In the period from 1961 to 1971 twenty cases of a primary idiopathic stop of backflow of the axillary veins were observed at our Surgical University Hospital in Innsbruck. The conservative therapy outweighs by far the surgical one (relation 19:1). Two patients were treated with conservative means only; with fourteen patients the conservative therapy was enlarged by the usage of anticoagulants. Fibrinolytic therapy was tried on three patients but it did not produce a remarkable phlebographical drainage. After a foudroyantic development in one case, an operation (thrombectomy) was necessary in spite of a combined streptocinaseheparintherapy. Nine patients were painless out of a group of nineteen controlled ones, eight showed a swelling at work and other efforts, after an observation-period from 3 months to 10 years. One patient had pains, another one had pains and was swollen when making struggles. Relying on our own experiences and on informations scientific works, a treatment schedule of the primary stop of backflow of the axillary vein is to be discussed.
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DOI: 10.1007/BF01239174
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ISTEX:8E8270B6B662E918D493B303E0BB1ED46D7A051BLe document en format XML
<record><TEI wicri:istexFullTextTei="biblStruct"><teiHeader><fileDesc><titleStmt><title xml:lang="de">Primärer Achselvenenstau (Paget-von Schroetter-Syndrom)</title><author><name sortKey="Holzl, H R" sort="Holzl, H R" uniqKey="Holzl H" first="H. R." last="Hölzl">H. R. Hölzl</name><affiliation><mods:affiliation>Chirurg. Univ.-Klinik, Anichstraße 35, A-6020, Innsbruck, Österreich</mods:affiliation></affiliation></author><author><name sortKey="Flora, G" sort="Flora, G" uniqKey="Flora G" first="G." last="Flora">G. Flora</name><affiliation><mods:affiliation>Chirurg. Univ.-Klinik, Anichstraße 35, A-6020, Innsbruck, Österreich</mods:affiliation></affiliation></author><author><name sortKey="Marberger, M" sort="Marberger, M" uniqKey="Marberger M" first="M." last="Marberger">M. Marberger</name><affiliation><mods:affiliation>Chirurg. 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Univ.-Klinik, Anichstraße 35, A-6020, Innsbruck, Österreich</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j">Langenbecks Archiv für Chirurgie</title><title level="j" type="abbrev">Langenbecks Arch Chiv</title><idno type="ISSN">0023-8236</idno><idno type="eISSN">1435-2451</idno><imprint><publisher>Springer-Verlag</publisher><pubPlace>Berlin/Heidelberg</pubPlace><date type="published" when="1972-12-01">1972-12-01</date><biblScope unit="volume">331</biblScope><biblScope unit="issue">4</biblScope><biblScope unit="page" from="295">295</biblScope><biblScope unit="page" to="314">314</biblScope></imprint><idno type="ISSN">0023-8236</idno></series></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0023-8236</idno></seriesStmt></fileDesc><profileDesc><textClass></textClass><langUsage><language ident="de">de</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Summary: The primary stop of backflow of the axillary vein marked by swelling, pains, cyanosis and increasing superficial vein-prominence of the arm and of the neighboured shoulder-chest region. This pathological symptom has been wellknown under many synonyma since a 100 years, mostly induced by thrombotic closing of the axillary vein, as a result of irritation on locus minoris resistentiae (Costoclavicular region). The etiology is manyfold. Therefore in literature this syndrom is not always strictly separable from many other illnesses with the same or similar clinical symptoms (secundary disturbance of the backflow of the axillary vein, lymphedema). In the period from 1961 to 1971 twenty cases of a primary idiopathic stop of backflow of the axillary veins were observed at our Surgical University Hospital in Innsbruck. The conservative therapy outweighs by far the surgical one (relation 19:1). Two patients were treated with conservative means only; with fourteen patients the conservative therapy was enlarged by the usage of anticoagulants. Fibrinolytic therapy was tried on three patients but it did not produce a remarkable phlebographical drainage. After a foudroyantic development in one case, an operation (thrombectomy) was necessary in spite of a combined streptocinaseheparintherapy. Nine patients were painless out of a group of nineteen controlled ones, eight showed a swelling at work and other efforts, after an observation-period from 3 months to 10 years. One patient had pains, another one had pains and was swollen when making struggles. Relying on our own experiences and on informations scientific works, a treatment schedule of the primary stop of backflow of the axillary vein is to be discussed.</div></front></TEI><istex><corpusName>springer</corpusName><author><json:item><name>Dr. H. R. Hölzl</name><affiliations><json:string>Chirurg. Univ.-Klinik, Anichstraße 35, A-6020, Innsbruck, Österreich</json:string></affiliations></json:item><json:item><name>Doz. Dr. G. Flora</name><affiliations><json:string>Chirurg. Univ.-Klinik, Anichstraße 35, A-6020, Innsbruck, Österreich</json:string></affiliations></json:item><json:item><name>Dr. M. Marberger</name><affiliations><json:string>Chirurg. Univ.-Klinik, Anichstraße 35, A-6020, Innsbruck, Österreich</json:string></affiliations></json:item></author><articleId><json:string>BF01239174</json:string><json:string>Art3</json:string></articleId><language><json:string>ger</json:string></language><originalGenre><json:string>OriginalPaper</json:string></originalGenre><abstract>Summary: The primary stop of backflow of the axillary vein marked by swelling, pains, cyanosis and increasing superficial vein-prominence of the arm and of the neighboured shoulder-chest region. This pathological symptom has been wellknown under many synonyma since a 100 years, mostly induced by thrombotic closing of the axillary vein, as a result of irritation on locus minoris resistentiae (Costoclavicular region). The etiology is manyfold. Therefore in literature this syndrom is not always strictly separable from many other illnesses with the same or similar clinical symptoms (secundary disturbance of the backflow of the axillary vein, lymphedema). In the period from 1961 to 1971 twenty cases of a primary idiopathic stop of backflow of the axillary veins were observed at our Surgical University Hospital in Innsbruck. The conservative therapy outweighs by far the surgical one (relation 19:1). Two patients were treated with conservative means only; with fourteen patients the conservative therapy was enlarged by the usage of anticoagulants. Fibrinolytic therapy was tried on three patients but it did not produce a remarkable phlebographical drainage. After a foudroyantic development in one case, an operation (thrombectomy) was necessary in spite of a combined streptocinaseheparintherapy. Nine patients were painless out of a group of nineteen controlled ones, eight showed a swelling at work and other efforts, after an observation-period from 3 months to 10 years. One patient had pains, another one had pains and was swollen when making struggles. 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Univ.-Klinik, Anichstraße 35, A-6020, Innsbruck, Österreich</affiliation></author><author xml:id="author-0001"><persName><forename type="first">G.</forename><surname>Flora</surname></persName><roleName type="degree">Doz. Dr.</roleName><affiliation>Chirurg. Univ.-Klinik, Anichstraße 35, A-6020, Innsbruck, Österreich</affiliation></author><author xml:id="author-0002"><persName><forename type="first">M.</forename><surname>Marberger</surname></persName><roleName type="degree">Dr.</roleName><affiliation>Chirurg. Univ.-Klinik, Anichstraße 35, A-6020, Innsbruck, Österreich</affiliation></author><idno type="istex">8E8270B6B662E918D493B303E0BB1ED46D7A051B</idno><idno type="DOI">10.1007/BF01239174</idno><idno type="ArticleID">BF01239174</idno><idno type="ArticleID">Art3</idno></analytic><monogr><title level="j">Langenbecks Archiv für Chirurgie</title><title level="j" type="abbrev">Langenbecks Arch Chiv</title><idno type="pISSN">0023-8236</idno><idno type="eISSN">1435-2451</idno><idno type="journal-ID">true</idno><idno type="issue-article-count">8</idno><idno type="volume-issue-count">4</idno><imprint><publisher>Springer-Verlag</publisher><pubPlace>Berlin/Heidelberg</pubPlace><date type="published" when="1972-12-01"></date><biblScope unit="volume">331</biblScope><biblScope unit="issue">4</biblScope><biblScope unit="page" from="295">295</biblScope><biblScope unit="page" to="314">314</biblScope></imprint></monogr></biblStruct></sourceDesc></fileDesc><profileDesc><creation><date>1972-03-28</date></creation><langUsage><language ident="de">de</language></langUsage><abstract xml:lang="en"><p>Summary: The primary stop of backflow of the axillary vein marked by swelling, pains, cyanosis and increasing superficial vein-prominence of the arm and of the neighboured shoulder-chest region. This pathological symptom has been wellknown under many synonyma since a 100 years, mostly induced by thrombotic closing of the axillary vein, as a result of irritation on locus minoris resistentiae (Costoclavicular region). The etiology is manyfold. Therefore in literature this syndrom is not always strictly separable from many other illnesses with the same or similar clinical symptoms (secundary disturbance of the backflow of the axillary vein, lymphedema). In the period from 1961 to 1971 twenty cases of a primary idiopathic stop of backflow of the axillary veins were observed at our Surgical University Hospital in Innsbruck. The conservative therapy outweighs by far the surgical one (relation 19:1). Two patients were treated with conservative means only; with fourteen patients the conservative therapy was enlarged by the usage of anticoagulants. Fibrinolytic therapy was tried on three patients but it did not produce a remarkable phlebographical drainage. After a foudroyantic development in one case, an operation (thrombectomy) was necessary in spite of a combined streptocinaseheparintherapy. Nine patients were painless out of a group of nineteen controlled ones, eight showed a swelling at work and other efforts, after an observation-period from 3 months to 10 years. One patient had pains, another one had pains and was swollen when making struggles. Relying on our own experiences and on informations scientific works, a treatment schedule of the primary stop of backflow of the axillary vein is to be discussed.</p></abstract><abstract xml:lang="de"><p>Zusammenfassung: Der primäre Achselvenenstau ist im wesentlichen gekennzeichnet durch Schwellung, Schmerzen, Cyanose und vermehrte oberflächliche Venenzeichnung eines Armes und der zugehörigen Schulter-Brustregion. Seit etwa 100 Jahren ist dieses Krankheitsbild unter vielen Synonyma bekannt und ist in der Mehrzahl der Fälle durch thrombotischen Verschluß der Achselvene als Irritationsfolge am Locus minoris resistentiae (Costoclavicularraum) bedingt. Seine Ätiologie ist vielgestaltig, so daß es von vielen anderen Erkrankungen mit gleichen oder ähnlichen klinischen Erscheinungsformen (sekundärer Achselvenenstau, Lymphödem) in der Literatur nicht immer streng getrennt wird. Im Zeitraum von 1961 bis 1971 wurden an der Chirurgischen Universitätsklinik Innsbruck 20 Fälle eines primären idiopathischen Achselvenenstaus beobachtet. Die konservative Therapie überwiegt bei weitem die chirurgische (19:1). 2 Patienten wurden nur konservativ behandelt; bei 14 Patienten wurde die konservative Therapie durch Anwendung von Anticoagulantien erweitert. Eine Fibrinolysetherapie wurde an 3 Patienten versucht, brachte jedoch keine phlebographisch nachweisbare Rekanalisation. Nach foudroyantem Verlauf eines Falles trotz einer kombinierten Streptokinase-Reparintherapie war ein operatives Vorgehen (Thrombektomie) notwendig. Nach einem Beobachtungszeitraum von 3 Monaten bis 10 Jahre waren von 19 nachuntersuchten Patienten 9 beschwerdefrei, 8 zeigten Schwellung bei Anstrengung. Bei einem Patienten bestehen Schmerzen bei Anstrengung, bei einem weiteren Schmerzen bei Anstrengung sowie Schwellung. Gestützt auf eigene Erfahrungen und Mitteilungen in der Literatur wird ein Behandlungsschema des primären Achselvenenstaus zur Diskussion gestellt.</p></abstract><textClass><keywords scheme="Journal Subject"><list><head>Medicine & Public Health</head><item><term>Abdominal Surgery</term></item><item><term>Cardiac Surgery</term></item><item><term>General Surgery</term></item><item><term>Thoracic Surgery</term></item><item><term>Traumatic Surgery</term></item><item><term>Vascular Surgery</term></item></list></keywords></textClass></profileDesc><revisionDesc><change when="1972-03-28">Created</change><change when="1972-12-01">Published</change></revisionDesc></teiHeader></istex:fulltextTEI><json:item><extension>txt</extension><original>false</original><mimetype>text/plain</mimetype><uri>https://api.istex.fr/document/8E8270B6B662E918D493B303E0BB1ED46D7A051B/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Springer, Publisher found" wicri:toSee="no header"><istex:xmlDeclaration>version="1.0" encoding="UTF-8"</istex:xmlDeclaration><istex:docType PUBLIC="-//Springer-Verlag//DTD A++ V2.4//EN" URI="http://devel.springer.de/A++/V2.4/DTD/A++V2.4.dtd" name="istex:docType"></istex:docType><istex:document><Publisher><PublisherInfo><PublisherName>Springer-Verlag</PublisherName><PublisherLocation>Berlin/Heidelberg</PublisherLocation></PublisherInfo><Journal><JournalInfo JournalProductType="ArchiveJournal" NumberingStyle="Unnumbered"><JournalID>423</JournalID><JournalPrintISSN>0023-8236</JournalPrintISSN><JournalElectronicISSN>1435-2451</JournalElectronicISSN><JournalTitle>Langenbecks Archiv für Chirurgie</JournalTitle><JournalAbbreviatedTitle>Langenbecks Arch Chiv</JournalAbbreviatedTitle><JournalSubjectGroup><JournalSubject Type="Primary">Medicine & Public Health</JournalSubject><JournalSubject Type="Secondary">Abdominal Surgery</JournalSubject><JournalSubject Type="Secondary">Cardiac Surgery</JournalSubject><JournalSubject Type="Secondary">General Surgery</JournalSubject><JournalSubject Type="Secondary">Thoracic Surgery</JournalSubject><JournalSubject Type="Secondary">Traumatic Surgery</JournalSubject><JournalSubject Type="Secondary">Vascular Surgery</JournalSubject></JournalSubjectGroup></JournalInfo><Volume><VolumeInfo VolumeType="Regular" TocLevels="0"><VolumeIDStart>331</VolumeIDStart><VolumeIDEnd>331</VolumeIDEnd><VolumeIssueCount>4</VolumeIssueCount></VolumeInfo><Issue IssueType="Regular"><IssueInfo TocLevels="0"><IssueIDStart>4</IssueIDStart><IssueIDEnd>4</IssueIDEnd><IssueArticleCount>8</IssueArticleCount><IssueHistory><CoverDate><DateString>1972</DateString><Year>1972</Year><Month>12</Month></CoverDate></IssueHistory><IssueCopyright><CopyrightHolderName>Springer-Verlag</CopyrightHolderName><CopyrightYear>1972</CopyrightYear></IssueCopyright></IssueInfo><Article ID="Art3"><ArticleInfo Language="De" ArticleType="OriginalPaper" NumberingStyle="Unnumbered" TocLevels="0" ContainsESM="No"><ArticleID>BF01239174</ArticleID><ArticleDOI>10.1007/BF01239174</ArticleDOI><ArticleSequenceNumber>3</ArticleSequenceNumber><ArticleTitle Language="De">Primärer Achselvenenstau (Paget-von Schroetter-Syndrom)</ArticleTitle><ArticleTitle Language="En">Primary thrombosis of the axillary vein</ArticleTitle><ArticleFirstPage>295</ArticleFirstPage><ArticleLastPage>314</ArticleLastPage><ArticleHistory><RegistrationDate><Year>2005</Year><Month>2</Month><Day>14</Day></RegistrationDate><Received><Year>1972</Year><Month>3</Month><Day>28</Day></Received></ArticleHistory><ArticleCopyright><CopyrightHolderName>Springer-Verlag</CopyrightHolderName><CopyrightYear>1972</CopyrightYear></ArticleCopyright><ArticleGrants Type="Regular"><MetadataGrant Grant="OpenAccess"></MetadataGrant><AbstractGrant Grant="OpenAccess"></AbstractGrant><BodyPDFGrant Grant="Restricted"></BodyPDFGrant><BodyHTMLGrant Grant="Restricted"></BodyHTMLGrant><BibliographyGrant Grant="Restricted"></BibliographyGrant><ESMGrant Grant="Restricted"></ESMGrant></ArticleGrants><ArticleContext><JournalID>423</JournalID><VolumeIDStart>331</VolumeIDStart><VolumeIDEnd>331</VolumeIDEnd><IssueIDStart>4</IssueIDStart><IssueIDEnd>4</IssueIDEnd></ArticleContext></ArticleInfo><ArticleHeader><AuthorGroup><Author AffiliationIDS="Aff1"><AuthorName DisplayOrder="Western"><Prefix>Dr.</Prefix><GivenName>H.</GivenName><GivenName>R.</GivenName><FamilyName>Hölzl</FamilyName></AuthorName></Author><Author AffiliationIDS="Aff1"><AuthorName DisplayOrder="Western"><Prefix>Doz. Dr.</Prefix><GivenName>G.</GivenName><FamilyName>Flora</FamilyName></AuthorName></Author><Author AffiliationIDS="Aff1"><AuthorName DisplayOrder="Western"><Prefix>Dr.</Prefix><GivenName>M.</GivenName><FamilyName>Marberger</FamilyName></AuthorName></Author><Affiliation ID="Aff1"><OrgName>Chirurg. Univ.-Klinik</OrgName><OrgAddress><Street>Anichstraße 35</Street><Postcode>A-6020</Postcode><City>Innsbruck</City><Country>Österreich</Country></OrgAddress></Affiliation></AuthorGroup><Abstract ID="Abs1" Language="En"><Heading>Summary</Heading><Para>The primary stop of backflow of the axillary vein marked by swelling, pains, cyanosis and increasing superficial vein-prominence of the arm and of the neighboured shoulder-chest region. This pathological symptom has been wellknown under many synonyma since a 100 years, mostly induced by thrombotic closing of the axillary vein, as a result of irritation on locus minoris resistentiae (Costoclavicular region). The etiology is manyfold. Therefore in literature this syndrom is not always strictly separable from many other illnesses with the same or similar clinical symptoms (secundary disturbance of the backflow of the axillary vein, lymphedema). In the period from 1961 to 1971 twenty cases of a primary idiopathic stop of backflow of the axillary veins were observed at our Surgical University Hospital in Innsbruck. The conservative therapy outweighs by far the surgical one (relation 19:1). Two patients were treated with conservative means only; with fourteen patients the conservative therapy was enlarged by the usage of anticoagulants. Fibrinolytic therapy was tried on three patients but it did not produce a remarkable phlebographical drainage. After a foudroyantic development in one case, an operation (thrombectomy) was necessary in spite of a combined streptocinaseheparintherapy. Nine patients were painless out of a group of nineteen controlled ones, eight showed a swelling at work and other efforts, after an observation-period from 3 months to 10 years. One patient had pains, another one had pains and was swollen when making struggles. Relying on our own experiences and on informations scientific works, a treatment schedule of the primary stop of backflow of the axillary vein is to be discussed.</Para></Abstract><Abstract ID="Abs2" Language="De"><Heading>Zusammenfassung</Heading><Para>Der primäre Achselvenenstau ist im wesentlichen gekennzeichnet durch Schwellung, Schmerzen, Cyanose und vermehrte oberflächliche Venenzeichnung eines Armes und der zugehörigen Schulter-Brustregion. Seit etwa 100 Jahren ist dieses Krankheitsbild unter vielen Synonyma bekannt und ist in der Mehrzahl der Fälle durch thrombotischen Verschluß der Achselvene als Irritationsfolge am Locus minoris resistentiae (Costoclavicularraum) bedingt. Seine Ätiologie ist vielgestaltig, so daß es von vielen anderen Erkrankungen mit gleichen oder ähnlichen klinischen Erscheinungsformen (sekundärer Achselvenenstau, Lymphödem) in der Literatur nicht immer streng getrennt wird.</Para><Para>Im Zeitraum von 1961 bis 1971 wurden an der Chirurgischen Universitätsklinik Innsbruck 20 Fälle eines primären idiopathischen Achselvenenstaus beobachtet. Die konservative Therapie überwiegt bei weitem die chirurgische (19:1). 2 Patienten wurden nur konservativ behandelt; bei 14 Patienten wurde die konservative Therapie durch Anwendung von Anticoagulantien erweitert. Eine Fibrinolysetherapie wurde an 3 Patienten versucht, brachte jedoch keine phlebographisch nachweisbare Rekanalisation. Nach foudroyantem Verlauf eines Falles trotz einer kombinierten Streptokinase-Reparintherapie war ein operatives Vorgehen (Thrombektomie) notwendig.</Para><Para>Nach einem Beobachtungszeitraum von 3 Monaten bis 10 Jahre waren von 19 nachuntersuchten Patienten 9 beschwerdefrei, 8 zeigten Schwellung bei Anstrengung. Bei einem Patienten bestehen Schmerzen bei Anstrengung, bei einem weiteren Schmerzen bei Anstrengung sowie Schwellung.</Para><Para>Gestützt auf eigene Erfahrungen und Mitteilungen in der Literatur wird ein Behandlungsschema des primären Achselvenenstaus zur Diskussion gestellt.</Para></Abstract><KeywordGroup Language="En"><Heading>Key words</Heading><Keyword>Primary Thrombosis of the Axillary Vein</Keyword><Keyword>Anticoagulant and Fibrinolytic Agents</Keyword><Keyword>Thrombectomy</Keyword><Keyword>Regional Sympathetic Blockade</Keyword></KeywordGroup></ArticleHeader><NoBody></NoBody></Article></Issue></Volume></Journal></Publisher></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="de"><title>Primärer Achselvenenstau (Paget-von Schroetter-Syndrom)</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="de"><title>Primärer Achselvenenstau (Paget-von Schroetter-Syndrom)</title></titleInfo><titleInfo lang="en"><title>Primary thrombosis of the axillary vein</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>Primary thrombosis of the axillary vein</title></titleInfo><name type="personal"><namePart type="termsOfAddress">Dr.</namePart><namePart type="given">H.</namePart><namePart type="given">R.</namePart><namePart type="family">Hölzl</namePart><affiliation>Chirurg. Univ.-Klinik, Anichstraße 35, A-6020, Innsbruck, Österreich</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="termsOfAddress">Doz. Dr.</namePart><namePart type="given">G.</namePart><namePart type="family">Flora</namePart><affiliation>Chirurg. Univ.-Klinik, Anichstraße 35, A-6020, Innsbruck, Österreich</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="termsOfAddress">Dr.</namePart><namePart type="given">M.</namePart><namePart type="family">Marberger</namePart><affiliation>Chirurg. Univ.-Klinik, Anichstraße 35, A-6020, Innsbruck, Österreich</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="research-article" displayLabel="OriginalPaper"></genre><originInfo><publisher>Springer-Verlag</publisher><place><placeTerm type="text">Berlin/Heidelberg</placeTerm></place><dateCreated encoding="w3cdtf">1972-03-28</dateCreated><dateIssued encoding="w3cdtf">1972-12-01</dateIssued><copyrightDate encoding="w3cdtf">1972</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">de</languageTerm><languageTerm type="code" authority="iso639-2b">ger</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType></physicalDescription><abstract lang="en">Summary: The primary stop of backflow of the axillary vein marked by swelling, pains, cyanosis and increasing superficial vein-prominence of the arm and of the neighboured shoulder-chest region. This pathological symptom has been wellknown under many synonyma since a 100 years, mostly induced by thrombotic closing of the axillary vein, as a result of irritation on locus minoris resistentiae (Costoclavicular region). The etiology is manyfold. Therefore in literature this syndrom is not always strictly separable from many other illnesses with the same or similar clinical symptoms (secundary disturbance of the backflow of the axillary vein, lymphedema). In the period from 1961 to 1971 twenty cases of a primary idiopathic stop of backflow of the axillary veins were observed at our Surgical University Hospital in Innsbruck. The conservative therapy outweighs by far the surgical one (relation 19:1). Two patients were treated with conservative means only; with fourteen patients the conservative therapy was enlarged by the usage of anticoagulants. Fibrinolytic therapy was tried on three patients but it did not produce a remarkable phlebographical drainage. After a foudroyantic development in one case, an operation (thrombectomy) was necessary in spite of a combined streptocinaseheparintherapy. Nine patients were painless out of a group of nineteen controlled ones, eight showed a swelling at work and other efforts, after an observation-period from 3 months to 10 years. One patient had pains, another one had pains and was swollen when making struggles. Relying on our own experiences and on informations scientific works, a treatment schedule of the primary stop of backflow of the axillary vein is to be discussed.</abstract><abstract lang="de">Zusammenfassung: Der primäre Achselvenenstau ist im wesentlichen gekennzeichnet durch Schwellung, Schmerzen, Cyanose und vermehrte oberflächliche Venenzeichnung eines Armes und der zugehörigen Schulter-Brustregion. Seit etwa 100 Jahren ist dieses Krankheitsbild unter vielen Synonyma bekannt und ist in der Mehrzahl der Fälle durch thrombotischen Verschluß der Achselvene als Irritationsfolge am Locus minoris resistentiae (Costoclavicularraum) bedingt. Seine Ätiologie ist vielgestaltig, so daß es von vielen anderen Erkrankungen mit gleichen oder ähnlichen klinischen Erscheinungsformen (sekundärer Achselvenenstau, Lymphödem) in der Literatur nicht immer streng getrennt wird. Im Zeitraum von 1961 bis 1971 wurden an der Chirurgischen Universitätsklinik Innsbruck 20 Fälle eines primären idiopathischen Achselvenenstaus beobachtet. Die konservative Therapie überwiegt bei weitem die chirurgische (19:1). 2 Patienten wurden nur konservativ behandelt; bei 14 Patienten wurde die konservative Therapie durch Anwendung von Anticoagulantien erweitert. Eine Fibrinolysetherapie wurde an 3 Patienten versucht, brachte jedoch keine phlebographisch nachweisbare Rekanalisation. Nach foudroyantem Verlauf eines Falles trotz einer kombinierten Streptokinase-Reparintherapie war ein operatives Vorgehen (Thrombektomie) notwendig. Nach einem Beobachtungszeitraum von 3 Monaten bis 10 Jahre waren von 19 nachuntersuchten Patienten 9 beschwerdefrei, 8 zeigten Schwellung bei Anstrengung. Bei einem Patienten bestehen Schmerzen bei Anstrengung, bei einem weiteren Schmerzen bei Anstrengung sowie Schwellung. Gestützt auf eigene Erfahrungen und Mitteilungen in der Literatur wird ein Behandlungsschema des primären Achselvenenstaus zur Diskussion gestellt.</abstract><relatedItem type="host"><titleInfo><title>Langenbecks Archiv für Chirurgie</title></titleInfo><titleInfo type="abbreviated"><title>Langenbecks Arch Chiv</title></titleInfo><genre type="journal" displayLabel="Archive Journal"></genre><originInfo><dateIssued encoding="w3cdtf">1972-12-01</dateIssued><copyrightDate encoding="w3cdtf">1972</copyrightDate></originInfo><subject><genre>Medicine & Public Health</genre><topic>Abdominal Surgery</topic><topic>Cardiac Surgery</topic><topic>General Surgery</topic><topic>Thoracic Surgery</topic><topic>Traumatic Surgery</topic><topic>Vascular Surgery</topic></subject><identifier type="ISSN">0023-8236</identifier><identifier type="eISSN">1435-2451</identifier><identifier type="JournalID">423</identifier><identifier type="IssueArticleCount">8</identifier><identifier type="VolumeIssueCount">4</identifier><part><date>1972</date><detail type="volume"><number>331</number><caption>vol.</caption></detail><detail type="issue"><number>4</number><caption>no.</caption></detail><extent unit="pages"><start>295</start><end>314</end></extent></part><recordInfo><recordOrigin>Springer-Verlag, 1972</recordOrigin></recordInfo></relatedItem><identifier type="istex">8E8270B6B662E918D493B303E0BB1ED46D7A051B</identifier><identifier type="DOI">10.1007/BF01239174</identifier><identifier type="ArticleID">BF01239174</identifier><identifier type="ArticleID">Art3</identifier><accessCondition type="use and reproduction" contentType="copyright">Springer-Verlag, 1972</accessCondition><recordInfo><recordContentSource>SPRINGER</recordContentSource><recordOrigin>Springer-Verlag, 1972</recordOrigin></recordInfo></mods></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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