Genetic polymorphisms of the N-acetyltransferase genes and risk of Parkinson's disease.
Identifieur interne : 000221 ( PubMed/Corpus ); précédent : 000220; suivant : 000222Genetic polymorphisms of the N-acetyltransferase genes and risk of Parkinson's disease.
Auteurs : J Van ; E Martin ; W Scott ; F. Zhang ; M Nance ; R Watts ; J Hubble ; J Haines ; W Koller ; K. Lyons ; R. Pahwa ; M Stern ; A. Colcher ; B Hiner ; J. Jankovic ; W Ondo ; F Allen ; C Goetz ; G Small ; F. Mastaglia ; A Roses ; J Stajich ; M Booze ; K. Fujiwara ; R Gibson ; L Middleton ; B Scott ; M Pericak ; J VanceSource :
- Neurology ; 2003.
English descriptors
- KwdEn :
- Aged, Alleles, Arylamine N-Acetyltransferase (genetics), Chromosomes, Human, Pair 8 (genetics), Female, Gene Frequency, Genetic Linkage, Genetic Predisposition to Disease, Genotype, Haplotypes, Humans, Isoenzymes (genetics), Male, Middle Aged, Parkinson Disease (enzymology), Parkinson Disease (genetics), Polymorphism, Genetic, Polymorphism, Single Nucleotide (genetics), Risk Assessment.
- MESH :
- chemical , genetics : Arylamine N-Acetyltransferase, Isoenzymes.
- enzymology : Parkinson Disease.
- genetics : Chromosomes, Human, Pair 8, Parkinson Disease, Polymorphism, Single Nucleotide.
- Aged, Alleles, Female, Gene Frequency, Genetic Linkage, Genetic Predisposition to Disease, Genotype, Haplotypes, Humans, Male, Middle Aged, Polymorphism, Genetic, Risk Assessment.
Abstract
Recently, the authors demonstrated linkage in idiopathic PD to a region on chromosome 8p that contains the N-acetyltransferase genes, NAT1 and NAT2. The authors examined NAT1 and NAT2 for association with PD using family-based association methods and single nucleotide polymorphisms (SNPs). The authors did not find evidence for association with increased risk for PD between any individual NAT1 or NAT2 SNP or acetylation haplotype (N = 397 families, 1,580 individuals).
PubMed: 12682333
Links to Exploration step
pubmed:12682333Le document en format XML
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Zhang</name></author><author><name sortKey="Nance, M A" sort="Nance, M A" uniqKey="Nance M" first="M" last="Nance">M Nance</name></author><author><name sortKey="Watts, R L" sort="Watts, R L" uniqKey="Watts R" first="R" last="Watts">R Watts</name></author><author><name sortKey="Hubble, J P" sort="Hubble, J P" uniqKey="Hubble J" first="J" last="Hubble">J Hubble</name></author><author><name sortKey="Haines, J L" sort="Haines, J L" uniqKey="Haines J" first="J" last="Haines">J Haines</name></author><author><name sortKey="Koller, W C" sort="Koller, W C" uniqKey="Koller W" first="W" last="Koller">W Koller</name></author><author><name sortKey="Lyons, K" sort="Lyons, K" uniqKey="Lyons K" first="K" last="Lyons">K. Lyons</name></author><author><name sortKey="Pahwa, R" sort="Pahwa, R" uniqKey="Pahwa R" first="R" last="Pahwa">R. 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Zhang</name></author><author><name sortKey="Nance, M A" sort="Nance, M A" uniqKey="Nance M" first="M" last="Nance">M Nance</name></author><author><name sortKey="Watts, R L" sort="Watts, R L" uniqKey="Watts R" first="R" last="Watts">R Watts</name></author><author><name sortKey="Hubble, J P" sort="Hubble, J P" uniqKey="Hubble J" first="J" last="Hubble">J Hubble</name></author><author><name sortKey="Haines, J L" sort="Haines, J L" uniqKey="Haines J" first="J" last="Haines">J Haines</name></author><author><name sortKey="Koller, W C" sort="Koller, W C" uniqKey="Koller W" first="W" last="Koller">W Koller</name></author><author><name sortKey="Lyons, K" sort="Lyons, K" uniqKey="Lyons K" first="K" last="Lyons">K. Lyons</name></author><author><name sortKey="Pahwa, R" sort="Pahwa, R" uniqKey="Pahwa R" first="R" last="Pahwa">R. Pahwa</name></author><author><name sortKey="Stern, M B" sort="Stern, M B" uniqKey="Stern M" first="M" last="Stern">M Stern</name></author><author><name sortKey="Colcher, A" sort="Colcher, A" uniqKey="Colcher A" first="A" last="Colcher">A. Colcher</name></author><author><name sortKey="Hiner, B C" sort="Hiner, B C" uniqKey="Hiner B" first="B" last="Hiner">B Hiner</name></author><author><name sortKey="Jankovic, J" sort="Jankovic, J" uniqKey="Jankovic J" first="J" last="Jankovic">J. Jankovic</name></author><author><name sortKey="Ondo, W G" sort="Ondo, W G" uniqKey="Ondo W" first="W" last="Ondo">W Ondo</name></author><author><name sortKey="Allen, F H" sort="Allen, F H" uniqKey="Allen F" first="F" last="Allen">F Allen</name></author><author><name sortKey="Goetz, C G" sort="Goetz, C G" uniqKey="Goetz C" first="C" last="Goetz">C Goetz</name></author><author><name sortKey="Small, G W" sort="Small, G W" uniqKey="Small G" first="G" last="Small">G Small</name></author><author><name sortKey="Mastaglia, F" sort="Mastaglia, F" uniqKey="Mastaglia F" first="F" last="Mastaglia">F. Mastaglia</name></author><author><name sortKey="Roses, A D" sort="Roses, A D" uniqKey="Roses A" first="A" last="Roses">A Roses</name></author><author><name sortKey="Stajich, J M" sort="Stajich, J M" uniqKey="Stajich J" first="J" last="Stajich">J Stajich</name></author><author><name sortKey="Booze, M W" sort="Booze, M W" uniqKey="Booze M" first="M" last="Booze">M Booze</name></author><author><name sortKey="Fujiwara, K" sort="Fujiwara, K" uniqKey="Fujiwara K" first="K" last="Fujiwara">K. Fujiwara</name></author><author><name sortKey="Gibson, R A" sort="Gibson, R A" uniqKey="Gibson R" first="R" last="Gibson">R Gibson</name></author><author><name sortKey="Middleton, L T" sort="Middleton, L T" uniqKey="Middleton L" first="L" last="Middleton">L Middleton</name></author><author><name sortKey="Scott, B L" sort="Scott, B L" uniqKey="Scott B" first="B" last="Scott">B Scott</name></author><author><name sortKey="Pericak Vance, M A" sort="Pericak Vance, M A" uniqKey="Pericak Vance M" first="M" last="Pericak">M Pericak</name></author><author><name sortKey="Vance, J M" sort="Vance, J M" uniqKey="Vance J" first="J" last="Vance">J Vance</name></author></analytic><series><title level="j">Neurology</title><idno type="e-ISSN">1526-632X</idno><imprint><date when="2003" type="published">2003</date></imprint></series></biblStruct></sourceDesc></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Aged</term><term>Alleles</term><term>Arylamine N-Acetyltransferase (genetics)</term><term>Chromosomes, Human, Pair 8 (genetics)</term><term>Female</term><term>Gene Frequency</term><term>Genetic Linkage</term><term>Genetic Predisposition to Disease</term><term>Genotype</term><term>Haplotypes</term><term>Humans</term><term>Isoenzymes (genetics)</term><term>Male</term><term>Middle Aged</term><term>Parkinson Disease (enzymology)</term><term>Parkinson Disease (genetics)</term><term>Polymorphism, Genetic</term><term>Polymorphism, Single Nucleotide (genetics)</term><term>Risk Assessment</term></keywords><keywords scheme="MESH" type="chemical" qualifier="genetics" xml:lang="en"><term>Arylamine N-Acetyltransferase</term><term>Isoenzymes</term></keywords><keywords scheme="MESH" qualifier="enzymology" xml:lang="en"><term>Parkinson Disease</term></keywords><keywords scheme="MESH" qualifier="genetics" xml:lang="en"><term>Chromosomes, Human, Pair 8</term><term>Parkinson Disease</term><term>Polymorphism, Single Nucleotide</term></keywords><keywords scheme="MESH" xml:lang="en"><term>Aged</term><term>Alleles</term><term>Female</term><term>Gene Frequency</term><term>Genetic Linkage</term><term>Genetic Predisposition to Disease</term><term>Genotype</term><term>Haplotypes</term><term>Humans</term><term>Male</term><term>Middle Aged</term><term>Polymorphism, Genetic</term><term>Risk Assessment</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Recently, the authors demonstrated linkage in idiopathic PD to a region on chromosome 8p that contains the N-acetyltransferase genes, NAT1 and NAT2. The authors examined NAT1 and NAT2 for association with PD using family-based association methods and single nucleotide polymorphisms (SNPs). The authors did not find evidence for association with increased risk for PD between any individual NAT1 or NAT2 SNP or acetylation haplotype (N = 397 families, 1,580 individuals).</div></front></TEI><pubmed><MedlineCitation Owner="NLM" Status="MEDLINE"><PMID Version="1">12682333</PMID><DateCreated><Year>2003</Year><Month>04</Month><Day>08</Day></DateCreated><DateCompleted><Year>2003</Year><Month>04</Month><Day>23</Day></DateCompleted><DateRevised><Year>2010</Year><Month>11</Month><Day>18</Day></DateRevised><Article PubModel="Print"><Journal><ISSN IssnType="Electronic">1526-632X</ISSN><JournalIssue CitedMedium="Internet"><Volume>60</Volume><Issue>7</Issue><PubDate><Year>2003</Year><Month>Apr</Month><Day>8</Day></PubDate></JournalIssue><Title>Neurology</Title><ISOAbbreviation>Neurology</ISOAbbreviation></Journal><ArticleTitle>Genetic polymorphisms of the N-acetyltransferase genes and risk of Parkinson's disease.</ArticleTitle><Pagination><MedlinePgn>1189-91</MedlinePgn></Pagination><Abstract><AbstractText>Recently, the authors demonstrated linkage in idiopathic PD to a region on chromosome 8p that contains the N-acetyltransferase genes, NAT1 and NAT2. The authors examined NAT1 and NAT2 for association with PD using family-based association methods and single nucleotide polymorphisms (SNPs). The authors did not find evidence for association with increased risk for PD between any individual NAT1 or NAT2 SNP or acetylation haplotype (N = 397 families, 1,580 individuals).</AbstractText></Abstract><AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>van der Walt</LastName><ForeName>J M</ForeName><Initials>JM</Initials><AffiliationInfo><Affiliation>Department of Medicine and Center for Human Genetics, Institute for Genome Sciences and Policy, Duke University Medical Center, Durham, NC 27710, USA.</Affiliation></AffiliationInfo></Author><Author ValidYN="Y"><LastName>Martin</LastName><ForeName>E R</ForeName><Initials>ER</Initials></Author><Author ValidYN="Y"><LastName>Scott</LastName><ForeName>W K</ForeName><Initials>WK</Initials></Author><Author ValidYN="Y"><LastName>Zhang</LastName><ForeName>F</ForeName><Initials>F</Initials></Author><Author ValidYN="Y"><LastName>Nance</LastName><ForeName>M A</ForeName><Initials>MA</Initials></Author><Author ValidYN="Y"><LastName>Watts</LastName><ForeName>R L</ForeName><Initials>RL</Initials></Author><Author ValidYN="Y"><LastName>Hubble</LastName><ForeName>J P</ForeName><Initials>JP</Initials></Author><Author ValidYN="Y"><LastName>Haines</LastName><ForeName>J L</ForeName><Initials>JL</Initials></Author><Author ValidYN="Y"><LastName>Koller</LastName><ForeName>W C</ForeName><Initials>WC</Initials></Author><Author ValidYN="Y"><LastName>Lyons</LastName><ForeName>K</ForeName><Initials>K</Initials></Author><Author ValidYN="Y"><LastName>Pahwa</LastName><ForeName>R</ForeName><Initials>R</Initials></Author><Author ValidYN="Y"><LastName>Stern</LastName><ForeName>M B</ForeName><Initials>MB</Initials></Author><Author ValidYN="Y"><LastName>Colcher</LastName><ForeName>A</ForeName><Initials>A</Initials></Author><Author ValidYN="Y"><LastName>Hiner</LastName><ForeName>B C</ForeName><Initials>BC</Initials></Author><Author ValidYN="Y"><LastName>Jankovic</LastName><ForeName>J</ForeName><Initials>J</Initials></Author><Author ValidYN="Y"><LastName>Ondo</LastName><ForeName>W G</ForeName><Initials>WG</Initials></Author><Author ValidYN="Y"><LastName>Allen</LastName><ForeName>F H</ForeName><Initials>FH</Initials><Suffix>Jr</Suffix></Author><Author ValidYN="Y"><LastName>Goetz</LastName><ForeName>C G</ForeName><Initials>CG</Initials></Author><Author ValidYN="Y"><LastName>Small</LastName><ForeName>G W</ForeName><Initials>GW</Initials></Author><Author ValidYN="Y"><LastName>Mastaglia</LastName><ForeName>F</ForeName><Initials>F</Initials></Author><Author ValidYN="Y"><LastName>Roses</LastName><ForeName>A D</ForeName><Initials>AD</Initials></Author><Author ValidYN="Y"><LastName>Stajich</LastName><ForeName>J M</ForeName><Initials>JM</Initials></Author><Author ValidYN="Y"><LastName>Booze</LastName><ForeName>M W</ForeName><Initials>MW</Initials></Author><Author ValidYN="Y"><LastName>Fujiwara</LastName><ForeName>K</ForeName><Initials>K</Initials></Author><Author ValidYN="Y"><LastName>Gibson</LastName><ForeName>R A</ForeName><Initials>RA</Initials></Author><Author ValidYN="Y"><LastName>Middleton</LastName><ForeName>L T</ForeName><Initials>LT</Initials></Author><Author ValidYN="Y"><LastName>Scott</LastName><ForeName>B L</ForeName><Initials>BL</Initials></Author><Author ValidYN="Y"><LastName>Pericak-Vance</LastName><ForeName>M A</ForeName><Initials>MA</Initials></Author><Author ValidYN="Y"><LastName>Vance</LastName><ForeName>J M</ForeName><Initials>JM</Initials></Author></AuthorList><Language>eng</Language><GrantList CompleteYN="Y"><Grant><GrantID>1R01-AG-20135-01</GrantID><Acronym>AG</Acronym><Agency>NIA NIH HHS</Agency><Country>United States</Country></Grant><Grant><GrantID>2 P50 NS39764-02</GrantID><Acronym>NS</Acronym><Agency>NINDS NIH HHS</Agency><Country>United States</Country></Grant><Grant><GrantID>P01 NS26630</GrantID><Acronym>NS</Acronym><Agency>NINDS NIH HHS</Agency><Country>United States</Country></Grant></GrantList><PublicationTypeList><PublicationType UI="D016428">Journal Article</PublicationType><PublicationType UI="D013485">Research Support, Non-U.S. Gov't</PublicationType><PublicationType UI="D013487">Research Support, U.S. Gov't, P.H.S.</PublicationType></PublicationTypeList></Article><MedlineJournalInfo><Country>United States</Country><MedlineTA>Neurology</MedlineTA><NlmUniqueID>0401060</NlmUniqueID><ISSNLinking>0028-3878</ISSNLinking></MedlineJournalInfo><ChemicalList><Chemical><RegistryNumber>0</RegistryNumber><NameOfSubstance UI="D007527">Isoenzymes</NameOfSubstance></Chemical><Chemical><RegistryNumber>EC 2.3.1.5</RegistryNumber><NameOfSubstance UI="D001191">Arylamine N-Acetyltransferase</NameOfSubstance></Chemical><Chemical><RegistryNumber>EC 2.3.1.5</RegistryNumber><NameOfSubstance UI="C089384">N-acetyltransferase 1</NameOfSubstance></Chemical><Chemical><RegistryNumber>EC 2.3.1.5</RegistryNumber><NameOfSubstance UI="C478900">NAT2 protein, human</NameOfSubstance></Chemical></ChemicalList><CitationSubset>AIM</CitationSubset><CitationSubset>IM</CitationSubset><MeshHeadingList><MeshHeading><DescriptorName MajorTopicYN="N" UI="D000368">Aged</DescriptorName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D000483">Alleles</DescriptorName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D001191">Arylamine N-Acetyltransferase</DescriptorName><QualifierName MajorTopicYN="Y" UI="Q000235">genetics</QualifierName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D002898">Chromosomes, Human, Pair 8</DescriptorName><QualifierName MajorTopicYN="N" UI="Q000235">genetics</QualifierName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D005260">Female</DescriptorName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D005787">Gene Frequency</DescriptorName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D008040">Genetic Linkage</DescriptorName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D020022">Genetic Predisposition to Disease</DescriptorName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D005838">Genotype</DescriptorName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D006239">Haplotypes</DescriptorName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D006801">Humans</DescriptorName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D007527">Isoenzymes</DescriptorName><QualifierName MajorTopicYN="N" UI="Q000235">genetics</QualifierName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D008297">Male</DescriptorName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D008875">Middle Aged</DescriptorName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D010300">Parkinson Disease</DescriptorName><QualifierName MajorTopicYN="Y" UI="Q000201">enzymology</QualifierName><QualifierName MajorTopicYN="Y" UI="Q000235">genetics</QualifierName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="Y" UI="D011110">Polymorphism, Genetic</DescriptorName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D020641">Polymorphism, Single Nucleotide</DescriptorName><QualifierName MajorTopicYN="N" UI="Q000235">genetics</QualifierName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D018570">Risk Assessment</DescriptorName></MeshHeading></MeshHeadingList></MedlineCitation><PubmedData><History><PubMedPubDate PubStatus="pubmed"><Year>2003</Year><Month>4</Month><Day>12</Day><Hour>5</Hour><Minute>0</Minute></PubMedPubDate><PubMedPubDate PubStatus="medline"><Year>2003</Year><Month>4</Month><Day>24</Day><Hour>5</Hour><Minute>0</Minute></PubMedPubDate><PubMedPubDate 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