Rimonabant improves cholesterol, insulin resistance and markers of non‐alcoholic fatty liver in morbidly obese patients: a retrospective cohort study
Identifieur interne : 002955 ( Istex/Corpus ); précédent : 002954; suivant : 002956Rimonabant improves cholesterol, insulin resistance and markers of non‐alcoholic fatty liver in morbidly obese patients: a retrospective cohort study
Auteurs : A. S. Wierzbicki ; S. Pendleton ; Z. Mcmahon ; A. Dar ; J. Oben ; M. A. Crook ; A. J. BothaSource :
- International Journal of Clinical Practice [ 1368-5031 ] ; 2011-06.
Url:
DOI: 10.1111/j.1742-1241.2011.02683.x
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Botha</name><affiliation><mods:affiliation>Departments of Bariatric & upper gastrointestinal surgery, Guy’s & St Thomas’ NHS Trust, St Thomas Hospital, London, UKEmail: anthony.wierzbicki@kcl.ac.uk</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j" type="main">International Journal of Clinical Practice</title><title level="j" type="alt">INTERNATIONAL JOURNAL OF CLINICAL PRACTICE</title><idno type="ISSN">1368-5031</idno><idno type="eISSN">1742-1241</idno><imprint><biblScope unit="vol">65</biblScope><biblScope unit="issue">6</biblScope><biblScope unit="page" from="713">713</biblScope><biblScope unit="page" to="715">715</biblScope><biblScope unit="page-count">3</biblScope><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="2011-06">2011-06</date></imprint><idno type="ISSN">1368-5031</idno></series></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">1368-5031</idno></seriesStmt></fileDesc><profileDesc><textClass></textClass></profileDesc></teiHeader></TEI><istex><corpusName>wiley</corpusName><keywords><teeft><json:string>rimonabant</json:string><json:string>mmol</json:string><json:string>clin</json:string><json:string>insulin resistance</json:string><json:string>glucose</json:string><json:string>rimonabant therapy</json:string><json:string>blood pressure</json:string><json:string>clin pract</json:string><json:string>metabolic syndrome</json:string><json:string>weight loss</json:string><json:string>iron absorption</json:string><json:string>thomas hospital</json:string><json:string>gout</json:string><json:string>vascular risk</json:string><json:string>blackwell publishing</json:string><json:string>hepatic steatosis</json:string><json:string>total cholesterol</json:string><json:string>coffee consumption</json:string><json:string>cardiovascular risk</json:string><json:string>metabolic</json:string><json:string>insulin</json:string><json:string>fatty liver</json:string><json:string>retrospective cohort study</json:string><json:string>gouty arthritis</json:string><json:string>coffee intake</json:string><json:string>bariatric surgery</json:string><json:string>clin nutr</json:string><json:string>synovial membrane</json:string><json:string>iron overload</json:string><json:string>framingham heart study</json:string><json:string>surrogate markers</json:string><json:string>incident gout</json:string><json:string>inverse association</json:string><json:string>metabolic changes</json:string><json:string>framingham study</json:string><json:string>uric acid</json:string><json:string>free radic</json:string><json:string>cardiometabolic risk evaluation</json:string><json:string>homeostasis index</json:string><json:string>syndrome</json:string></teeft></keywords><author><json:item><name>A. 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S.</forename><surname>Wierzbicki</surname></persName><affiliation>Departments of Metabolic Medicine/Chemical Pathology, Guy’s & St Thomas’ NHS Trust, St Thomas Hospital, London, UK<address><country key="GB"></country></address></affiliation></author><author xml:id="author-0001"><persName><forename type="first">S.</forename><surname>Pendleton</surname></persName><affiliation>Departments of Nutrition & Dietetics, Guy’s & St Thomas’ NHS Trust, St Thomas Hospital, London, UK<address><country key="GB"></country></address></affiliation></author><author xml:id="author-0002"><persName><forename type="first">Z.</forename><surname>McMahon</surname></persName><affiliation>Departments of Metabolic Medicine/Chemical Pathology, Guy’s & St Thomas’ NHS Trust, St Thomas Hospital, London, UK<address><country key="GB"></country></address></affiliation></author><author xml:id="author-0003"><persName><forename type="first">A.</forename><surname>Dar</surname></persName><affiliation>Departments of Gastroenterology, Guy’s & St Thomas’ NHS Trust, St Thomas Hospital, London, UK<address><country key="GB"></country></address></affiliation></author><author xml:id="author-0004"><persName><forename type="first">J.</forename><surname>Oben</surname></persName><affiliation>Departments of Gastroenterology, Guy’s & St Thomas’ NHS Trust, St Thomas Hospital, London, UK<address><country key="GB"></country></address></affiliation></author><author xml:id="author-0005"><persName><forename type="first">M. 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Departments of Bariatric & upper gastrointestinal surgery, Guy’s & St Thomas’ NHS Trust, St Thomas Hospital, London, UK
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S.</givenNames><familyName>Wierzbicki</familyName></personName></creator><creator creatorRole="author" xml:id="cr2" affiliationRef="#a2"><personName><givenNames>S.</givenNames><familyName>Pendleton</familyName></personName></creator><creator creatorRole="author" xml:id="cr3" affiliationRef="#a1"><personName><givenNames>Z.</givenNames><familyName>McMahon</familyName></personName></creator><creator creatorRole="author" xml:id="cr4" affiliationRef="#a3"><personName><givenNames>A.</givenNames><familyName>Dar</familyName></personName></creator><creator creatorRole="author" xml:id="cr5" affiliationRef="#a3"><personName><givenNames>J.</givenNames><familyName>Oben</familyName></personName></creator><creator creatorRole="author" xml:id="cr6" affiliationRef="#a1"><personName><givenNames>M. A.</givenNames><familyName>Crook</familyName></personName></creator><creator creatorRole="author" xml:id="cr7" affiliationRef="#a4"><personName><givenNames>A. J.</givenNames><familyName>Botha</familyName></personName></creator></creators><affiliationGroup><affiliation xml:id="a1" countryCode="GB"><unparsedAffiliation>Departments of Metabolic Medicine/Chemical Pathology, Guy’s & St Thomas’ NHS Trust, St Thomas Hospital, London, UK</unparsedAffiliation></affiliation><affiliation xml:id="a2" countryCode="GB"><unparsedAffiliation>Departments of Nutrition & Dietetics, Guy’s & St Thomas’ NHS Trust, St Thomas Hospital, London, UK</unparsedAffiliation></affiliation><affiliation xml:id="a3" countryCode="GB"><unparsedAffiliation>Departments of Gastroenterology, Guy’s & St Thomas’ NHS Trust, St Thomas Hospital, London, UK</unparsedAffiliation></affiliation><affiliation xml:id="a4" countryCode="GB"><unparsedAffiliation>
Departments of Bariatric & upper gastrointestinal surgery, Guy’s & St Thomas’ NHS Trust, St Thomas Hospital, London, UK
Email: <email>anthony.wierzbicki@kcl.ac.uk</email></unparsedAffiliation></affiliation></affiliationGroup><supportingInformation><p><b>Figure S1.</b> Database characteristics of the retrospective cohort analysis of rimonabant therapy.</p><p><b>Table S1.</b> Baseline characteristics of patients adherent to treatment pathway and prescribed rimonabant therapy. Comparisons were performed between the continued and discontinued therapy groups.</p><p><b>Table S2.</b> Discontinuations associated with rimonabant therapy. Patients could have more than one reason for discontinuing treatment.</p><supportingInfoItem><mediaResource alt="supporting info item" href="urn-x:wiley:13685031:media:ijcp2683:IJCP_2683_sm_RimonabantSupplementarydata"></mediaResource><caption>Supporting info item</caption></supportingInfoItem></supportingInformation></contentMeta></header></component></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Rimonabant improves cholesterol, insulin resistance and markers of non‐alcoholic fatty liver in morbidly obese patients: a retrospective cohort study</title></titleInfo><titleInfo type="abbreviated" lang="en"><title>Letters</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>Rimonabant improves cholesterol, insulin resistance and markers of non‐alcoholic fatty liver in morbidly obese patients: a retrospective cohort study</title></titleInfo><name type="personal"><namePart type="given">A. S.</namePart><namePart type="family">Wierzbicki</namePart><affiliation>Departments of Metabolic Medicine/Chemical Pathology, Guy’s & St Thomas’ NHS Trust, St Thomas Hospital, London, UK</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">S.</namePart><namePart type="family">Pendleton</namePart><affiliation>Departments of Nutrition & Dietetics, Guy’s & St Thomas’ NHS Trust, St Thomas Hospital, London, UK</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Z.</namePart><namePart type="family">McMahon</namePart><affiliation>Departments of Metabolic Medicine/Chemical Pathology, Guy’s & St Thomas’ NHS Trust, St Thomas Hospital, London, UK</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">A.</namePart><namePart type="family">Dar</namePart><affiliation>Departments of Gastroenterology, Guy’s & St Thomas’ NHS Trust, St Thomas Hospital, London, UK</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">J.</namePart><namePart type="family">Oben</namePart><affiliation>Departments of Gastroenterology, Guy’s & St Thomas’ NHS Trust, St Thomas Hospital, London, UK</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">M. A.</namePart><namePart type="family">Crook</namePart><affiliation>Departments of Metabolic Medicine/Chemical Pathology, Guy’s & St Thomas’ NHS Trust, St Thomas Hospital, London, UK</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">A. J.</namePart><namePart type="family">Botha</namePart><affiliation>Departments of Bariatric & upper gastrointestinal surgery, Guy’s & St Thomas’ NHS Trust, St Thomas Hospital, London, UKEmail: anthony.wierzbicki@kcl.ac.uk</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="review-article" displayLabel="letter"></genre><originInfo><publisher>Blackwell Publishing Ltd</publisher><place><placeTerm type="text">Oxford, UK</placeTerm></place><dateIssued encoding="w3cdtf">2011-06</dateIssued><copyrightDate encoding="w3cdtf">2011</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType><extent unit="tables">1</extent></physicalDescription><relatedItem type="host"><titleInfo><title>International Journal of Clinical Practice</title></titleInfo><genre type="journal">journal</genre><note type="content"> Figure S1. Database characteristics of the retrospective cohort analysis of rimonabant therapy. Table S1. Baseline characteristics of patients adherent to treatment pathway and prescribed rimonabant therapy. Comparisons were performed between the continued and discontinued therapy groups. Table S2. Discontinuations associated with rimonabant therapy. Patients could have more than one reason for discontinuing treatment. Figure S1. Database characteristics of the retrospective cohort analysis of rimonabant therapy. Table S1. Baseline characteristics of patients adherent to treatment pathway and prescribed rimonabant therapy. Comparisons were performed between the continued and discontinued therapy groups. Table S2. Discontinuations associated with rimonabant therapy. Patients could have more than one reason for discontinuing treatment. Figure S1. Database characteristics of the retrospective cohort analysis of rimonabant therapy. Table S1. Baseline characteristics of patients adherent to treatment pathway and prescribed rimonabant therapy. Comparisons were performed between the continued and discontinued therapy groups. Table S2. Discontinuations associated with rimonabant therapy. Patients could have more than one reason for discontinuing treatment.Supporting Info Item: Supporting info item - </note><subject><genre>article-category</genre><topic>LETTER</topic></subject><identifier type="ISSN">1368-5031</identifier><identifier type="eISSN">1742-1241</identifier><identifier type="DOI">10.1111/(ISSN)1742-1241</identifier><identifier type="PublisherID">IJCP</identifier><part><date>2011</date><detail type="volume"><caption>vol.</caption><number>65</number></detail><detail type="issue"><caption>no.</caption><number>6</number></detail><extent unit="pages"><start>713</start><end>715</end><total>3</total></extent></part></relatedItem><identifier type="istex">59A5D8CC8FA3C7E7FF43AD83060BF922C456A22C</identifier><identifier type="DOI">10.1111/j.1742-1241.2011.02683.x</identifier><identifier type="ArticleID">IJCP2683</identifier><accessCondition type="use and reproduction" contentType="copyright">© 2011 Blackwell Publishing Ltd</accessCondition><recordInfo><recordContentSource>WILEY</recordContentSource><recordOrigin>Blackwell Publishing Ltd</recordOrigin></recordInfo></mods></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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