Apolipoprotein E controls the risk and age at onset of Parkinson disease.
Identifieur interne : 000073 ( Ncbi/Curation ); précédent : 000072; suivant : 000074Apolipoprotein E controls the risk and age at onset of Parkinson disease.
Auteurs : Y Li [États-Unis] ; M Hauser ; W Scott ; E Martin ; M Booze ; X Qin ; J Walter ; M Nance ; J Hubble ; W Koller ; R. Pahwa ; M Stern ; B Hiner ; Joseph Jankovic [États-Unis] ; C Goetz ; G Small ; F. Mastaglia ; J Haines ; M Pericak ; J VanceSource :
- Neurology ; 2004.
Descripteurs français
- Wicri :
- geographic : Australie, États-Unis.
English descriptors
- KwdEn :
- Adult, Age of Onset, Aged, Alleles, Apolipoprotein E3, Apolipoprotein E4, Apolipoproteins E (genetics), Apolipoproteins E (physiology), Australia (epidemiology), Cognition, Dementia (epidemiology), Dementia (genetics), Female, Gene Frequency, Genetic Predisposition to Disease, Genotype, Humans, Male, Middle Aged, Neuropsychological Tests, Parkinson Disease (epidemiology), Parkinson Disease (genetics), Parkinson Disease (psychology), Pedigree, Risk, United States (epidemiology).
- MESH :
- chemical , genetics : Apolipoproteins E.
- chemical , physiology : Apolipoproteins E.
- chemical : Apolipoprotein E3, Apolipoprotein E4.
- geographic , epidemiology : Australia, United States.
- epidemiology : Dementia, Parkinson Disease.
- genetics : Dementia, Parkinson Disease.
- psychology : Parkinson Disease.
- Adult, Age of Onset, Aged, Alleles, Cognition, Female, Gene Frequency, Genetic Predisposition to Disease, Genotype, Humans, Male, Middle Aged, Neuropsychological Tests, Pedigree, Risk.
Abstract
Similarities between Alzheimer disease (AD) and Parkinson disease (PD) suggest a possible role for apolipoprotein E (APOE) in PD. Most previous studies seeking to establish such a link used case-control datasets and results have been inconsistent.
PubMed: 15184605
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pubmed:15184605Le document en format XML
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<author><name sortKey="Li, Y J" sort="Li, Y J" uniqKey="Li Y" first="Y" last="Li">Y Li</name>
<affiliation wicri:level="2"><nlm:affiliation>Department of Medicine and Center for Human Genetics, Duke University Medical Center, Durham, NC 27710, USA. yiju.li@duke.edu</nlm:affiliation>
<country xml:lang="fr">États-Unis</country>
<wicri:regionArea>Department of Medicine and Center for Human Genetics, Duke University Medical Center, Durham, NC 27710</wicri:regionArea>
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<affiliation wicri:level="2"><nlm:affiliation>Department of Medicine and Center for Human Genetics, Duke University Medical Center, Durham, NC 27710, USA. yiju.li@duke.edu</nlm:affiliation>
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<term>Age of Onset</term>
<term>Aged</term>
<term>Alleles</term>
<term>Apolipoprotein E3</term>
<term>Apolipoprotein E4</term>
<term>Apolipoproteins E (genetics)</term>
<term>Apolipoproteins E (physiology)</term>
<term>Australia (epidemiology)</term>
<term>Cognition</term>
<term>Dementia (epidemiology)</term>
<term>Dementia (genetics)</term>
<term>Female</term>
<term>Gene Frequency</term>
<term>Genetic Predisposition to Disease</term>
<term>Genotype</term>
<term>Humans</term>
<term>Male</term>
<term>Middle Aged</term>
<term>Neuropsychological Tests</term>
<term>Parkinson Disease (epidemiology)</term>
<term>Parkinson Disease (genetics)</term>
<term>Parkinson Disease (psychology)</term>
<term>Pedigree</term>
<term>Risk</term>
<term>United States (epidemiology)</term>
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<keywords scheme="MESH" type="chemical" qualifier="genetics" xml:lang="en"><term>Apolipoproteins E</term>
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<keywords scheme="MESH" type="chemical" qualifier="physiology" xml:lang="en"><term>Apolipoproteins E</term>
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<keywords scheme="MESH" type="chemical" xml:lang="en"><term>Apolipoprotein E3</term>
<term>Apolipoprotein E4</term>
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<keywords scheme="MESH" type="geographic" qualifier="epidemiology" xml:lang="en"><term>Australia</term>
<term>United States</term>
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<keywords scheme="MESH" qualifier="epidemiology" xml:lang="en"><term>Dementia</term>
<term>Parkinson Disease</term>
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<keywords scheme="MESH" qualifier="genetics" xml:lang="en"><term>Dementia</term>
<term>Parkinson Disease</term>
</keywords>
<keywords scheme="MESH" qualifier="psychology" xml:lang="en"><term>Parkinson Disease</term>
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<keywords scheme="MESH" xml:lang="en"><term>Adult</term>
<term>Age of Onset</term>
<term>Aged</term>
<term>Alleles</term>
<term>Cognition</term>
<term>Female</term>
<term>Gene Frequency</term>
<term>Genetic Predisposition to Disease</term>
<term>Genotype</term>
<term>Humans</term>
<term>Male</term>
<term>Middle Aged</term>
<term>Neuropsychological Tests</term>
<term>Pedigree</term>
<term>Risk</term>
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<term>États-Unis</term>
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<front><div type="abstract" xml:lang="en">Similarities between Alzheimer disease (AD) and Parkinson disease (PD) suggest a possible role for apolipoprotein E (APOE) in PD. Most previous studies seeking to establish such a link used case-control datasets and results have been inconsistent.</div>
</front>
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